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PAIN, ISSN 0304-3959, 10/2019, Volume 160, Issue 10, pp. 2358 - 2364
Naldemedine improves opioid-induced constipation in a timely manner in patients using opioids for noncancer pain; most experienced spontaneous bowel movement... 
PAMORA | Research Paper | Naldemedine | Constipation | Chronic noncancer pain | Opioids
Journal Article
Cancer Science, ISSN 1347-9032, 07/2017, Volume 108, Issue 7, pp. 1378 - 1385
Small‐cell lung cancer ( SCLC ) accounts for approximately 15% of all lung cancers, and is characterized as extremely aggressive, often displaying rapid tumor... 
Animal model | targeted therapy | MET | small‐cell lung cancer | HGF | small-cell lung cancer | TYROSINE KINASE-ACTIVITY | EFFICACY | ACQUIRED-RESISTANCE | T790M MUTATION | GEFITINIB RESISTANCE | FACTOR RECEPTOR | AMPLIFICATION | METASTASIS MODEL | ONCOLOGY | JUXTAMEMBRANE DOMAIN | C-MET | Proto-Oncogene Proteins c-met - metabolism | Cell Survival - drug effects | Apoptosis - drug effects | Humans | Lung Neoplasms - metabolism | In Situ Hybridization, Fluorescence | Mice, SCID | Blotting, Western | Hepatocyte Growth Factor - metabolism | Small Cell Lung Carcinoma - metabolism | Xenograft Model Antitumor Assays | Animals | Signal Transduction - drug effects | Cell Line, Tumor | Mice | Protein Kinase Inhibitors - pharmacology | Tyrosine | Killer cells | Chemotherapy | Lung cancer | Analysis | Cancer cells | Development and progression | Metastasis | Cancer | Cell culture | Phosphorylation | Copy number | AKT protein | Kinases | Cancer therapies | Clinical outcomes | Metastases | c-Met protein | Proteins | Signal transduction | Cell activation | Cell cycle | Protein-tyrosine kinase receptors | Natural killer cells | Growth factors | Protein-tyrosine kinase | Immunoglobulins | Small cell lung carcinoma | Hepatocyte growth factor | Research & development--R&D | Secretion | Extracellular signal-regulated kinase | siRNA | Patients | Cell lines | Mutation | Tumors | Original
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 06/2017, Volume 23, Issue 12, pp. 3139 - 3149
Purpose: The BIM deletion polymorphism is associated with apoptosis resistance to EGFR tyrosine kinase inhibitors (EGFR-TKI), such as gefitinib and erlotinib,... 
NSCLC | ONCOLOGY | MESSENGER-RNA EXPRESSION | MUTATION | GROWTH | ACQUIRED-RESISTANCE | DIFFERENTIATION | ERLOTINIB | AZD9291 | CHEMOTHERAPY | TKI RESISTANCE | Carcinoma, Non-Small-Cell Lung - pathology | Piperazines - administration & dosage | Sequence Deletion | Histone Deacetylases - genetics | Apoptosis - drug effects | Carcinoma, Non-Small-Cell Lung - genetics | Humans | Hydroxamic Acids - adverse effects | ErbB Receptors - genetics | Histone Deacetylase Inhibitors - administration & dosage | Drug Resistance, Neoplasm | Bcl-2-Like Protein 11 - genetics | Piperazines - adverse effects | Protein Kinase Inhibitors - administration & dosage | Vorinostat | Cell Line, Tumor | Hydroxamic Acids - administration & dosage | Histone Deacetylases - administration & dosage | Carcinoma, Non-Small-Cell Lung - drug therapy | Alternative splicing | Biotechnology | Histone deacetylase | Lung cancer | mRNA | Gene deletion | Kinases | Gene polymorphism | Risk factors | Western blotting | Clonal deletion | Xenografts | Deletion | Inhibition | Gefitinib | Protein-tyrosine kinase | Tyrosine | Splicing | Epidermal growth factor receptors | Tumor cells | Non-small cell lung carcinoma | Regression analysis | Gene expression | Polymerase chain reaction | Inhibitors | Lungs | Experimental design | Cell lines | Mutation | BIM protein | Tumors | Cancer | Apoptosis | Polymorphism
Journal Article
Journal Article
The Journal of neuroscience : the official journal of the Society for Neuroscience, ISSN 0270-6474, 08/2019, Volume 39, Issue 39, pp. 7689 - 7702
Epidemiological studies suggest that poor nutrition during pregnancy influences offspring predisposition to experience developmental and psychiatric disorders.... 
