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Journal Article
Nature, ISSN 0028-0836, 2013, Volume 494, Issue 7436, pp. 201 - 206
The lysosomal degradation pathway of autophagy has a crucial role in defence against infection, neurodegenerative disorders, cancer and ageing. Accordingly,... 
PATHOGENESIS | LOCALIZES | COMPLEX | PROTEIN | BECLIN-1 | PATHWAY | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | DISEASE | INFECTION | BINDING | Apoptosis Regulatory Proteins - pharmacology | Recombinant Fusion Proteins - pharmacology | Humans | Cell Membrane Permeability | Molecular Sequence Data | Peptide Fragments - pharmacology | Membrane Proteins - pharmacology | Membrane Proteins - therapeutic use | Recombinant Fusion Proteins - metabolism | Autophagy - drug effects | HIV-1 - physiology | nef Gene Products, Human Immunodeficiency Virus - metabolism | Membrane Proteins - metabolism | Beclin-1 | Amino Acid Sequence | HIV-1 - metabolism | Virus Replication - drug effects | Peptide Fragments - metabolism | HIV-1 - drug effects | tat Gene Products, Human Immunodeficiency Virus - genetics | Apoptosis Regulatory Proteins - chemistry | Cells, Cultured | West Nile virus - drug effects | Macrophages - cytology | Recombinant Fusion Proteins - chemistry | Apoptosis Regulatory Proteins - metabolism | Chikungunya virus - drug effects | Peptide Fragments - chemistry | Animals | Membrane Proteins - chemistry | Mice | HeLa Cells | Apoptosis Regulatory Proteins - therapeutic use | tat Gene Products, Human Immunodeficiency Virus - metabolism | Proteins | Peptides | Microscopy | Rodents | Mortality | Amino acids | Infections | Autophagy | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 01/2012, Volume 481, Issue 7382, pp. 511 - 515
Exercise has beneficial effects on human health, including protection against metabolic disorders such as diabetes(1). However, the cellular mechanisms... 
DISRUPTION | BECLIN-1 | GENE | DIET | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | CAUSES INSULIN-RESISTANCE | MOUSE | SENSITIVITY | MICE | TUMORIGENESIS | Physical Exertion - genetics | Protein Binding - genetics | Physical Exertion - physiology | Male | Muscle, Skeletal - metabolism | Autophagy - physiology | Muscle, Skeletal - cytology | Physical Endurance - physiology | Physical Endurance - genetics | Autophagy - drug effects | Running - physiology | Glucose Intolerance - prevention & control | Phosphorylation - genetics | Leptin - blood | Muscle, Skeletal - drug effects | Myocardium - metabolism | Apoptosis Regulatory Proteins - genetics | Glucose Intolerance - chemically induced | Autophagy - genetics | Homeostasis - drug effects | Beclin-1 | Proto-Oncogene Proteins - metabolism | Dietary Fats - adverse effects | Glucose Tolerance Test | Cells, Cultured | Mice, Transgenic | Proto-Oncogene Proteins - genetics | Physical Conditioning, Animal - physiology | Apoptosis Regulatory Proteins - metabolism | Gene Knock-In Techniques | Myocardium - cytology | Adiponectin - blood | Food Deprivation - physiology | Animals | Glucose - metabolism | Mice | Mutation | Proto-Oncogene Proteins c-bcl-2 | Prevention | Autophagy (Cytology) | Metabolic diseases | Influence | Exercise | Health aspects | Muscular system | Glucose | Kinases | Rodents | Index Medicus
Journal Article
Journal Article
Journal Article
eLife, ISSN 2050-084X, 02/2015, Volume 4, Issue 4
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 04/2018, Volume 115, Issue 16, pp. 4176 - 4181
Allelic loss of the autophagy gene, beclin 1/BECN1, increases the risk of patients developing aggressive, including human epidermal growth factor receptor 2... 
breast cancer | HER2 | Autophagy | Beclin 1 | ACTIVATION | autophagy | MECHANISM | MULTIDISCIPLINARY SCIENCES | INDUCTION | TRASTUZUMAB RESISTANCE | CANCER | EGFR | INHIBITION | BECLIN-1 | GROWTH | MUTATIONS | Beclin-1 - deficiency | Humans | Neoplasm Proteins - physiology | Antineoplastic Agents - therapeutic use | Molecular Targeted Therapy | Autophagy - drug effects | Receptor, ErbB-2 - physiology | Protein Binding - drug effects | Female | Antineoplastic Agents - pharmacology | Receptor, ErbB-2 - antagonists & inhibitors | Lapatinib | Neoplasm Proteins - genetics | Beclin-1 - physiology | Mice, Inbred C57BL | Mice, Transgenic | Random Allocation | Gene Knock-In Techniques | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - pathology | Protein Kinase Inhibitors - therapeutic use | Beclin-1 - genetics | Cell Line, Tumor | Mice | Neoplasm Proteins - deficiency | Peptide Fragments - therapeutic use | Protein Kinase Inhibitors - pharmacology | Mutation | Quinazolines - pharmacology | Amino Acid Substitution | Physiological aspects | Autophagy (Cytology) | Development and progression | Breast cancer | Health aspects | Carcinogenesis | Binding | Tyrosine | Transcription | Xenotransplantation | Pharmacology | ErbB-2 protein | Epidermal growth factor | Xenografts | Biomarkers | Breast | Tumorigenesis | Genetic engineering | Inhibition | Growth factors | Protein-tyrosine kinase | Phagocytosis | Cancer | Tumors | Index Medicus | Biological Sciences
Journal Article