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Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2013, Volume 133, Issue 2, pp. 520 - 528.e5
Journal Article
Journal Article
Journal Article
Molecular Cancer, ISSN 1476-4598, 10/2010, Volume 9, Issue 1, pp. 273 - 273
Journal Article
British Journal of Haematology, ISSN 0007-1048, 02/2018, Volume 180, Issue 3, pp. 391 - 394
Summary Predominantly diffuse t(14;18) negative follicular lymphoma (FL) with 1p36 deletion shows distinctive clinical, morphological and molecular features... 
STAT6 | exome | diffuse | follicular lymphoma | 1p36 | ABNORMALITIES | STAT6 MUTATIONS | HEMATOLOGY | EXPRESSION | CANCER | Lymphomas | Nucleotide sequencing | Gene mutations | Analysis | DNA sequencing | Gene frequency | Clonal deletion | Stat6 protein | Deletion | Mutation | Lymphoma | Membrane proteins
Journal Article
Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13, pp. 3544 - 3559
Terminal tissue differentiation and function of slan monocytes in cancer is largely unexplored. Our recent studies demonstrated that slan monocytes... 
Lymph Nodes - pathology | Humans | Middle Aged | Antibody-Dependent Cell Cytotoxicity - immunology | Child, Preschool | Male | Monocytes - metabolism | Monocytes - immunology | Leukocytes, Mononuclear | Young Adult | Rituximab - therapeutic use | Aged, 80 and over | Lymphoma, Large B-Cell, Diffuse - immunology | Adult | Female | Retrospective Studies | Child | Macrophages - immunology | Tumor Microenvironment - drug effects | Antigens, CD20 - immunology | Tumor Suppressor Proteins - metabolism | Lymphoma, Large B-Cell, Diffuse - drug therapy | Lymphoma, Large B-Cell, Diffuse - pathology | Cells, Cultured | Antigens, CD20 - metabolism | Lymph Nodes - immunology | Lymph Nodes - cytology | Antineoplastic Agents, Immunological - pharmacology | Cytophagocytosis - drug effects | Monocytes - drug effects | Rituximab - pharmacology | Tumor Microenvironment - immunology | Macrophages - metabolism | Antineoplastic Agents, Immunological - therapeutic use | Biopsy | Adolescent | Macrophages - drug effects | Aged | Cytophagocytosis - immunology | Primary Cell Culture | Antibody-Dependent Cell Cytotoxicity - drug effects | Cohort Studies | Toxicity | Differentiation (biology) | Cytotoxicity | Macrophages | CD14 antigen | Lymph nodes | Metastases | Homing | Peripheral blood | Conditioning | Phenotypes | Dendritic cells | Tumor cells | Rituximab | Patients | Lymphoma | Drainage | Monocytes | CD16 antigen | Lymphocytes B | Effectors | CD20 antigen | Lymphomas | Cell migration | Phagocytosis | Cancer | Tumors | Apoptosis | B-cell lymphoma
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2013, Volume 8, Issue 4, p. e60136
Journal Article
Cancer Cell, ISSN 1535-6108, 04/2015, Volume 27, Issue 4, pp. 516 - 532
Journal Article
Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13, pp. 3413 - 3420
Journal Article
Nature Medicine, ISSN 1078-8956, 01/2019, Volume 25, Issue 1, pp. 130 - 140
In T lymphocytes, the Wiskott-Aldrich Syndrome protein (WASP) and WASP-interacting-protein (WIP) regulate T cell antigen receptor (TCR) signaling, but their... 
ALK | MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | ACTIN POLYMERIZATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | RHOA | DEFICIENCY | CELL BIOLOGY | CDC42 | MUTATIONS | RAC1 | WIP | Wiskott-Aldrich Syndrome Protein - deficiency | Cell Proliferation | Humans | cdc42 GTP-Binding Protein - metabolism | Guanosine Triphosphate - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Lymphoma, T-Cell - metabolism | MAP Kinase Signaling System | Lymphoma, T-Cell - enzymology | Wiskott-Aldrich Syndrome Protein - metabolism | Anaplastic Lymphoma Kinase - metabolism | Cytoskeletal Proteins - metabolism | STAT3 Transcription Factor - metabolism | Tumor Suppressor Proteins - metabolism | Cell Survival | Down-Regulation | Kaplan-Meier Estimate | CCAAT-Enhancer-Binding Protein-beta - metabolism | Animals | Lymphoma, T-Cell - pathology | Cell Line, Tumor | Protein Binding | T-Lymphocytes - immunology | Mice | Enzyme Activation | Research | B cells | T cells | Protein kinases | Cdc42 protein | GTP | Stat3 protein | MAP kinase | Lymphocytes T | CCAAT/enhancer-binding protein | Kinases | Lymphoma | Suppressors | Proteins | T-cell receptor | Inhibitors | Clonal deletion | Protein kinase | Lymphocytes | Deletion | Tumor suppressor genes | Lymphomas | Anaplastic large-cell lymphoma | Protein-tyrosine kinase | T-cell lymphoma | Tumors
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 01/2018, Volume 28, Issue 1, pp. 1 - 14
Aims: Iron overload (IO) is a life-threatening complication of chronic hemolytic disorders such as β-thalassemia. IO results in severe cellular oxidative... 
Original Research Communications | Iron overload | Hepcidin | Peroxiredoxin-2 | iron overload | hepcidin | OXIDATIVE STRESS | peroxiredoxin-2 | MEMBRANE | BIOCHEMISTRY & MOLECULAR BIOLOGY | RED-CELLS | BETA-THALASSEMIC MICE | AMELIORATES ANEMIA | METABOLISM | MOUSE MODEL | IN-VIVO | ENDOCRINOLOGY & METABOLISM | EXPRESSION | Erythropoiesis | Hepcidins - metabolism | Liver - pathology | Oxidative Stress | Peroxiredoxins - metabolism | Homeostasis | Recombinant Proteins | Iron Overload - etiology | Anemia - drug therapy | Peroxiredoxins - genetics | Iron Overload - metabolism | Bone Marrow - metabolism | Anemia - etiology | STAT3 Transcription Factor - metabolism | Disease Models, Animal | Peroxiredoxins - pharmacology | Signal Transduction | Liver - metabolism | Transcription Factors - genetics | Iron - metabolism | Mice, Knockout | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Anemia - metabolism | Animals | Models, Biological | Bone Marrow - pathology | Hepcidins - genetics | Cytoprotection - genetics | Mice | Complications and side effects | Peroxidase | Research | Blood diseases | Peroxiredoxin | Toxicity | Gene regulation | Dietary supplements | Stat3 protein | Innovations | Cytotoxicity | Thalassemia | Activation | Iron | Lipopolysaccharides | Interleukin 6 | Signaling | Rodents | Janus kinase 2 | Supplementation | Recombinant | Index Medicus
Journal Article
Muscle & Nerve, ISSN 0148-639X, 11/2019, Volume 60, Issue 5, pp. 586 - 590
Background Several viruses have been described as causes of acquired inflammatory myopathies; however, the mechanisms by which they cause muscle disease are... 
inflammatory myopathies | polymyositis | rhabdomyolysis | viral myositis | myositis‐specific antibodies | Coxsackie virus | Viral antibodies |