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Gastroenterology, ISSN 0016-5085, 04/2017, Volume 152, Issue 5, pp. S1297 - S1297
Journal Article
Human Pathology, ISSN 0046-8177, 2007, Volume 38, Issue 9, pp. 1361 - 1371
Journal Article
Nature Cell Biology, ISSN 1465-7392, 09/2002, Volume 4, Issue 9, pp. 699 - 704
Journal Article
Pediatric Blood & Cancer, ISSN 1545-5009, 07/2016, Volume 63, Issue 7, pp. 1163 - 1167
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2015, Volume 10, Issue 2, pp. e0117000 - e0117000
Insulin resistance is an important contributing factor in non-alcoholic fatty liver disease. AKT and mTORC1 are key components of the insulin pathway, and play... 
COMPLEX | ACTIVATION | INHIBITION | NRF2 | LIPOGENESIS | SUBSTRATE | MULTIDISCIPLINARY SCIENCES | FATTY LIVER | NONALCOHOLIC STEATOHEPATITIS | CELL-SURVIVAL | SREBP-1C | Non-alcoholic Fatty Liver Disease - pathology | PTEN Phosphohydrolase - genetics | Liver - pathology | Tumor Suppressor Proteins - metabolism | Non-alcoholic Fatty Liver Disease - genetics | TOR Serine-Threonine Kinases - metabolism | Liver - metabolism | PTEN Phosphohydrolase - metabolism | Non-alcoholic Fatty Liver Disease - metabolism | Mechanistic Target of Rapamycin Complex 1 | Mice, Knockout | Multiprotein Complexes - metabolism | Ribosomal Protein S6 Kinases, 90-kDa - metabolism | Animals | Diet, High-Fat | Gene Deletion | Ribosomal Protein S6 Kinases, 90-kDa - genetics | Tumor Suppressor Proteins - genetics | Mice | Lipogenesis | Proto-Oncogene Proteins c-akt - metabolism | Glucose metabolism | Insulin resistance | Glucose | Liver | Body weight | Dextrose | Animal models | Phosphorylation | Transcription factors | Hyperactivity | AKT protein | Lipids | Feedback inhibition | Kinases | Metabolic syndrome | TSC1 protein | High fat diet | Proteins | Fatty liver | Clonal deletion | Feedback | Rodents | Surgery | Gastroenterology | Deletion | Oxidation | Tumorigenesis | Inhibition | Lipid metabolism | Liver diseases | Rapamycin | Gene expression | Metabolism | Insulin | Fatty acids | Steatosis | Glucose tolerance | Nutrition research | Diet | In vivo methods and tests | PTEN protein | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 3, pp. e18075 - e18075
Non-alcoholic fatty liver disease (NAFLD) is causally linked to type 2 diabetes, insulin resistance and dyslipidemia. In a normal liver, insulin suppresses... 
MAMMALIAN TARGET | GENE | STEATOSIS | INSULIN-RESISTANCE | BIOLOGY | FATTY LIVER | MICE | CELL-GROWTH | INDUCED OBESITY | MTOR | ADIPOSE-TISSUE | Hepatomegaly - pathology | Organ Specificity - drug effects | Liver - enzymology | Fatty Liver - pathology | Hepatomegaly - enzymology | Multiprotein Complexes | Fatty Liver - complications | Hepatocytes - pathology | Glucose - biosynthesis | RNA, Messenger - metabolism | Mechanistic Target of Rapamycin Complex 1 | Liver - drug effects | Dietary Fats - pharmacology | Tumor Suppressor Proteins - deficiency | Gene Deletion | Dietary Fats - administration & dosage | Fatty Liver - enzymology | Lipid Metabolism - genetics | Proto-Oncogene Proteins c-akt - metabolism | Hepatocytes - drug effects | PTEN Phosphohydrolase - deficiency | Tumor Suppressor Proteins - metabolism | Liver - metabolism | RNA, Messenger - genetics | Insulin Resistance | PTEN Phosphohydrolase - metabolism | Hepatomegaly - complications | Sirolimus - pharmacology | Gene Expression Regulation - drug effects | Animals | Proteins - metabolism | Diet | Lipid Metabolism - drug effects | Feedback, Physiological - drug effects | Mice | TOR Serine-Threonine Kinases | Proteins - antagonists & inhibitors | Hepatocytes - enzymology | Type 2 diabetes | Liver diseases | Liver | Tuberous sclerosis | Insulin resistance | Triglycerides | Glucose | Insulin | Dextrose | Transcription factors | Pathogenesis | Dyslipidemia | AKT protein | Bifurcations | TSC1 protein | Sclerosis | Proteins | Cell growth | Lipid metabolism | Lipogenesis | Gluconeogenesis | Immunoglobulins | Nutrient deficiency | Rapamycin | Gene expression | Metabolism | Ablation | Fatty acids | Hepatocytes | Animal models | Phosphorylation | Lipids | Hamartin | Kinases | Metabolic syndrome | Accumulation | High fat diet | Fatty liver | Clonal deletion | Rodents | Atherosclerosis | Deletion | Diabetes mellitus (non-insulin dependent) | Phenotypes | Diabetes mellitus | Breast cancer | Steatosis | Gastrointestinal surgery | Diabetes | PTEN protein | Index Medicus
Journal Article
Gastroenterology, ISSN 0016-5085, 04/2017, Volume 152, Issue 5, pp. S1236 - S1237
Journal Article
American Journal of Pathology, ISSN 0002-9440, 2010, Volume 177, Issue 4, pp. 1765 - 1778
The tuberous sclerosis complex 2 (TSC2) gene encodes the protein tuberin, which functions as a key negative regulator of both mammalian target of rapamycin... 
