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1. Full Text Unsuspected Protumorigenic Signaling Role for the Oncometabolite GABA in Advanced Prostate Cancer
by Taylor, RA and Watt, MJ
CANCER RESEARCH, ISSN 0008-5472, 09/2019, Volume 79, Issue 18, pp. 4580 - 4581
Elucidating the events that underpin the transition from androgen-dependent to castrate-resistant prostate cancer (CRPC) remains a clinical challenge. In this... 
ONCOLOGY | GROWTH
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2. Full Text 287-OR: Acute Exercise Enhances the Differentiation Rate of Human Adipocyte Precursor Cells
by VARSHNEY, PALLAVI and GUTH, LISA M and ANDERSON, CARA M and RAAJENDIRAN, ARTHE and WATT, MATTHEW J and HOROWITZ, JEFFREY F
Diabetes, ISSN 0012-1797, 06/2019, Volume 68, Issue Supplement 1, p. 287
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3. Full Text The role of Ap2a2 in PPARα-mediated regulation of lipolysis in adipose tissue
by Montgomery, Magdalene K and Bayliss, Jacqueline and Keenan, Stacey and Rhost, Sarah and Ting, Stephen B and Watt, Matthew J
The FASEB Journal, ISSN 0892-6638, 12/2019, Volume 33, Issue 12, pp. 13267 - 13279
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4. Full Text Impact of Lipotoxicity on Tissue “Cross Talk” and Metabolic Regulation
by Montgomery, Magdalene K and De Nardo, William and Watt, Matthew J
Physiology, ISSN 1548-9213, 03/2019, Volume 34, Issue 2, pp. 134 - 149
Obesity-associated comorbidities include non-alcoholic fatty liver disease, Type 2 diabetes, and cardiovascular disease. These diseases are associated with... 
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5. Full Text Invited talk: Do factors secreted from the fatty liver cause diabetes?
by Watt, Matthew J
Obesity Research & Clinical Practice, ISSN 1871-403X, 01/2019, Volume 13, Issue 1, pp. 33 - 33
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6. Full Text Hepatokines: Linking nonalcoholic fatty liver disease and insulin resistance
by Meex, Ruth C. R and Watt, Matthew J
Nature Reviews Endocrinology, ISSN 1759-5029, 09/2017, Volume 13, Issue 9, pp. 509 - 520
Hepatic steatosis is an underlying feature of nonalcoholic fatty liver disease (NAFLD), which is the most common form of liver disease and is present in up to... 
ADIPOSE TRIGLYCERIDE LIPASE | INDUCED HEPATIC STEATOSIS | ENDOCRINOLOGY & METABOLISM | FIBROBLAST GROWTH FACTOR-21 | IMPROVES GLUCOSE-TOLERANCE | ENDOGENOUS SEX-HORMONES | HORMONE-BINDING GLOBULIN | TYPE-2 DIABETES-MELLITUS | CORONARY-ARTERY-DISEASE | INDUCED OBESE MICE | CARDIOVASCULAR RISK-FACTORS | Obesity | Humans | Insulin Resistance | Non-alcoholic Fatty Liver Disease - physiopathology | Liver - physiopathology | Retinol-Binding Proteins, Plasma - physiology | alpha-2-HS-Glycoprotein - physiology | Overweight | Animals | Hypertriglyceridemia | Blood Proteins - physiology | Diet, High-Fat | Diabetes Mellitus, Type 2 | Fetuin-B - physiology | Selenoprotein P - physiology | Lipid Metabolism - physiology | Insulin resistance | Care and treatment | Fatty liver | Research | Selenoproteins | Liver cells | Phenotypes | Liver diseases | Secretion | Diabetes mellitus | Body weight | Metabolism | Insulin | Skeletal muscle | Steatosis | Glucose tolerance | Disease resistance | Intolerance | Vitamin A | Hepatocytes | Diabetes mellitus (non-insulin dependent) | Lipoproteins (high density) | Retinol-binding protein
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7. Perilipin 5 is dispensable for normal substrate metabolism and in the adaptation of skeletal muscle to exercise training
by Ruzaidi A M Mohktar and Magda K Montgomery and Robyn M Murphy and Matthew J Watt
American Journal of Physiology, ISSN 0193-1849, 07/2016, Volume 311, Issue 1, p. E128
  Cytoplasmic lipid droplets provide a reservoir for triglyceride storage and are a central hub for fatty acid trafficking in cells. The protein perilipin 5... 
