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Muscle & Nerve, ISSN 0148-639X, 07/2018, Volume 58, Issue 1, pp. 42 - 48
Introduction : More than 90% of amyotrophic lateral sclerosis (ALS) patients have muscle cramps, but evidence‐based treatments have not been available. Methods... 
neuromuscular | quality of life | amyotrophic lateral sclerosis | Muscle cramp | fasciculations | RILUZOLE | NERVE | PLACEBO | EFFICACY | SAFETY | ALS | MOTOR-NEURON DISEASE | NEUROSCIENCES | CLINICAL NEUROLOGY | EXCITABILITY PROPERTIES | SYMPTOMATIC TREATMENT | Reduction | Cramps | Muscles | Amyotrophic lateral sclerosis | Muscle pain | Patients | Sclerosis
Journal Article
by van Rheenen, Wouter and Shatunov, Aleksey and Dekker, Annelot M and McLaughlin, Russell L and Diekstra, Frank P and Pulit, Sara L and van der Spek, Rick A. A and Võsa, Urmo and de Jong, Simone and Robinson, Matthew R and Yang, Jian and Fogh, Isabella and van Doormaal, Perry Tc and Tazelaar, Gijs H. P and Koppers, Max and Blokhuis, Anna M and Sproviero, William and Jones, Ashley R and Kenna, Kevin P and van Eijk, Kristel R and Harschnitz, Oliver and Schellevis, Raymond D and Brands, William J and Medic, Jelena and Menelaou, Anoniki and Vajda, Alice and Ticozzi, Nicola and Lin, Kuang and Rogelj, Boris and Vrabec, Katarina and Ravnik-Glavač, Metka and Koritnik, Blaž and Zidar, Janez and Leonardis, Lea and Grošelj, Leja Dolenc and Millecamps, Stéphanie and Salachas, François and Meininger, Vincent and de Carvalho, Mamede and Pinto, Susana and Mora, Jesus S and Rojas-García, Ricardo and Polak, Meraida and Chanan, Siddharthan and Colville, Shuna and Swingler, Robert and Morrison, Karen E and Shaw, Pamela J and Hardy, John and Orrell, Richard W and Pittman, Alan and Sidle, Katie and Fratta, Pietro and Malaspina, Anea and Topp, Simon and Petri, Susanne and Abdulla, Susanne and pper, Carsten and Sendtner, Michael and Meyer, Thomas and Ophoff, Roel A and Staats, Kim A and Wiedau-Pazos, Martina and Lomen-Hoerth, Catherine and van Deerlin, Vivianna M and Trojanowski, John Q and Elman, Lauren and McCluskey, Leo and Basak, A. Nazli and Tunca, Ceren and Hamzeiy, Hamid and Parman, Yesim and Meitinger, Thomas and Lichtner, Peter and Radivojkov-Blagojevic, Milena and Anes, Christian R and Maurel, Cindy and Bensimon, Gilbert and Landwehrmeyer, Bernhard and Brice, Alexis and Payan, Christine A. M and Saker-Delye, Safaa and Dürr, Alexana and Wood, Nicholas W and Tittmann, Lukas and Lieb, Wolfgang and Franke, Ane and Rietschel, Marcella and Cichon, Sven and Nöthen, Markus M and Amouyel, Philippe and Tzourio, Christophe and Dartigues, Jean-François and Uitterlinden, Ane G and Rivadeneira, Fernando and Estrada, Karol and Hofman, Albert and Curtis, Charles and Blauw, Hylke M and van der Kooi, Anneke J and ... and PARALS Registry and FALS Sequencing Consortium and SLALOM Grp and SLAGEN Consortium and NNIPPS Study Grp and SLAP Registry and SLALOM Group and NNIPPS Study Group and Medicinska fakulteten and Institutionen för farmakologi och klinisk neurovetenskap and Klinisk neurovetenskap and Umeå universitet
Nature genetics, ISSN 1061-4036, 2016, Volume 48, Issue 9, pp. 1043 - 1048
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 10/2009, Volume 111, Issue 2, pp. 332 - 343
In amyotrophic lateral sclerosis, down-regulation of the astrocyte-specific glutamate excitatory amino acid transporter 2 is hypothesized to increase... 
