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1. Full Text Investigating the Causal Relationship of C-Reactive Protein with 32 Complex Somatic and Psychiatric Outcomes: A Large-Scale Cross-Consortium Mendelian Randomization Study
by Prins, B.P and Abbasi, A and Wong, A and Vaez, Ahmad and Nolte, Ilja and Franceschini, Nora and Stuart, P.E and Guterriez Achury, J and Mistry, V and Bradfield, Jonathan and Valdes, Ana Maria and Bras, Jose and Shatunov, Aleksey and Lu, Chao and Han, B and Raychaudhuri, Soumya and Bevan, Steve and Mayes, M.D and Tsoi, L.C and Evangelou, Evangelos and Nair, Rajan P and Grant, Struan and Polychronakos, Constantin and Radstake, Timothy and Heel, David and Dunstan, M.L and Wood, Nicholas and Al-Chalabi, Ammar and Dehghan, Abbas and Hakonarson, Hakon and Markus, H.S and Elder, James and Knight, J and Arking, Dan and Spector, Timothy and Koeleman, Bobby and Duijn, Cornelia and Martín, Javier and Morris, Anew and Weersma, Rinse K and Wijmenga, Cisca and Munroe, Patricia and Perry, John and Pouget, J.G and Jamshidi, Yalda and Snieder, Harold and Alizadeh, Behrooz and Inflammation Working Grp CHARGE Co and Autism Spectrum Disorder Working G and PAGE Consortium and Int Consortium Blood Pressure and Int Parkinson Dis Genomics Consort and Schizophrenia Working Grp Psychiat and DIAGRAM Consortium and CARDIoGRAMplusC4d Consortium and Int Stroke Genetics Consortium and Treat OA Consortium and ALS Consortium and CKDGen Consortium and Systemic Sclerosis Consortium and GERAD1 Consortium and International Consortium for Blood Pressure and International Stroke Genetics Consortium and Treat OA consortium and ALS consortium and CKDGen consortium and Schizophrenia Working Group of the Psychiatric Genomics Consortium and Systemic Sclerosis consortium and Inflammation Working Group of the CHARGE Consortium and International Parkinson’s Disease Genomics Consortium and CARDIoGRAMplusC4D Consortium and Autism Spectrum Disorder Working Group of the Psychiatric Genomics Consortium
PLoS Medicine, ISSN 1549-1277, 06/2016, Volume 13, Issue 6, p. e1001976
textabstractBackground: C-reactive protein (CRP) is associated with immune, cardiometabolic, and psychiatric traits and diseases. Yet it is inconclusive... 
ALL-CAUSE MORTALITY | COMMON VARIANTS | MEDICINE, GENERAL & INTERNAL | CARDIOVASCULAR-DISEASE | BODY-MASS INDEX | GENETIC-VARIANTS | SUSCEPTIBILITY LOCUS | CORONARY-HEART-DISEASE | BIPOLAR DISORDER | GENOME-WIDE ASSOCIATION | INFLAMMATORY MARKERS | Mental Disorders - genetics | Genome-Wide Association Study | C-Reactive Protein - metabolism | Immune System Diseases - genetics | Humans | Mendelian Randomization Analysis | Metabolic Diseases - genetics | Genotype | C-Reactive Protein - genetics | Polymorphism, Single Nucleotide | Heart Diseases - genetics | Type 2 diabetes | C-reactive protein | Psoriasis | Genomics | Albumin | Schizophrenia | Crohn's disease | Coronary heart disease | Investigations | Consortia | Rheumatoid factor | Analysis | Genetic aspects | Single nucleotide polymorphisms | Osteoarthritis | Chromosomes | Blood proteins | Outcome and process assessment (Health Care) | Research | Mental illness | Risk factors | Studies | Medical research | Bipolar disorder | Health risk assessment
