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11/2014, Protein Reviews Ser., ISBN 9781489998798, Volume 2
Annotation The current year (2004) marks the Silver Anniversary of the discovery of the p53 tumor suppressor. The emerging ?eld ?rst considered p53 as a viral... 
Web Resource
2005, 1. Aufl., Protein reviews, ISBN 0387241353, Volume 2, x, 246
Book
Journal of Molecular Cell Biology, ISSN 1759-4685, 07/2019
Journal Article
Journal of molecular cell biology, 07/2019
Journal Article
01/2010, Protein Reviews Ser., ISBN 9781280945052
Annotation The p53 tumor suppressor gene is mutated in approximately half of all human malignancies, including colon, lung, and breast cancers. It is well... 
Web Resource
2005, Protein reviews, ISBN 0387241353, Volume 2
Web Resource
2005, Protein reviews, ISBN 0387241353, Volume 2
Web Resource
Cancer Research, ISSN 0008-5472, 01/2015, Volume 75, Issue 1 Supplement, pp. A52 - A52
Journal Article
Cell Cycle, ISSN 1538-4101, 08/2011, Volume 10, Issue 16, pp. 2619 - 2619
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 05/2014, Volume 205, Issue 3, p. 301
The etoposide-induced protein Ei24 was initially identified as a p53-responsive, proapoptotic factor, but no clear function has been described. Here, we use a... 
Physiological aspects | Research | Carrier proteins | Tumor proteins | Protein binding | Genetics | Cellular biology | Gene expression | Embryos | Rodents | Proteomics
Journal Article
Science, ISSN 0036-8075, 12/2010, Volume 330, Issue 6009, pp. 1390 - 1393
Although the proteins BAX and BAK are required for initiation of apoptosis at the mitochondria, how BAX and BAK are activated remains unsettled. We provide in... 
T lymphocytes | Mitochondria | Cytokines | Thymocytes | Neurons | Cell death | REPORTS | Cytochromes | Mice | Potassium | Apoptosis | NEURONAL APOPTOSIS | CYTOCHROME-C | MITOCHONDRIAL APOPTOSIS | MECHANISM | MULTIDISCIPLINARY SCIENCES | BH3 DOMAINS | RELEASE | JNK PATHWAY | PROTEINS | BCL-2 FAMILY-MEMBERS | MEMBRANE PERMEABILIZATION | BH3 Interacting Domain Death Agonist Protein - deficiency | T-Lymphocytes - physiology | bcl-2-Associated X Protein - chemistry | Protein Multimerization | Stress, Physiological | bcl-2 Homologous Antagonist-Killer Protein - genetics | BH3 Interacting Domain Death Agonist Protein - genetics | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Membrane Proteins - deficiency | Caspases - metabolism | Bcl-2-Like Protein 11 | Apoptosis Regulatory Proteins - deficiency | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | Neurons - physiology | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | bcl-2-Associated X Protein - genetics | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cytochromes c - metabolism | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Proto-Oncogene Proteins - genetics | Mitochondria - metabolism | Permeability | Proto-Oncogene Proteins - deficiency | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | Models, Biological | Cerebellum - cytology | Intracellular Membranes - metabolism | bcl-2 Homologous Antagonist-Killer Protein - chemistry | Protein research | Genetic aspects | Mitochondrial DNA | Biochemical genetics | Research | Properties | Methods | Index Medicus
Journal Article
Molecular Cell, ISSN 1097-2765, 11/2009, Volume 36, Issue 3, pp. 487 - 499
While activation of BAX/BAK by BH3-only molecules (BH3s) is essential for mitochondrial apoptosis, the underlying mechanisms remain unsettled. Here we... 
CELLCYCLE | CYTOCHROME-C | PROAPOPTOTIC BAX | OLIGOMERIZES BAK | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOPLASMIC-RETICULUM | SUBCELLULAR LOCATION | PROTEINS | BCL-2 FAMILY-MEMBERS | BH3 DOMAIN | CELL-DEATH | MEMBRANE PERMEABILIZATION | CELL BIOLOGY | bcl-2-Associated X Protein - chemistry | Immunoprecipitation | Apoptosis - drug effects | Protein Multimerization | bcl-2 Homologous Antagonist-Killer Protein - genetics | Immunoblotting | BH3 Interacting Domain Death Agonist Protein - genetics | Green Fluorescent Proteins - genetics | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Bcl-2-Like Protein 11 | Protein Binding - drug effects | Tumor Suppressor Proteins - genetics | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | bcl-2-Associated X Protein - genetics | Fibroblasts - metabolism | Proto-Oncogene Proteins - metabolism | Green Fluorescent Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Etoposide - pharmacology | Proto-Oncogene Proteins - genetics | Mitochondria - metabolism | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | Models, Biological | Tunicamycin - pharmacology | Fibroblasts - drug effects | Thapsigargin - pharmacology | Fibroblasts - cytology | Mice | Mutation | Microscopy, Fluorescence | Staurosporine - pharmacology | bcl-2 Homologous Antagonist-Killer Protein - chemistry | Monomers | Apoptosis | Oligomers | Index Medicus
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2009, Volume 284, Issue 39, pp. 26591 - 26602
Free fatty acids (FFA) induce hepatocyte lipoapoptosis by a c-Jun N-terminal kinase (JNK)-dependent mechanism. However, the cellular processes by which JNK... 
