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JAMA, ISSN 0098-7484, 02/2016, Volume 315, Issue 8, pp. 788 - 800
IMPORTANCE: Limited information exists about the epidemiology, recognition, management, and outcomes of patients with the acute respiratory distress syndrome... 
PRESSURE | MEDICINE, GENERAL & INTERNAL | UNDERRECOGNITION | STATES | BERLIN DEFINITION | VENTILATION | ACUTE LUNG INJURY | CLINICIAN | UNDERUSE | COHORT | Severity of Illness Index | Prone Position | Prevalence | Prospective Studies | Hospital Mortality | Neuromuscular Blockade - methods | Patient Positioning | Humans | Middle Aged | Intensive Care Units - statistics & numerical data | Male | Treatment Outcome | Positive-Pressure Respiration - statistics & numerical data | Positive-Pressure Respiration - methods | Respiratory Distress Syndrome, Adult - mortality | Respiratory Distress Syndrome, Adult - diagnosis | Incidence | Neuromuscular Blockade - statistics & numerical data | Algorithms | Global Health - statistics & numerical data | Female | Organ Dysfunction Scores | Respiratory Distress Syndrome, Adult - epidemiology | Respiratory Distress Syndrome, Adult - therapy | Acute respiratory distress syndrome | Intensive care units | Care and treatment | Patient outcomes | Analysis | Management | Epidemiology | Risk factors | Global health | Intensive care | Respiratory distress syndrome | Mortality | Clinical outcomes | Clinical Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Anestesi och intensivvård | Anesthesiology and Intensive Care | Lungmedicin och allergi | Klinisk medicin | Respiratory Medicine and Allergy
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2016, Volume 22, Issue 7, pp. 744 - 753
MYC oncoproteins are involved in the genesis and maintenance of the majority of human tumors but are considered undruggable. By using a direct in vivo shRNA... 
MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | NEUROENDOCRINE PROSTATE-CANCER | TUMOR SUPPRESSION | REGENERATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | INHIBITION PROFILE | C-MYC | IDENTIFICATION | CELL BIOLOGY | N-MYC | HUMAN HEPATOCELLULAR-CARCINOMA | Humans | Hepatocytes - metabolism | Aurora Kinase A - antagonists & inhibitors | Molecular Targeted Therapy | Aurora Kinase A - metabolism | Oncogene Protein p21(ras) - metabolism | Tumor Suppressor Protein p53 - genetics | Liver Neoplasms, Experimental - metabolism | Carcinoma, Hepatocellular - drug therapy | Carcinoma, Hepatocellular - genetics | Gene Deletion | Liver Neoplasms, Experimental - genetics | Liver Neoplasms - genetics | Liver Neoplasms - drug therapy | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Monomeric GTP-Binding Proteins - genetics | Pyrimidines - pharmacology | Proto-Oncogene Proteins c-myc - metabolism | Mice, Knockout | Azepines - pharmacology | Xenograft Model Antitumor Assays | Cell Cycle Checkpoints | Animals | Liver Neoplasms, Experimental - drug therapy | Liver Neoplasms - metabolism | Mice | Phenylurea Compounds - pharmacology | Protein Kinase Inhibitors - pharmacology | RNA, Small Interfering | Mutation | Carcinoma, Hepatocellular - metabolism | Liver cancer | Care and treatment | Tumor suppressor genes | Development and progression | Genetic aspects | Properties | Gene expression | Phosphotransferases | Health aspects | Proteins | Kinases | Drug therapy
Journal Article
Intensive Care Medicine, ISSN 0342-4642, 5/2018, Volume 44, Issue 5, pp. 564 - 577
Journal Article
Gut, ISSN 0017-5749, 12/2012, Volume 61, Issue 12, pp. 1733 - 1743
Background Hepatocellular carcinoma (HCC) is a typical inflammation-associated cancer, but may also provoke antitumour immune responses whose significance and... 
FIBROSIS | HEPATOCELLULAR-CARCINOMA PATIENTS | GENE-EXPRESSION | INNATE | MONOCYTES | RECEPTOR | GASTROENTEROLOGY & HEPATOLOGY | NF-KAPPA-B | T-CELLS | MOUSE MODELS | HEPATOCARCINOGENESIS | Carcinoma, Hepatocellular - chemically induced | Carcinoma, Hepatocellular - mortality | Oligonucleotide Array Sequence Analysis | Humans | Liver Neoplasms, Experimental - chemically induced | Liver Neoplasms - chemically induced | Male | Liver Neoplasms - mortality | Liver Neoplasms, Experimental - immunology | Carcinogens | Diethylnitrosamine | Liver Neoplasms - pathology | Precancerous Conditions - pathology | Carcinoma, Hepatocellular - immunology | Chemokine CCL2 - metabolism | Chemokine CCL5 - metabolism | Liver Neoplasms, Experimental - pathology | B-Lymphocytes - metabolism | Adaptive Immunity | Biomarkers - metabolism | Mice, Inbred C57BL | Liver Neoplasms - immunology | Disease Progression | Mice, Knockout | Macrophages - metabolism | Animals | Precancerous Conditions - immunology | T-Lymphocytes, Cytotoxic - metabolism | Chemokine CXCL9 - metabolism | Carcinoma, Hepatocellular - pathology | Survival Analysis | Mice | Precancerous Conditions - chemically induced | Leukocytes - metabolism | Liver cancer | Development and progression | Prognosis | Immune response | Research | Genes | Mortality | Cytotoxicity | T cell receptors | Inflammation | Hepatitis | Lymphocytes | Rodents | Medical prognosis | Bone marrow | Regulation | Mutation | Chemokines | Immune system | Tumors
Journal Article
27.