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1. Full Text Utility of mammaglobin immunohistochemistry as a proxy marker for the ETV6-NTRK3 translocation in the diagnosis of salivary mammary analogue secretory carcinoma
by Bishop, Justin A., MD and Yonescu, Raluca, MD and Batista, Denise, PhD and Begum, Shahnaz, MBBS, PhD and Eisele, David W., MD and Westra, William H., MD
Human Pathology, ISSN 0046-8177, 2013, Volume 44, Issue 10, pp. 1982 - 1988
Summary Mammary analogue secretory carcinoma is a recently described salivary gland neoplasm defined by ETV6-NTRK3 gene fusion. Mammary analogue secretory... 
Pathology | Mammary analogue secretory carcinoma | Acinic cell carcinoma | ETV6-NTRK3 | Mammaglobin | GCDFP-15 | ENTITY | DUCT CARCINOMA | PAROTID-GLAND | PATHOLOGY | VARIANT | Translocation, Genetic | Oncogene Proteins, Fusion - metabolism | Carcinoma, Adenoid Cystic - pathology | Tissue Array Analysis | Carcinoma, Adenoid Cystic - metabolism | Humans | Carcinoma, Acinar Cell - pathology | Adenoma, Pleomorphic - metabolism | Carcinoma, Mucoepidermoid - metabolism | Breast Neoplasms - metabolism | Carcinoma, Mucoepidermoid - pathology | Cystadenocarcinoma - metabolism | Immunohistochemistry - methods | Carcinoma, Acinar Cell - metabolism | Cystadenocarcinoma - pathology | Breast Neoplasms - pathology | Mammaglobin A - metabolism | Adenoma, Pleomorphic - pathology | Salivary Gland Neoplasms - metabolism | Biomarkers, Tumor - metabolism | Female | Carcinoma - metabolism | Carcinoma - pathology | Salivary Gland Neoplasms - pathology | Immunohistochemistry | Cytogenetics | Breast cancer | Carcinoma | Diagnosis | Cancer | Cytoplasm | Tumors | acinic cell carcinoma | mammaglobin
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2. Full Text Most Nonparotid “Acinic Cell Carcinomas” Represent Mammary Analog Secretory Carcinomas
by Bishop, Justin A and Yonescu, Raluca and Batista, Denise and Eisele, David W and Westra, William H
The American Journal of Surgical Pathology, ISSN 0147-5185, 07/2013, Volume 37, Issue 7, pp. 1053 - 1057
Acinic cell carcinoma (ACC) is a low-grade salivary gland malignancy characterized by serous acinar differentiation. Most ACCs arise in the parotid gland, but... 
S100 | Minor salivary gland carcinoma | Acinic cell carcinoma | ETV6-NTRK3 translocation | Mammaglobin | Mammary analog secretory carcinoma
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3. Full Text A subset of prostatic basal cell carcinomas harbor the MYB rearrangement of adenoid cystic carcinoma
by Bishop, Justin A., MD and Yonescu, Raluca, MD and Epstein, Jonathan I., MD and Westra, William H., MD
Human Pathology, ISSN 0046-8177, 2015, Volume 46, Issue 8, pp. 1204 - 1208
Summary Adenoid cystic carcinoma (ACC) is a basaloid tumor consisting of myoepithelial and ductal cells typically arranged in a cribriform pattern. Adenoid... 
