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1. Of men in mice
: twoards reconstituting the human immune system in mice
by Yeung, Rae S. M. (Rae Suk-Man)
ISBN 9780612189355, 242 leaves.
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2. Full Text Reply
by Aeschlimann, Florence A and Yeung, Rae S. M and Barra, Lillian and Benseler, Susanne M
Arthritis & Rheumatology, ISSN 2326-5191, 05/2019, Volume 71, Issue 5, pp. 836 - 838
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3. Reply
by Florence A Aeschlimann and Rae S M Yeung and Lillian Barra and Susanne M Benseler
Arthritis & Rheumatology, ISSN 2326-5191, 05/2019, Volume 71, Issue 5, pp. 836 - 838
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4. Full Text Reply
by Aeschlimann, Florence A and Yeung, Rae S. M and Barra, Lillian and Benseler, Susanne M
Arthritis & Rheumatology, ISSN 2326-5191, 05/2019, Volume 71, Issue 5, pp. 836 - 838
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5. Full Text Kawasaki disease: update on pathogenesis
by Yeung, Rae SM
Current Opinion in Rheumatology, ISSN 1040-8711, 09/2010, Volume 22, Issue 5, pp. 551 - 560
PURPOSE OF REVIEWThis review will highlight recent advances in our understanding of the pathogenesis of Kawasaki disease, highlighting the molecular players... 
pathogenesis | genetics | Kawasaki disease | animal model | T-cell activation | SOLUBLE CD40 LIGAND | TAIWANESE CHILDREN | FACTOR-ALPHA LEVELS | PROMOTER POLYMORPHISM | BINDING LECTIN GENOTYPE | RHEUMATOLOGY | CORONARY-ARTERY LESIONS | KOREAN CHILDREN | ANIMAL-MODEL | RECEPTOR GENE POLYMORPHISMS | CONVERTING-ENZYME GENE | Mucocutaneous Lymph Node Syndrome - genetics | Genetic Predisposition to Disease | Humans | Asian Continental Ancestry Group - genetics | Mucocutaneous Lymph Node Syndrome - immunology | Phosphotransferases (Alcohol Group Acceptor) - genetics | T-Lymphocytes - immunology | Mucocutaneous Lymph Node Syndrome - etiology | Child
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6. Full Text Revisiting the role of steroids and aspirin in the management of acute Kawasaki disease
by Dhanrajani, Anita and Yeung, Rae S.M
Current Opinion in Rheumatology, ISSN 1040-8711, 09/2017, Volume 29, Issue 5, pp. 547 - 552
PURPOSE OF REVIEWKawasaki disease is an acute multisystem childhood vasculitis with a predilection for the coronary arteries. The role of corticosteroids and... 
corticosteroids | aspirin | Kawasaki disease | THERAPY | EFFICACY | INTRAVENOUS IMMUNOGLOBULIN | CORONARY-ARTERY ABNORMALITIES | PREVENTION | RANDOMIZED-TRIAL | ANEURYSM | RHEUMATOLOGY | Mucocutaneous Lymph Node Syndrome - drug therapy | Immunoglobulins, Intravenous - therapeutic use | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Humans | Aspirin - therapeutic use | Adrenal Cortex Hormones - therapeutic use | Child
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7. Full Text Genome-wide association study identifies FCGR2A as a susceptibility locus for Kawasaki disease
