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2014, Chu ban., Yin ke wen xue, ISBN 9789865823917, Volume 413., 190 pages
Book
Canadian Journal of Physiology and Pharmacology, ISSN 0008-4212, 2014, Volume 92, Issue 2, pp. 132 - 139
Journal Article
2015, ISBN 7010145423
Book
Journal Article
2010, Chu ban., Guo fang guan li cong shu., ISBN 9789860241938, ix, 273
Book
2011, Chu ban., Gu dai li shi wen hua yan jiu ji kan. Wu bian, ISBN 9862544228, Volume di 8 ce, [1], 3, 160
Book
Cell Death and Disease, ISSN 2041-4889, 2014, Volume 5, Issue 3, pp. e1137 - e1137
Hepatocellular carcinoma (HCC) is the most common form of primary liver cancer, and is also highly resistant to conventional chemotherapy treatments. In this... 
Cell cycle arrest | Hepatocellular carcinoma | Skp2 | Longikaurin A | Apoptosis | KINASE | CYCLE ARREST | P38 MAPK | apoptosis | KAPPA-B | cell cycle arrest | P21 | CELL BIOLOGY | ISODON-ERIOCALYX | COLON-CANCER CELLS | REACTIVE OXYGEN | SIGNALING PATHWAY | ROS | hepatocellular carcinoma | CDC2 Protein Kinase | Phosphorylation | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Cyclin B1 - metabolism | Dose-Response Relationship, Drug | Carcinoma, Hepatocellular - drug therapy | G2 Phase Cell Cycle Checkpoints - drug effects | Transfection | Time Factors | Carcinoma, Hepatocellular - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Liver Neoplasms - pathology | Cyclin-Dependent Kinases | Liver Neoplasms - enzymology | Diterpenes, Kaurane - pharmacology | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Liver Neoplasms - genetics | Down-Regulation | Liver Neoplasms - drug therapy | Carcinoma, Hepatocellular - enzymology | S-Phase Kinase-Associated Proteins - metabolism | Hep G2 Cells | Xenograft Model Antitumor Assays | Animals | Signal Transduction - drug effects | Tumor Burden - drug effects | Mice, Nude | Proto-Oncogene Proteins c-jun - metabolism | Carcinoma, Hepatocellular - pathology | Cyclin B - metabolism | S-Phase Kinase-Associated Proteins - genetics | Mice | Mice, Inbred BALB C | Protein Kinase Inhibitors - pharmacology | Antineoplastic Agents, Phytogenic - pharmacology | Original
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 2000, Volume 106, Issue 5, pp. 663 - 670
.... UV inhibition of type I procollagen synthesis is mediated in part by c-Jun, which is induced by IV irradiation and interferes with procollagen... 
NORMAL HUMAN-SKIN | MEDICINE, RESEARCH & EXPERIMENTAL | GROWTH-FACTOR-BETA | HAIRLESS MOUSE | I PROCOLLAGEN | ALPHA-2(I) COLLAGEN GENE | KERATINOCYTES | IDENTIFICATION | N-PROTEINASE | EXPRESSION | FIBROBLASTS
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 7522 - 9
Transcription factors c-Jun and Fra-1 have been reported to play a role during the initiation and progression in oral squamous cell carcinoma (OSCC... 
HEAD | APOPTOSIS | TUMOR-DEVELOPMENT | MULTIDISCIPLINARY SCIENCES | CYCLE PROGRESSION | FOS | CANCER | AP-1 | EXPRESSION | Survival analysis | Squamous cell carcinoma | Antagonism | Transcription factors | Prognosis | Medical prognosis | c-Jun protein | Oral squamous cell carcinoma | Fra1 protein
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 11/2015, Volume 100, Issue 11, pp. E1404 - E1414
Context: Endometriosis is an estrogen-dependent disease. P38 and C-jun NH2-terminal kinase (JNK... 
BREAST-CANCER | CELLS | CYCLIC-AMP | ROSTRAL VENTROLATERAL MEDULLA | BRAIN-STEM DEATH | ENDOCRINOLOGY & METABOLISM | PROGESTERONE-RECEPTOR | DIFFERENTIATION | ACTIVATED PROTEIN-KINASES | PROLIFE ROLE | EXPRESSION | Ovarian Diseases - metabolism | Ovarian Diseases - drug therapy | Mitogen-Activated Protein Kinase 14 - antagonists & inhibitors | Stromal Cells - pathology | Humans | Endometrium - drug effects | Aromatase - metabolism | JNK Mitogen-Activated Protein Kinases - metabolism | Estradiol - agonists | Promoter Regions, Genetic - drug effects | Aromatase - chemistry | Estrogen Receptor beta - metabolism | Aromatase - genetics | Estrogen Receptor beta - agonists | Estrogen Receptor beta - genetics | Mitogen-Activated Protein Kinase 14 - metabolism | RNA Interference | Stromal Cells - drug effects | Female | Ovary - drug effects | JNK Mitogen-Activated Protein Kinases - genetics | Mitogen-Activated Protein Kinase 14 - chemistry | Estrogen Receptor beta - antagonists & inhibitors | Disease Models, Animal | Endometrium - metabolism | Ovary - metabolism | Dinoprostone - pharmacology | Estradiol - metabolism | Ovary - pathology | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | JNK Mitogen-Activated Protein Kinases - chemistry | Stromal Cells - metabolism | Cells, Cultured | Endometriosis - pathology | Endometriosis - drug therapy | Mitogen-Activated Protein Kinase 14 - genetics | Ovarian Diseases - pathology | Random Allocation | Dinoprostone - metabolism | Gene Expression Regulation - drug effects | Estradiol - chemistry | Animals | Mice, Nude | Protein Kinase Inhibitors - therapeutic use | Endometriosis - metabolism | Protein Kinase Inhibitors - pharmacology | Endometrium - pathology
Journal Article
Oncogene, ISSN 0950-9232, 11/2006, Volume 25, Issue 53, pp. 7070 - 7077
Journal Article