Pathology | Basal cell carcinoma | MYB-NFIB | Prostate | Adenoid cystic carcinoma | Fluorescence in situ hybridization | NEOPLASMS | TUMOR | CLASSIFICATION | PATHOLOGY | HYPERPLASIA | BREAST | CHROMOSOMAL REARRANGEMENTS | GENE FUSIONS | NFIB | EXPRESSION | GLAND | Prostatic Neoplasms - pathology | Carcinoma, Adenoid Cystic - pathology | Carcinoma, Basal Cell - genetics | Genes, myb - genetics | Humans | In Situ Hybridization, Fluorescence | Male | Carcinoma, Basal Cell - pathology | Prostatic Neoplasms - genetics | Carcinoma, Adenoid Cystic - genetics | Oncogene Proteins, Fusion - genetics | Aged, 80 and over | Aged | Carcinoma | Harbors | Analysis | Cancer | Studies | Transcription factors | Classification schemes | Sunscreen | Genes | Morphology | Classification | Gangrene | Hybridization | Tumors
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4. Full Text Whole‐exome sequencing of pancreatic neoplasms with acinar differentiation
by Jiao, Yuchen and Yonescu, Raluca and Offerhaus, G Johan A and Klimstra, David S and Maitra, Anirban and Eshleman, James R and Herman, James G and Poh, Weijie and Pelosof, Lorraine and Wolfgang, Christopher L and Vogelstein, Bert and Kinzler, Kenneth W and Hruban, Ralph H and Papadopoulos, Nickolas and Wood, Laura D
The Journal of Pathology, ISSN 0022-3417, 03/2014, Volume 232, Issue 4, pp. 428 - 435
Pancreatic carcinomas with acinar differentiation, including acinar cell carcinoma, pancreatoblastoma and carcinomas with mixed differentiation, are distinct... 
genetics | pancreatoblastoma | carcinoma | pancreas | acinar cell carcinoma | sequencing | METHYLATION | HUMAN BREAST | INHIBITION | ONCOLOGY | PATHWAY | MUTATIONS | PATHOLOGY | PANCREATOBLASTOMA | HETEROZYGOSITY | CELL-CARCINOMA | ALLELOTYPE | INSIGHTS | Genetic Predisposition to Disease | Humans | Middle Aged | Pancreatic Neoplasms - pathology | Pancreatic Neoplasms - surgery | Carcinoma, Acinar Cell - pathology | Gene Expression Regulation, Neoplastic | In Situ Hybridization, Fluorescence | Male | Pancreatic Neoplasms - genetics | Carcinoma, Acinar Cell - genetics | Exome | Cell Differentiation - genetics | Phenotype | Acinar Cells - pathology | Acinar Cells - chemistry | Carcinoma, Acinar Cell - surgery | DNA Mutational Analysis | Female | Biomarkers, Tumor - genetics | Chromosomes, Human | DNA, Neoplasm - analysis | Mutation | Fluorescence | Tumor proteins | Analysis | Genes | Tumors
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5. Full Text MYB rearrangement and clinicopathologic characteristics in head and neck adenoid cystic carcinoma
by Rettig, Eleni M and Tan, Marietta and Ling, Shizhang and Yonescu, Raluca and Bishop, Justin A and Fakhry, Carole and Ha, Patrick K
The Laryngoscope, ISSN 0023-852X, 09/2015, Volume 125, Issue 9, pp. E292 - E299
Objectives Salivary gland adenoid cystic carcinoma (ACC) is rare, aggressive, and challenging to treat. Many ACCs have a t(6;9) chromosomal translocation... 
MYB | survival | disease‐free survival | MYB‐NFIB fusion gene | salivary gland neoplasms | Adenoid cystic carcinoma | minor salivary glands | disease-free survival | MYB-NFIB fusion gene | SURGERY | MEDICINE, RESEARCH & EXPERIMENTAL | FUSION | SALIVARY-GLANDS | RADIATION | THERAPY | NFIB | PREDICTORS | OTORHINOLARYNGOLOGY | RECURRENCE | EXPRESSION | Salivary Gland Neoplasms - mortality | Survival Rate - trends | Carcinoma, Adenoid Cystic - pathology | Follow-Up Studies | Salivary Gland Neoplasms - genetics | Humans | Middle Aged | In Situ Hybridization, Fluorescence | Male | Young Adult | Disease-Free Survival | Carcinoma, Adenoid Cystic - mortality | Carcinoma, Adenoid Cystic - genetics | Oncogene Proteins, Fusion - genetics | Adolescent | Aged, 80 and over | Adult | Female | Aged | Retrospective Studies | DNA, Neoplasm - genetics | Gene Rearrangement - genetics | Neoplasm Staging | Salivary Gland Neoplasms - pathology | Development and progression | Carcinoma | Cancer
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6. Full Text Identification of der(1;19)(q10;p10) in Five Oligodendrogliomas Suggests Mechanism of Concurrent 1p and 19q Loss
by Griffin, Constance A and Burger, Peter and Morsberger, Laura and Yonescu, Raluca and Swierczynski, Sharon and Weingart, Jon D and Murphy, Kathleen M
Journal of Neuropathology and Experimental Neurology, ISSN 0022-3069, 10/2006, Volume 65, Issue 10, pp. 988 - 994
Deletions of portions of chromosomes 1p and 19q are closely associated with the oligodendroglioma histologic phenotype. In most cases, 1p and 19q are... 
