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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2006, Volume 103, Issue 3, pp. 511 - 518
Journal Article
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 08/2012, Volume 111, Issue 6, pp. 708 - 717
RATIONALE:Atrial fibrillation (AF) is the most common cardiac arrhythmia, however the mechanism(s) causing AF remain poorly understood and therapy is... 
atrial fibrillation | Ca2sparks | ryanodine receptor | CPVT | SR Ca2leak | arrhythmia | CARDIAC & CARDIOVASCULAR SYSTEMS | RISK-FACTORS | Ca2+ sparks | PHOSPHORYLATION | HEART-FAILURE | SARCOPLASMIC-RETICULUM | FKBP12.6 DEFICIENCY | RELEASE | CHANNEL | SUDDEN CARDIAC DEATH | PERIPHERAL VASCULAR DISEASE | MICE | SR Ca2+ leak | HEMATOLOGY | CA2+ LEAK | Calcium - metabolism | Sarcoplasmic Reticulum - drug effects | Humans | Immunoblotting | Ryanodine Receptor Calcium Release Channel - metabolism | Tacrolimus Binding Proteins - metabolism | Atrial Fibrillation - physiopathology | Tachycardia, Ventricular - metabolism | Caffeine - pharmacology | Tachycardia, Ventricular - genetics | Sarcoplasmic Reticulum - metabolism | Disease Models, Animal | Heart - physiopathology | Myocytes, Cardiac - cytology | Cells, Cultured | Electrocardiography - drug effects | Thiazepines - pharmacology | Cardiac Pacing, Artificial | Atrial Fibrillation - genetics | Physical Conditioning, Animal - physiology | Gene Knock-In Techniques | Mice, Knockout | Tacrolimus Binding Proteins - genetics | Atrial Fibrillation - metabolism | Tachycardia, Ventricular - physiopathology | Animals | Ryanodine Receptor Calcium Release Channel - genetics | Heart - drug effects | Myocytes, Cardiac - metabolism | Mice | Mutation | Epinephrine - pharmacology | Index Medicus
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2015, Volume 21, Issue 11, pp. 1262 - 1271
Cancer-associated muscle weakness is a poorly understood phenomenon, and there is no effective treatment. Here we find that seven different mouse models of... 
MEDICINE, RESEARCH & EXPERIMENTAL | CANCER CACHEXIA | CELLS | INTRACELLULAR CALCIUM | BIOCHEMISTRY & MOLECULAR BIOLOGY | RELEASE | CELL BIOLOGY | DYSTROPHIC MUSCLE | GROWTH-FACTOR-BETA | HORMONE-RELATED PROTEIN | RYANODINE RECEPTOR | CAMURATI-ENGELMANN-DISEASE | IN-VIVO | Neoplasms - metabolism | Prostatic Neoplasms - metabolism | Up-Regulation | Calcium - metabolism | Humans | Lung Neoplasms - metabolism | NADPH Oxidases - metabolism | Bone Neoplasms - secondary | Lung Neoplasms - pathology | Male | Muscle, Skeletal - metabolism | Osteolysis - etiology | Ryanodine Receptor Calcium Release Channel - metabolism | X-Ray Microtomography | Bone Neoplasms - metabolism | Breast Neoplasms - metabolism | Neoplasms - complications | MCF-7 Cells | Osteolysis - diagnostic imaging | Muscle Proteins - metabolism | NADPH Oxidases - genetics | Female | Camurati-Engelmann Syndrome - metabolism | Muscle Strength | Calcium Signaling | Disease Models, Animal | Muscle Weakness - etiology | Prostatic Neoplasms - pathology | Oxidation-Reduction | Bone Neoplasms - diagnostic imaging | NADPH Oxidase 4 | Mice, SCID | Multiple Myeloma - metabolism | Absorptiometry, Photon | Osteolysis - metabolism | Multiple Myeloma - pathology | Animals | Muscle Contraction | Breast Neoplasms - pathology | Mice, Nude | Muscle Weakness - metabolism | Cell Line, Tumor | Mice | Neoplasms - pathology | Transforming Growth Factor beta - metabolism | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2015, Volume 21, Issue 11, p. 1262
  Cancer-associated muscle weakness is a poorly understood phenomenon, and there is no effective treatment. Here we find that seven different mouse models of... 
Genetically modified mice | Care and treatment | Analysis | Muscles | Research | Bone diseases | Transforming growth factors | Prostate cancer | Musculoskeletal system | Signal transduction | Bones | Metastasis | Growth factors | Cancer
Journal Article
Cell Metabolism, ISSN 1550-4131, 05/2012, Volume 15, Issue 5, pp. 739 - 751
Journal Article
ISSN 1091-6490, 2016
Endothelial cells (ECs) are critical mediators of blood pressure (BP) regulation, primarily via the generation and release of vasorelaxants, including nitric... 
Physiology | Blood pressure--Regulation | Medical sciences | Molecular biology | Calcium--Physiological effect | Endothelial cells
Journal Article
Circulation Research, ISSN 0009-7330, 07/2011, Volume 109, Issue 3, pp. 281 - 290
Journal Article
ISSN 0021-9738, 2014
Drugs currently approved to coat stents used in percutaneous coronary interventions do not discriminate between proliferating vascular smooth muscle cells... 
Medicine | Drugs | Genetics
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2010, Volume 120, Issue 12, p. 4375
Increased sarcoplasmic reticulum (SR) [Ca.sup.2+] leak via the cardiac ryanodine receptor/calcium release channel (RyR2) is thought to play a role in heart... 
Heart failure | Alkaloids | Care and treatment | Cell receptors | Development and progression | Properties | Health aspects
Journal Article