Neuroimaging | Brain | Ethanolamine | Intravenous administration | Mental disorders | Nutrition | Hyperactivity | Cognitive ability | Impairment | Mass spectroscopy | Biosynthesis | Exposure | Phospholipids | Injection | Epidemiology | Undernutrition | Pregnancy | Progeny | Offspring | Rodents | Neostriatum | Genetic crosses | Prenatal experience | Mass spectrometry
Journal Article
Cancer Science, ISSN 1347-9032, 01/2017, Volume 108, Issue 1, pp. 53 - 60
Journal Article
Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 259 - 14
A novel EGFR-tyrosine kinase inhibitor (TKI), osimertinib, has marked efficacy in patients with EGFR-mutated lung cancer. However, some patients show intrinsic... 
TYROSINE KINASE INHIBITORS | BREAST-CANCER | HETEROGENEITY | LUNG-CANCER | ACTIVATION | MULTIDISCIPLINARY SCIENCES | THERAPEUTIC TARGET | ACQUIRED-RESISTANCE | LEUKEMIA-CELLS | MEDIATES RESISTANCE | EGFR | Lung Neoplasms - drug therapy | Pyrazoles - therapeutic use | Receptor, ErbB-3 - metabolism | Adenocarcinoma of Lung - pathology | Humans | Cell Survival - genetics | Male | Gene Knockdown Techniques | Neoplasm Recurrence, Local - pathology | Adenocarcinoma of Lung - genetics | Adenocarcinoma of Lung - drug therapy | Receptor Protein-Tyrosine Kinases - antagonists & inhibitors | Cell Survival - drug effects | ErbB Receptors - antagonists & inhibitors | Piperazines - therapeutic use | Receptor, ErbB-3 - genetics | Receptor Protein-Tyrosine Kinases - metabolism | Piperazines - pharmacology | Heterocyclic Compounds, 2-Ring - pharmacology | Drug Resistance, Neoplasm - genetics | Cell Line, Tumor | Mice, Inbred NOD | Mice | Mutation | RNA, Small Interfering - metabolism | ErbB Receptors - genetics | Lung Neoplasms - pathology | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Heterocyclic Compounds, 2-Ring - therapeutic use | Adult | Female | Pyrazoles - pharmacology | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins - antagonists & inhibitors | Lung - pathology | ErbB Receptors - metabolism | Neoplasm Recurrence, Local - prevention & control | Carcinoma, Non-Small-Cell Lung - genetics | Proto-Oncogene Proteins - genetics | Treatment Outcome | Xenograft Model Antitumor Assays | Disease-Free Survival | Animals | Receptor Protein-Tyrosine Kinases - genetics | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Protein Kinase Inhibitors - therapeutic use | Neoplasm Recurrence, Local - genetics | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Tyrosine | Cell survival | Epidermal growth factor receptors | Lung cancer | Feedback loops | Patients | Axl protein | Negative feedback | Inhibitors | Emergence | Inhibition | Viability | Protein-tyrosine kinase | Tumors | Cancer
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2013, Volume 8, Issue 12, p. e84700
Purpose: Although EGF receptor tyrosine kinase inhibitors (EGFR-TKI) have shown dramatic effects against EGFR mutant lung cancer, patients ultimately develop... 
CELL LUNG-CANCER | AMPLIFICATION | GENE | MULTIDISCIPLINARY SCIENCES | HEPATOCYTE GROWTH-FACTOR | PHASE-III | ACQUIRED-RESISTANCE | T790M MUTATION | RECEPTOR | GEFITINIB RESISTANCE | ERLOTINIB | Proto-Oncogene Proteins c-met - metabolism | Lung Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - genetics | Phosphorylation | Humans | Lung Neoplasms - pathology | Mutation, Missense | Quinazolines | Receptor, Epidermal Growth Factor - metabolism | Female | Acrylamides - pharmacology | Pyrazoles - pharmacology | Lung Neoplasms - genetics | Lung Neoplasms - enzymology | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Pyrimidines - pharmacology | Mice, SCID | Proto-Oncogene Proteins c-met - genetics | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Animals | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Mice | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Amino Acid Substitution | Drug Resistance, Neoplasm - drug effects | Tyrosine | Development and progression | Genetic aspects | Epidermal growth factor | Lung cancer | Biotechnology | Laboratories | Clinical trials | Oncology | Kinases | Drug resistance | Cancer therapies | c-Met protein | Rodents | Penicillin | Protein-tyrosine kinase receptors | Inhibition | Protein-tyrosine kinase | Medical research | Epidermal growth factor receptors | Tumor cell lines | Amplification | Gene amplification | Inhibitors | Experimental design | Cell lines | Combined treatment | Mutation | Tumors | Cancer
Journal Article