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2012, Minguo ji cui, 161
Book
2017, Yong yuan de chang zheng, ISBN 9787541749766
Book
Journal Article
Nature genetics, ISSN 1546-1718, 2019, Volume 51, Issue 6, pp. 999 - 1010
Journal Article
by Xu, Yan
09/2012, ISBN 7545415477
" Jie fang jun wei shen me neng ying " yi shu shi wei ji nian zhong guo ren min jie fang jun jian jun 85 zhou nian,ying jie dang de shi ba da sheng li zhao kai,gai shu yi jie fang jun 85 nian hui... 
Book
Nature communications, ISSN 2041-1723, 2016, Volume 7, Issue 1, p. 11422
.... Functionally, miR-216b directly targets c-Jun, thereby reducing AP-1-dependent transcription and sensitizing cells to ER... 
MESSENGER-RNA TRANSLATION | GLUCOSE-HOMEOSTASIS | UNFOLDED-PROTEIN RESPONSE | ALPHA-SUBUNIT | ER-STRESS | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | ENDOPLASMIC-RETICULUM STRESS | TRANSCRIPTION FACTOR | MAMMALIAN-CELLS | TRANSMEMBRANE PROTEIN
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2001, Volume 98, Issue 24, pp. 13681 - 13686
Jun N-terminal kinase (JNK) is a stress-activated protein kinase that can be induced by inflammatory cytokines, bacterial endotoxin, osmotic shock, UV radiation, and hypoxia... 
T lymphocytes | Enzymes | Biological Sciences | Phosphorylation | Cell growth | Nervous system diseases | Messenger RNA | Cytokines | Thymocytes | Inhibitory concentration 50 | Apoptosis | SIGNAL-TRANSDUCTION | APOPTOSIS | TUMOR-NECROSIS-FACTOR | ACTIVATION | MESSENGER-RNA | ANTICANCER AGENTS | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | EXPRESSION | BINDING | T-CELLS | Gene Expression - drug effects | Monocytes - cytology | Humans | Monocytes - metabolism | Structure-Activity Relationship | Pyrazolones | CD4-Positive T-Lymphocytes - immunology | Adenosine Triphosphate - metabolism | Enzyme Inhibitors - chemistry | JNK Mitogen-Activated Protein Kinases | Female | Molecular Structure | Binding, Competitive | Cytokines - metabolism | Enzyme Inhibitors - metabolism | Anthracenes - chemistry | Jurkat Cells | Pyrazoles | CD4-Positive T-Lymphocytes - cytology | Mice, Inbred C57BL | Cells, Cultured | Enzyme Inhibitors - pharmacology | Anthracenes - pharmacology | Protein Kinase Inhibitors | Animals | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Anthraquinones | Cell Differentiation - drug effects | Lymphocyte Activation - drug effects | Anthracenes - metabolism | Mice | Tumor Necrosis Factor-alpha - biosynthesis | CD4-Positive T-Lymphocytes - drug effects | Physiological aspects | Protein kinases | Enzyme inhibitors
Journal Article
2008, Di 1 ban., Zhongguo xian ming jia zhuan ji cong shu, ISBN 7508720679, 2, 256
Book
2012, ISBN 750656341X, 1 v.
Book
2012, Di 1 ban., 3, 399 p., 18 p. of plates
Book
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 05/2015, Volume 19, Issue 5, pp. 1151 - 1161
Extracellular high‐mobility group box‐1 (HMGB1) acts as a signalling molecule during inflammation, cell differentiation and angiogenesis. Increased abundance... 
HMGB1 | proliferation | inflammation | pulmonary hypertension | alarmins | Alarmins | Inflammation | Proliferation | Pulmonary hypertension | FIBROSIS | MEDICINE, RESEARCH & EXPERIMENTAL | PROTEIN | PULMONARY-ARTERIAL-HYPERTENSION | SYSTEMIC-SCLEROSIS | CUTTING EDGE | CELL BIOLOGY | PATHOGENESIS | LUNG-DISEASE | SMOOTH-MUSCLE-CELLS | Immunohistochemistry | Humans | Middle Aged | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Familial Primary Pulmonary Hypertension - metabolism | HMGB1 Protein - pharmacology | RNA Interference | HMGB1 Protein - metabolism | Female | Vascular Remodeling - physiology | JNK Mitogen-Activated Protein Kinases - genetics | Receptor for Advanced Glycation End Products - genetics | Vascular Remodeling - drug effects | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Familial Primary Pulmonary Hypertension - pathology | Cell Proliferation - genetics | Endothelial Cells - metabolism | Receptor for Advanced Glycation End Products - metabolism | Cells, Cultured | Toll-Like Receptor 4 - genetics | Familial Primary Pulmonary Hypertension - genetics | Hypertension, Pulmonary - genetics | Hypertension, Pulmonary - metabolism | Signal Transduction - genetics | Enzyme Activation - drug effects | Muscle, Smooth, Vascular - cytology | Reverse Transcriptase Polymerase Chain Reaction | Toll-Like Receptor 4 - metabolism | Blotting, Western | Pulmonary Artery - cytology | Signal Transduction - drug effects | Vascular Remodeling - genetics | Aged | Cell Proliferation - drug effects | Hypertension, Pulmonary - pathology | HMGB1 Protein - blood | Endothelial Cells - drug effects | Lung diseases, Obstructive | Analysis | Myosin | Physiological aspects | Muscle proteins | Cell differentiation | Hypertension | Medical research | Chronic obstructive pulmonary disease | Rodents | Cell proliferation | Phosphorylation | Thoracic surgery | Physiological effects | Disease | Laboratories | Pathogenesis | Differentiation (biology) | Mobility | Smooth muscle | Activation | Remodeling | Kinases | HMGB1 protein | Angiogenesis | Antigens | Immunoglobulins | Pulmonary arteries | Lung diseases | Muscles | Extracellular signal-regulated kinase | Blood vessels | Ablation | Patients | Endothelial cells | Asthma | Serum levels | Lungs | Obstructive lung disease | Original
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 02/2018, Volume 22, Issue 2, pp. 1214 - 1223
Preeclampsia (PE), a pregnancy‐specific disorder, is associated with impaired uterine spiral artery remodelling, which is related to the dysfunction of... 
Jun‐B | long non‐coding RNAs | MVIH | Preeclampsia | Jun-B | long non-coding RNAs | MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | ACTIVATION | LONG NONCODING RNAS | INITIATION | CELL BIOLOGY | MOUSE DEVELOPMENT | EVOLUTION | DISEASE | EPIGENETIC REGULATION | RNA | Mass spectrometry | Analysis | Original
Journal Article
Journal Article
Journal Article