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Cancer Research, ISSN 0008-5472, 07/2016, Volume 76, Issue 14 Supplement, pp. 932 - 932
Journal Article
Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13 Supplement, pp. 1514 - 1514
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 10/2017, Volume 70, Issue 16, pp. C34 - C34
Objectives Calcium-activated chloride channel (CaCC) is an important ion channel of vascular smooth muscle cells (VSMCs). In this study, we further observed... 
Hypertension | Cell proliferation | Leukocyte migration | Rodents | Rats | Smooth muscle | Cell migration | Cell adhesion & migration
Journal Article
Journal Article
Life Sciences, ISSN 0024-3205, 08/2018, Volume 207, p. 145
Objective Although angiogenesis plays an important role in coronary collateral circulation (CCC) formation and there are many determinants of coronary... 
Proteins | Plasma | Sensitivity | Correlation | Angiography | Coronary artery | Biomarkers | Cardiovascular disease | Regression analysis | Ribonucleic acid--RNA | Coronary artery disease
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 09/2017, Volume 23, Issue 9, pp. 1055 - 1062
Bromodomain and extraterminal domain (BET) protein inhibitors are emerging as promising anticancer therapies. The gene encoding the E3 ubiquitin ligase... 
SELECTIVE-INHIBITION | TARGET | MEDICINE, RESEARCH & EXPERIMENTAL | ANDROGEN RECEPTOR | STEM-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | ACUTE MYELOID-LEUKEMIA | ENHANCERS | CELL BIOLOGY | RNA-SEQ | BROMODOMAIN INHIBITION | MUTATIONS | BRD4 | Prostatic Neoplasms - metabolism | Immunoprecipitation | TOR Serine-Threonine Kinases - metabolism | Humans | Drug Resistance, Neoplasm | Male | Gene Expression Profiling | Molecular Targeted Therapy | Mechanistic Target of Rapamycin Complex 1 | Transcription Factors - drug effects | Multiprotein Complexes - metabolism | Prostatic Neoplasms - genetics | Proteasome Endopeptidase Complex - drug effects | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Nuclear Proteins - drug effects | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | TOR Serine-Threonine Kinases - drug effects | Multiprotein Complexes - drug effects | Prostatic Neoplasms - drug therapy | Protein-Serine-Threonine Kinases - metabolism | Repressor Proteins - metabolism | RNA-Binding Proteins - antagonists & inhibitors | Triazoles - therapeutic use | Cell Survival | Repressor Proteins - genetics | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Azepines - therapeutic use | RNA-Binding Proteins - drug effects | Azepines - pharmacology | Transcription Factors - metabolism | Triazoles - pharmacology | Nuclear Proteins - antagonists & inhibitors | Protein-Serine-Threonine Kinases - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Mutation | RNA-Binding Proteins - metabolism | rac1 GTP-Binding Protein - metabolism | Proto-Oncogene Proteins c-akt - drug effects | rac1 GTP-Binding Protein - genetics | Gene mutations | Physiological aspects | Genetic aspects | Research | Drug resistance | Drug therapy | Prostate cancer | Ubiquitin | Inhibitor drugs | Stabilization | AKT protein | Activation | Biosynthesis | Degradation | Proteins | Ubiquitination | Transcription activation | Ubiquitin-protein ligase | Binding | Rac1 protein | Tumor cell lines | Gene expression | Cholesterol | Mutants | Inhibitors | Proteasomes | Biomarkers | Bet protein | Prostate | Cancer | Guanosinetriphosphatase
Journal Article
Journal Article
Journal of Controlled Release, ISSN 0168-3659, 08/2017, Volume 259, pp. e158 - e158
Journal Article
International Journal of Cancer, ISSN 0020-7136, 08/2019, Volume 145, Issue 4, pp. 1055 - 1067
Standard therapy for advanced Prostate Cancer (PCa) consists of antiandrogens, which provide respite from disease progression, but ultimately fail resulting in... 
CELLS | GENE | ONCOLOGY | PATHWAY | DNA-DAMAGE RESPONSE | GROWTH | NEK1 | ATM | CANCER PROGRESSION | INHIBITOR | Antiandrogens | Development and progression | Prostate cancer | DNA repair | Tumors | Phenothiazine | Nek1 protein | Medical research | Therapy | Deprivation | Toxicity | DNA damage | Xenotransplantation | CHK1 protein | Clinical trials | Kinases | Androgens | Thioridazine | Cell cycle | Xenografts | Inhibition | Prostate | Deoxyribonucleic acid--DNA | Apoptosis | Molecular Cancer Biology
Journal Article