by Khor, Chiea Chuen and Davila, Sonia and Breunis, Willemijn B and Lee, Yi-Ching and Shimizu, Chisato and Wright, Victoria J and Yeung, Rae S. M and Tan, Dennis E. K and Sim, Kar Seng and Wang, Jie Jin and Wong, Tien Yin and Pang, Junxiong and Mitchell, Paul and Cimaz, Rolando and Dahdah, Nagib and Cheung, Yiu-Fai and Huang, Guo-Ying and Yang, Wanling and Park, In-Sook and Lee, Jong-Keuk and Wu, Jer-Yuarn and Levin, Michael and Burns, Jane C and Burgner, David and Kuijpers, Taco W and Hibberd, Martin L and Lau, Yu-Lung and Zhang, Jing and Ma, Xiao-Jing and Liu, Fang and Wu, Lin and Yoo, Jeong-Jin and Hong, Soo-Jong and Kim, Kwi-Joo and Kim, Jae-Jung and Park, Young-Mi and Mi Hong, Young and Sohn, Sejung and Young Jang, Gi and Ha, Kee-Soo and Nam, Hyo-Kyoung and Byeon, Jung-Hye and Weon Yun, Sin and Ki Han, Myung and Lee, Kyung-Yil and Hwang, Ja-Young and Kuipers, Irene M and Ottenkamp, Jaap J and Biezeveld, Maarten and Tacke, Carline and Korean Kawasaki Dis Genetics Conso and Hong Kong-Shanghai Kawasaki Dis Ge and Int Kawasaki Dis Genetics Consor and US Kawasaki Dis Genetics Consortiu and Blue Mt Eye Study and Taiwan Kawasaki Dis Genetics Conso and Korean Kawasaki Disease Genetics Consortium and Taiwan Kawasaki Disease Genetics Consortium and US Kawasaki Disease Genetics Consortium and International Kawasaki Disease Genetics Consortium and Blue Mountains Eye Study and Hong Kong–Shanghai Kawasaki Disease Genetics Consortium
Nature genetics, ISSN 1061-4036, 2011, Volume 43, Issue 12, pp. 1241 - 1246
Kawasaki disease is a systemic vasculitis of unknown etiology, with clinical observations suggesting a substantial genetic contribution to disease... 
POPULATION | VARIANTS | INTRAVENOUS IMMUNOGLOBULIN | ULCERATIVE-COLITIS | GENETICS & HEREDITY | POLYMORPHISM | COPY NUMBER VARIATION | LINKAGE | RECEPTORS | TRAITS | CHILDREN | Haplotypes | Genetic Predisposition to Disease | Genome-Wide Association Study | Multigene Family | Humans | Genetic Loci | Case-Control Studies | Linkage Disequilibrium | Mucocutaneous Lymph Node Syndrome - genetics | Chromosomes, Human, Pair 19 | Receptors, IgG - genetics | Chromosomes, Human, Pair 1 | Polymorphism, Single Nucleotide | Principal Component Analysis | Disease susceptibility | Genetic aspects | Kawasaki disease | Research | Single nucleotide polymorphisms | Risk factors | Disease | Pathogenesis | Quality control | Data collection | Genomes | Manuscripts | Independent sample
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8. Full Text Loss of the Arp2/3 complex component ARPC1B causes platelet abnormalities and predisposes to inflammatory disease
by Kahr, Walter H. A and Pluthero, Fred G and Elkadri, Abdul and Warner, Neil and Drobac, Marko and Chen, Chang Hua and Lo, Richard W and Li, Ling and Li, Ren and Li, Qi and Thoeni, Cornelia and Pan, Jie and Leung, Gabriella and Lara-Corrales, Irene and Murchie, Ryan and Cutz, Ernest and Laxer, Ronald M and Upton, Julia and Roifman, Chaim M and Yeung, Rae S.M and Brumell, John H and Muise, Aleixo M
Nature Communications, ISSN 2041-1723, 04/2017, Volume 8, Issue 1, p. 14816
Human actin-related protein 2/3 complex (Arp2/3), required for actin filament branching, has two ARPC1 component isoforms, with ARPC1B prominently expressed in... 
