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Hypertension, ISSN 0194-911X, 01/2019, Volume 73, Issue 1, pp. 92 - 101
Atrial fibrillation (AF) is the most common type of cardiac arrhythmia and increases the risk of stroke, heart failure, and death. Ang II (angiotensin II)... 
AT1R associated protein | atrial fibrillation | immunoproteasome | catalytic subunit β5 | mice | angiotensin II
Journal Article
by Li, J and Wang, S and Zhang, YL and Bai, J and Lin, QY and Liu, RS and Yu, XH and Li, HH
HYPERTENSION, ISSN 0194-911X, 01/2019, Volume 73, Issue 1, pp. 92 - 101
Atrial fibrillation (AF) is the most common type of cardiac arrhythmia and increases the risk of stroke, heart failure, and death. Ang II (angiotensin II)... 
immunoproteasome | ACTIVATION | N-ACETYLCYSTEINE | KAPPA-B | mice | AT1R associated protein | PRESSURE | atrial fibrillation | CARDIAC PROTEASOME | angiotensin II | INFLAMMATION | catalytic subunit 5 | PERIPHERAL VASCULAR DISEASE
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, p. e35506
Background: The inflammatory response plays a critical role in hypertension-induced cardiac remodeling. We aimed to study how interaction among inflammatory... 
FIBROBLAST PRECURSORS | SYSTEM | CYTOKINE PROFILES | INTERFERON-GAMMA | MULTIDISCIPLINARY SCIENCES | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | ANGIOTENSIN-II | MYOCARDITIS | HYPERTENSION | NF-KAPPA-B | EXPRESSION | Inflammation - chemically induced | Angiotensin II - pharmacology | Myocardium - immunology | Interferon-gamma - metabolism | Fibrosis - metabolism | Mice, Knockout | Inflammation - metabolism | Animals | T-Lymphocytes - metabolism | Myocardium - metabolism | Fibrosis - immunology | Chemokine CCL2 - metabolism | Mice | Interferon-gamma - genetics | Body Weight - physiology | Heart | Cell culture | Collagen (type I) | Transcription factors | Disease | Leukocyte migration | Smooth muscle | Nervous system | Leukocytes | Recruitment | Metastases | Proteins | Ischemia | Education | Fibroblasts | Blood pressure | Cytokines | Cardiac muscle | Tumor necrosis factor-α | Low level | Hospitals | Monocyte chemoattractant protein | Nitric oxide | γ-Interferon | Fibrosis | Infiltration | Cell migration | Monocyte chemoattractant protein 1 | Flow cytometry | Laboratories | Inflammatory response | Lymphocytes T | Insulin-like growth factors | Macrophages | Remodeling | Infusion | Lymphocytes | Actin | Rodents | Tumor necrosis factor-TNF | Angiotensin II | Heart diseases | Hypertension | Kidneys | Muscles | Inflammation | White blood cells | Angiotensin | Interferon | gamma -Interferon | Tumor necrosis factor- alpha
Journal Article
Cancer Letters, ISSN 0304-3835, 2016, Volume 376, Issue 2, pp. 268 - 277
Highlights • ACE2 expression is downregulated in breast cancer • ACE2 expression is negatively correlated with metastasis from breast cancer • Inhibition of... 
Hematology, Oncology and Palliative Medicine | ACE2/Ang-(1–7)/Mas axis | Breast cancer metastasis | Store-operated calcium entry (SOCE) | E-cadherin | PAK1/NF-κB/Snail1 pathways | ACE2/Ang-(1-7)/Mas axis | CONVERTING ENZYME 2 | PAK1/NF-kappa B/Snail1 pathways | ANGIOGENESIS | FACTOR-KAPPA-B | ANGIOTENSIN-(1-7) | LUNG-CANCER | INHIBITION | ONCOLOGY | CELL-MIGRATION | PROSTATE-CANCER | EXPRESSION | Neoplasm Transplantation | Cadherins - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Gene Expression Regulation, Neoplastic | NF-kappa B - metabolism | Peptidyl-Dipeptidase A - genetics | Breast Neoplasms - enzymology | Neoplasm Metastasis | Heterografts | Neoplasm Grading | Transfection | MCF-7 Cells | RNA Interference | Peptidyl-Dipeptidase A - metabolism | Female | Calcium Signaling | Proto-Oncogene Proteins - metabolism | Peptide Fragments - metabolism | Snail Family Transcription Factors - metabolism | Down-Regulation | Angiotensin I - metabolism | Mice, SCID | p21-Activated Kinases - metabolism | Gene Expression Regulation, Enzymologic | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Mice, Inbred NOD | Cell Movement | Prevention | Breast cancer | Metastasis | Cancer | Angiotensin | Physiological aspects | Development and progression | Proteins | Studies | Phosphorylation | Lymphatic system | Calcium | Prostate cancer | Tumors
Journal Article
Inflammation Research, ISSN 1023-3830, 03/2015, Volume 64, Issue 3-4, pp. 253 - 260
Journal Article
CNS Neuroscience & Therapeutics, ISSN 1755-5930, 05/2014, Volume 20, Issue 5, pp. 452 - 459
Summary Background The angiotensin (Ang) converting enzyme 2 (ACE2)/Ang‐(1‐7)/Mas receptor pathway is an important component of the renin–angiotensin system... 
