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Journal Article
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY, ISSN 0363-6143, 09/2017, Volume 313, Issue 3, pp. C340 - C351
Oxidized modifications of LDL (oxLDL) play a key role in the development of endothelial dysfunction and atherosclerosis. However, the underlying mechanisms of... 
MULTIPLE FUNCTIONS | NADPH OXIDASE | PHYSIOLOGY | PROTEIN | Rho kinase | PHOSPHORYLATION | MEMBRANE | LOW-DENSITY-LIPOPROTEIN | CELL BIOLOGY | oxidized modifications of low-density lipoproteins | RHOA ACTIVATION | STIFFNESS | CIRCULATING OXIDIZED LDL | INHIBITOR
Journal Article
STEM CELLS, ISSN 1066-5099, 06/2015, Volume 33, Issue 6, pp. 1719 - 1729
Adhesion of embryonic stem cells (ESCs) to the extracellular matrix may influence differentiation potential and cell fate decisions. Here, we investigated the... 
MIGRATION | ACTIVATION | Stem cell endothelial differentiation | Gene regulation | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Laminin-1 | MESODERM | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | Stem cells | EXPANSION | STEM-CELL | FOCAL ADHESION KINASE | ALPHA-3-BETA-1 | HEMATOLOGY | ASSOCIATION | ALPHA-6-BETA-1 | Integrins | Tetraspanins | TETRASPANIN CD151
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2017, Volume 292, Issue 50, pp. 20785 - 20798
A key feature of acute myocardial infarction (AMI) is an alteration in cardiac architecture. Signaling events that result in the inhibition of glycogen... 
CELLS | PROTECTION | angiogenesis | endothelial cell | BIOCHEMISTRY & MOLECULAR BIOLOGY | myocardial infarction | GSK-3-BETA | INJURY | ISCHEMIA | allosteric regulation | TIDEGLUSIB | glycogen synthase kinase 3 (GSK-3) | STRATEGIES | Wnt signaling | CARDIOPROTECTION | EXPRESSION | MODULATION | Neovascularization, Physiologic - drug effects | Apoptosis - drug effects | Atrial Remodeling - drug effects | Male | Aorta - metabolism | Angiogenesis Inducing Agents - therapeutic use | Ligation | Protein Processing, Post-Translational - drug effects | Thiadiazoles - therapeutic use | Myocardial Infarction - pathology | Female | Myocardial Infarction - physiopathology | Angiogenesis Inducing Agents - pharmacology | Phosphorylation - drug effects | Heart Ventricles - pathology | Disease Models, Animal | Thiadiazoles - pharmacology | Allosteric Regulation - drug effects | Coronary Vessels - drug effects | Coronary Vessels - pathology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Aorta - drug effects | Mice, Inbred C57BL | Myocardial Infarction - metabolism | Glycogen Synthase Kinase 3 - metabolism | Aorta - pathology | Animals | Myocardial Infarction - drug therapy | Heart Ventricles - physiopathology | Protein Kinase Inhibitors - therapeutic use | Aorta - surgery | Heart Ventricles - metabolism | Mice, Inbred BALB C | Protein Kinase Inhibitors - pharmacology | In Vitro Techniques | Heart Ventricles - drug effects | Index Medicus | Gene Regulation
Journal Article
CELL, ISSN 0092-8674, 09/1998, Volume 94, Issue 5, pp. 625 - 634
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 1768, Volume 289, Issue 19, pp. 13476 - 13491
Journal Article
Journal Article
PLOS ONE, ISSN 1932-6203, 12/2013, Volume 8, Issue 12
Rationale: Induced pluripotent stem (iPS) cells have emerged as a source of potentially unlimited supply of autologous endothelial cells (ECs) for... 
PLURIPOTENT STEM-CELLS | PROGENITOR CELLS | MESODERM | PRECURSOR | LETHALITY | VASCULOGENESIS | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | INDUCTION | EXPRESSION | BLOOD
Journal Article
STEM CELLS, ISSN 1066-5099, 06/2014, Volume 32, Issue 6, pp. 1538 - 1552
Endothelial cell (EC) dedifferentiation in relation to neovascularization is a poorly understood process. In this report, we addressed the role of Wnt... 