Brain tumor | Chromosome | Karyotype | Fluorescent in situ hybridization (FISH) | Oligodendroglioma | fluorescent in situ hybridization (FISH) | DIAGNOSIS | oligodendroglioma | chromosome | ASTROCYTOMAS | brain tumor | PATHOLOGY | NEUROSCIENCES | CLINICAL NEUROLOGY | BRAIN-TUMORS | CHROMOSOME-ABNORMALITIES | karyotype | ALPHA-SATELLITE DNA | WHOLE-ARM TRANSLOCATION | NERVOUS-SYSTEM TUMORS | HUMAN GLIOMAS | COMPARATIVE GENOMIC HYBRIDIZATION | MIXED OLIGOASTROCYTOMAS | Chromosome Deletion | Translocation, Genetic | Oligodendroglioma - genetics | Humans | Brain Neoplasms - genetics | In Situ Hybridization, Fluorescence | Male | Loss of Heterozygosity | Chromosomes, Human, Pair 19 | Chromosomes, Human, Pair 1 | Chromosome Aberrations | Adult | Female
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7. Full Text Genetic analyses of isolated high-grade pancreatic intraepithelial neoplasia (HG-PanIN) reveal paucity of alterations in TP53 and SMAD4
by Hosoda, Waki and Chianchiano, Peter and Griffin, James F and Pittman, Meredith E and Brosens, Lodewijk Aa and Noë, Michaël and Yu, Jun and Shindo, Koji and Suenaga, Masaya and Rezaee, Neda and Yonescu, Raluca and Ning, Yi and Albores-Saavea, Jorge and Yoshizawa, Naohiko and Harada, Kenichi and Yoshizawa, Akihiko and Hanada, Keiji and Yonehara, Shuji and Shimizu, Michio and Uehara, Takeshi and Samra, Jaswinder S and Gill, Anthony J and Wolfgang, Christopher L and Goggins, Michael G and Hruban, Ralph H and Wood, Laura D
Journal of Pathology, ISSN 0022-3417, 05/2017, Volume 242, Issue 1, pp. 16 - 23
High-grade pancreatic intraepithelial neoplasia (HG-PanIN) is the major precursor of pancreatic ductal adenocarcinoma (PDAC) and is an ideal target for early... 
SMAD4 | cancerization | targeted next-generation sequencing | pancreas | Multicenter Study | HG-PanIN | pancreatic intraepithelial neoplasia | whole-exome sequencing | TP53 | Journal Article | pancreatic ductal adenocarcinoma | HG‐PanIN | whole‐exome sequencing | targeted next‐generation sequencing | PATHWAYS | DUCTAL ADENOCARCINOMA | TUMOR | PATHOLOGY | CANCER | LESIONS | INACTIVATION | ONCOLOGY | JUICE | MUTATIONS | EXPRESSION | PROGRESSION | Pancreatic Neoplasms - metabolism | Carcinoma in Situ - pathology | Humans | Pancreatic Neoplasms - pathology | Tumor Suppressor Protein p53 - metabolism | Pancreatic Neoplasms - genetics | Genes, p53 - genetics | Neoplasm Proteins - metabolism | Smad4 Protein - metabolism | Genome, Human - genetics | Carcinoma in Situ - genetics | Neoplasm Grading | Smad4 Protein - genetics | Mutation | Carcinoma in Situ - metabolism | High-Throughput Nucleotide Sequencing - methods | Neoplasm Proteins - genetics
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8. Full Text Pancreatic undifferentiated carcinoma with osteoclast-like giant cells is genetically similar to, but clinically distinct from, conventional ductal adenocarcinoma
by Luchini, Claudio and Pea, Antonio and Lionheart, Gemma and Mafficini, Anea and Nottegar, Alessia and Veronese, Nicola and Brosens, L.A.A and Scarpa, Aldo and Wood, Laura D
Journal of Pathology, ISSN 0022-3417, 2017, Volume 243, Issue 2, pp. 148 - 154
Undifferentiated carcinoma of the pancreas with osteoclast‐like giant cells (UCOGC) is currently considered a morphologically and clinically distinct variant... 
