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Circulation Journal, ISSN 1346-9843, 2018, Volume 82, Issue 2, pp. 486 - 493
Background:This study aimed to evaluate the diagnostic performance of coronary flow reserve (CFR), hyperemic myocardial blood flow (hMBF), and CFR ratio for... 
Coronary artery disease | Positron emission tomography | Myocardial perfusion imaging
Journal Article
JOURNAL OF BONE AND MINERAL RESEARCH, ISSN 0884-0431, 05/2014, Volume 29, Issue 5, pp. 1170 - 1182
Cherubism is a genetic disorder of the craniofacial skeleton caused by gain-of-function mutations in the signaling adaptor protein, SH3-domain binding protein... 
RHEUMATOID-ARTHRITIS | CHERUBISM | APOPTOSIS | CROHNS-DISEASE | EFFICACY | SERIOUS INFECTION | NECROSIS-FACTOR-ALPHA | ADAPTER PROTEIN 3BP2 | TNF | BONE LOSS | THERAPY | INFLAMMATION | MOUSE MODEL | ENDOCRINOLOGY & METABOLISM | DIFFERENTIATION | ETANERCEPT | Infants (Newborn) | Inflammation | Etanercept | Tumor necrosis factor | Analysis | Protein binding
Journal Article
Modern Rheumatology, ISSN 1439-7595, 2/2010, Volume 20, Issue 1, pp. 11 - 17
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2014, Volume 9, Issue 8, p. e105518
Objective: SH3BP2 is a signaling adapter protein which regulates immune and skeletal systems. Gain-of-function mutations in SH3BP2 cause cherubism,... 
RHEUMATOID-ARTHRITIS | CHERUBISM | COLONY-STIMULATING FACTOR | M-CSF | NUCLEAR-FACTOR | MULTIDISCIPLINARY SCIENCES | RECEPTOR ACTIVATOR | ADAPTER PROTEIN 3BP2 | MICE | T-CELLS | KAPPA-B LIGAND | Tumor Necrosis Factor-alpha - metabolism | Arthritis, Experimental - genetics | Cell Proliferation | RANK Ligand - metabolism | Cartilage - pathology | Tumor Necrosis Factor-alpha - genetics | Arthritis, Experimental - diagnosis | X-Ray Microtomography | Arthritis, Experimental - pathology | RANK Ligand - pharmacology | Bone Resorption - genetics | Gene Expression | Cytokines - metabolism | NFATC Transcription Factors - metabolism | Lymphoid Tissue - metabolism | Mice, Transgenic | Arthritis, Experimental - immunology | Osteoclasts - metabolism | Macrophages - metabolism | Animals | Adaptor Proteins, Signal Transducing - genetics | Macrophages - drug effects | Mice | Mutation | Adaptor Proteins, Signal Transducing - metabolism | Amino Acid Substitution | Rheumatoid factor | Analysis | Collagen | Arthritis | Genetic aspects | Inflammation | Macrophage colony stimulating factor | B cells | Gene expression | Cell culture | Transcription factors | Collagens | Jawbone | Proteins | Cell activation | Bone growth | Computed tomography | Bone marrow | Fibroblasts | Biocompatibility | Cytokines | Adapters | Rheumatology | Macrophage colony-stimulating factor | T cell receptors | Tumor necrosis factor-α | Joint diseases | Interleukin 17 | Osteoclastogenesis | Rheumatoid arthritis | γ-Interferon | Lymph | Health sciences | Phosphorylation | Collagen (type II) | Activation | Macrophages | Lymph nodes | Biomedical materials | Rodents | Tumor necrosis factor-TNF | Bone loss | TRANCE protein | Localization | Pharmaceutical sciences | Dentistry | Medicine | Signaling | Government agencies | Lymphocytes B | Drainage measurement | Osteoclasts | Laboratory animals
Journal Article
Journal of Orthopaedic Research, ISSN 0736-0266, 12/2013, Volume 31, Issue 12, pp. 1992 - 1998
Journal Article
Journal of Bone and Mineral Research, ISSN 0884-0431, 12/2014, Volume 29, Issue 12, p. 2618
Cherubism (OMIM# 118400) is a genetic disorder with excessive jawbone resorption caused by mutations in SH3 domain binding protein 2 (SH3BP2), a signaling... 
Genetic aspects | Macrophage colony stimulating factor | Tumor necrosis factor | Analysis | Protein binding
Journal Article
Cell Reports, ISSN 2211-1247, 09/2014, Volume 8, Issue 6, pp. 1752 - 1766
Cherubism is caused by mutations in SH3BP2. Studies of cherubism mice showed that tumor necrosis factor α (TNF-α)-dependent autoinflammation is a major cause... 
RESPONSES | ACTIVATION | BACTERIAL-INFECTIONS | DISRUPTION | INFLAMMATION | ADAPTER PROTEIN 3BP2 | VAV | MUTATIONS | PATHOGEN RECOGNITION | PATHOPHYSIOLOGY | CELL BIOLOGY | Tumor Necrosis Factor-alpha - metabolism | Cherubism - etiology | Liver - pathology | Protein-Tyrosine Kinases - metabolism | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | NF-kappa B - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Protein-Tyrosine Kinases - deficiency | RNA, Messenger - metabolism | Intracellular Signaling Peptides and Proteins - deficiency | Protein-Tyrosine Kinases - genetics | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Disease Models, Animal | Signal Transduction | Jaw - diagnostic imaging | Mice, Inbred C57BL | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Inflammation | Toll-Like Receptor 4 - chemistry | Macrophages - cytology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Radiography | Macrophages - metabolism | Animals | Myeloid Differentiation Factor 88 - deficiency | Adaptor Proteins, Signal Transducing - deficiency | Adaptor Proteins, Signal Transducing - genetics | Mice | Adaptor Proteins, Signal Transducing - metabolism | Myeloid Differentiation Factor 88 - metabolism | Toll-Like Receptor 2 - chemistry
Journal Article