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2. Full Text Association between C reactive protein and coronary heart disease: mendelian randomisation analysis based on individual participant data
by Wensley, Frances and Gao, Pei and Burgess, Stephen and Kaptoge, Stephen and Di Angelantonio, Emanuele and Shah, Tina and Engert, James C and Clarke, Robert and Davey-Smith, George and Nordestgaard, Børge G and Saleheen, Danish and Samani, Nilesh J and Sandhu, Manjinder and Anand, Sonia and Pepys, Mark B and Smeeth, Liam and Whittaker, John and Casas, Juan Pablo and Thompson, Simon G and Hingorani, Aroon D and Danesh, John and Eiriksdottir, G and Harris, T. B and Launer, L. J and Gudnason, V and Folsom, A. R and Anews, G and Ballantyne, C. M and Samani, N. J and Hall, A. S and Braund, P. S and Balmforth, A. J and Whincup, P. H and Morris, R and Lawlor, D. A and Lowe, G. D. O and Timpson, N and Ebrahim, S and Ben-Shlomo, Y and Davey-Smith, G and Nordestgaard, B. J and Tybjærg-Hansen, A and Zacho, J and Brown, M and Sandhu, M and Ricketts, S. L and Ashford, S and Lange, L and Reiner, A and Cushman, M and Tracy, R and Wu, C and Ge, J and Zou, Y and Sun, A and Hung, J and McQuillan, B and Thompson, P and Beilby, J and Warrington, N and Palmer, L. J and Wanner, C and chsler, C and Hoffmann, M. M and Fowkes, F. G. R and Tzoulaki, I and Kumari, M and Miller, M and Marmot, M and Onland-Moret, C and van der Schouw, Y. T and Boer, J. M and Wijmenga, C and Khaw, K.-T and Vasan, R. S and Schnabel, R. B and Yamamoto, J. F and Benjamin, E. J and Schunkert, H and Erdmann, J and König, I. R and Hengstenberg, C and Chiodini, B and Franzosi, M. G and Pietri, S and Gori, F and Rudock, M and Liu, Y and Lohman, K and Humphries, S. E and Hamsten, A and Norman, P. E and Hankey, G. J and Jamrozik, K and Rimm, E. B and Pai, J. K and Psaty, B. M and Heckbert, S. R and Bis, J. C and Yusuf, S and ... and EAS and INTERHEART and CRPHANS and LOLIPOP and FRAMOFF and PROSPER and SPEED and NPHSII and GERMIFS and HVHS and CHAOS and ISIS and PENNCATH and ROTTERDAM and CCHS CGPS CIHDS and ELSA and C Reactive Prot Coronary Heart Dis and HPFS and HIFMECH and 4D and EPICNOR and BWHHS and GISSI-P and MALMO and ARIC and PROMIS and SHEEP and WOSCOPS and HEALTHABC and WHIOS and BHF-FHS and EPICNL and NHS and LURIC and PROCARDIS and AGES and UCP and BRHS and CHS and CAPS and HIMS and CUDAS CUPID and MONICAKORA and NSC and WHITEII and C Reactive Protein Coronary Heart Disease Genetics Collaboration (CCGC)
BMJ, ISSN 0959-8138, 2011, Volume 342, Issue 7794, pp. 425 - 425
Objective To use genetic variants as unconfounded proxies of C reactive protein concentration to study its causal role in coronary heart disease.Design... 
RESEARCH | Disease risk | Coronary artery disease | Medical genetics | Predisposing factors | MEDICINE, GENERAL & INTERNAL | METAANALYSIS | POLYMORPHISMS | MYOCARDIAL-INFARCTION | GENE | INSTRUMENTAL VARIABLES | INFLAMMATION | CARDIOVASCULAR-DISEASE | ATHEROSCLEROSIS | MARKERS | RISK | Coronary Disease - blood | Prospective Studies | Gene Frequency | Humans | Middle Aged | Risk Factors | Male | C-Reactive Protein - genetics | C-Reactive Protein - metabolism | Coronary Disease - genetics | Mendelian Randomization Analysis | Polymorphism, Single Nucleotide - genetics | Female
