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The New England Journal of Medicine, ISSN 0028-4793, 04/2016, Volume 374, Issue 14, pp. 1321 - 1331
Journal Article
Experimental Brain Research, ISSN 0014-4819, 2002, Volume 145, Issue 3, pp. 286 - 296
Journal Article
Archives of Physical Medicine and Rehabilitation, ISSN 0003-9993, 12/2002, Volume 83, Issue 12, pp. 1732 - 1735
Journal Article
Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques, ISSN 0317-1671, 02/1997, Volume 24, Issue 1, pp. 171 - 172
Journal Article
Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques, ISSN 0317-1671, 08/1997, Volume 24, Issue 3, pp. 261 - 262
Journal Article
Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques, ISSN 0317-1671, 02/1994, Volume 21, Issue 1, pp. 81 - 82
Journal Article
Canadian Journal of Neurological Sciences, ISSN 0317-1671, 1989, Volume 16, Issue 1, pp. 67 - 70
A 53 year old man developed symptoms of motor neuron disease in childhood. There was a family history of a similar disorder and it was felt to represent a form... 
CLINICAL NEUROLOGY | Pedigree | Humans | Middle Aged | Neurons, Afferent - pathology | Male | Nerve Degeneration | Neuromuscular Diseases - pathology | Motor Neurons - pathology | Neuromuscular Diseases - genetics | Index Medicus
Journal Article
by Kasner, Scott E and Kasner, SE and Swaminathan, Balakumar and Lavados, Pablo and Lavados, P and Sharma, M and Sharma, Mukul and Muir, Keith and Muir, KW and Veltkamp, Roland and Veltkamp, R and Ameriso, Sebastian F and Ameriso, SF and Endres, M and Endres, Matthias and Lutsep, H and Lutsep, Helmi and Messé, Steven R and Spence, J David and Spence, D and Nedeltechev, Krassen and Perera, Kanjana and Perera, K and Santo, G and Santo, Gustavo and Olavarria, Veronica and Olavarria, V and Lindgren, Arne and Lindgren, A and Bangdiwala, Shrikant and Shoamanesh, Ashkan and Shoamanesh, A and Berkowitz, Scott D and Berkowitz, SD and Mundl, Hardi and Mundl, H and Connolly, Stuart J and Connolly, SJ and Hart, Robert G and Hart, RG and Abdelhamid, N and Abdul Rahman, D and Abdul-Saheb, M and Abreu, P and Abroskina, M and Abu Ahmad, F and Accassat, S and Acciaresi, M and Adami, A and Ahmad, N and Ahmed, F and Alberto Hawkes, M and Alemseged, F and Ali, A and Altavilla, R and Alwis, L and Amarenco, P and Amaro, S and Amaya Sanchez, LE and Amelia Pinto, A and Amin, H and Amino, T and Amjad, AK and Anagnostou, E and Andersen, G and Anderson, C and Anderson, DC and Andrea Falco, M and Andres Mackinnon, F and Andreu, D and Androulakis, M and Angel Gamero, M and Angel Saredo, G and Angeles Diaz, R and Angels Font, M and Anticoli, S and Arauz, A and Arauz Gongora, AA and Araya, P and Arenillas Lara, JF and Arias Rivas, S and Arnold, M and Augustin, S and Avelar, W and Azevedo, E and Babikian, V and Bacellar, A and Badalyan, K and Bae, HJ and Baez Martinez, EM and Bagelmann, H and Bailey, P and Bak, Z and Baker, M and Balazs, A and Baldaranov, D and Balogun, I and Balueva, T and Bankuti, Z and Bar, M and ... and NAVIGATE ESUS Investigators and Klinisk strokeforskning and EpiHealth: Epidemiology for Health and Clinical Stroke Research Group and Lund University and Lunds universitet
The Lancet Neurology, ISSN 1474-4422, 12/2018, Volume 17, Issue 12, pp. 1053 - 1060
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, pp. e45323 - e45323
Ca2+/calmodulin-dependent protein kinase II (CaMKII) is abundant in striatal medium spiny neurons (MSNs). CaMKII is dynamically regulated by changes in... 
AMPA RECEPTORS | GLUR-A-DEFICIENT | BASAL GANGLIA | MULTIDISCIPLINARY SCIENCES | POSTSYNAPTICALLY SILENT SYNAPSES | DEVELOPMENTAL-CHANGES | LONG-TERM POTENTIATION | RAT-BRAIN | D-ASPARTATE RECEPTOR | PROTEIN-KINASE-II | SQUID GIANT SYNAPSE | Synaptic Transmission - physiology | Synaptic Transmission - genetics | Peptides - genetics | Male | Mice, Transgenic | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - antagonists & inhibitors | Corpus Striatum - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - genetics | Excitatory Postsynaptic Potentials - physiology | Animals | Peptides - metabolism | Female | Mice | Neurons - metabolism | Receptors, AMPA - genetics | Excitatory Postsynaptic Potentials - genetics | Receptors, AMPA - metabolism | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Neurosciences | Phosphorylation | Parkinson's disease | Calcium | Laboratories | Physicians | Parkinsons disease | Kinases | α-Amino-3-hydroxy-5-methyl-4-isoxazole propionic acid receptors | Proteins | Depolarization | Dendritic spines | Clonal deletion | Synaptic transmission | Transgenic animals | α-Amino-3-hydroxy-5-methyl-4-isoxazole propionic acid | Rodents | Calcium-binding protein | Neostriatum | Deletion | Physiology | Inhibition | Excitation | Calmodulin | Movement disorders | Dopamine | Neurodegenerative diseases | Neurons | Alcoholism | Transgenic mice | Excitability | Biophysics | Addictions | Spiny neurons | Medicine | Signaling | Excitatory postsynaptic potentials | Addiction | Cocaine | Hippocampus | Synapses | Ca2+/calmodulin-dependent protein kinase II | Index Medicus
Journal Article