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Journal Article
PLoS ONE, ISSN 1932-6203, 04/2016, Volume 11, Issue 4, p. e0153462
Glucose-6-phosphate dehydrogenase (G6PD) provides the reducing agent NADPH to meet the cellular needs for reductive biosynthesis and the maintenance of redox... 
Gene Expression Regulation, Enzymologic - drug effects | Virus Replication - drug effects | Epithelial Cells - metabolism | Epithelial Cells - drug effects | Humans | NADPH Oxidases - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | NF-kappa B - metabolism | Lung - cytology | Gene Knockdown Techniques | Dinoprostone - metabolism | Tumor Necrosis Factor-alpha - pharmacology | MAP Kinase Signaling System - drug effects | Cyclooxygenase 2 - genetics | Epithelial Cells - virology | Cyclooxygenase 2 - metabolism | Cell Line, Tumor | Glucosephosphate Dehydrogenase - metabolism | Glucosephosphate Dehydrogenase - genetics | Phosphorylation - drug effects | Epithelial Cells - cytology | Coronavirus - physiology | Phosphates | Oxidative stress | Phosphorylation | Dehydrogenases | Transcription | Laboratories | Epithelial cells | Homeostasis | Prostaglandin E2 | Inflammatory response | Viruses | Stimulation | Infections | Biosynthesis | Glucose | Kinases | NAD(P)H oxidase | Prostaglandin endoperoxide synthase | Signal transduction | Glucose 6 phosphate dehydrogenase | Glucose dehydrogenase | CYBB protein | Antiviral activity | Tumor necrosis factor-TNF | Physiology | Pretreatment | Glucosephosphate dehydrogenase | Pathogens | NF-κB protein | Immunoglobulins | Cytokines | Extracellular signal-regulated kinase | MAP kinase | Inflammation | siRNA | Tumor necrosis factor-α | Gene expression | Medicine | Signaling | Lungs | Cyclooxygenase-2 | Viral infections | Apoptosis
Journal Article
Circulation Research, ISSN 0009-7330, 04/2009, Volume 104, Issue 7, pp. 842 - 850
Abnormal proliferation and migration of vascular smooth muscle cells (VSMCs) are important pathogenic mechanisms in atherosclerosis and restenosis after... 
Restenosis | NQO1 | AMPK | Vascular smooth muscle cell | β-lapachone | restenosis | ENERGY | CARDIAC & CARDIOVASCULAR SYSTEMS | beta-lapachone | CANCER CELLS | PROTEIN-KINASE ACTIVITY | NEOINTIMA-FORMATION | INHIBITION | PATHWAY | PERIPHERAL VASCULAR DISEASE | vascular smooth muscle cell | CYCLE PROGRESSION | HEMATOLOGY | EXPRESSION | Enzyme Activators - pharmacology | AMP-Activated Protein Kinases - metabolism | Enzyme Activators - toxicity | Phosphorylation | Humans | Hyperplasia | Carotid Stenosis - drug therapy | Myocytes, Smooth Muscle - pathology | Carotid Artery Injuries - drug therapy | Male | Secondary Prevention | NAD(P)H Dehydrogenase (Quinone) - genetics | Dose-Response Relationship, Drug | RNA Interference | Time Factors | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Myocytes, Smooth Muscle - drug effects | Protein-Serine-Threonine Kinases - metabolism | Carotid Artery Injuries - pathology | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Myocytes, Smooth Muscle - enzymology | AMP-Activated Protein Kinases - antagonists & inhibitors | Retinoblastoma Protein - metabolism | Enzyme Inhibitors - pharmacology | Tumor Suppressor Protein p53 - metabolism | Rats | Naphthoquinones - pharmacology | Tunica Intima - enzymology | Carotid Stenosis - pathology | Naphthoquinones - toxicity | Rats, Sprague-Dawley | NAD(P)H Dehydrogenase (Quinone) - antagonists & inhibitors | Tunica Intima - drug effects | Muscle, Smooth, Vascular - pathology | Platelet-Derived Growth Factor - metabolism | Animals | Carotid Artery Injuries - enzymology | Tunica Intima - pathology | NAD(P)H Dehydrogenase (Quinone) - metabolism | Carotid Stenosis - enzymology | Cell Proliferation - drug effects | Enzyme Activation | HeLa Cells | Cell Cycle - drug effects | Muscle, Smooth, Vascular - enzymology | Acetyl-CoA Carboxylase - metabolism | RNA, Small Interfering - metabolism | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e61629
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, p. e77063
Background: We sought to quantify plasma high sensitivity cardiac troponin (hs-cTnT) levels, their determinants, and their associations with left ventricular... 
INDUCED CARDIOTOXICITY | GENERAL-POPULATION | NAD(P)H OXIDASE | OLDER-ADULTS | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | PEDIATRIC-PATIENTS | CARDIOVASCULAR-DISEASE | ISOVOLUMIC ACCELERATION | MYOCARDIAL INJURY | FORCE-FREQUENCY RELATIONSHIPS | Heart - physiopathology | Acute Disease | Leukemia - blood | Gene Frequency | Humans | Echocardiography - methods | Neoplasm Recurrence, Local | Survivors | Leukemia - therapy | Genotype | Linear Models | Male | Chemoradiotherapy | Stem Cell Transplantation | Young Adult | Troponin T - blood | Myocardium - metabolism | Adolescent | NADPH Oxidases - genetics | Adult | Multidrug Resistance-Associated Proteins - genetics | Anthracyclines - therapeutic use | Child | Leukemia - genetics | Oxidases | Heart | Drug resistance in microorganisms | Anthracyclines | Stem cells | Physiological aspects | Transplantation | Plasma | Deformation | Syngeneic grafts | Leukemia | Radiation | Cardiovascular disease | Biochemistry | Males | NAD(P)H oxidase | Calcium-binding protein | Children | Heart diseases | Strain rate | Heart failure | Hypertension | Echocardiography | Congenital diseases | Heart transplantation | Mortality | Multidrug resistance | Metabolism | Troponin T | Strain | NAD | Medicine | Genetic variance | Sensitivity | Troponin | Chemotherapy | Hospitals | Anthracycline | Genotyping | Medical prognosis | Irradiation | Determinants | Ventricle | Females | Cancer
Journal Article
Journal Article
Journal Article
Annals of Biomedical Engineering, ISSN 0090-6964, 11/2005, Volume 33, Issue 11, pp. 1546 - 1554
Journal Article