F-ACTIN | CELL-MIGRATION | ACTIN POLYMERIZATION | LEADING-EDGE | MULTIDISCIPLINARY SCIENCES | WISKOTT-ALDRICH-SYNDROME | SYNDROME PROTEIN | MEGAKARYOCYTE | LAMELLIPODIA | PROPLATELET FORMATION | WASP | Blood Platelets - pathology | Inflammation - pathology | Disease Susceptibility | Actin-Related Protein 2-3 Complex - deficiency | Humans | Vasculitis - pathology | Actins - metabolism | Bone Marrow Cells - pathology | Gene Knockout Techniques | Mutation - genetics | Actin-Related Protein 2-3 Complex - metabolism | Megakaryocytes - pathology | Blood Platelets - metabolism | Blood Platelets - ultrastructure | Cell Shape | Bone Marrow Cells - drug effects | Fibrinogen - pharmacology | Wiskott-Aldrich Syndrome - pathology | Blood Platelets - drug effects | Megakaryocytes - drug effects | Megakaryocytes - metabolism | Blood Platelet Disorders - metabolism | Bone Marrow Cells - metabolism
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9. Full Text Genetic architecture distinguishes systemic juvenile idiopathic arthritis from other forms of juvenile idiopathic arthritis: clinical and therapeutic implications
by Ombrello, Michael J and Arthur, Victoria L and Remmers, Elaine F and Hinks, Anne and Tachmazidou, Ioanna and Grom, Alexei A and Foell, Dirk and Martini, Alberto and Gattorno, Marco and Özen, Seza and Prahalad, Sampath and Zeft, Andrew S and Bohnsack, John F and Ilowite, Norman T and Mellins, Elizabeth D and Russo, Ricardo and Len, Claudio and Hilario, Maria Odete E and Oliveira, Sheila and Yeung, Rae S M and Rosenberg, Alan M and Wedderburn, Lucy R and Anton, Jordi and Haas, Johannes-Peter and Rosen-Wolff, Angela and Minden, Kirsten and Tenbrock, Klaus and Demirkaya, Erkan and Cobb, Joanna and Baskin, Elizabeth and Signa, Sara and Shuldiner, Emily and Duerr, Richard H and Achkar, Jean-Paul and Kamboh, M Ilyas and Kaufman, Kenneth M and Kottyan, Leah C and Pinto, Dalila and Scherer, Stephen W and Alarcón-Riquelme, Marta E and Docampo, Elisa and Estivill, Xavier and Gül, Ahmet and Langefeld, Carl D and Thompson, Susan and Zeggini, Eleftheria and Kastner, Daniel L and Woo, Patricia and Thomson, Wendy and Childhood Arthrit Prospective Stud and Randomized Placebo Phase Study Ril and Biologically Based Outcome Predict and Inception Cohort Newly Diagnosed P and Sparks-Childhood Arthrit Response and British Soc Pediat Adolescent Rheu and British Society of Pediatric and Adolescent Rheumatology (BSPAR) Study Group, Inception Cohort of Newly Diagnosed Patients with Juvenile Idiopathic Arthritis (ICON-JIA) Study Group, Childhood Arthritis Prospective Study (CAPS) Group, Randomized Placebo Phase Study of Rilonacept in sJIA (RAPPORT) Investigators, Sparks-Childhood Arthritis Response to Medication Study (CHARMS) Group, Biologically Based Outcome Predictors in JIA (BBOP) Group
Annals of the Rheumatic Diseases, ISSN 0003-4967, 05/2017, Volume 76, Issue 5, pp. 906 - 913
ObjectivesJuvenile idiopathic arthritis (JIA) is a heterogeneous group of conditions unified by the presence of chronic childhood arthritis without an... 
Gene Polymorphism | Adult Onset Still's Disease | Juvenile Idiopathic Arthritis | IMPUTATION | VARIANTS | SUSCEPTIBILITY | RHEUMATOLOGY | LOCUS | ABATACEPT | GENOME-WIDE ASSOCIATION | CHILDREN | Genome-Wide Association Study | Major Histocompatibility Complex - genetics | Humans | Risk Factors | Genotype | Polymorphism, Single Nucleotide | Chromosomes, Human, Pair 1 - genetics | Arthritis, Juvenile - genetics | Case-Control Studies | Arthritis, Juvenile - drug therapy | Clinical trials | Treatment outcome | Research | Juvenile arthritis | Analysis | Basic and Translational Research | 1506
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10. Full Text Childhood Takayasu arteritis: Disease course and response to therapy
by Aeschlimann, Florence A and Eng, Simon W.M and Sheikh, Shehla and Laxer, Ronald M and Hebert, Diane and Noone, Damien and Twilt, Marinka and Pagnoux, Christian and Benseler, Susanne M and Yeung, Rae S.M
Arthritis Research and Therapy, ISSN 1478-6354, 11/2017, Volume 19, Issue 1, pp. 255 - 10
Background: Takayasu arteritis (TAK) is a large vessel vasculitis that rarely affects children. Data on childhood TAK are scarce. The aim of this study was to... 
