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Gut, ISSN 0017-5749, 06/2013, Volume 62, Issue 6, pp. 824 - 832
Objective Eosinophilic oesophagitis (EoE) and gastro-oesophageal reflux disease (GORD) can have similar clinical and histological features. Proton pump... 
INTERLEUKIN-4 | ACTIVATION | EPITHELIAL-CELLS | GASTROESOPHAGEAL-REFLUX DISEASE | FOOD ALLERGY | GENE-EXPRESSION | ADULTS | MECHANISMS | BARRETTS-ESOPHAGUS | GASTROENTEROLOGY & HEPATOLOGY | CONSENSUS RECOMMENDATIONS | Epithelial Cells - metabolism | Th2 Cells - physiology | Interleukin-13 - antagonists & inhibitors | Epithelial Cells - drug effects | Humans | Middle Aged | Male | RNA, Messenger - metabolism | Chemokine CCL26 | Interleukin-4 - pharmacology | Adult | Female | Gastroesophageal Reflux - drug therapy | Real-Time Polymerase Chain Reaction | Eosinophilic Esophagitis - metabolism | Chemokines, CC - drug effects | Gastroesophageal Reflux - metabolism | Omeprazole - therapeutic use | Interleukin-4 - antagonists & inhibitors | Enzyme-Linked Immunosorbent Assay | Cells, Cultured | Gene Expression Regulation | Interleukin-13 - pharmacology | Proton Pump Inhibitors - therapeutic use | Esophagus - pathology | Chemokines, CC - genetics | Eosinophilic Esophagitis - drug therapy | Chemokines, CC - metabolism | Omeprazole | Gastroesophageal reflux | Patient outcomes | Physiological aspects | Esophagitis | Genetic aspects | Dosage and administration | Research | Drug therapy | Chemokines | Cell culture | Transcription | Veterans | Gene regulation | Lymphocytes T | Eotaxin | Interleukin 4 | Dysphagia | Endoscopy | Telomerase | Squamous cells | Food allergies | Cytokines | Review boards | Secretion | Leukocytes (eosinophilic) | Interleukin 13 | Gene expression | Eosinophilia | Esophagus | Studies | Molecular modelling | Acids | Cell lines | Clinical medicine | proton pump inhibitors | Th2 cytokines | eosinophilic oesophagitis | GORD | eotaxin-3
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e50037
Background: Patients who have esophageal eosinophilia without gastroesophageal reflux disease (GERD) nevertheless can respond to proton pump inhibitors (PPIs),... 
ACTIVATION | EPITHELIAL-CELLS | GASTROESOPHAGEAL-REFLUX DISEASE | ATPASE | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | ADULTS | ACIDIFICATION | CONSENSUS RECOMMENDATIONS | ASSOCIATION | CHILDREN | Eosinophilic Esophagitis - metabolism | Eosinophils - metabolism | Eosinophils - drug effects | Humans | Interleukin-4 - metabolism | Omeprazole - administration & dosage | DNA-Binding Proteins - drug effects | Eosinophilic Esophagitis - pathology | STAT6 Transcription Factor - genetics | RNA Polymerase II - metabolism | STAT6 Transcription Factor - metabolism | Promoter Regions, Genetic - drug effects | Chemokine CCL26 | Gene Expression Regulation - drug effects | Gastric Acid - secretion | Proton Pump Inhibitors - administration & dosage | Interleukin-4 - administration & dosage | Chemokines, CC - genetics | Chemokines, CC - biosynthesis | Eosinophilic Esophagitis - drug therapy | Cell Culture Techniques | Eosinophils - cytology | Chemokines, CC - metabolism | Gastroesophageal reflux | Omeprazole | Care and treatment | Messenger RNA | Cytokines | Esophagitis | Enzyme-linked immunosorbent assay | Lansoprazole | Phosphorylation | Disease | Veterans | Acidification | Lymphocytes T | DNA-directed RNA polymerase | Western blotting | Eotaxin | Interleukin 4 | Dysphagia | Telomerase | Binding | Translocation | Squamous cells | Internal medicine | Secretion | Leukocytes (eosinophilic) | Cultures | Inflammation | RNA polymerase | Gene expression | Patients | Eosinophilia | Nuclear transport | Esophagus | Asthma | Medicine | Polymerase | Molecular modelling | Acids | Ribonucleic acids | Biopsy | Production methods | Ulcers | Stat6 protein | RNA polymerase II | mRNA stability | Cancer | RNA | Ribonucleic acid
Journal Article
AIAA Journal, ISSN 0001-1452, 11/2019, Volume 57, Issue 11, pp. 4684 - 4697
Journal Article
Journal of the American Chemical Society, ISSN 0002-7863, 09/2018, Volume 140, Issue 37, pp. 11726 - 11734
Light-driven H2 generation using semiconductor nanocrystal heterostructures has attracted intense recent interest because of the ability to rationally improve... 
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 6, p. e0157376
Background Although most studies on treatments for eosinophilic esophagitis (EoE) have focused on effects in the epithelium, EoE is a transmural disease.... 