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3. Full Text Plasma HDL cholesterol and risk of myocardial infarction: a mendelian randomisation study
by Voight, Benjamin F, PhD and Peloso, Gina M, PhD and Orho-Melander, Marju, PhD and Frikke-Schmidt, Ruth, DMSc and Barbalic, Maja, PhD and Jensen, Majken K, PhD and Hindy, George, MD and Hólm, Hilma, MD and Ding, Eric L, ScD and Johnson, Toby, PhD and Schunkert, Heribert, Prof and Samani, Nilesh J, Prof and Clarke, Robert, MD and Hopewell, Jemma C, PhD and Thompson, John F, Prof and Li, Mingyao, PhD and Thorleifsson, Gudmar, PhD and Newton-Cheh, Christopher, MD and Musunuru, Kiran, MD and Pirruccello, James P, BS and Saleheen, Danish, MBBS and Chen, Li, MSc and Stewart, Alexandre FR, PhD and Schillert, Arne, PhD and Thorsteinsdottir, Unnur, PhD and Thorgeirsson, Gudmundur, MD and Anand, Sonia, Prof and Engert, James C, PhD and Morgan, Thomas, MD and Spertus, John, Prof and Stoll, Monika, Prof and Berger, Klaus, Prof and Martinelli, Nicola, MD and Girelli, Domenico, Prof and McKeown, Pascal P, Prof and Patterson, Christopher C, PhD and Epstein, Stephen E, MD and Devaney, Joseph, PhD and Burnett, Mary-Susan, PhD and Mooser, Vincent, MD and Ripatti, Samuli, PhD and Surakka, Ida, BSci and Nieminen, Markku S, Prof and Sinisalo, Juha, MD and Lokki, Marja-Liisa, PhD and Perola, Markus, Prof and Havulinna, Aki, PhD and de Faire, Ulf, Prof and Gigante, Bruna, MD and Ingelsson, Erik, PhD and Zeller, Tanja, PhD and Wild, Philipp, MD and de Bakker, Paul I W, Prof and Klungel, Olaf H, PhD and Maitland-van der Zee, Anke-Hilse, PhD and Peters, Bas J M, PhD and de Boer, Anthonius, Prof and Grobbee, Diederick E, Prof and Kamphuisen, Pieter W, PhD and Deneer, Vera H M, PhD and Elbers, Clara C, PhD and Onland-Moret, N Charlotte, PhD and Hofker, Marten H, Prof and Wijmenga, Cisca, Prof and Verschuren, WM Monique, PhD and Boer, Jolanda MA, PhD and van der Schouw, Yvonne T, Prof and Rasheed, Asif and Frossard, Philippe and Demissie, Serkalem, PhD and Willer, Cristen, PhD and Do, Ron, PhD and Ordovas, Jose M, Prof and Abecasis, Gonçalo R, Prof and Boehnke, Michael, Prof and Mohlke, Karen L, PhD and Daly, Mark J, PhD and Guiducci, Candace, BS and Burtt, Noël P, BS and Surti, Aarti, BS and Gonzalez, Elena, BS and Purcell, Shaun, PhD and Gabriel, Stacey, PhD and Marrugat, Jaume, PhD and Peden, John, PhD and Erdmann, Jeanette, PhD and Diemert, Patrick, MD and Willenborg, Christina, MSc and König, Inke R, PhD and Fischer, Marcus, MD and Hengstenberg, Christian, MD and Ziegler, Andreas, PhD and Buysschaert, Ian, MD and Lambrechts, Diether, PhD and Van de Werf, Frans, Prof and Fox, Keith A, Prof and El Mokhtari, Nour Eddine, MD and Rubin, Diana, MD and Schrezenmeir, Jürgen, PhD and Schreiber, Stefan, Prof and ...
Lancet, The, ISSN 0140-6736, 08/2012, Volume 380, Issue 9841, pp. 572 - 580
Summary Background High plasma HDL cholesterol is associated with reduced risk of myocardial infarction, but whether this association is causal is unclear.... 