Vasculitis | Children | Takayasu arteritis | Biologic therapy | LARGE-VESSEL VASCULITIS | CLASSIFICATION | RHEUMATOLOGY | SURGICAL-TREATMENT | RHEUMATOLOGY 1990 CRITERIA | INITIAL VALIDATION | TOCILIZUMAB | CASE SERIES | EXPERIENCE | OF-THE-LITERATURE | Development and progression | Care and treatment | Takayasu's arteritis | Diseases | Pediatrics | Medical imaging | Cytokines | Laboratories | Rheumatology | Data collection | Systematic review | Inflammation | Arthritis | Blood pressure | Family medical history | Patients
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11. Full Text Outcome of kidney transplantation in pediatric patients with ANCA-associated glomerulonephritis: a single-center experience
by Noone, Damien and Yeung, Rae S. M and Hebert, Diane
Pediatric Nephrology, ISSN 0931-041X, 12/2017, Volume 32, Issue 12, pp. 2343 - 2350
Data on kidney transplant outcomes for pediatric patients with end-stage renal disease (ESRD) secondary to anti-neutrophil cytoplasmic antibody... 
Glomerulonephritis | Pediatrics | Nephrology | Medicine & Public Health | Outcome | Anti-neutrophil cytoplasmic antibody | Children | Survival | Urology | Transplant | ANTIBODY-ASSOCIATED VASCULITIS | RENAL-TRANSPLANTATION | GRANULOMATOSIS | SMALL-VESSEL VASCULITIS | HISTOPATHOLOGIC CLASSIFICATION | UROLOGY & NEPHROLOGY | PEDIATRICS | RECURRENCE | Kidney Failure, Chronic - mortality | Glomerulonephritis - therapy | Humans | Glomerulonephritis - mortality | Male | Treatment Outcome | Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis - therapy | Kidney Transplantation - methods | Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis - mortality | Kidney Failure, Chronic - therapy | Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis - complications | Adolescent | Female | Retrospective Studies | Glomerulonephritis - etiology | Child | Kidney - physiopathology | Antibodies, Antineutrophil Cytoplasmic - blood | Kidney Failure, Chronic - etiology | Kidney Transplantation - adverse effects | Kidneys | Patient outcomes | Transplantation | Diagnosis | Kidney diseases | Health aspects | End-stage renal disease | Epidermal growth factor receptors | Transplants & implants | Syngeneic grafts | Granulomatosis | Patients | Glomerular filtration rate | Graft rejection | Cyclophosphamide | Immunosuppression | Vasculitis | Antineutrophil cytoplasmic antibodies | Steroid hormones | Kidney transplantation
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12. Full Text Inositol-Triphosphate 3-Kinase C Mediates Inflammasome Activation and Treatment Response in Kawasaki Disease
by Alphonse, Martin Prince and Duong, Trang T and Shumitzu, Chisato and Hoang, Truong Long and McCrindle, Brian W and Franco, Alessana and Schurmans, Stéphane and Philpott, Dana J and Hibberd, Martin L and Burns, Jane and Kuijpers, Taco W and Yeung, Rae S. M
Journal of immunology (Baltimore, Md, ISSN 0022-1767, 2016, Volume 197, Issue 9, pp. 3481 - 3489
Kawasaki disease (KD) is a multisystem vasculitis that predominantly targets the coronary arteries in children. Phenotypic similarities between KD and... 