XCT | Amyotrophic lateral sclerosis | Excitatory amino acid transporter | Ceftriaxone | Nrf2 | Glutathione | OXIDATIVE-STRESS | glutathione | BIOCHEMISTRY & MOLECULAR BIOLOGY | xCT | AMYOTROPHIC-LATERAL-SCLEROSIS | GLUTAMATE TRANSPORTER EXPRESSION | CEREBROSPINAL-FLUID | amyotrophic lateral sclerosis | NEUROSCIENCES | ceftriaxone | excitatory amino acid transporter | ANTIOXIDANT RESPONSE ELEMENT | MOTOR-NEURON DEGENERATION | GENE-EXPRESSION | HUNTINGTONS-DISEASE | SUPEROXIDE-DISMUTASE | CELL-LINE | Superoxide Dismutase - genetics | Glutathione - metabolism | Humans | Ceftriaxone - pharmacology | Stem Cells - cytology | Amino Acid Transport Systems, Acidic | Amino Acid Transport System y+ - metabolism | Neuroprotective Agents - pharmacology | Motor Neurons - cytology | Spinal Cord - cytology | Motor Neurons - drug effects | Superoxide Dismutase - metabolism | Fibroblasts - metabolism | Astrocytes - cytology | Astrocytes - drug effects | Cell Line | Rats | Hippocampus - cytology | Mice, Knockout | Motor Neurons - metabolism | Animals | Fibroblasts - drug effects | NF-E2-Related Factor 2 - metabolism | Anti-Bacterial Agents - pharmacology | Fibroblasts - cytology | Mice | Oxidative Stress - drug effects | Superoxide Dismutase-1 | Glutamic Acid - toxicity | In Vitro Techniques | Astrocytes - metabolism | Cysteine | Neurons | Superoxide dismutase | Superoxide | Glutamate | Cystine | Antioxidants | Messenger RNA | Antibiotics | Ceftriaxone sodium | Neurology | Amino acids | Biochemistry | Index Medicus
Journal Article
Journal of Neuroinflammation, ISSN 1742-2094, 11/2010, Volume 7, Issue 1, pp. 76 - 76
Journal Article
Neuron, ISSN 0896-6273, 2002, Volume 34, Issue 4, pp. 509 - 519
Pathologic alterations in the microtubule-associated protein tau have been implicated in a number of neurodegenerative disorders, including Alzheimer's disease... 
GLYCOGEN-SYNTHASE KINASE-3-BETA | SEGMENT POLARITY GENE | ALZHEIMERS-DISEASE | FRONTOTEMPORAL DEMENTIA | PAIRED HELICAL FILAMENTS | HUMAN BRAIN-TAU | DISEASE-LIKE PHOSPHORYLATION | NUCLEAR LAMIN ISOFORMS | BETA-CATENIN | NEUROSCIENCES | TRANSGENIC MICE | Neurofibrillary Tangles - genetics | Cytoskeletal Proteins - genetics | Humans | tau Proteins - metabolism | Apoptosis - genetics | Glycogen Synthase Kinase 3 | Photoreceptor Cells, Invertebrate - ultrastructure | Drosophila melanogaster - metabolism | Glycogen Synthase Kinases | tau Proteins - genetics | Armadillo Domain Proteins | Nervous System Malformations - metabolism | Cytoskeletal Proteins - metabolism | Insect Proteins - ultrastructure | Nervous System Malformations - genetics | Eye Abnormalities - metabolism | Insect Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Repressor Proteins - metabolism | Disease Models, Animal | Neurofibrillary Tangles - pathology | tau Proteins - ultrastructure | Drosophila Proteins | Calcium-Calmodulin-Dependent Protein Kinases - genetics | High Mobility Group Proteins - metabolism | Inhibitor of Apoptosis Proteins | beta Catenin | Animals, Genetically Modified | Nervous System Malformations - pathology | Trans-Activators | Transgenes - genetics | Protein-Serine-Threonine Kinases - genetics | Insect Proteins - genetics | Repressor Proteins - genetics | Drosophila melanogaster - ultrastructure | Eye Abnormalities - genetics | Mutation - genetics | Phenotype | Animals | Eye Abnormalities - pathology | Photoreceptor Cells, Invertebrate - pathology | Photoreceptor Cells, Invertebrate - abnormalities | Drosophila melanogaster - growth & development | Neurofibrillary Tangles - ultrastructure | Transcription Factors | Calcium-Calmodulin-Dependent Protein Kinases - metabolism | High Mobility Group Proteins - genetics | Proteins | Pathology | Scanning electron microscopy | Genotype & phenotype | Phosphorylation | Insects | Neurodegeneration | Mutation | Kinases | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 2006, Volume 51, Issue 5, pp. 549 - 560
Journal Article