Internal Medicine | ENDOTHELIAL LIPASE | MEDICINE, GENERAL & INTERNAL | CONTRIBUTE | SUSCEPTIBILITY | CARDIOVASCULAR-DISEASE | ATHEROSCLEROSIS | GENETIC-VARIANTS | LOCI | ASSOCIATION | HIGH-DENSITY-LIPOPROTEIN | Mendelian Randomization Analysis - methods | Myocardial Infarction - blood | Myocardial Infarction - genetics | Genetic Predisposition to Disease | Myocardial Infarction - epidemiology | Prospective Studies | Gene Frequency | Humans | Risk Factors | Biomarkers - blood | Case-Control Studies | Cholesterol, HDL - blood | Cholesterol, LDL - blood | Polymorphism, Single Nucleotide | Myocardial Infarction - etiology | Lipase - genetics | Genetic aspects | High density lipoproteins | Health aspects | Heart attack | Risk factors | Studies | Plasma | Heart attacks | Diet | Cardiovascular disease | Lipids | Cholesterol | Medical and Health Sciences | Medicin och hälsovetenskap
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4. Full Text Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease
by Jostins, Luke and Ripke, Stephan and Weersma, Rinse K and Duerr, Richard H and McGovern, Dermot P and Hui, Ken Y and Lee, James C and Schumm, L. Philip and Sharma, Yashoda and Anderson, Carl A and Essers, Jonah and Mitrovic, Mitja and Ning, Kaida and Cleynen, Isabelle and Theatre, Emilie and Spain, Sarah L and Raychaudhuri, Soumya and Goyette, Philippe and Wei, Zhi and Abraham, Clara and Achkar, Jean-Paul and Ahmad, Tariq and Amininejad, Leila and Ananthakrishnan, Ashwin N and Andersen, Vibeke and Anews, Jane M and Baidoo, Leonard and Balschun, Tobias and Bampton, Peter A and Bitton, Alain and Boucher, Gabrielle and Brand, Stephan and Büning, Carsten and Cohain, Ariella and Cichon, Sven and D'Amato, Mauro and de Jong, Dirk and Devaney, Kathy L and Dubinsky, Marla and Edwards, Cathryn and Ellinghaus, David and Ferguson, Lynnette R and Franchimont, Denis and Fransen, Karin and Gearry, Richard and Georges, Michel and Gieger, Christian and Glas, Jürgen and Haritunians, Talin and Hart, Ailsa and Hawkey, Chris and Hedl, Matija and Hu, Xinli and Karlsen, Tom H and Kupcinskas, Limas and Kugathasan, Subra and Latiano, Anna and Laukens, Debby and Lawrance, Ian C and Lees, Charlie W and Louis, Edouard and Mahy, Gillian and Mansfield, John and Morgan, Angharad R and Mowat, Craig and Newman, William and Palmieri, Orazio and Ponsioen, Cyriel Y and Potocnik, Uros and Prescott, Natalie J and Regueiro, Miguel and Rotter, Jerome I and Russell, Richard K and Sanderson, Jeremy D and Sans, Miquel and Satsangi, Jack and Schreiber, Stefan and Simms, Lisa A and Sventoraityte, Jurgita and Targan, Stephan R and Taylor, Kent D and Tremelling, Mark and Verspaget, Hein W and de Vos, Martine and Wijmenga, Cisca and Wilson, David C and Winkelmann, Juliane and Xavier, Ramnik J and Zeissig, Sebastian and Zhang, Bin and Zhang, Clarence K and Zhao, Hongyu and Silverberg, Mark S and Annese, Vito and Hakonarson, Hakon and Brant, Steven R and Radford-Smith, Graham and Mathew, Christopher G and Rioux, John D and Schadt, Eric E and ... and Int IBD Genetics Consortium IIBDGC and International IBD Genetics Consortium (IIBDGC) and The International IBD Genetics Consortium (IIBDGC) and Region Örebro län and Örebro universitet and Institutionen för medicinska vetenskaper
Nature, ISSN 0028-0836, 2012, Volume 491, Issue 7422, pp. 119 - 124
Crohn's disease and ulcerative colitis, the two common forms of inflammatory bowel disease (IBD), affect over 2.5 million people of European ancestry, with... 