Aging | Ischemic stroke | NADPH oxidase | Angiotensin | Angiotensin‐converting enzyme 2 | Angiotensin-converting enzyme 2 | RENIN | NITRIC-OXIDE SYNTHASE | RECEPTOR | NEUROSCIENCES | STROKE | ACE2 | OVEREXPRESSION | PHARMACOLOGY & PHARMACY | MICE | DYSFUNCTION | CONTRIBUTES | EXPRESSION | Infarction, Middle Cerebral Artery - physiopathology | Neurons - pathology | Reactive Oxygen Species - metabolism | Age Factors | Membrane Glycoproteins - metabolism | NADPH Oxidases - metabolism | Male | Stroke - physiopathology | Hypoxia, Brain - pathology | Stroke - pathology | Infarction, Middle Cerebral Artery - complications | Neurons - physiology | Peptidyl-Dipeptidase A - metabolism | Female | Peptide Fragments - metabolism | Angiotensin II - metabolism | Brain Edema - etiology | Tissue Culture Techniques | Brain - physiopathology | Brain Edema - pathology | Mice, Transgenic | Brain Ischemia - physiopathology | Angiotensin II - analogs & derivatives | Brain Ischemia - etiology | Infarction, Middle Cerebral Artery - pathology | NADPH Oxidase 4 | Angiotensin I - metabolism | NADPH Oxidase 2 | Hypoxia, Brain - physiopathology | Glucose - deficiency | Stroke - etiology | Animals | Brain Edema - physiopathology | Cell Death - physiology | Brain Ischemia - pathology | Brain - pathology | Oxidases | Stroke (Disease) | Glucose metabolism | Angiotensin converting enzyme | Genetic engineering | Glucose | Dextrose | Enzymes | Stroke | Rodents | Dependence | Apoptosis | Index Medicus
Journal Article
by Fang, L and Zhang, PF and Wang, KK and Xiao, ZL and Yang, M and Yu, ZX
INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE, ISSN 1107-3756, 04/2019, Volume 43, Issue 4, pp. 1597 - 1610
The current study aimed to clarify the role of nucleolin in the phenotypic transformation of vascular smooth muscle cells (VSMCs) and to preliminarily explore... 
MEDICINE, RESEARCH & EXPERIMENTAL | PROTEIN | STABILIZATION | phenotypic transformation | PROLIFERATION | MECHANISMS | MODEL | CANCER | INHIBITION | angiotensin II | GROWTH-FACTOR | DIFFERENTIATION | vascular smooth muscle cells | EXPRESSION | nucleolin
Journal Article
Cancer Chemotherapy and Pharmacology, ISSN 0344-5704, 4/2017, Volume 79, Issue 4, pp. 747 - 758
Journal Article
Journal Article
JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2014, Volume 64, Issue 16, pp. C46 - C46
  Objectives To investigate the effects of angiotensinII (AngII) and angiotensin-(1-7) [Ang-(1-7)] on scavenger receptor class B type1 (SR-BI), ATP-binding... 
Cardiovascular | Internal Medicine | Cholesterol
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 14369 - 11
MicroRNA-21 (mir-21) induced by angiotensin II (AngII) plays a vital role in the development of pulmonary fibrosis, and the NLRP3 inflammasome is known to be... 
APOPTOSIS | DISEASES | PATHWAY | AXIS PROTECTS | MULTIDISCIPLINARY SCIENCES | PROLIFERATION | CARDIAC FIBROSIS | KAPPA-B | IDIOPATHIC PULMONARY-FIBROSIS | SYSTEMIC-SCLEROSIS | MICRORNA-21 | NF-κB protein | Lung diseases | MiRNA | Extracellular signal-regulated kinase | Bleomycin | Pulmonary fibrosis | Overexpression | Peptidyl-dipeptidase A | Collagen | Rodents | Fibrosis | Angiotensin | Fibroblasts | Angiotensin II | Apoptosis
Journal Article
American Journal of Translational Research, ISSN 1943-8141, 2018, Volume 10, Issue 7, pp. 1976 - 1989
Myocardial fibrosis (MF) plays an important part in cardiovascular diseases. The main cytological characteristics of MF is the increased number of... 
Pericytes | Myofibroblasts | Valproic acid | Fibrosis | FIBROBLAST PRECURSORS | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | pericytes | ANGIOTENSIN-II | MECHANISMS | PROTEIN PHOSPHATASE 2A | HYPERTROPHY | IN-VITRO | ONCOLOGY | INFLAMMATION | fibrosis | CARDIAC FIBROSIS | HISTONE DEACETYLASE INHIBITORS | myofibroblasts
Journal Article