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Cancer Cell, ISSN 1535-6108, 2011, Volume 19, Issue 2, pp. 257 - 272
Journal Article
Clinical Gastroenterology and Hepatology, ISSN 1542-3565, 2010, Volume 8, Issue 1, pp. 36 - 41.e1
Journal Article
STEM CELLS, ISSN 1066-5099, 11/2016, Volume 34, Issue 11, pp. 2613 - 2624
The network of stemness genes and oncogenes in human patient‐specific reprogrammed cancer stem cells (CSCs) remains elusive, especially in liver cancer.... 
OCT4 | c‐JUN | Liver cancer | Reprogramming | Cancer stem cells | Positive feedback | Pluripotency | c-JUN | CELLS | DEFINED FACTORS | OVARIAN-CANCER | CELL & TISSUE ENGINEERING | CELL BIOLOGY | BREAST-CANCER | HEPATOCELLULAR-CARCINOMA | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | IN-VIVO | EPIGENETIC REGULATION | DIMERIZATION PROTEIN-2 | HEMATOLOGY | EXPRESSION | Neoplastic Stem Cells - drug effects | Humans | Middle Aged | Transcriptional Activation | Gene Expression Regulation, Neoplastic | Spheroids, Cellular - pathology | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Cellular Reprogramming | Octamer Transcription Factor-3 - genetics | Neoplastic Stem Cells - metabolism | Neoplastic Stem Cells - pathology | Female | Liver Neoplasms - pathology | Antineoplastic Agents - pharmacology | Cell Differentiation | Spheroids, Cellular - drug effects | JNK Mitogen-Activated Protein Kinases - genetics | Induced Pluripotent Stem Cells - metabolism | Induced Pluripotent Stem Cells - pathology | Induced Pluripotent Stem Cells - drug effects | Liver Neoplasms - genetics | Signal Transduction | Spheroids, Cellular - metabolism | Liver Neoplasms - drug therapy | Cisplatin - pharmacology | Mice, SCID | Hep G2 Cells | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Feedback, Physiological | Animals | Tumor Burden - drug effects | Octamer Transcription Factor-3 - metabolism | Liver Neoplasms - metabolism | Aged | Fluorouracil - pharmacology | Mice | Doxorubicin - pharmacology | Development and progression | Stem cells | Transcription factors | Liver | Oct-4 protein | c-Myc protein | Chemoresistance | Stem cell transplantation | Transplantation | Feedback loops | Myc protein | Allografts | Cell fate | Feedback | Xenografts | Tumorigenesis | Tumor markers | Colonies | c-Jun protein | Gene expression | Hepatocytes | Tumors | Cancer | Index Medicus
Journal Article
Cancer Research, ISSN 0008-5472, 11/2010, Volume 70, Issue 21, pp. 8435 - 8445
Chronic infectious diseases, such as Helicobacter pylori infection, can promote cancer in a large part through induction of chronic inflammation. Oncogenic... 
STEM-CELLS | IN-VITRO | ONCOLOGY | HELICOBACTER-PYLORI INFECTION | GENE-EXPRESSION | TUMOR-CELLS | ACTIVATED MACROPHAGES | STROMAL FIBROBLASTS | GROWTH-FACTOR | INTESTINAL EPITHELIAL-CELLS | TRANSGENIC MICE | Inflammation - pathology | Precancerous Conditions - etiology | Proto-Oncogene Proteins p21(ras) | Cell Proliferation | Stromal Cells - pathology | Humans | Helicobacter Infections - pathology | Immunoenzyme Techniques | Stromal Cells - virology | Bone Marrow Transplantation | Helicobacter Infections - metabolism | Stomach Neoplasms - etiology | Carcinoma in Situ - pathology | Mesenchymal Stromal Cells - metabolism | Mice, Transgenic | Mutation - genetics | Reverse Transcriptase Polymerase Chain Reaction | Inflammation - etiology | Metaplasia - virology | Blotting, Western | Metaplasia - pathology | Stem Cells - pathology | Chemokines - metabolism | Mice | Actins - metabolism | Carcinoma in Situ - etiology | Metaplasia - etiology | Stomach Neoplasms - pathology | Stomach - metabolism | Stem Cells - metabolism | Flow Cytometry | In Situ Hybridization | Precancerous Conditions - pathology | Stomach - virology | ras Proteins - physiology | Helicobacter Infections - virology | Stem Cells - virology | Stomach - pathology | Cytokines - metabolism | Stomach Neoplasms - virology | Neoplasm Invasiveness | Mice, Inbred C57BL | RNA, Messenger - genetics | Stromal Cells - metabolism | Hyperplasia - virology | Muscle, Smooth - metabolism | Hyperplasia - etiology | Hyperplasia - pathology | Keratin-19 - genetics | Animals | Fluorescent Antibody Technique | Proto-Oncogene Proteins - physiology | Helicobacter felis | Mesenchymal Stromal Cells - pathology | Muscle, Smooth - cytology | Index Medicus | Bone marrow derived cells | Kras mutation | Gastric cancer | Gastric epithelial stem cells
Journal Article
Food and Chemical Toxicology, ISSN 0278-6915, 12/2012, Volume 50, Issue 12, pp. 4333 - 4339
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 09/2009, Volume 119, Issue 9, pp. 2691 - 2701
Journal Article
Journal of Antimicrobial Chemotherapy, ISSN 0305-7453, 01/2013, Volume 68, Issue 1, pp. 222 - 228
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 05/2016, Volume 20, Issue 5, pp. 962 - 972
Epidemiologic data show the incidence of gastric cancer in men is twofold higher than in women worldwide. Oestrogen is reported to have the capacity against... 
