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Cellular Signalling, ISSN 0898-6568, 08/2018, Volume 48, pp. 64 - 68
Dengue virus (DENV) infection is a disease that is endemic to many parts of the world, and its increasing prevalence ranks it among the diseases considered to... 
Dengue virus infection | Pathogenesis | Mitogen-activated protein kinase signaling | PATHWAYS | CELLS | P38 MAPK | HUMAN MONOCYTES | CELL BIOLOGY | REPLICATION | HEMORRHAGIC-FEVER | GENE-EXPRESSION | ANTIVIRAL ACTIVITY | INHIBITOR | NF-KAPPA-B | Hemorrhagic fever | Analysis | Dengue viruses | Liver | Permeability | Mitogens | Health aspects | Protein kinases
Journal Article
Antiviral Research, ISSN 0166-3542, 06/2019, Volume 166, pp. 42 - 55
Liver injury is one of the hallmark features of severe dengue virus (DENV) infection since DENV can replicate in the liver and induce hepatocytes to undergo... 
N-acetyl cysteine | Antiviral response | Dengue virus | Dengue replication | Liver injury | Virus diseases | Antioxidants | Enzymes | Cysteine | Dengue viruses | Liver | Interferon | Biological response modifiers | Health aspects | Cystine
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 1 - 11
Abstract Cellular immunotherapy is a promising new therapeutic approach for hepatocellular carcinoma (HCC), which has a high recurrence rate, irrespective of... 
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 01/2017, Volume 483, Issue 1, pp. 58 - 63
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2016, Volume 11, Issue 2, p. e0149486
Dengue virus (DENV) infection causes organ injuries, and the liver is one of the most important sites of DENV infection, where viral replication generates a... 
PRO-INFLAMMATORY CYTOKINES | HEAT-SHOCK PROTEINS | D IMMUNE GLOBULIN | ISCHEMIA/REPERFUSION INJURY | ACTIVATED PROTEIN-KINASE | HEMORRHAGIC-FEVER | MULTIDISCIPLINARY SCIENCES | GAMMA-GLUTAMYL-TRANSFERASE | INHIBITOR SB203580 | TNF-ALPHA | CELL-DEATH | Liver - virology | Liver Diseases - virology | Male | Intracellular Signaling Peptides and Proteins - metabolism | Activating Transcription Factor 2 - metabolism | Liver - injuries | Animals | MAP Kinase Signaling System - drug effects | Liver - drug effects | Dengue Virus - pathogenicity | HSP27 Heat-Shock Proteins - metabolism | Liver Diseases - drug therapy | Mice | Mice, Inbred BALB C | p38 Mitogen-Activated Protein Kinases - metabolism | Imidazoles - therapeutic use | Phosphorylation - drug effects | Protein-Serine-Threonine Kinases - metabolism | Pyridines - therapeutic use | Complications and side effects | Liver failure | Care and treatment | Dengue | Heat shock proteins | Physiological aspects | Development and progression | Genetic aspects | Research | Mitogen-activated protein kinases | Phosphorylation | Transcription factors | RANTES | Liver | Viral diseases | Viruses | Infections | Kinases | Experiments | Caspase-3 | Interleukin 6 | Proteins | Histopathology | Rodents | IP-10 protein | Tumor necrosis factor-TNF | Activating transcription factor 2 | Vector-borne diseases | Thrombocytopenia | Cytokines | Hematology | Dengue fever | Caspase | MAP kinase | Tumor necrosis factor-α | Gene expression | Medicine | Caspase-8 | Injury prevention | Hospitals | Transaminases | Hsp27 protein | Interleukin 10 | Caspase-9 | Laboratory animals | Chemokines | Apoptosis
Journal Article