undifferentiated carcinoma with osteoclast‐like giant cells | PDAC variants | whole exome sequencing | undifferentiated carcinoma with osteoclast-like giant cells | GENOMIC ANALYSES | PATHOLOGY | INTRAEPITHELIAL NEOPLASIA | CANCER | MOLECULAR ANALYSIS | GENE | ONCOLOGY | LIVER | MUTATIONS | TP53 | Immunohistochemistry | Osteoclasts - pathology | Humans | Middle Aged | Pancreatic Neoplasms - pathology | Male | Pancreatic Neoplasms - genetics | Mutation - genetics | Carcinoma, Pancreatic Ductal - pathology | Carcinoma, Pancreatic Ductal - genetics | Exome - genetics | Aged, 80 and over | Carcinoma, Pancreatic Ductal - mortality | Female | Aged | Pancreatic Neoplasms - mortality | Neoplasm Proteins - genetics | Adenocarcinoma | Tumor proteins | Analysis | Pancreatic cancer | Sequences | Pancreatic carcinoma | p53 Protein | Tumor suppressor genes | Biocompatibility | Giant cells | Mutation | Pancreas | Smad4 protein | K-Ras protein
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9. Full Text Most Nonparotid "Acinic Cell Carcinomas" Represent Mammary Analog Secretory Carcinomas
by Bishop, JA and Yonescu, R and Batista, D and Eisele, DW and Westra, WH
AMERICAN JOURNAL OF SURGICAL PATHOLOGY, ISSN 0147-5185, 07/2013, Volume 37, Issue 7, pp. 1053 - 1057
Acinic cell carcinoma (ACC) is a low-grade salivary gland malignancy characterized by serous acinar differentiation. Most ACCs arise in the parotid gland, but... 
SURGERY | ORIGIN | mammary analog secretory carcinoma | minor salivary gland carcinoma | mammaglobin | ETV6-NTRK3 GENE FUSION | S100 | acinic cell carcinoma | PATHOLOGY | ETV6-NTRK3 translocation | FIBROSARCOMA | MINOR SALIVARY-GLANDS | Translocation, Genetic | Humans | Middle Aged | Carcinoma, Acinar Cell - pathology | Male | Mouth Neoplasms - metabolism | Breast Neoplasms - metabolism | Submandibular Gland Neoplasms - metabolism | Young Adult | Carcinoma, Acinar Cell - metabolism | Salivary Gland Neoplasms - metabolism | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Female | Retrospective Studies | Acinar Cells - metabolism | Submandibular Gland Neoplasms - pathology | S100 Proteins - metabolism | Acinar Cells - pathology | Carcinoma, Acinar Cell - surgery | Breast Neoplasms - pathology | Mammaglobin A - metabolism | Mouth Neoplasms - pathology | Aged | Salivary Gland Neoplasms - pathology | Salivary Gland Neoplasms - surgery
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10. Full Text Cytopathologic features of mammary analogue secretory carcinoma
by Bishop, Justin A and Yonescu, Raluca and Batista, Denise A. S and Westra, William H and Ali, Syed Z
Cancer Cytopathology, ISSN 1934-662X, 05/2013, Volume 121, Issue 5, pp. 228 - 233
BACKGROUND Mammary analogue secretory carcinoma (MASC) is a recently described salivary gland neoplasm that is defined by ETV6‐NTRK3 gene fusion. To the best... 