mesenchymal stem cell | gastric cancer | Src | 17β‐estradiol | cell invasion motility | Mesenchymal stem cell | 17β-estradiol | IL-8 | Gastric cancer | Cell invasion motility | Paxillin - metabolism | Interleukin-8 - genetics | Paxillin - genetics | Epithelial Cells - metabolism | Coculture Techniques | Epithelial Cells - drug effects | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Gene Expression Regulation, Neoplastic | Crk-Associated Substrate Protein - metabolism | Stomach - metabolism | Proto-Oncogene Proteins c-akt - genetics | Mesenchymal Stromal Cells - cytology | MAP Kinase Kinase 4 - metabolism | Mitogen-Activated Protein Kinase 1 - genetics | Bone Marrow Cells - drug effects | Female | MAP Kinase Kinase 4 - antagonists & inhibitors | Interleukin-8 - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Estradiol - pharmacology | Mesenchymal Stromal Cells - drug effects | Stomach - pathology | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Bone Marrow Cells - cytology | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Mesenchymal Stromal Cells - metabolism | Epithelial Cells - pathology | Proto-Oncogene Proteins pp60(c-src) - antagonists & inhibitors | Interleukin-8 - antagonists & inhibitors | Paxillin - antagonists & inhibitors | Crk-Associated Substrate Protein - genetics | Cell Movement - drug effects | Mitogen-Activated Protein Kinase 3 - metabolism | Crk-Associated Substrate Protein - antagonists & inhibitors | Proto-Oncogene Proteins pp60(c-src) - genetics | Proto-Oncogene Proteins pp60(c-src) - metabolism | Cell Line, Tumor | MAP Kinase Kinase 4 - genetics | Bone Marrow Cells - metabolism | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus | Original | IL‐8
Journal Article
Journal Article
STEM CELLS, ISSN 1066-5099, 10/2017, Volume 35, Issue 10, pp. 2115 - 2128
Reprogramming of cancer cells into induced pluripotent stem cells (iPSCs) is a compelling idea for inhibiting oncogenesis, especially through modulation of... 
HoxA transcript at the distal tip | Jun dimerization protein 2 | HoxA transcript antisense RNA | Long noncoding RNA | Gastric cancer | HOXA13 | Bone morphogenetic protein 7 | iPS‐like cells | Octamer‐binding protein 4 | Octamer-binding protein 4 | iPS-like cells | HOTAIR | CHROMATIN | RETINOIC-ACID | HOX GENES | BMP7 | CELL & TISSUE ENGINEERING | CELL BIOLOGY | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | STEM-CELL | DIFFERENTIATION | HEMATOLOGY | EXPRESSION | CARCINOMA | PROGRESSION | Stomach Neoplasms - genetics | Promoter Regions, Genetic | Cell Proliferation | Cellular Reprogramming Techniques - methods | Bone Morphogenetic Protein 7 - genetics | Homeodomain Proteins - metabolism | Humans | Stomach Neoplasms - metabolism | Stomach Neoplasms - pathology | RNA, Long Noncoding - genetics | Homeodomain Proteins - genetics | Bone Morphogenetic Protein 7 - metabolism | Cell Line, Tumor | RNA, Long Noncoding - metabolism | Epigenetic inheritance | Lysine | Leukemia | Cancer cells | Stem cells | Bone morphogenetic proteins | Antisense RNA | Stomach cancer | Protein binding | Transcription | Methyltransferase | Homology | Kinases | Recruitment | Homeobox | Proteins | N-Methyltransferase | Tumorigenesis | Inhibition | Dimerization | Ribonucleic acid--RNA | Switching | Methylation | Pluripotency | Histone H3 | Cancer | Inhibitory postsynaptic potentials | Index Medicus
Journal Article