TARGET | SCLEROSIS COMPLEX | LYMPHANGIOLEIOMYOMATOSIS | TSC2 | PATHOLOGY | BETA-CATENIN | CELL | EXPRESSION | CARCINOMA | LYMPHANGIOMYOMATOSIS | TRANSPLANTATION | Kidney Neoplasms - genetics | Phosphorylation | Cell Proliferation | Cadherins - metabolism | Microtubule-Associated Proteins - genetics | Microtubule-Associated Proteins - metabolism | Epithelial-Mesenchymal Transition - physiology | Humans | Multiprotein Complexes | Kidney Neoplasms - metabolism | Lymphangioleiomyomatosis - pathology | Neoplasm Proteins - metabolism | Immunoenzyme Techniques | Proto-Oncogene Proteins c-akt - genetics | Mechanistic Target of Rapamycin Complex 1 | Tuberous Sclerosis - pathology | Flow Cytometry | Cadherins - genetics | Neoplasm Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Lymphangioleiomyomatosis - metabolism | RNA, Messenger - genetics | Rats | Angiomyolipoma - pathology | Reverse Transcriptase Polymerase Chain Reaction | Angiomyolipoma - genetics | Cell Adhesion | Blotting, Western | Mice, Knockout | Tuberous Sclerosis - genetics | Proteins - genetics | Tumor Suppressor Proteins - physiology | Animals | Proteins - metabolism | Tuberous Sclerosis - metabolism | Fluorescent Antibody Technique | Anoikis | Kidney Neoplasms - pathology | Mice | TOR Serine-Threonine Kinases | Angiomyolipoma - metabolism | Apoptosis | Cell Movement | Lymphangioleiomyomatosis - genetics | Index Medicus | Abridged Index Medicus | Regular
Journal Article
Gastrointestinal Endoscopy, ISSN 0016-5107, 2016, Volume 83, Issue 5, pp. AB270 - AB271
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, pp. e92925 - e92925
Chronic liver injury leads to fibrosis, cirrhosis, and loss of liver function. Liver cirrhosis is the 12th leading cause of death in the United States, and it... 
RAT HEPATIC LIPOCYTES | HEPATOCELLULAR-CARCINOMA | SMOOTH-MUSCLE ACTIN | MULTIDISCIPLINARY SCIENCES | PDGFR-ALPHA | IN-VIVO | CARBON-TETRACHLORIDE | EXPRESSION | PULMONARY-FIBROSIS | TRANSGENIC MICE | BETA | Liver - pathology | Humans | Gene Expression Regulation, Neoplastic | Hepatic Stellate Cells - metabolism | Male | RNA, Messenger - metabolism | Receptor, Platelet-Derived Growth Factor beta - genetics | Liver - drug effects | Carcinoma, Hepatocellular - genetics | Liver Cirrhosis - metabolism | Chemical and Drug Induced Liver Injury - pathology | Liver Cirrhosis - genetics | Cell Line | Liver Cirrhosis - drug therapy | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Liver Neoplasms - genetics | Liver - metabolism | RNA, Messenger - genetics | Gene Dosage | Chemical and Drug Induced Liver Injury - genetics | Animals | Chemical and Drug Induced Liver Injury - metabolism | Receptor, Platelet-Derived Growth Factor alpha - genetics | Alleles | Liver Cirrhosis - pathology | Mice | Fluorescence | Liver cancer | Platelet-derived growth factor | Health aspects | Liver cirrhosis | Fibrosis | Liver | Carbon tetrachloride | Smooth muscle | Activation | Adipocytes | Kinases | Risk factors | Signal transduction | Cell growth | Rodents | Surgery | Growth factors | Stellate cells | Liver diseases | Health risks | Pathology | Cirrhosis | Signaling | Injury prevention | Pulmonary fibrosis | Hepatocytes | Collagen | Green fluorescent protein | Laboratory animals | Cancer | Index Medicus
Journal Article