mammary analogue secretory carcinoma | salivary glands | acinic cell carcinoma | fine‐needle aspiration | cytopathology | Mammary analogue secretory carcinoma | Cytopathology | Fine-needle aspiration | Acinic cell carcinoma | Salivary glands | FINE-NEEDLE-ASPIRATION | ONCOLOGY | PAROTID-GLAND | ETV6-NTRK3 GENE FUSION | fine-needle aspiration | PATHOLOGY | LESIONS | Adenoma, Pleomorphic - genetics | Diagnosis, Differential | Carcinoma, Mucoepidermoid - diagnosis | Salivary Gland Neoplasms - genetics | Humans | Middle Aged | Carcinoma, Acinar Cell - pathology | Adenoma, Pleomorphic - diagnosis | In Situ Hybridization, Fluorescence | Male | Carcinoma, Acinar Cell - genetics | Salivary Gland Neoplasms - diagnosis | Carcinoma, Mucoepidermoid - pathology | Young Adult | Biopsy, Fine-Needle | Carcinoma, Mucoepidermoid - genetics | Carcinoma, Acinar Cell - diagnosis | Oncogene Proteins, Fusion - genetics | Adenoma, Pleomorphic - pathology | Adult | Female | Aged | Salivary Gland Neoplasms - pathology | Research | Diagnosis | Salivary gland tumors | Pathology, Cellular | Distribution | Cancer
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11. Full Text Whole-exome sequencing of duodenal neuroendocrine tumors in patients with neurofibromatosis type 1
by Noe, Michael and Pea, Antonio and Luchini, Claudio and Felsenstein, Matthaus and Barbi, Stefano and Bhaijee, Feriyl and Brosens, L.A.A and Roberts, Nicholas J and Wood, Laura D
Modern Pathology, ISSN 0893-3952, 2018, Volume 31, Issue 10, pp. 1532 - 1538
Neurofibromatosis type 1 (NF1) is a hereditary cancer predisposition syndrome characterized by frequent cutaneous and nervous system abnormalities. Patients... 
NF1 INACTIVATION | GENE | PHEOCHROMOCYTOMA | LANDSCAPE | DISEASE | SOMATIC MUTATION | PANCREATITIS | PATHOLOGY | GASTROINTESTINAL STROMAL TUMORS | NERVE SHEATH TUMORS | MULTIPLE | Copy number | Genes | Neurofibromin 1 | Nervous system | Chromosome deletion | Recklinghausen's disease | Heterozygosity | Clonal deletion | Neurofibromatosis | Loss of heterozygosity | Chromosome 22 | Tumor suppressor genes | Somatostatin | Neuroendocrine tumors | Tumors | Cancer
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12. Full Text GATA6 activates Wnt signaling in pancreatic cancer by negatively regulating the Wnt antagonist Dickkopf-1
by Zhong, Yi and Wang, Zheng and Fu, Baojin and Pan, Fan and Yachida, Shinichi and Dhara, Mousumi and Albesiano, Emilia and Li, Li and Naito, Yoshiki and Vilardell, Felip and Cummings, Christopher and Martinelli, Paola and Li, Ang and Yonescu, Raluca and Ma, Qingyong and Griffin, Constance A and Real, Francisco X and Iacobuzio-Donahue, Christine A
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 7, p. e22129
Pancreatic ductal adenocarcinoma (PDAC) is a highly lethal disease characterized by late diagnosis and treatment resistance. Recurrent genetic alterations in... 
