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The Journal of Physiology, ISSN 0022-3751, 02/2019, Volume 597, Issue 4, pp. 983 - 983
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 05/2013, Volume 304, Issue 10, pp. 678 - 688
Mineralocorticoid receptor (MR) activation stimulates systemic vascular and left ventricular remodeling. We hypothesized that MR contributes to pulmonary... 
Pulmonary vascular remodeling | Hypoxia | Spironolactone | Aldosterone | Monocrotaline | FIBROSIS | hypoxia | OXIDATIVE STRESS | PHYSIOLOGY | aldosterone | monocrotaline | HEART-FAILURE | ANGIOTENSIN-II | spironolactone | HYPERTROPHIC CARDIOMYOPATHY | pulmonary vascular remodeling | RESPIRATORY SYSTEM | ARTERIAL-HYPERTENSION | SMOOTH-MUSCLE-CELLS | MICE | GENE-TRANSCRIPTION | Fibrosis - drug therapy | Hypoxia - drug therapy | Receptors, Mineralocorticoid - metabolism | Myocytes, Smooth Muscle - pathology | Body Weight - drug effects | Fibrosis - metabolism | Hypoxia - metabolism | Pulmonary Artery - metabolism | Spironolactone - pharmacology | Arterial Pressure - drug effects | Mineralocorticoid Receptor Antagonists - pharmacology | Vascular Resistance - drug effects | Hypertension, Pulmonary - drug therapy | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Heart Ventricles - pathology | Cardiac Output - drug effects | Rats | Aldosterone - pharmacology | Hypertension, Pulmonary - metabolism | Pulmonary Artery - drug effects | Platelet-Derived Growth Factor - metabolism | Animals | Hypoxia - pathology | Cell Proliferation - drug effects | Heart Ventricles - metabolism | Mice | Fibrosis - pathology | Hypertension, Pulmonary - pathology | Ventricular Remodeling - drug effects | Pulmonary Artery - pathology | Heart Ventricles - drug effects | Prevention | Physiological aspects | Mineralocorticoids | Smooth muscle | Muscle cells | Health aspects | Pulmonary hypertension | Pulmonary artery | Hypertension | Pathogenesis | Respiratory diseases | Cells | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 11/2018, Volume 138, Issue Suppl_1 Suppl 1, pp. A16053 - A16053
IntroductionPrevious studies showed that the mineralocorticoid receptor (MR), a known regulator of blood pressure that contributes to systemic vascular... 
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 11/2017, Volume 313, Issue 5, pp. L752 - L762
Journal Article
Journal of Applied Physiology, ISSN 8750-7587, 08/2015, Volume 119, Issue 4, pp. 412 - 419
Anthrax is associated with severe vascular leak, which is caused by the bacterial lethal toxin (LeTx). Pleural effusions and pulmonary edema that occur in... 
Endothelial | Acute lung injury | Or acute respiratory distress syndrome | Permeability | MK2 | Heat shock protein 27 | PATHWAYS | CELLS | SPORT SCIENCES | PHYSIOLOGY | PROTEIN | permeability | heat shock protein 27 | HYPOXIA | VIRULENCE | acute lung injury | RESPIRATORY-DISTRESS-SYNDROME | ENDOTHELIAL PERMEABILITY | BERLIN DEFINITION | endothelial | BACILLUS-ANTHRACIS | or acute respiratory distress syndrome | EDEMA TOXIN | Enzyme Activators - pharmacology | Phosphorylation | Rats, Inbred F344 | Capillary Permeability - drug effects | Intracellular Signaling Peptides and Proteins - metabolism | Acute Lung Injury - enzymology | Lung - enzymology | Transfection | RNA Interference | Time Factors | Acute Lung Injury - prevention & control | Bacterial Toxins | Pulmonary Edema - prevention & control | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Cytoprotection | Disease Models, Animal | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Rats | Antigens, Bacterial | Peptides - pharmacology | Pulmonary Edema - chemically induced | Animals | Acute Lung Injury - chemically induced | Pulmonary Edema - enzymology | Signal Transduction - drug effects | Lung - drug effects | HSP27 Heat-Shock Proteins - metabolism | Enzyme Activation | Lung - blood supply | Endothelial Cells - enzymology | Endothelial Cells - drug effects | Toxins | Physiology | Respiratory distress syndrome | Peptides | Anthrax | Index Medicus
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 06/2012, Volume 302, Issue 12, pp. L1273 - L1279
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 10/2014, Volume 307, Issue 7, pp. L576 - L585
We previously reported that transglutaminase 2 (TG2) activity is markedly elevated in lungs of hypoxia-exposed rodent models of pulmonary hypertension (PH).... 
