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Journal of Neurochemistry, ISSN 0022-3042, 02/2012, Volume 120, Issue 3, pp. 461 - 472
J. Neurochem. (2012) 120, 461–472. Activation of microglia, the resident macrophages of the brain, around the amyloid plaques is a key hallmark of Alzheimer’s... 
NF‐κB | Alzheimer’s disease | microglia | resveratrol | TLR4 | NF-κB | Alzheimer's disease | Tumor Necrosis Factor-alpha - metabolism | Stilbenes - therapeutic use | Amyloid beta-Peptides - pharmacology | Humans | Tumor Necrosis Factor-alpha - genetics | Male | I-kappa B Proteins - metabolism | Stilbenes - pharmacology | I-kappa B Proteins - genetics | Alzheimer Disease - pathology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Drug Interactions | Time Factors | Inflammation Mediators - pharmacology | Statistics, Nonparametric | Toll-Like Receptors - metabolism | Disease Models, Animal | NF-KappaB Inhibitor alpha | Cytokines - metabolism | Microglia - drug effects | Alzheimer Disease - drug therapy | Presenilin-1 - genetics | Mice, Transgenic | Amyloid beta-Protein Precursor - genetics | Animals | Signal Transduction - drug effects | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Toll-Like Receptors - genetics | Lipopolysaccharides - pharmacology | Mice | Alzheimer Disease - genetics | Cell Line, Transformed | Brain | Cell culture | Animal models | Phosphorylation | Transcription | Clinical trials | beta -Amyloid | Macrophages | IKK protein | Lipopolysaccharides | Interleukin 6 | Cell activation | Resveratrol | Microglial cells | Polyphenols | Toll-like receptors | Amyloid | Stat1 protein | Plaques | Antiinflammatory agents | Oligomerization | Neurodegenerative diseases | Stat3 protein | Inflammation | TLR4 protein | Microglia | NF- Kappa B protein | Lymphocytes B | Tumor necrosis factor- alpha | STAT | lipopolysaccharide
Journal Article
Journal Article
Journal Article
Journal Article
Current Oncology Reports, ISSN 1523-3790, 11/2017, Volume 19, Issue 11, pp. 1 - 9
Diffuse large B cell lymphoma (DLBCL) is the most common type of non-Hodgkin lymphoma (NHL), accounting for approximately 25% of NHL cases. It is a... 
DLBCL | Medicine & Public Health | BCL6 | MYC | BCL2 | Oncology | Double-hit lymphoma | SURVIVAL | PROGNOSTIC IMPACT | C-MYC | FEATURES | CLINICAL-PRACTICE GUIDELINES | BURKITT-LYMPHOMA | ONCOLOGY | CHOP | INHIBITOR | EXPRESSION | Medical colleges | Chemotherapy | Lymphomas | Gene mutations | Cancer | Lymphocytes B | Clinical trials | Myc protein | Bcl-6 protein | B-cell lymphoma
Journal Article
Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research, ISSN 0884-0431, 05/2019
Skeletal abnormalities are common comorbidities of inflammatory bowel disease (IBD). Patients suffering from IBD, including ulcerative colitis and Crohn's... 
Journal Article
SCIENCE TRANSLATIONAL MEDICINE, ISSN 1946-6234, 02/2019, Volume 11, Issue 481, p. eaav4319
Tendon disorders represent the most common musculoskeletal complaint for which patients seek medical attention; inflammation drives tendon degeneration before... 
MEDICINE, RESEARCH & EXPERIMENTAL | SUPRASPINATUS TENDON | POPULATION | APOPTOSIS | REPAIR INTEGRITY | INFLAMMATION | ROTATOR CUFF TEARS | MECHANISMS | TENDINOPATHY | INTERLEUKIN-6 | EXPRESSION | CELL BIOLOGY
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e91421
Pathologic conditions impair bone homeostasis. The transcription factor NF-kappa B regulates bone homeostasis and is central to bone pathologies. Whereas... 
RHEUMATOID-ARTHRITIS | SCLEROSTIN | INHIBITION | INFLAMMATORY DISEASES | MULTIDISCIPLINARY SCIENCES | IKK-BETA | DICKKOPF-1 | DIFFERENTIATION | NF-KAPPA-B | EXPRESSION | INDUCED BONE LOSS | Bone and Bones - pathology | Chondrocytes - cytology | Osteogenesis - physiology | Bone Development - physiology | NF-kappa B - metabolism | I-kappa B Kinase - metabolism | Bone and Bones - metabolism | Models, Animal | Cell Differentiation | Osteoblasts - cytology | Chondrogenesis | Chondrocytes - metabolism | Gene Expression | Bone Density | Stromal Cells - metabolism | Mice, Transgenic | I-kappa B Kinase - genetics | Phenotype | Animals | NF-kappa B - genetics | Models, Biological | Biomarkers | Mice | Enzyme Activation | Osteoblasts - metabolism | Stromal Cells - cytology | Alkaline phosphatase | Disease | Multiple myeloma | IKK2 protein | Homeostasis | Heterozygotes | Activation | Biochemistry | Kinases | Bone (trabecular) | Osteoblasts | Epiphysis | Inflammatory diseases | Ossification | Osteoporosis | Biomedical materials | Bone growth | Computed tomography | Tibia | Rodents | Biocompatibility | Bones | Skeleton | NF-κB protein | Vertebrae | Ethanol | Deformation mechanisms | Markers | Histology | Bone turnover | Homozygotes | Medicine | Studies | Osteoblastogenesis | Hedgehog protein | Orthopedics | Rheumatoid arthritis | Collagen | Chondrocytes | Bone | Aggrecan | Differentiation | Osteogenesis
Journal Article