CORONARY ARTERITIS | CELLS | INTERLEUKIN-18 | MANAGEMENT | CA2 | SUSCEPTIBILITY | MICE | NLRP3 INFLAMMASOME | MODEL | IMMUNOLOGY | ASSOCIATION | Coronary Vessels - pathology | Inflammasomes - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Humans | Mice, Inbred C57BL | Child, Preschool | Genotype | Mucocutaneous Lymph Node Syndrome - immunology | Phosphotransferases (Alcohol Group Acceptor) - genetics | Risk | Treatment Outcome | Interleukin-1beta - genetics | Coronary Vessels - metabolism | Mice, Knockout | Mucocutaneous Lymph Node Syndrome - genetics | Animals | Interleukin-1beta - metabolism | Mice | Polymorphism, Single Nucleotide | Interleukin-18 - genetics | Interleukin-18 - metabolism | Cohort Studies
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13. Full Text Enhancing translational research in paediatric rheumatology through standardization
by Yeung, Rae S M and Albani, S and Feldman, B.M and Mellins, Elizabeth D and Prakken, ABJ and Wedderburn, Lucy R
Nature Reviews. Rheumatology, ISSN 1759-4790, 11/2016, Volume 11, p. 684
The past decade has seen many successes in translational rheumatology, from dramatic improvements in outcomes brought about by novel biologic therapies, to the... 
Review | Journal Article
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14. Full Text Epidemiology and Management of Kawasaki Disease
by Luca, Nadia J. C and Yeung, Rae S. M
Drugs, ISSN 0012-6667, 5/2012, Volume 72, Issue 8, pp. 1029 - 1038
Kawasaki disease (KD) is an acute systemic vasculitis affecting young children and is rising in incidence worldwide. It is most common in children <5 years of... 
Pharmacotherapy | Internal Medicine | Medicine & Public Health | Pharmacology/Toxicology | Anticoagulants | Aspirin | Immunosuppressants | Abciximab | Corticosteroids | Myocarditis | Cyclophosphamide | Systemic-vasculitis | Antiplatelets | Ciclosporin | Heparin | Mucocutaneous-lymph-node-syndrome | Immune-globulin | Haemophagocytic-lymphohistiocytosis | ADDITIONAL IMMUNE GLOBULIN | CORONARY-ARTERY INVOLVEMENT | LONG-TERM MANAGEMENT | INTRAVENOUS GAMMA-GLOBULIN | DIFFERENTIAL-DIAGNOSIS | MACROPHAGE ACTIVATION SYNDROME | PHARMACOLOGY & PHARMACY | TOXICOLOGY | STEROID PULSE THERAPY | AMERICAN-HEART-ASSOCIATION | HEMOPHAGOCYTIC LYMPHOHISTIOCYTOSIS | NATIONWIDE SURVEY | Animals | Humans | Mucocutaneous Lymph Node Syndrome - therapy | Female | Male | Disease Management | Child | Mucocutaneous Lymph Node Syndrome - epidemiology | Randomized Controlled Trials as Topic | Care and treatment | Kawasaki disease | Medical case management | Causes of | Dosage and administration | Drug therapy | Heart diseases | Immunosuppressive agents | Risk factors | Methods
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15. Full Text Comparing Presenting Clinical Features in 48 Children With Microscopic Polyangiitis to 183 Children Who Have Granulomatosis With Polyangiitis (Wegener's): An ARChiVe Cohort Study
by Cabral, David A and Canter, Debra L and Muscal, Eyal and Nanda, Kabita and Wahezi, Dawn M and Spalding, Steven J and Twilt, Marinka and Benseler, Susanne M and Campillo, Sarah and Charuvanij, Sirirat and Dancey, Paul and Eberhard, Barbara A and Elder, Melissa E and Hersh, Aimee and Higgins, Gloria C and Huber, Adam M and Khubchandani, Raju and Kim, Susan and Klein‐Gitelman, Marisa and Kostik, Mikhail M and Lawson, Erica F and Lee, Tzielan and Lubieniecka, Joanna M and McCurdy, Deborah and Moorthy, Lakshmi N and Morishita, Kimberly A and Nielsen, Susan M and O'Neil, Kathleen M and Reiff, Andreas and Ristic, Goran