CaSR | HIF-1α | TRPV4 | TG2 | Pulmonary artery smooth muscle cells | COBALT CHLORIDE | INDUCIBLE FACTOR-1-ALPHA | PHYSIOLOGY | HIF-1 alpha | RECEPTOR | pulmonary artery smooth muscle cells | INTRACELLULAR CA2 | RESPIRATORY SYSTEM | CALCIUM | HYPERTENSION | TISSUE TRANSGLUTAMINASE | EXPRESSION | VASOCONSTRICTION | MAMMARY EPITHELIAL-CELLS | Cell Proliferation | GTP-Binding Proteins - antagonists & inhibitors | Humans | Receptors, Calcium-Sensing - metabolism | Muscle, Smooth, Vascular - physiopathology | Cell Hypoxia | TRPV Cation Channels - metabolism | Cattle | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | TRPV Cation Channels - antagonists & inhibitors | Calcium Signaling | Hypertension, Pulmonary - enzymology | Receptors, Calcium-Sensing - antagonists & inhibitors | Transglutaminases - antagonists & inhibitors | GTP-Binding Proteins - physiology | Myocytes, Smooth Muscle - enzymology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Myocytes, Smooth Muscle - physiology | Enzyme Induction | Pulmonary Artery - physiopathology | Muscle, Smooth, Vascular - pathology | Animals | Transglutaminases - physiology | Enzyme Activation | Hypertension, Pulmonary - pathology | Pulmonary Artery - pathology | Cell proliferation | Cell research | Smooth muscle | Muscle cells | Research | Health aspects | Pulmonary artery | Glutamine | Lungs | Pulmonary arteries | Rodents | Hypoxia | Gene expression | Enzyme kinetics | Cells | Index Medicus
Journal Article
FASEB Journal, ISSN 0892-6638, 09/2007, Volume 21, Issue 11, pp. 2725 - 2734
Serotonin ( 5- HT) stimulates smooth muscle cell growth through 5- HT receptors and the 5- HT transporter ( 5- HTT), and has been associated with pulmonary... 
Serotonin | Pulmonary hypertension | SEROTONIN TRANSPORTER | ACTIVATION | pulmonary hypertension | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROTEIN-TYROSINE PHOSPHATASES | HYPERPLASIA | CELL BIOLOGY | serotonin | IN-VITRO | SIGNALING PATHWAY | BIOLOGY | MESANGIAL CELLS | COUPLED RECEPTORS | GROWTH-FACTOR | HYPERTENSION
Journal Article
Thrombosis Research, ISSN 0049-3848, 12/2017, Volume 160, pp. 58 - 65
One of the major contributors to sickle cell disease (SCD) pathobiology is the hemolysis of sickle red blood cells (RBCs), which release free hemoglobin and... 
Aggregation | Hypertension | Hypoxia | Calpain-1 | Platelets | Sickle cell disease | HEMOGLOBIN | ACTIVATION | PULMONARY-HYPERTENSION | HEMOLYSIS | TARGETED GENE INACTIVATION | INFLAMMATION | TRANSGENIC SAD MOUSE | COAGULATION | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | REVEALS | Index Medicus | hypoxia | aggregation | hypertension | platelets | sickle cell disease
Journal Article
Journal Article
American Journal of Physiology, ISSN 1040-0605, 02/2006, Volume 34, Issue 2, p. L367
  The 12-lipoxygenase (12-LO) pathway of arachidonic acid metabolism stimulates cell growth and metastasis of various cancer cells and the 12-LO metabolite,... 
Proteins | Hypertension | Enzymes | Pulmonary arteries | Rodents | Muscular system | Gene expression | Cells
Journal Article
Journal Article