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细胞研究:英文版, ISSN 1001-0602, 2015, Volume 25, Issue 9, pp. 997 - 1012
Organ size determination is one of the most intriguing unsolved mysteries in biology. Aberrant activation of the major effector and transcription co-activator... 
正反馈机制 | 活性抑制剂 | 直接诱导 | 信号通路 | 激活因子 | 肿瘤发生 | 反馈回路 | microRNA | YAP | tumorigenesis | organ size | Hippo | GROWTH-CONTROL | HIPPO SIGNALING PATHWAY | STEM-CELL PROLIFERATION | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER CELLS | IN-VIVO | YAP ONCOPROTEIN | BANTAM MICRORNA | TUMOR-SUPPRESSOR PATHWAY | TRANSCRIPTIONAL COMPLEX | Cell Proliferation | MicroRNAs - antagonists & inhibitors | Humans | Male | MicroRNAs - metabolism | Transplantation, Heterologous | Phosphoproteins - antagonists & inhibitors | Drosophila Proteins - metabolism | Phosphoproteins - metabolism | Carcinogenesis | Cysteine-Rich Protein 61 - metabolism | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Base Sequence | Organ Size - genetics | Signal Transduction | Cysteine-Rich Protein 61 - genetics | Transcription Factors - antagonists & inhibitors | Phosphoproteins - genetics | Transcription Factors - genetics | Cysteine-Rich Protein 61 - antagonists & inhibitors | Mice, Inbred ICR | Hep G2 Cells | Transcription Factors - metabolism | Sequence Alignment | Animals | Mice, Nude | Adaptor Proteins, Signal Transducing - genetics | Cell Line, Tumor | Mice | Drosophila - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Index Medicus | Original
Journal Article
Journal Article
Genes and Development, ISSN 0890-9369, 07/2008, Volume 22, Issue 14, pp. 1962 - 1971
The YAP transcription coactivator has been implicated as an oncogene and is amplified in human cancers. Recent studies have established that YAP is... 
YAP | TEAD | Transcription | Hippo | Cancer | CTGF | YES-ASSOCIATED PROTEIN | DOMAIN | transcription | HIPPO PATHWAY | CELL-PROLIFERATION | DEVELOPMENTAL BIOLOGY | DROSOPHILA | CELL BIOLOGY | ORGAN SIZE | NF2 TUMOR-SUPPRESSOR | GENETICS & HEREDITY | cancer | SIZE-CONTROL | PROMOTES APOPTOSIS | NIH 3T3 Cells | Kidney Neoplasms - genetics | Cell Proliferation | Epithelial Cells - metabolism | Humans | Transcriptional Activation | Carcinoma, Renal Cell - genetics | Kidney Neoplasms - metabolism | Mesoderm - cytology | Phosphoproteins - metabolism | Breast Neoplasms - metabolism | DNA-Binding Proteins - metabolism | Transcription, Genetic | Nuclear Proteins - genetics | Promoter Regions, Genetic | Signal Transduction | Trans-Activators | Cells, Cultured | Gene Expression Regulation | Nuclear Proteins - metabolism | Phosphoproteins - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Transcription Factors - metabolism | Carcinoma, Renal Cell - metabolism | Animals | Breast Neoplasms - genetics | Cell Transformation, Neoplastic | Adaptor Proteins, Signal Transducing - genetics | Mesoderm - metabolism | Mice | Adaptor Proteins, Signal Transducing - metabolism | Drosophila | Analysis | Physiological aspects | Genetic aspects | Genetic transcription | Research | DNA binding proteins | Gene expression | Index Medicus | Research Paper
Journal Article
Genes and Development, ISSN 0890-9369, 11/2007, Volume 21, Issue 21, pp. 2747 - 2761
The Hippo pathway plays a key role in organ size control by regulating cell proliferation and apoptosis in Drosophila. Although recent genetic studies have... 
