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Nature, ISSN 0028-0836, 10/2010, Volume 467, Issue 7318, pp. 972 - 976
Reciprocity of inflammation, oxidative stress and neovascularization is emerging as an important mechanism underlying numerous processes from tissue healing... 
SYSTEM | TOLL-LIKE RECEPTOR-2 | CELLS | PATTERN-RECOGNITION | MULTIDISCIPLINARY SCIENCES | MACULAR DEGENERATION | CD36 | CHRONIC INFLAMMATION | CANCER | INNATE IMMUNITY | Neovascularization, Physiologic - drug effects | Toll-Like Receptor 2 - agonists | Melanoma - blood supply | Humans | Oxidative Stress - physiology | Vascular Endothelial Growth Factor A - metabolism | Inflammation - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Wound Healing - drug effects | Receptors, Scavenger - metabolism | Melanoma - metabolism | Cell Line | Pyrroles - metabolism | Oxidation-Reduction | Endothelial Cells - metabolism | Aorta - drug effects | Mice, Inbred C57BL | Ischemia - metabolism | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Immunity, Innate - immunology | Pyrroles - pharmacology | Animals | Signal Transduction - drug effects | Propionates | Pyrroles - chemistry | Ligands | Mice | Neovascularization, Pathologic - metabolism | Wound Healing - physiology | Aorta - cytology | Hindlimb - metabolism | Myeloid Differentiation Factor 88 - metabolism | rac1 GTP-Binding Protein - metabolism | Aging - metabolism | Cell Movement | Oxidative stress | Complications and side effects | Cell receptors | Physiological aspects | Genetic aspects | Research | Neovascularization | Vascular endothelial growth factor | Angiogenesis | Wound healing | Rodents | Atherosclerosis
Journal Article
Circulation Research, ISSN 0009-7330, 01/2013, Volume 112, Issue 1, pp. 103 - 112
RATIONALE:A prothrombotic state and increased platelet reactivity are common in pathophysiological conditions associated with oxidative stress and infections.... 
carboxy(alkylpyrrole) protein adducts | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | HUMAN PLASMA | MACROPHAGES | DEPENDENT PROTEIN-KINASE | MACULAR DEGENERATION | Toll-like receptor 9 | platelets | LOW-DENSITY LIPOPROTEINS | INFLAMMATION | PERIPHERAL VASCULAR DISEASE | OXIDIZED PHOSPHOLIPIDS | CD36 | HEMATOLOGY | oxidative stress | B SCAVENGER RECEPTORS | Toll-Like Receptor 9 - blood | CD36 Antigens - deficiency | Phosphorylation | Blood Platelets - immunology | CD36 Antigens - genetics | Toll-Like Receptor 2 - genetics | Oxidative Stress | Humans | Toll-Like Receptor 9 - genetics | Toll-Like Receptor 2 - deficiency | Transfection | src-Family Kinases - blood | Time Factors | Toll-Like Receptor 6 - genetics | Phosphatidylinositol 3-Kinase - blood | Thrombosis - blood | Genes, Reporter | Disease Models, Animal | Cell Line | Scavenger Receptors, Class B - deficiency | Signal Transduction | Mice, Inbred C57BL | Toll-Like Receptor 9 - deficiency | Myeloid Differentiation Factor 88 - genetics | Thrombosis - immunology | Scavenger Receptors, Class B - genetics | Immunity, Innate | Toll-Like Receptor 6 - deficiency | Mice, Knockout | Platelet Aggregation | Animals | Myeloid Differentiation Factor 88 - deficiency | Blood Platelets - metabolism | Platelet Activation | Proto-Oncogene Proteins c-akt - blood | Ligands | Thrombosis - genetics | Interleukin-1 Receptor-Associated Kinases - blood | Mice | Serum Albumin - metabolism
Journal Article
Nature Communications, ISSN 2041-1723, 03/2016, Volume 7, Issue 1, p. 10960
Journal Article
Heart, ISSN 1355-6037, 10/2012, Volume 98, Issue Suppl 2, p. E7
Objectives Cardiovascular disease resulting in myocardial infarction (MI) is the leading cause of mortality in developed countries; however, the mechanisms... 