Rodents | Sports training | Protein expression | Metabolism | Stress response | Fatty acids | Cytoplasm
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8. Perilipin 5 is dispensable for normal substrate metabolism and in the adaptation of skeletal muscle to exercise training
by Mohktar, Ruzaidi A M and Montgomery, Magda K and Murphy, Robyn M and Watt, Matthew J
American Journal of Physiology: Endocrinology and Metabolism, ISSN 0193-1849, 07/2016, Volume 311, Issue 1, pp. E128 - E128
Cytoplasmic lipid droplets provide a reservoir for triglyceride storage and are a central hub for fatty acid trafficking in cells. The protein perilipin 5... 
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9. Full Text Radical gastric cancer surgery results in widespread upregulation of pro-tumourigenic intraperitoneal cytokines
: Pro-tumourigenic cytokines during gastric surgery
by Long, Andrew J and Burton, Paul R and De Veer, Michael J and Ooi, Geraldine J and Laurie, Cheryl P and Nottle, Peter D and Watt, Matthew J and Brown, Wendy A
ANZ Journal of Surgery, ISSN 1445-1433, 05/2018, Volume 88, Issue 5, pp. E370 - E376
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10. Full Text CDK4/6 inhibition triggers anti-tumour immunity
by Goel, Shom and Decristo, Molly J and Watt, April C and Brinjones, Haley and Sceneay, Jaclyn and Li, Ben B and Khan, Naveed and Ubellacker, Jessalyn M and Xie, Shaozhen and Metzger-Filho, Otto and Hoog, Jeremy and Ellis, Matthew J and Ma, Cynthia X and Ramm, Susanne and Krop, Ian E and Winer, Eric P and Roberts, Thomas M and Kim, Hye-Jung and McAllister, Sandra S and Zhao, Jean J
Nature, ISSN 0028-0836, 08/2017, Volume 548, Issue 7668, pp. 471 - 475
Cyclin-dependent kinases 4 and 6 (CDK4/6) are fundamental drivers of the cell cycle and are required for the initiation and progression of various... 
BREAST-CANCER | CELLS | METHYLATION | MULTIDISCIPLINARY SCIENCES | SENESCENCE | EXPRESSION | REQUIREMENT | Cyclin-Dependent Kinase 6 - antagonists & inhibitors | Viruses - genetics | Breast Neoplasms - immunology | Humans | Transcriptome | T-Lymphocytes, Regulatory - immunology | T-Lymphocytes, Regulatory - cytology | Female | Biological Mimicry - drug effects | Phosphorylation - drug effects | Cyclin-Dependent Kinase 4 - antagonists & inhibitors | Disease Models, Animal | Viruses - drug effects | Viruses - immunology | Antigen Presentation - immunology | Breast Neoplasms - drug therapy | T-Lymphocytes, Regulatory - drug effects | Animals | Breast Neoplasms - genetics | Repressor Proteins - biosynthesis | Signal Transduction - drug effects | Breast Neoplasms - pathology | Cell Cycle Checkpoints - drug effects | Protein Kinase Inhibitors - therapeutic use | RNA, Double-Stranded - genetics | Cell Line, Tumor | Interferons - metabolism | Antigen Presentation - drug effects | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Oncology, Experimental | Cancer cells | Physiological aspects | Research | Protein kinases | Immunity | Cyclins | Cancer | Cell proliferation | Flow cytometry | Animal models | Phosphorylation | Senescence | Peptides | Genomics | Immune clearance | Cytotoxicity | Lymphocytes T | Genomes | Kinases | Cancer therapies | Cyclin-dependent kinase 4 | E2F protein | Breast carcinoma | Cell growth | Lymphocytes | New combinations | Cell cycle | Inhibition | Deoxyribonucleic acid--DNA | Immune system | Antigen presentation | Medical research | Immune response | Immunoregulation | Intracellular levels | Double-stranded RNA | Breast cancer | Pharmacology | Gene expression | Ribonucleic acid--RNA | Inhibitors | Immune checkpoint | Immunogenicity | Breast | DNA methyltransferase | Interferon | Retinoblastoma | Tumors | Apoptosis
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11. Full Text Obesity does not promote tumorigenesis of localized patientderived prostate cancer xenografts
by Lo, Jennifer C.Y and Clark, Ashlee K and Ascui, Natasha and Frydenberg, Mark and Risbridger, Gail P and Taylor, Renea A and Watt, Matthew J
Oncotarget, 2016, Volume 7, Issue 30, pp. 47650 - 47662
Human | Obesity | Xenograft | Periprostatic adipose tissue | Prostate cancer
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12. Full Text Lipid metabolism in skeletal muscle: Generation of adaptive and maladaptive intracellular signals for cellular function
by Watt, Matthew J and Hoy, Andrew J
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 06/2012, Volume 302, Issue 11, pp. E1315 - E1328
Watt MJ, Hoy AJ. Lipid metabolism in skeletal muscle: generation of adaptive and maladaptive intracellular signals for cellular function. Am J Physiol... 