and Robinson, Angela B and Sarmiento, Angelyne and Shenoi, Susan and Toth, Mary B and Van Mater, Heather A and Wagner‐Weiner, Linda and Weiss, Jennifer E and White, Andrew J and Yeung, Rae S. M and Cabral, David A and Sarmiento, Angelyne and Yang, Qun and Espinosa, Victor and Lubieniecki, Joanna and Guzman, Jaime and Houghton, Kristin and Morishita, Kimberly and Petty, Ross and Tucker, Lori and Toth, Mary B and Benseler, Susanne and Twilt, Marinka and Beresford, Michael and Baildam, Eileen and Klein‐Gitelman, Marisa and Miller, Michael and Curran, Megan and Southwood, Taunton and Khubchandani, Raju and Ilowite, Norman T and Wahezi, Dawn M and Kim, Susan and Dedeoglu, Fatma and Fuhlbrigge, Robert and Hazen, Melissa and Son, Mary Beth and Sundel, Robert and Reiff, Andreas and Brown, Diane and Marzan, Katherine and Ramanathan, Anusha and Shaham, Bracha and Duffy, Ciaran and Adams, Matthew and Valentini, Rudolf and Rosenkranz, Margalit and Kietz, Daniel and Cassidy, Elaine and Torok, Kathryn and Becker, Mara and Jung, Lawrence K and Spalding, Steven and Zeft, Andrew and Eberhard, Anne and Gottlieb, Bett and Toruner, Cagri and Wagner‐Weiner, Linda and Onel, Karen and Spencer, Charles and De Ranieri, Deidre and Tesher, Melissa and Eichenfield, Andrew and Imundo, Lisa and Van Mater, Heather and Egla Rabinovich, C and Schanberg, Laura and Dvergsten, Jeffery and Friswell, Mark and Yeung, Rae and Feldman, Brian and ... and ARChiVe Investigators Network Ped and ARChiVe Investigators Network within the PedVas Initiative
Arthritis & Rheumatology, ISSN 2326-5191, 10/2016, Volume 68, Issue 10, pp. 2514 - 2526
Objective To uniquely classify children with microscopic polyangiitis (MPA), to describe their demographic characteristics, presenting clinical features, and... 
CHURG-STRAUSS-SYNDROME | SYSTEMIC VASCULITIS | CHILDHOOD GRANULOMATOSIS | CONSENSUS CONFERENCE | ANCA-ASSOCIATED VASCULITIS | CLASSIFICATION | ANTINEUTROPHIL CYTOPLASMIC ANTIBODIES | POLYARTERITIS-NODOSA | RHEUMATOLOGY | RHEUMATOLOGY 1990 CRITERIA | EPIDEMIOLOGY | United States - epidemiology | Azathioprine - therapeutic use | Age Distribution | Granulomatosis with Polyangiitis - therapy | Humans | Immunosuppressive Agents - therapeutic use | Child, Preschool | Respiratory Insufficiency - etiology | Infant | Male | Microscopic Polyangiitis - epidemiology | Methotrexate - therapeutic use | Microscopic Polyangiitis - complications | Adrenal Cortex Hormones - therapeutic use | Cyclophosphamide - therapeutic use | Lung Diseases - etiology | Antibodies, Antineutrophil Cytoplasmic | Rituximab - therapeutic use | Canada - epidemiology | Hemorrhage - etiology | Female | Granulomatosis with Polyangiitis - physiopathology | Drug Therapy, Combination | Child | Proteinuria - etiology | Kidney Failure, Chronic - physiopathology | Microscopic Polyangiitis - physiopathology | Respiratory Insufficiency - physiopathology | Hemorrhage - physiopathology | Europe - epidemiology | Granulomatosis with Polyangiitis - epidemiology | Respiratory Insufficiency - therapy | Oxygen Inhalation Therapy | Plasmapheresis | Kidney Failure, Chronic - therapy | Asia - epidemiology | Microscopic Polyangiitis - therapy | Mycophenolic Acid - therapeutic use | Nephrotic Syndrome - physiopathology | Adolescent | Lung Diseases - physiopathology | Nephrotic Syndrome - etiology | Cohort Studies | Granulomatosis with Polyangiitis - complications | Kidney Failure, Chronic - etiology | Renal Dialysis | Archives & records | Algorithms
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