Journal Article
Blood, ISSN 0006-4971, 07/2018, Volume 132, Issue 1, pp. 78 - 88
Key Points Recruitment of neutrophils generates phospholipid oxidation and formation of CEP adducts with extracellular matrix proteins. CEP–protein adducts... 
Phagocytes, Granulocytes, and Myelopoiesis
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 2, p. e31071
Integrins mediate cell adhesion, migration, and survival by connecting intracellular machinery with the surrounding extracellular matrix. Previous studies... 
Humans | Integrin beta3 - chemistry | Molecular Sequence Data | Extracellular Signal-Regulated MAP Kinases - metabolism | Phosphotyrosine - metabolism | Peptides - metabolism | Protein Binding - drug effects | Phosphorylation - drug effects | Vascular Endothelial Growth Factor A - pharmacology | Amino Acid Sequence | Human Umbilical Vein Endothelial Cells - drug effects | Magnetic Resonance Spectroscopy | Peptides - chemistry | Mice, Inbred C57BL | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Enzyme Activation - drug effects | Amino Acid Motifs | Peptides - pharmacology | Integrin beta3 - metabolism | Vascular Endothelial Growth Factor Receptor-2 - chemistry | Animals | Human Umbilical Vein Endothelial Cells - enzymology | Signal Transduction - drug effects | Mice | In Vitro Techniques | Neovascularization, Physiologic | Receptor Cross-Talk - drug effects | Fibroblast growth factor | Phosphorylation | Leukocyte migration | Peptides | Molecular structure | Crosstalk | Kinases | Machinery | Mimicry | Cell adhesion & migration | Machinery and equipment | Angiogenesis | Spectrum analysis | Cell adhesion | Extracellular matrix | Cardiology | Vascular endothelial growth factor | Pharmaceutical sciences | Tyrosine | Cell survival | Endothelial cells | Vascular endothelial growth factor receptors | Studies | Pharmacy | Data models | Mutation | Adapter proteins | Cell migration | Conformation | Integrins
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 07/2015, Volume 117, Issue 4, pp. 321 - 332
RATIONALE:Oxidative stress is an important contributing factor in several human pathologies ranging from atherosclerosis to cancer progression; however, the... 
Antigens | Wound healing | CD36 | Angiogenesis inducing agents | Macrophages | Toll-like receptor 2 | wound healing | CARDIAC & CARDIOVASCULAR SYSTEMS | GENOMIC BIOMARKERS | LOW-DENSITY-LIPOPROTEIN | antigens, CD36 | MACULAR DEGENERATION | ENDOGENOUS LIGANDS | VASCULAR-DISEASE | angiogenesis inducing agents | macrophages | ACCESSORY MOLECULES | PERIPHERAL VASCULAR DISEASE | OXIDIZED PHOSPHOLIPIDS | STRESS | HEMATOLOGY | MACROPHAGE SUBSETS | toll-like receptor 2 | CD36 Antigens - deficiency | CD36 Antigens - genetics | Toll-Like Receptor 2 - genetics | Apolipoproteins E - deficiency | Oxidative Stress | Atherosclerosis - genetics | Humans | Melanoma, Experimental - metabolism | Wound Healing | Toll-Like Receptor 2 - deficiency | Transfection | CD36 Antigens - metabolism | RNA Interference | Time Factors | HEK293 Cells | Antigens, Differentiation - metabolism | Lipoproteins, LDL - metabolism | Disease Models, Animal | Pyrroles - metabolism | Signal Transduction | Mice, Inbred C57BL | Macrophages, Peritoneal - immunology | Melanoma, Experimental - pathology | Toll-Like Receptor 2 - metabolism | Tumor Burden | Atherosclerosis - metabolism | Mice, Knockout | Phenotype | Animals | Apolipoproteins E - genetics | Macrophages, Peritoneal - metabolism | Neovascularization, Physiologic | Oxidized lipids | angiogenesis | macrophage | CEP | TLR2
Journal Article
Journal Article
Heart, ISSN 1355-6037, 10/2012, Volume 98, Issue Suppl 2, p. E62
Objectives Cardiovascular disease resulting in myocardial infarction (MI) is the leading cause of mortality in developed countries; however, the mechanisms... 
Journal Article