ADIPOSE TRIGLYCERIDE LIPASE | PHYSIOLOGY | ACTIVATED PROTEIN-KINASE | HORMONE-SENSITIVE LIPASE | RECEPTOR-GAMMA | FATTY-ACID OXIDATION | OBESE ZUCKER RATS | ACYL-COA SYNTHETASE | ENDOCRINOLOGY & METABOLISM | PHOSPHATIDIC-ACID | INDUCED INSULIN-RESISTANCE | SPHINGOSINE 1-PHOSPHATE | Animals | Insulin Resistance - physiology | Humans | Lipids - physiology | Adaptation, Physiological - physiology | Muscle, Skeletal - metabolism | Signal Transduction - physiology | Muscle, Skeletal - physiology | Fatty Acids - metabolism | Lipid Metabolism - physiology | Muscle Fibers, Skeletal - physiology | Triglycerides - metabolism | Physiological aspects | Muscles | Fatty acid metabolism | Health aspects
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13. Lipid metabolism in skeletal muscle: generation of adaptive and maladaptive intracellular signals for cellular function
by Matthew J Watt and Andrew J Hoy
American Journal of Physiology, ISSN 0193-1849, 06/2012, Volume 302, Issue 11, p. E1315
Fatty acids derived from adipose tissue lipolysis, intramyocellular triacylglycerol lipolysis, or de novo lipogenesis serve a variety of functions in skeletal... 
Musculoskeletal system | Metabolites | Lipids | Oxidation | Metabolism | Fatty acids
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14. Full Text Impact of endurance exercise training on adipocyte miRNA expression in overweight men
by Tsiloulis, Thomas and Pike, Joshua and Powell, David and Rossello, Fernando J and Canny, Benedict J and Meex, Ruth C.R and Watt, Matthew J
Obesity Research & Clinical Practice, ISSN 1871-403X, 01/2019, Volume 13, Issue 1, pp. 53 - 53
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15. Full Text Perilipin 5 deletion in hepatocytes remodels lipid metabolism and causes hepatic insulin resistance in mice
by Keenan, Stacey N and Meex, Ruth C and Lo, Jen C.Y and Montgomery, Magdalene K and Watt, Matthew J
Obesity Research & Clinical Practice, ISSN 1871-403X, 05/2019, Volume 13, Issue 3, pp. 271 - 271
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16. Full Text The role of Ap2a2 in PPARα-mediated regulation of lipolysis in adipose tissue
by Montgomery, Magdalene K and Bayliss, Jacqueline and Ting, Stephen and Raajendiran, Arthe and Watt, Matthew J
Obesity Research & Clinical Practice, ISSN 1871-403X, 05/2019, Volume 13, Issue 3, pp. 276 - 276
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17. Full Text Overexpression of Carnitine Palmitoyltransferase-1 in Skeletal Muscle Is Sufficient to Enhance Fatty Acid Oxidation and Improve High-Fat Diet–Induced Insulin Resistance
by Clinton R. Bruce and Andrew J. Hoy and Nigel Turner and Matthew J. Watt and Tamara L. Allen and Kevin Carpenter and Gregory J. Cooney and Mark A. Febbraio and Edward W. Kraegen
Diabetes, ISSN 0012-1797, 03/2009, Volume 58, Issue 3, pp. 550 - 558
Overexpression of Carnitine Palmitoyltransferase-1 in Skeletal Muscle Is Sufficient to Enhance Fatty Acid Oxidation and Improve High-Fat Diet–Induced Insulin... 
