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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 06/2013, Volume 110, Issue 26, pp. 10723 - 10728
textabstractPsoriasis is an autoinflammatory skin disease of unknown etiology. Topical application of Aldara cream containing the Toll-like receptor (TLR)7... 
Biological Sciences
Journal Article
Nature, ISSN 0028-0836, 08/2012, Volume 488, Issue 7410, pp. 222 - 225
The herpes virus entry mediator (HVEM), a member of the tumour-necrosis factor receptor family, has diverse functions, augmenting or inhibiting the immune... 
INTESTINAL INFLAMMATION | PROTECTS MICE | HERPESVIRUS ENTRY MEDIATOR | NK CELLS | MULTIDISCIPLINARY SCIENCES | ULCERATIVE-COLITIS | RECEPTOR | CITROBACTER-RODENTIUM | CELL-DIFFERENTIATION | NF-KAPPA-B | INNATE IMMUNITY | Antigens, CD - immunology | Immunity, Mucosal - immunology | Lung - microbiology | Epithelial Cells - metabolism | Enteropathogenic Escherichia coli | Citrobacter rodentium - pathogenicity | Intestines - immunology | Antigens, CD - metabolism | Streptococcus pneumoniae - immunology | Enterobacteriaceae Infections - immunology | Pneumococcal Infections - immunology | Lymphocytes - immunology | Escherichia coli Infections | Pneumococcal Infections - microbiology | Receptors, Immunologic - immunology | Protein-Serine-Threonine Kinases - metabolism | Receptors, Tumor Necrosis Factor, Member 14 - immunology | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Lymphocytes - metabolism | Receptors, Tumor Necrosis Factor, Member 14 - genetics | Cell Line | Citrobacter rodentium - immunology | GPI-Linked Proteins - immunology | Signal Transduction | Enterobacteriaceae Infections - microbiology | Mice, Inbred C57BL | Survival Rate | Bacterial Load | GPI-Linked Proteins - metabolism | Mucous Membrane - metabolism | Receptors, Tumor Necrosis Factor, Member 14 - metabolism | Animals | Intestines - microbiology | Mucous Membrane - immunology | Epithelial Cells - immunology | Receptors, Tumor Necrosis Factor, Member 14 - deficiency | Ligands | Mice | Lung - immunology | Mucous Membrane - microbiology | Receptors, Immunologic - metabolism | Physiological aspects | Bacteria | Cellular signal transduction | Research | Health aspects | Mucous membrane | Immune system | Phosphorylation | Bacterial infections | Cytokines | Rodents | Kinases | Gene expression | Chemokines | Streptococcus infections
Journal Article
JOURNAL OF EXPERIMENTAL MEDICINE, ISSN 0022-1007, 02/2018, Volume 215, Issue 2, pp. 415 - 422
Dermatitis is often associated with an allergic reaction characterized by excessive type 2 responses leading to epidermal acanthosis, hyperkeratosis, and... 
PATHOGENESIS | MEDICINE, RESEARCH & EXPERIMENTAL | ATOPIC-DERMATITIS | HERPESVIRUS ENTRY MEDIATOR | MOLECULE LIGHT | SUSCEPTIBILITY LOCI | ASTHMA | SUPERFAMILY | SKIN | IMMUNOLOGY | PERIOSTIN | THYMIC STROMAL LYMPHOPOIETIN | RNA, Small Interfering - genetics | Cell Proliferation | Humans | Male | Tumor Necrosis Factor Ligand Superfamily Member 14 - genetics | Dermatitis, Atopic - etiology | Tumor Necrosis Factor Ligand Superfamily Member 14 - deficiency | Dermatitis, Atopic - immunology | HEK293 Cells | Disease Models, Animal | Receptors, Tumor Necrosis Factor, Member 14 - genetics | Signal Transduction | Mice, Inbred C57BL | Tumor Necrosis Factor Ligand Superfamily Member 14 - metabolism | Cell Adhesion Molecules - metabolism | Keratinocytes - immunology | Mice, Knockout | Keratinocytes - pathology | Receptors, Tumor Necrosis Factor, Member 14 - metabolism | Animals | Keratinocytes - metabolism | Receptors, Tumor Necrosis Factor, Member 14 - deficiency | Tumor Necrosis Factor Ligand Superfamily Member 14 - antagonists & inhibitors | Mice | Antigens, Dermatophagoides - adverse effects | Dermatitis, Atopic - metabolism | Receptors, Tumor Necrosis Factor, Member 14 - antagonists & inhibitors | Lymphotoxin | Allergens | Hyperplasia | Homology | Lymphocytes T | Dermatitis | Thymus | Proteins | Interleukin 4 | Clonal deletion | Lymphocytes | Atopic dermatitis | Light | Deletion | Skin diseases | Tumor necrosis factor-TNF | Scleroderma | Thickening | Glycoprotein D | Cytokines | Hypersensitivity | House dust | Glycoprotein | Keratinocytes | Interleukin 13 | Disease control | Signaling | Thymic stromal lymphopoietin | Tumor necrosis factor | Collagen | Reagents | 319
Journal Article
Journal of Immunology, ISSN 0022-1767, 09/2011, Volume 187, Issue 6, pp. 3104 - 3110
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 11/2018, Volume 9, p. 2585
Over 1.5 million individuals in the United States are afflicted with inflammatory bowel disease (IBD). While the progression of IBD is multifactorial, chronic,... 