MITOCHONDRIAL ABNORMALITIES | JNK PHOSPHORYLATION | BAX ACTIVATION | FATTY LIVER-DISEASE | INSULIN-RESISTANCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | N-TERMINAL KINASE | BH3-ONLY PROTEIN | NONALCOHOLIC STEATOHEPATITIS | BCL-2 FAMILY-MEMBERS | CELL-DEATH | Gene Expression - drug effects | Mitogen-Activated Protein Kinase 8 - antagonists & inhibitors | Apoptosis - drug effects | Fatty Liver - pathology | Humans | Apoptosis - genetics | Immunoblotting | Hepatocytes - metabolism | Dose-Response Relationship, Drug | Hepatocytes - cytology | RNA Interference | Palmitates - pharmacology | Tumor Suppressor Proteins - genetics | Apoptosis Regulatory Proteins - genetics | Mitogen-Activated Protein Kinase 8 - genetics | Phosphorylation - drug effects | Hepatocytes - drug effects | Fatty Liver - genetics | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Fatty Liver - metabolism | Mice, Inbred C57BL | Mitogen-Activated Protein Kinase 8 - metabolism | bcl-2-Associated X Protein - metabolism | Proto-Oncogene Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Anthracenes - pharmacology | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | Cell Line, Tumor | Mice | Apoptosis - physiology | Microscopy, Fluorescence | Index Medicus | Mechanisms of Signal Transduction
Journal Article
Cancer Research, ISSN 0008-5472, 06/2010, Volume 70, Issue 11, pp. 4666 - 4675
MicroRNAs (miRNAs) act at the posttranscriptional level to control gene expression in virtually every biological process, including oncogenesis. Here, we... 
OVEREXPRESSION | FACTOR-II | ONCOLOGY | UPSTREAM | RECEPTOR | CELL-PROLIFERATION | EXPRESSION PROFILES | CARCINOMA | CONTRIBUTES | HUMAN OVARIAN-CANCER | MTOR | Humans | Cell Growth Processes - physiology | MicroRNAs - metabolism | Transplantation, Heterologous | Intracellular Signaling Peptides and Proteins - metabolism | Adrenocortical Carcinoma - pathology | Adrenocortical Carcinoma - metabolism | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Adrenocortical Adenoma - genetics | Female | Everolimus | Adrenal Cortex Neoplasms - metabolism | Protein-Serine-Threonine Kinases - metabolism | Somatomedins - metabolism | Sirolimus - analogs & derivatives | Adrenocortical Carcinoma - genetics | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | Adrenal Cortex Neoplasms - pathology | Mice, SCID | Adrenocortical Adenoma - pathology | Sirolimus - pharmacology | Adrenocortical Adenoma - metabolism | Adrenal Cortex Neoplasms - genetics | Animals | Mice, Inbred NOD | Mice | MicroRNAs - genetics | TOR Serine-Threonine Kinases | Index Medicus | Somatomedins | Sirolimus | Protein-Serine-Threonine Kinases | Biochemistry, Molecular Biology | Adrenocortical Adenoma | Adrenocortical Carcinoma | Intracellular Signaling Peptides and Proteins | Cell Growth Processes | Life Sciences | Adrenal Cortex Neoplasms | MicroRNAs
Journal Article
Cell, ISSN 0092-8674, 2005, Volume 123, Issue 4, pp. 641 - 653
In response to DNA damage, the p53 tumor suppressor can elicit either apoptosis or cell-cycle arrest and repair, but how this critical decision is made in... 
BREAST-CANCER | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION FACTORS SNAIL | ZINC-FINGER PROTEIN | C-ELEGANS | COLORECTAL-CANCER CELLS | BCL-2 FAMILY | EXPRESSION | RADIATION-THERAPY | P53 | CELL BIOLOGY | Gamma Rays | Protein Binding - genetics | Transcriptional Activation - genetics | Gene Expression - genetics | Humans | Apoptosis - genetics | Bone Marrow - radiation effects | Myeloid Progenitor Cells - radiation effects | Tumor Suppressor Protein p53 - genetics | Tumor Suppressor Protein p53 - physiology | Proto-Oncogene Proteins c-bcl-2 - metabolism | Chromatin Immunoprecipitation | Myelopoiesis - radiation effects | Tumor Suppressor Proteins - genetics | Bone Marrow Transplantation | Apoptosis Regulatory Proteins - genetics | Hematopoietic Stem Cells - physiology | Mitochondria - radiation effects | Myeloid Progenitor Cells - cytology | Hematopoietic Stem Cells - radiation effects | Snail Family Transcription Factors | Myeloid Progenitor Cells - metabolism | Transcription Factors - physiology | Tumor Suppressor Proteins - metabolism | Introns - genetics | Tumor Suppressor Protein p53 - metabolism | Proto-Oncogene Proteins - genetics | Transcription Factors - genetics | Gene Expression - radiation effects | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Transcription Factors - metabolism | Animals | Models, Biological | Survival Analysis | Cell Line, Tumor | Apoptosis Regulatory Proteins - physiology | Mice | Apoptosis - physiology | Mitochondria - physiology | Cell Line, Transformed | Gamma rays | Research | Genetic transcription | Apoptosis | Tumor proteins | Radiation | Index Medicus
Journal Article
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