HYPER-IGE SYNDROME | RISK LOCI | NETWORK | NUMBER | METAANALYSIS | TUBERCULOSIS | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | MUTATIONS | EXPRESSION | Crohn Disease - genetics | Humans | Inflammatory Bowel Diseases - immunology | Colitis, Ulcerative - genetics | Mycobacterium tuberculosis - immunology | Inflammatory Bowel Diseases - physiopathology | Colitis, Ulcerative - immunology | Host-Pathogen Interactions - immunology | Mycobacterium tuberculosis - pathogenicity | Haplotypes - genetics | Mycobacterium - pathogenicity | Mycobacterium Infections - genetics | Inflammatory Bowel Diseases - genetics | Crohn Disease - microbiology | Mycobacterium Infections - microbiology | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Reproducibility of Results | Mycobacterium - immunology | Crohn Disease - immunology | Genome, Human - genetics | Phenotype | Colitis, Ulcerative - microbiology | Host-Pathogen Interactions - genetics | Polymorphism, Single Nucleotide - genetics | Inflammatory Bowel Diseases - microbiology | Crohn Disease - physiopathology | Colitis, Ulcerative - physiopathology | Inflammatory bowel disease | Candidates | Tuberculosis | Architecture | Genetics | Genomes | Diabetes | Gene expression | Health risk assessment | Immune system | Rheumatology and Autoimmunity | Basic Medicine | Medical Genetics | Medicinsk genetik | Clinical Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Reumatologi och inflammation | Medicinska och farmaceutiska grundvetenskaper | Klinisk medicin
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5. Full Text Discovery and refinement of loci associated with lipid levels
by Willer, Cristen J and Schmidt, Ellen M and Sengupta, Sebanti and Peloso, Gina M and Gustafsson, Stefan and Kanoni, Stavroula and Ganna, Anea and Chen, Jin and Buchkovich, Martin L and Mora, Samia and Beckmann, Jacques S and Bragg-Gresham, Jennifer L and Chang, Hsing-Yi and Demirkan, Ayşe and den Hertog, Heleen M and Do, Ron and Donnelly, Louise A and Ehret, Georg B and Esko, Tõnu and Feitosa, Mary F and Ferreira, Teresa and Fischer, Krista and Fontanillas, Pierre and Fraser, Ross M and Freitag, Daniel F and Gurdasani, Deepti and Heikkilä, Kauko and Hyppönen, Elina and Isaacs, Aaron and Jackson, Anne U and Johansson, Asa and Johnson, Toby and Kaakinen, Marika and Kettunen, Johannes and Kleber, Marcus E and Li, Xiaohui and Luan, Jian'an and Lyytikäinen, Leo-Pekka and Magnusson, Patrik K. E and Mangino, Massimo and Mihailov, Evelin and Montasser, May E and Müller-Nurasyid, Martina and Nolte, Ilja M and O'Connell, Jeffrey R and Palmer, Cameron D and Perola, Markus and Petersen, Ann-Kristin and Sanna, Serena and Saxena, Richa and Service, Susan K and Shah, Sonia and Shungin, Dmitry and Sidore, Carlo and Song, Ci and Strawbridge, Rona J and Surakka, Ida and Tanaka, Toshiko and Teslovich, Tanya M and Thorleifsson, Gudmar and van den Herik, Evita G and Voight, Benjamin F and Volcik, Kelly A and Waite, Lindsay L and Wong, Anew and Wu, Ying and Zhang, Weihua and Absher, Devin and Asiki, Gershim and Barroso, Inês and Been, Latonya F and Bolton, Jennifer L and Bonnycastle, Lori L and Brambilla, Paolo and Burnett, Mary S and Cesana, Giancarlo and Dimitriou, Maria and Doney, Alex S. F and Döring, Angela and Elliott, Paul and Epstein, Stephen E and Eyjolfsson, Gudmundur Ingi and Gigante, Bruna and Goodarzi, Mark O and Grallert, Harald and Gravito, Martha L and Groves, Christopher J and Hallmans, Göran and Hartikainen, Anna-Liisa and Hayward, Caroline and Hernandez, Dena and Hicks, Anew A and Holm, Hilma and Hung, Yi-Jen and Illig, Thomas and Jones, Michelle R and Kaleebu, Pontiano and Kastelein, John J. P and Khaw, Kay-Tee and Kim, Eric and ... and Global Lipids Genetics Consortium and Medicinska fakulteten and Institutionen för folkhälsa och klinisk medicin and Institutionen för odontologi and Näringsforskning and Umeå universitet and Medicin
Nature genetics, ISSN 1061-4036, 2013, Volume 45, Issue 11, pp. 1274 - 1283
Levels of low-density lipoprotein (LDL) cholesterol, high-density lipoprotein (HDL) cholesterol, triglycerides and total cholesterol are heritable, modifiable... 
DENSITY-LIPOPROTEIN CHOLESTEROL | HEART-DISEASE | HOMEOSTASIS | METAANALYSIS | PLASMA | GENE | GENETICS & HEREDITY | SUSCEPTIBILITY LOCI | RISK | IDENTIFIES 13 | TRAITS | Quantitative trait loci | Proteolipids | Lipoproteins | Genetic aspects | Blood lipoproteins | Biological control systems | Observations | Identification and classification | Studies | Lipids | Genes | Cholesterol | Basic Medicine | Medical Genetics | Medicinsk genetik | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
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