Colitis | DSS (dextran sulfate sodium) | Lymphotoxin (LT) | Light | TNF superfamily | CELLS | ACTIVATION | HVEM | IMMUNOLOGY | EXPRESSION | TL1A
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 02/2018, Volume 215, Issue 2, pp. 415 - 422
Journal Article
Journal Article
The Journal of investigative dermatology, ISSN 0022-202X, 06/2019
The cell adhesion molecule E-cadherin is a major component of adherens junctions and marks Langerhans cells (LC), the only dendritic cell (DC) population of... 
Journal Article
The Journal of Experimental Medicine, ISSN 0022-1007, 09/2013, Volume 210, Issue 9, pp. 1657 - 1664
textabstractLangerhans cells (LCs) constitute a network of immune sentinels in the skin epidermis that is seeded during embryogenesis. Whereas the development... 
SYSTEM | MEDICINE, RESEARCH & EXPERIMENTAL | STEM-CELLS | ORIGIN | DENDRITIC CELLS | MICROGLIA | IN-VIVO | MICE | SKIN | IMMUNOLOGY | EXPRESSION | DELETION | Cell Lineage | Inflammation - pathology | Animals | Langerhans Cells - pathology | Mice, Inbred C57BL | Color | Cytological Techniques - methods | Homeostasis | Mice, Transgenic | Mice | Imaging, Three-Dimensional | Brief Definitive Report
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 146, Issue 7, pp. 1752 - 1762.e4
Background & Aims The pathogenesis of inflammatory bowel disease (IBD) is associated with a dysregulated mucosal immune response. Expression of the tumor... 
Gastroenterology and Hepatology | IBD | Ulcerative Colitis | Innate Immunity | Mouse Model | DEXTRAN SULFATE SODIUM | APOPTOSIS | ACTIVATION | MURINE COLITIS | MOLECULE | INTERLEUKIN-1-BETA | LEADS | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | T-CELLS | Dextran Sulfate | Intestinal Mucosa - metabolism | Leukocyte Common Antigens - metabolism | Homeodomain Proteins - metabolism | Colitis - pathology | Colon - immunology | CD4-Positive T-Lymphocytes - pathology | Tumor Necrosis Factor Ligand Superfamily Member 14 - genetics | CD4-Positive T-Lymphocytes - immunology | Lymphotoxin beta Receptor - metabolism | Time Factors | Intestinal Mucosa - immunology | Colitis - chemically induced | Tumor Necrosis Factor Ligand Superfamily Member 14 - deficiency | Inflammation Mediators - metabolism | Colitis - immunology | Disease Models, Animal | Severity of Illness Index | Cytokines - metabolism | Signal Transduction | Colon - pathology | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Immunity, Innate | Tumor Necrosis Factor Ligand Superfamily Member 14 - metabolism | Colon - metabolism | Homeodomain Proteins - genetics | Mice, Knockout | Animals | CD4-Positive T-Lymphocytes - transplantation | Weight Loss | Colitis - metabolism | Mice | Colitis - prevention & control | Intestinal Mucosa - pathology | Tumor necrosis factor | Gastrointestinal diseases | Genetic engineering | Inflammation | T cells | Tumors | Necrosis | ulcerative colitis | mouse model | innate immunity
Journal Article
Cell Host & Microbe, ISSN 1931-3128, 08/2018, Volume 24, Issue 2, pp. 249 - 260.e4
Innate lymphoid cells (ILCs) are important regulators of early infection at mucosal barriers. ILCs are divided into three groups based on expression profiles,... 
Yersinia enterocolitica | ileum | IFN-γ | infection | HVEM | LIGHT | innate lymphoid cells | CCR6 | MUCOSAL BARRIERS | ROR-GAMMA-T | YERSINIA-ENTEROCOLITICA | RECEPTOR | MICROBIOLOGY | NF-KAPPA B | TL1A | INTESTINAL INFLAMMATION | VIROLOGY | HOST-DEFENSE | BTLA | NKP46(+) CELLS | PARASITOLOGY | Enterobacteriaceae Infections - prevention & control | Homeodomain Proteins - metabolism | Humans | Male | Adoptive Transfer | Interferon-gamma - metabolism | Spleen - microbiology | Host-Pathogen Interactions - immunology | Lymphocytes - immunology | Receptors, CCR6 - metabolism | Adult | Spleen - pathology | Receptors, Tumor Necrosis Factor, Member 14 - immunology | Disease Models, Animal | Lymphocytes - metabolism | Receptors, Tumor Necrosis Factor - metabolism | Cytokines - metabolism | Signal Transduction | Yersinia enterocolitica - pathogenicity | Mice, Inbred C57BL | Neuropeptides - metabolism | Enterobacteriaceae Infections - pathology | Tumor Necrosis Factor Ligand Superfamily Member 14 - metabolism | Homeodomain Proteins - genetics | Mice, Knockout | Protein Transport | Receptors, Tumor Necrosis Factor, Member 14 - metabolism | Animals | Receptors, CCR6 - genetics | Mice | Host-Pathogen Interactions - physiology | Interferon | Bacterial infections | Biological response modifiers | Health aspects | Mortality | IFNγ
Journal Article
Journal of immunology (Baltimore, Md, ISSN 0022-1767, 2011, Volume 187, Issue 10, pp. 5069 - 5076
Journal Article
Blood, ISSN 0006-4971, 01/2014, Volume 123, Issue 2, pp. 217 - 227
Langerhans cells (LCs) are dendritic cells (DCs) residing in epithelia, where they critically regulate immunity and tolerance. The p14 adaptor molecule is part... 
DENDRITIC CELLS | IN-VIVO | DISTINCT | SUBSETS | STEADY-STATE | MICE | CHEDIAK-HIGASHI-SYNDROME | SKIN | IDENTIFICATION | HEMATOLOGY | HERMANSKY-PUDLAK-SYNDROME | Animals, Newborn |