DUCTAL ADENOCARCINOMA | EPIGENETIC INACTIVATION | ZINC-FINGER | IN-VIVO | BIOLOGY | TUMOR-SUPPRESSOR GENE | E-CADHERIN | DIFFERENTIATION | BETA-CATENIN | EXPRESSION | TRANSCRIPTION FACTORS GATA-4 | Gene Dosage - genetics | Cell Proliferation | Signal Transduction | Carcinoma in Situ - pathology | Humans | Pancreatic Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Intercellular Signaling Peptides and Proteins - genetics | Pancreatic Neoplasms - genetics | Wnt Proteins - metabolism | Carcinoma, Pancreatic Ductal - pathology | Disease Progression | GATA6 Transcription Factor - metabolism | Carcinoma, Pancreatic Ductal - genetics | Gene Knockdown Techniques | Intercellular Signaling Peptides and Proteins - metabolism | Carcinoma in Situ - genetics | Cell Line, Tumor | Wnt Proteins - antagonists & inhibitors | Chromatin | Cellular signal transduction | DNA microarrays | RNA | Pancreatic cancer | Analysis | Adenocarcinoma | Cell proliferation | Cell culture | Biotechnology | Immunoprecipitation | Media (culture) | Wnt protein | Copy number | Fluorescence | Oncology | Activation | Metastasis | Tissues | Carcinogenesis | Signal transduction | Carcinogens | Transfection | Surgery | Deoxyribonucleic acid--DNA | Binding | Medical research | Cell survival | Secretion | Medical treatment | Agar | siRNA | Tumor cell lines | Gene expression | Medicine | Pathology | Signaling | Electrophoretic mobility | Cell lines | Culture media | Laboratory animals | Dkk1 protein | Tumors | Cancer | Malalties | Pàncrees | Fetge | Càncer | Pèptids | Deoxyribonucleic acid | DNA
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13. Full Text Tumor-infiltrating macrophages in post-transplant, relapsed classical Hodgkin lymphoma are donor-derived
by Crane, Genevieve M and Samols, Mark A and Morsberger, Laura A and Yonescu, Raluca and Thiess, Michele L and Batista, Denise A. S and Ning, Yi and Burns, Kathleen H and Vuica-Ross, Milena and Borowitz, Michael J and Gocke, Christopher D and Ambinder, Richard F and Duffield, Amy S
PLoS ONE, ISSN 1932-6203, 09/2016, Volume 11, Issue 9, p. e0163559
Tumor-associated inflammatory cells in classical Hodgkin lymphoma (CHL) typically out-number the neoplastic Hodgkin/Reed-Sternberg (H/RS) cells. The... 
SURVIVAL | REED-STERNBERG CELLS | DENDRITIC CELLS | CCL5/RANTES | PROGNOSIS | MULTIDISCIPLINARY SCIENCES | DISEASE | REGULATORY T-CELLS | MONOCYTES | EXPRESSION | EPSTEIN-BARR-VIRUS | Relapse | Analysis | Bone marrow | Transplantation | Non-Hodgkin's lymphomas | Macrophages | Diseases | Antigens | Animal models | Transplants & implants | Bone marrow transplantation | Inflammation | Patients | Lymphoma | Studies | Pathology | Monocytes | Medical prognosis | Lymphomas | Chimerism | Archives | Hodgkin's disease | Deoxyribonucleic acid--DNA | Cancer | Tumors | Deoxyribonucleic acid | DNA
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14. Full Text Utilization of a TFE3 Break-apart FISH Assay in a Renal Tumor Consultation Service
by Green, Whitney M and Yonescu, Raluca and Morsberger, Laura and Morris, Kerry and Netto, George J and Epstein, Jonathan I and Illei, Peter B and Allaf, Mohamad and Ladanyi, Marc and Griffin, Constance A and Argani, Pedram
The American Journal of Surgical Pathology, ISSN 0147-5185, 08/2013, Volume 37, Issue 8, pp. 1150 - 1163
Xp11 translocation renal cell carcinomas (RCCs) are characterized by chromosome translocations involving the Xp11.2 breakpoint, resulting in gene fusions... 
FISH | renal cell carcinoma | Xp11 translocation | TFE3 | SURGERY | TRANSLOCATION CARCINOMAS | NEOPLASMS | SOFT PART SARCOMA | PATHOLOGY | CELL CARCINOMA | FEATURES | GENE FUSIONS | CLEAR-CELL | CATHEPSIN-K | SPECTRUM | IMMUNOHISTOCHEMISTRY | Immunohistochemistry | Predictive Value of Tests | Translocation, Genetic | Kidney Neoplasms - genetics | Prognosis | Carcinoma, Renal Cell - chemistry | Humans | Middle Aged | Carcinoma, Renal Cell - genetics | Male | Kidney Neoplasms - chemistry | Chromosomes, Human, X | Young Adult | Adult | Female | Child | Genetic Predisposition to Disease | Biomarkers, Tumor - analysis | Carcinoma, Renal Cell - pathology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - analysis | In Situ Hybridization, Fluorescence | Pathology, Clinical - methods | Phenotype | Gene Rearrangement | Adolescent | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Kidney Neoplasms - pathology | Aged | Biomarkers, Tumor - genetics | Referral and Consultation
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