DIAGNOSIS | CYTOKINES | THERAPY | MULTIDISCIPLINARY SCIENCES | DISEASE | GENE-EXPRESSION | INTERLEUKIN-13 | SQUAMOUS-CELLS | FLUTICASONE | CHILDREN | MOTILITY | Eosinophils - metabolism | Phosphorylation | Epithelial Cells - metabolism | Eosinophils - drug effects | Interleukin-13 - antagonists & inhibitors | Epithelial Cells - drug effects | Humans | Eosinophilic Esophagitis - pathology | STAT6 Transcription Factor - genetics | Chemokine CCL26 | Th2 Cells - drug effects | Janus Kinase 1 - metabolism | Interleukin-4 - pharmacology | Janus Kinase 1 - genetics | Omeprazole - pharmacology | Eosinophils - pathology | STAT6 Transcription Factor - antagonists & inhibitors | Eosinophilic Esophagitis - genetics | Th2 Cells - pathology | Fibroblasts - metabolism | Pyrazoles - pharmacology | Eosinophilic Esophagitis - metabolism | Interleukin-4 - antagonists & inhibitors | Signal Transduction | Gene Expression Regulation | Epithelial Cells - pathology | Inflammation | Pyrimidines - pharmacology | STAT6 Transcription Factor - metabolism | Fibroblasts - pathology | Interleukin-13 - pharmacology | Janus Kinase 1 - antagonists & inhibitors | Th2 Cells - metabolism | Isoxazoles - pharmacology | Chemokines, CC - antagonists & inhibitors | Fibroblasts - drug effects | Chemokines, CC - genetics | Fibrosis | Eosinophilic Esophagitis - drug therapy | Primary Cell Culture | Cell Line, Transformed | Chemokines, CC - metabolism | Pediatrics | Transcription | Veterans | Epithelial cells | Lymphocytes T | Leflunomide | Cancer therapies | Eotaxin | Rodents | Fibroblasts | Janus kinase | Protein transport | Telomerase | Translocation | Cytokines | Internal medicine | Complications | Secretion | Leukocytes (eosinophilic) | Esophagitis | Gene expression | Epithelium | Eosinophilia | Nuclear transport | Esophagus | Omeprazole | Medicine | Pathology | Inhibitors | Stat6 protein | Eosinophils
Journal Article
Journal Article
Zhongguo Dianji Gongcheng Xuebao/Proceedings of the Chinese Society of Electrical Engineering, ISSN 0258-8013, 09/2015, Volume 35, pp. 197 - 204
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 02/2019, Volume 509, Issue 2, pp. 448 - 454
Thousands of lncRNAs have been identified but few have been functionally characterized in triple negative breast cancer (TNBC). LINC00152 was known as... 
LINC00152 | YY1 | PTEN | Triple negative breast cancer | Tumor progression | NEDD4-1 | UBIQUITIN LIGASE | SUBTYPES | ADENOCARCINOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | PATTERNS | PROLIFERATION | LUNG-CANCER | BIOPHYSICS | LONG NONCODING RNA | EXPRESSION | EZH2
Journal Article
International Journal of Modern Physics B, ISSN 0217-9792, 12/2019, p. 2040065
Daytime star sensor provides accuracy navigation information to air vehicles near the ground in the daytime by observing stars. It has been an important... 
Journal Article
Journal of Nursing Management, ISSN 0966-0429, 11/2019, Volume 27, Issue 8, pp. 1818 - 1825
Aim This cross‐sectional correlation study aimed to explore the associations among patient safety culture, organisational support, second victim‐related... 
absenteeism | turnover intentions | second victim | structure equation modelling | organisational support | second victim‐related distress | patient safety culture
Journal Article
Gut, ISSN 0017-5749, 09/2017, Volume 66, Issue 9, pp. 1542 - 1554
Journal Article
Journal of Pharmacy and Pharmacology, ISSN 0022-3573, 02/2019, Volume 71, Issue 2, pp. 176 - 184
Journal Article
by Li, K and Li, MM and Li, WL and Yu, HZ and Sun, X and Zhang, QY and Li, Y and Li, X and Abel, ED and Wu, Q and Chen, HY
CELL DEATH & DISEASE, ISSN 2041-4889, 11/2019, Volume 10, Issue 12, pp. 1 - 14
Efficient repair of injured epithelium by airway progenitor cells could prevent acute inflammation from progressing into chronic phase in lung. Here, we used... 
MAINTENANCE | STEM-CELLS | ACTIVATED PROTEIN-KINASE | ROLES | REPERFUSION | MAINTAINS | ISCHEMIA | AMPK | DIFFERENTIATION | INDUCTION | CELL BIOLOGY | Glucose transporter | Cell proliferation | Ovalbumin | Hypersensitivity | Inflammation | Glucose | Epithelium | Cell differentiation | Metabolism | Autophagy | Progenitor cells | Respiratory tract | Glucose metabolism | Molecular modelling | Organoids | Lungs | Stem cells | Glycolysis | Phagocytosis
Journal Article