6‐Bromoindirubin‐3′‐oxime | Hind limb ischemia | Neovascularization | Dedifferentiation | Endothelial cells | 6-Bromoindirubin-3′-oxime | PROGENITOR CELLS | YOLK-SAC | REGENERATION | MOUSE | CARDIOMYOCYTES | IDENTIFICATION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | PLURIPOTENT STEM-CELLS | HEART | 6-Bromoindirubin-3 '-oxime | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | HEMATOLOGY | PRECURSOR CELLS | EXPRESSION | Neovascularization, Physiologic - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Homeodomain Proteins - metabolism | Humans | Oximes - administration & dosage | Cell Aggregation - drug effects | Promoter Regions, Genetic - genetics | Indoles - administration & dosage | Dose-Response Relationship, Drug | Cell Nucleus - metabolism | Angiogenesis Inducing Agents - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Protein Binding - drug effects | Indoles - pharmacology | Vascular Endothelial Growth Factor A - pharmacology | Ischemia - pathology | Disease Models, Animal | Human Umbilical Vein Endothelial Cells - drug effects | Nanog Homeobox Protein | Cell Dedifferentiation - drug effects | Endothelial Cells - metabolism | T-Box Domain Proteins - genetics | beta Catenin - metabolism | Cell Division - drug effects | Pluripotent Stem Cells - metabolism | Cell Movement - drug effects | Phenotype | Animals | Endothelial Cells - cytology | Hindlimb - blood supply | Pluripotent Stem Cells - drug effects | Protein Stability - drug effects | Hindlimb - pathology | Oximes - pharmacology | Cell Proliferation - drug effects | Fetal Proteins - genetics | Mice | Cell Nucleus - drug effects | Endothelial Cells - drug effects | Cell division | Kinases | Vascular endothelial growth factor | Index Medicus | BIO
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, pp. e46158 - e46158
Background: Our previous studies indicated that MSCCXCR4 improved cardiac function after myocardial infarction (MI). This study was aimed to investigate the... 
CARDIAC-FUNCTION | PROGENITOR CELLS | IN-VITRO | REPAIR | MARROW-DERIVED CELLS | ETS-BINDING-SITES | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | INFARCTED MYOCARDIUM | HYPOXIA | EXPRESSION | Myocardial Infarction - genetics | Phosphorylation | Cadherins - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Antigens, CD - genetics | Antigens, CD - metabolism | Hypoxia - metabolism | Vascular Endothelial Growth Factor A - secretion | Mesenchymal Stromal Cells - cytology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Myocardium - metabolism | Lentivirus - genetics | Receptors, CXCR4 - genetics | Cell Differentiation | Cadherins - genetics | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Gene Expression | Genes, Transgenic, Suicide | Transduction, Genetic | Endothelial Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Mesenchymal Stromal Cells - metabolism | Rats | Myocardial Infarction - metabolism | Rats, Sprague-Dawley | Receptors, CXCR4 - metabolism | Hypoxia - genetics | Animals | Endothelial Cells - cytology | Genetic Vectors | Mesenchymal Stem Cell Transplantation | Neovascularization, Physiologic | Stem cells | Physiological aspects | Development and progression | Genetic aspects | Research | Neovascularization | Chemokine receptors | Heart attack | Myocardial infarction | Heart | Lipoproteins (low density) | Surgical implants | Transcription | Mesenchyme | Syngeneic grafts | Differentiation (biology) | Reversing | Kinases | Suicide | Arteries | Angiogenesis | Signal transduction | Ganciclovir | Allografts | Ischemia | Computed tomography | Restoration | Rodents | Vascular endothelial growth factor | Heart diseases | Medical research | Echocardiography | Heart transplantation | Stat3 protein | Blood vessels | Implantation | Pharmacology | Biophysics | Gene expression | Cadherin | CXCR4 protein | Endothelial cells | Medicine | Studies | Polymerase chain reaction | Pathology | Myocardium | Hypoxia | Infarction | In vivo methods and tests | Genetic engineering | Laboratory animals | Chemokines | Index Medicus
Journal Article