CELLS | PROTEIN-KINASE-C | OBESITY | ACTIVATION | METABOLISM | INTRAMYOCELLULAR LIPID-ACCUMULATION | GLUCOSE-TRANSPORTER | ENDOCRINOLOGY & METABOLISM | RATS | KAPPA-B | CERAMIDE | Palmitic Acid - metabolism | Carnitine O-Palmitoyltransferase - genetics | Dietary Fats - adverse effects | Muscle, Skeletal - enzymology | Mitochondria, Muscle - enzymology | Rats, Wistar | Humans | Rats | Electroporation | Male | Animals | Plasmids | Insulin Resistance - genetics | Polymerase Chain Reaction | Second Messenger Systems - physiology | Glucose Clamp Technique | Handling (Psychology) | Fatty Acids - metabolism | Hyperinsulinism | Muscle Fibers, Skeletal - enzymology | Control | Transferases | Physiological aspects | Insulin resistance | Development and progression | Biological oxidation (Metabolism) | Genetic aspects | Research | Health aspects | Ketogenic diet | Risk factors | Metabolism
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18. Fragments of the commentary on Matthew and Luke
by Philoxenus, Bishop of Hierapolis, approximately 440-523 and Watt, J. W
1978, Corpus scriptorum Christianorum Orientalium, ISBN 2801700827, Volume 392, 393 : Scriptores syri ; t. 171,172, 2 v. --
Bible. Commentaries
Book
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19. Reduced ATGL-mediated lipolysis attenuates beta -adrenergic-induced AMPK signaling, but not the induction of PKA-targeted genes, in adipocytes and adipose tissue
by MacPherson, Rebecca EK and Dragos, Steven M and Ramos, Sofhia and Sutton, Charles and Frendo-Cumbo, Scott and Castellani, Laura and Watt, Matthew J and Perry, Christopher GR and Mutch, David M and Wright, David C
American Journal of Physiology: Cell Physiology, ISSN 0363-6143, 08/2016, Volume 311, Issue 2, pp. C269 - C269
5'-AMP-activated protein kinase (AMPK) is activated as a consequence of lipolysis and has been shown to play a role in regulation of adipose tissue... 
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20. Full Text Regulation and function of triacylglycerol lipases in cellular metabolism
by Watt, Matthew J and Steinberg, Gregory R
Biochemical Journal, ISSN 0264-6021, 09/2008, Volume 414, Issue 3, pp. 313 - 325
The ability to store energy in the form of energy-dense TAG (triacylglycerol) and to mobilize these stores rapidly during times of low carbohydrate... 
Adipose triacylglycerol lipase (ATGL) | Type 2 diabetes | Obesity | Hormone-sensitive lipase (HSL) | Adipose tissue | Fatty acid metabolism | Skeletal muscle | ADIPOSE TRIGLYCERIDE LIPASE | HORMONE-SENSITIVE LIPASE | fatty acid metabolism | BIOCHEMISTRY & MOLECULAR BIOLOGY | adipose tissue | CHANARIN-DORFMAN-SYNDROME | HUMAN SKELETAL-MUSCLE | INDUCED INSULIN-RESISTANCE | LIPID STORAGE DISEASE | hormone-sensitive lipase (HSL) | ACID-BINDING PROTEIN | HSL-DEFICIENT MICE | FAMILIAL COMBINED HYPERLIPIDEMIA | skeletal muscle | adipose triacylglycerol lipase (ATGL) | ALPHA-INDUCED LIPOLYSIS | obesity | Muscle, Skeletal - enzymology | Phosphorylation | Liver - enzymology | Humans | Liver - metabolism | Sterol Esterase - metabolism | Lipase - physiology | Muscle, Skeletal - metabolism | Lipase - metabolism | Obesity - metabolism | Animals | Sterol Esterase - genetics | Models, Biological | Transcription, Genetic | Obesity - enzymology | Protein Processing, Post-Translational | Lipase - genetics
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