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1999, Di 1 ban., ISBN 7301040369, 2, 4, 5, 11, 2, 226
Book
2002, Di 1 ban., Hei hu, kong bu ti yan xi lie, ISBN 9787506323970, Volume 1, 247
Book
Advanced Materials Research, ISSN 1022-6680, 2012, Volume 472-475, pp. 427 - 431
Conference Proceeding
Science, ISSN 0036-8075, 4/2007, Volume 316, Issue 5824, pp. 575 - 579
The heart responds to diverse forms of stress by hypertrophic growth accompanied by fibrosis and eventual diminution of contractility, which results from... 
Heart | Untranslated regions | Up regulation | Messenger RNA | MicroRNA | Transgenic animals | Fibrosis | Mice | Gene expression regulation | Research Articles | Gene expression | HYPERTROPHY | MULTIDISCIPLINARY SCIENCES | CARDIOMYOPATHY | PHENOTYPE | PROLIFERATION | DIFFERENTIATION | THYROID-HORMONE RECEPTOR | HUMAN HEART | MYOSIN HEAVY-CHAIN | HYPOTHYROIDISM | TRANSGENIC MICE | Up-Regulation | Oligonucleotide Array Sequence Analysis | Hypothyroidism - physiopathology | Myosin Heavy Chains - genetics | Cardiomegaly - pathology | Triiodothyronine - metabolism | Gene Deletion | Myocardium - metabolism | Mediator Complex | Cardiac Myosins - metabolism | Heart - physiopathology | Myocardial Contraction | Signal Transduction | Heart Diseases - physiopathology | Introns | Cardiac Myosins - genetics | Gene Expression Regulation | Cardiomegaly - physiopathology | Rats | Mice, Transgenic | Myocardium - pathology | Stress, Physiological - physiopathology | Transcription Factors - genetics | Transcription Factors - metabolism | Myocytes, Cardiac - pathology | Animals | Ventricular Myosins - metabolism | MicroRNAs - genetics | MicroRNAs - physiology | Heart Diseases - genetics | Heart Diseases - pathology | Ventricular Myosins - genetics | Causes of | Research | Muscle contraction | Heart muscle | Proteins | Ribonucleic acid | Hormones | Ribonucleic acid--RNA | Stress | Index Medicus
Journal Article
Journal Article
Science, ISSN 0036-8075, 12/2009, Volume 326, Issue 5959, pp. 1549 - 1554
Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease characterized by loss of motor neurons, denervation of target muscles, muscle atrophy, and... 
Motor neurons | Up regulation | MicroRNA | Nerves | Muscles | Amyotrophic lateral sclerosis | Reports | T tests | Synapses | Skeletal muscle | Denervation | SKELETAL-MUSCLE | PROTEIN | MOTOR-NEURON DEGENERATION | RNA | TDP-43 | MULTIDISCIPLINARY SCIENCES | DISEASE | TARGETS | AMYOTROPHIC-LATERAL-SCLEROSIS | MUTATIONS | EXPRESSION | Up-Regulation | Amyotrophic Lateral Sclerosis - physiopathology | MyoD Protein - genetics | Transcriptional Activation | Muscle Denervation | MicroRNAs - metabolism | Muscle, Skeletal - innervation | Muscle, Skeletal - metabolism | Axons - physiology | Myogenin - genetics | Motor Neurons - pathology | Fibroblast Growth Factors - metabolism | RNA Interference | Neuromuscular Junction - pathology | Disease Models, Animal | Motor Neurons - physiology | Histone Deacetylases - genetics | Signal Transduction | Histone Deacetylases - metabolism | Mice, Transgenic | MyoD Protein - metabolism | Nerve Regeneration | Disease Progression | Carrier Proteins - genetics | Amyotrophic Lateral Sclerosis - pathology | Neuromuscular Junction - growth & development | Animals | Carrier Proteins - metabolism | Mice | MicroRNAs - genetics | Muscle, Skeletal - pathology | Myogenin - metabolism | Neuromuscular Junction - physiology | Fibroblast growth factors | Research | Regeneration (Biology) | Signal transduction | Cellular biology | Neurons | Rodents | Muscular system | Ribonucleic acid--RNA | Index Medicus
Journal Article
Genes and Development, ISSN 0890-9369, 07/2007, Volume 21, Issue 14, pp. 1790 - 1802
Journal Article
FASEB Journal, ISSN 0892-6638, 05/2017, Volume 31, Issue 5, pp. 1939 - 1952
Journal Article
Nature, ISSN 0028-0836, 2012, Volume 485, Issue 7400, pp. 599 - 604
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2013, Volume 110, Issue 34, pp. 13839 - 13844
The adult mammalian heart has limited potential for regeneration. Thus, after injury, cardiomyocytes are permanently lost, and contractility is diminished. In... 
Heart | Regeneration | Transcription factors | Ligation | Alleles | Fibrosis | Myocardium | Photoreceptors | Left coronary artery | Cardiac output | Cardiac fibrosis | Cardiomyopathy | Cell cycle | TRANSCRIPTION FACTORS | F-ACTIN | cell cycle | ORGAN SIZE CONTROL | MULTIDISCIPLINARY SCIENCES | CARDIOMYOCYTE PROLIFERATION | ZEBRAFISH HEART REGENERATION | MIR-15 FAMILY | TEAD/TEF FAMILY | cardiac fibrosis | SIGNALING PATHWAY | IN-VIVO | cardiomyopathy | HOLT-ORAM-SYNDROME | Echocardiography | Heart - physiology | Myocardial Contraction - physiology | Mice, Transgenic | DNA Primers - genetics | Phosphoproteins - genetics | Transcription Factors - genetics | Phosphoproteins - metabolism | Regeneration - physiology | Blotting, Western | Mutation, Missense - genetics | Tetrazolium Salts | Transcription Factors - metabolism | Myocardial Contraction - genetics | Animals | Histological Techniques | Myocytes, Cardiac - physiology | Adaptor Proteins, Signal Transducing - genetics | Myocytes, Cardiac - metabolism | Signal Transduction - physiology | Mice | Adaptor Proteins, Signal Transducing - metabolism | Protein-Serine-Threonine Kinases - metabolism | Proteins | Molecular genetics | Heart cells | Cellular control mechanisms | Physiological aspects | Genetic aspects | Regeneration (Biology) | Research | Signal transduction | Tissue engineering | Rodents | Cardiomyocytes | Insulin-like growth factors | Index Medicus | Biological Sciences
Journal Article
Journal Article