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Transplantation, ISSN 0041-1337, 09/2013, Volume 96, pp. 452 - 452
Presentation of graft-derived antigens with correct co-stimulation drives the allo-immune response. Without these factors graft-specific tolerance may result.... 
Journal Article
Nature Genetics, ISSN 1061-4036, 05/2015, Volume 47, Issue 5, pp. 435 - 444
Journal Article
Nature, ISSN 0028-0836, 12/2009, Volume 462, Issue 7275, pp. 868 - 874
Journal Article
Nature Genetics, ISSN 1061-4036, 2012, Volume 44, Issue 3, pp. 319 - 322
Journal Article
Gut, ISSN 0017-5749, 12/2018, Volume 67, Issue 12, pp. 2142 - 2155
ObjectiveExtensive molecular heterogeneity of pancreatic ductal adenocarcinoma (PDA), few effective therapies and high mortality make this disease a prime... 
molecular mechanisms | pancreatic cancer | cell cycle | extracellular matrix | KINASE 4/6 INHIBITOR | BREAST-CANCER | SURVIVAL | PALBOCICLIB | THERAPY | DUCTAL ADENOCARCINOMA | NAB-PACLITAXEL | FULVESTRANT | CELL-CYCLE | GASTROENTEROLOGY & HEPATOLOGY | GEMCITABINE | Phosphorylation | Pancreatic Neoplasms - metabolism | Prognosis | Humans | Pancreatic Neoplasms - pathology | Retinoblastoma Protein - metabolism | Carcinoma, Pancreatic Ductal - metabolism | Antineoplastic Agents - therapeutic use | Piperazines - therapeutic use | Carcinoma, Pancreatic Ductal - pathology | Pancreatic Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Carcinoma, Pancreatic Ductal - drug therapy | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Mice, Nude | Biomarkers, Tumor - metabolism | Mice, Inbred BALB C | Cyclin-Dependent Kinase 4 - antagonists & inhibitors | Molecular Targeted Therapy - methods | Disease Models, Animal | Pyridines - therapeutic use | Cell proliferation | Adenocarcinoma | Animal models | Genomes | Shear stresses | Metastasis | Kinases | Cancer therapies | Phase transitions | Cyclin-dependent kinase 4 | Metastases | Proteins | Cyclin-dependent kinase | Signal transduction | Cell cycle | Extracellular matrix | Cyclin D | Cyclin-dependent kinases | Retinoblastoma protein | Breast cancer | Patients | Chemotherapy | Molecular modelling | Pancreatic cancer | Biomarkers | Genetic engineering | Clinical medicine | Mutation | Retinoblastoma | Apoptosis | Tumors | 2312 | Pancreas | 1506
Journal Article
Science Translational Medicine, ISSN 1946-6234, 04/2017, Volume 9, Issue 384, pp. eaai8504 - eaai8504
The emerging standard of care for patients with inoperable pancreatic cancer is a combination of cytotoxic drugs gemcitabine and Abraxane, but patient response... 
MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | DUCTAL ADENOCARCINOMA | CELL INVASION | RHO-KINASE/ROCK | TUMOR | TENSION | EXTRACELLULAR-MATRIX | CLEAVAGE FURROW | REVEALS | CELL BIOLOGY | Liver - pathology | Humans | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Extracellular Matrix - metabolism | Deoxycytidine - pharmacology | Antineoplastic Agents - therapeutic use | rho-Associated Kinases - antagonists & inhibitors | CDC2 Protein Kinase - metabolism | Pancreatic Neoplasms - drug therapy | Deoxycytidine - therapeutic use | Neoplasm Metastasis | rho-Associated Kinases - metabolism | Actin Cytoskeleton - drug effects | src-Family Kinases - metabolism | Albumin-Bound Paclitaxel - therapeutic use | Antineoplastic Agents - pharmacology | Actin Cytoskeleton - metabolism | Neoplasm Invasiveness | Pancreatic Neoplasms - pathology | Albumin-Bound Paclitaxel - pharmacology | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - therapeutic use | Treatment Outcome | Disease Progression | Collagen - metabolism | Animals | Signal Transduction - drug effects | Protein Kinase Inhibitors - therapeutic use | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Biosensing Techniques | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Deoxycytidine - analogs & derivatives
Journal Article
NATURE IMMUNOLOGY, ISSN 1529-2908, 10/2019, Volume 20, Issue 10, pp. 1299 - 1299
Resisting and tolerating microbes are alternative strategies to survive infection, but little is known about the evolutionary mechanisms controlling this... 
ACTIVATION | GENOME SEQUENCE | VIRUS | TOLERANCE | INFLAMMATION | NEANDERTHAL | IMMUNOLOGY | ADMIXTURE | NF-KAPPA-B | T-CELLS | CELL-DEATH | Phosphorylation | Genetic aspects | Regulation | Immune response | Immunity | Ubiquitin | Biological evolution | Kinases | Immunological tolerance | Inflammatory diseases | Hominids | Lipopolysaccharides | Proteins | Inoculation | Ethyl nitrosourea | Alleles | Mice | A20 protein | Catalysis | Immune system
Journal Article
Journal Article