Contact inhibition | Lats | YAP | NF2 | Hippo | Mst | YES-ASSOCIATED PROTEIN | hippo | PROLIFERATION ARREST | SIZE CONTROL | lats | DEVELOPMENTAL BIOLOGY | mst | CELL BIOLOGY | ORGAN SIZE | SIGNALING PATHWAY | CYCLE EXIT | GENETICS & HEREDITY | IMAGINAL DISC | contact inhibition | BANTAM MICRORNA | TUMOR-SUPPRESSOR PATHWAY | PROMOTES APOPTOSIS | Protein Kinases - metabolism | NIH 3T3 Cells | Phosphorylation | Cell Proliferation | Cell Count | Humans | Drosophila Proteins - metabolism | Trans-Activators - chemistry | Contact Inhibition - genetics | Trans-Activators - physiology | Drosophila Proteins - physiology | Drosophila Proteins - antagonists & inhibitors | Drosophila - genetics | Amino Acid Sequence | Protein-Serine-Threonine Kinases - physiology | Cells, Cultured | Intracellular Signaling Peptides and Proteins | Nuclear Proteins - metabolism | Cell Communication - genetics | Drosophila Proteins - chemistry | Nuclear Proteins - chemistry | Amino Acid Motifs | Protein Transport | 14-3-3 Proteins - metabolism | Animals | Models, Biological | Nuclear Proteins - antagonists & inhibitors | Protein Binding | Signal Transduction - physiology | Trans-Activators - metabolism | Drosophila - growth & development | Mice | Nuclear Proteins - physiology | HeLa Cells | Trans-Activators - antagonists & inhibitors | Cell proliferation | Drosophila | Tumor suppressor genes | Genetic aspects | Research | Genetic transcription | Apoptosis | Index Medicus | Research Paper
Journal Article
Nature, ISSN 0028-0836, 02/2009, Volume 457, Issue 7231, pp. 910 - 914
Journal Article
Genes & Diseases, ISSN 2352-3042, 06/2015, Volume 2, Issue 2, pp. 144 - 151
FOXA1 (also known as hepatocyte nuclear factor 3α, or HNF-3α) is a protein of the FKHD family transcription factors. FOXA1 has been termed as a pioneer... 
FOXA1 | Chromatin accessibility | Pioneering factor | EMT | Prostate cancer | Androgen receptor
Journal Article
Science, ISSN 0036-8075, 12/2008, Volume 322, Issue 5908, pp. 1695 - 1699
Enhancer of zeste homolog 2 (EZH2) is a mammalian histone methyltransferase that contributes to the epigenetic silencing of target genes and regulates the... 
Cell growth | MicroRNA | Epithelial cells | Genomics | Stem cells | Cell lines | Small interfering RNA | Reports | Prostate cancer | Cancer | Tumors | MOUSE DEVELOPMENT | DEVELOPMENTAL REGULATORS | MULTIDISCIPLINARY SCIENCES | EMBRYONIC STEM-CELLS | IN-VIVO | H3 LYSINE-27 METHYLATION | ZESTE HOMOLOG-2 | ENHANCER | TARGETS | POLYCOMB | EXPRESSION | Neoplasms - metabolism | Prostatic Neoplasms - metabolism | RNA, Small Interfering - genetics | Epigenesis, Genetic | Humans | Gene Expression Regulation, Neoplastic | Stomach Neoplasms - metabolism | Male | MicroRNAs - metabolism | Breast Neoplasms - metabolism | DNA-Binding Proteins - metabolism | Neoplasm Metastasis | Prostatic Neoplasms - genetics | Neoplasms - genetics | Female | Lysine - metabolism | 3' Untranslated Regions | Stomach Neoplasms - genetics | Promoter Regions, Genetic | Prostatic Neoplasms - pathology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Disease Progression | Enhancer of Zeste Homolog 2 Protein | Transcription Factors - metabolism | Polycomb Repressive Complex 2 | Algorithms | Breast Neoplasms - genetics | Cell Line, Tumor | MicroRNAs - genetics | Histones - metabolism | Genome, Human | Methylation | Methyltransferases | RNA | Physiological aspects | Genetic aspects | Research | Health aspects | Oncology | Gene expression | Ribonucleic acid--RNA | Index Medicus
Journal Article