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1. Ge ming jun
by Zou, Rong, 1885-1905
2012, Minguo ji cui, 109
1644 - 1912 | Political science | Zou, Rong, 1885-1905 Views on revolution | China | Zou, Rong, 1885-1905 | China Politics and government | Revolution
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2. Jun chen dao
: sheng shi chao tang de cun wang fa ze
by Zou, Fanping
2006, Di 1 ban., ISBN 9787224076127, 8, 3, 315
China Kings and rulers | China History | China Politics and government
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3. Xi Jinping zhang jun da jue zhan
by Zou, Xiaojun
2014, ISBN 193689548X, 1 v.
Book
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4. Ge ming jun
by Zou, Rong, 1885-1905 and Feng, Xiaoqin
2002, Di 1 ban., Ying xiang Zhongguo jin dai shi de ming zhu, ISBN 7508027485, 95 p., [2] p. of plates
Zou, Rong, 1885-1905 Views on revolution | China Politics and government
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5. Ge ming jun
by Zou, Rong, 1885-1905
1954, 68
Revolutions
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6. Huangpu jun xiao shi liao
: Xu pian
by Zou, Zhihong and Chen, Yipei and Zhao, Liping and Huangpu jun xiao shi liao bian ji zu
1994, ISBN 7218012329, 2, 15, 605
Lu jun jun guan xue xiao History | Military education
Book
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7. Song shu zhou jun zhi hui shi
by Hu, Axiang, 1963 and Shen, Yue, 441-513 and Zou, Yilin and Zhou, Zhenhe, 1941 and Tan, Qixiang
2006, Di 1 ban., Zheng shi di li zhi hui shi cong kan, ISBN 7533647564, 2, 12, 2, 4, 382
Shen, Yue, 441-513. Song shu | China Historical geography
Book
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8. Zou Rong ji qi Ge ming jun
by Zou, Rong, 1885-1904 and Taibei shi Sichuan tong xiang hui. Sichuan cong shu bian ji wei yuan hui
1977, Sichuan cong shu, Volume di 1 zhong, 2, 1, 76 p., [1] leaf of plates
Zou, Rong, 1885-1904 | Biography | China Politics and government | Zou, Rong, 1885-1904. Ge ming jun | Revolutionaries
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9. Full Text Promotion of trophoblast invasion by lncRNA MVIH through inducing Jun‐B
by Zou, Yanfen and Li, Qian and Xu, Yetao and Yu, Xiang and Zuo, Qing and Huang, Shiyun and Chu, Yongli and Jiang, Ziyan and Sun, Lizhou
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 02/2018, Volume 22, Issue 2, pp. 1214 - 1223
Preeclampsia (PE), a pregnancy‐specific disorder, is associated with impaired uterine spiral artery remodelling, which is related to the dysfunction of... 
Jun‐B | long non‐coding RNAs | MVIH | Preeclampsia | Jun-B | long non-coding RNAs | MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | ACTIVATION | LONG NONCODING RNAS | INITIATION | CELL BIOLOGY | MOUSE DEVELOPMENT | EVOLUTION | DISEASE | EPIGENETIC REGULATION | RNA | Mass spectrometry | Analysis | Cell proliferation | Transcription | Pathogenesis | Mass spectroscopy | siRNA | Functional analysis | Pre-eclampsia | Gene expression | Tissues | Ribonucleic acid--RNA | JunB protein | Pregnancy | Angiogenesis | Cell growth | Transfection | Placenta | Uterus | Cell lines | Biomarkers | Bioindicators | Synergistic effect | Aberration | Cell migration | Original
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10. Role of Wnt/β-catenin,Wnt/c-Jun N-terminal kinase and Wnt/Ca2+ pathways in cisplatin-induced chemoresistance in ovarian cancer
by Huang, Lu and Jin, Ye and Feng, Shujun and Zou, Yuqing and Xu, Sainan and Qiu, Shuang and Li, Ling and Zheng, Jianhua
Experimental and Therapeutic Medicine, ISSN 1792-0981, 12/2016, Volume 12, Issue 6, pp. 3851 - 3858
The aim of the present study was to explore the expression of Wnt signaling proteins beta-catenin, c-Jun N-terminal kinase (JNK) and Ca2+/calmodulin-dependent... 
β-catenin | Cisplatin chemoresistance | C-Jun N-terminal kinase | calmodulin-dependent protein kinase II | Ovarian cancer | beta-catenin | MEDICINE, RESEARCH & EXPERIMENTAL | c-Jun N-terminal kinase | CELLS | CARBOPLATIN | METASTASIS | MUTATION PROFILES | ovarian cancer | CHEMOTHERAPY | SIGNALING PATHWAY | RESISTANCE | CARCINOMAS | cisplatin chemoresistance | EXPRESSION | Ca2+/calmodulin-dependent protein kinase II
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11. Full Text Follistatin‐like protein 1 contributes to dendritic cell and T‐lymphocyte activation in nasopharyngeal carcinoma patients by altering nuclear factor κb and Jun N‐terminal kinase expression
by Wang, Hong and Wu, Senyong and Huang, Shiping and Yin, Shaolin and Zou, Guilong and Huang, Kuan'en and Zhang, Zhe and Tang, Anzhou and Wen, Wensheng
Cell Biochemistry and Function, ISSN 0263-6484, 12/2016, Volume 34, Issue 8, pp. 554 - 562
Follistatin‐like protein 1 (FSTL1) is a newly characterized protein that can regulate the immune response in various ways. Dendritic cells (DCs) are central to... 
nasopharyngeal carcinoma | follistatin‐like protein 1 | nuclear factor κb | T lymphocyte | dendritic cell | Jun N‐terminal kinase | Jun N-terminal kinase | follistatin-like protein 1 | Follistatin-Related Proteins - metabolism | Nasopharyngeal Carcinoma | Carcinoma | Receptors, Antigen, T-Cell - metabolism | Lymphocyte Activation | Humans | Middle Aged | Cells, Cultured | JNK Mitogen-Activated Protein Kinases - metabolism | Male | NF-kappa B - metabolism | Blotting, Western | T-Lymphocytes - metabolism | Nasopharyngeal Neoplasms - immunology | Lymphocyte Count | Adult | Female | Real-Time Polymerase Chain Reaction | Dendritic Cells - metabolism | Interferon-gamma - biosynthesis
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12. Full Text Timeless-Stimulated miR-5188-FOXO1/β-Catenin-c-Jun Feedback Loop Promotes Stemness via Ubiquitination of β-Catenin in Breast Cancer
by Zou, Yujiao and Lin, Xian and Bu, Junguo and Lin, Zelong and Chen, Yanjuan and Qiu, Yunhui and Mo, Haiyue and Tang, Yao and Fang, Weiyi and Wu, Ziqing
Molecular Therapy, ISSN 1525-0016, 09/2019
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13. Full Text Transcription factor c-jun regulates 3Gn-T8 expression in gastric cancer cell line SGC-7901
by Jiang, Z and Liu, ZH and Zou, ST and Ni, JL and Shen, L and Zhou, YH and Hua, D and Wu, SL
ONCOLOGY REPORTS, ISSN 1021-335X, 09/2016, Volume 36, Issue 3, pp. 1353 - 1360
Aberrant glycosylation, a common feature of malignant alteration, is partly due to changes in the expression of glycosyltransferases, including... 
poly-LacNAc chains | METASTASIS | gastric cancer | CARCINOMA CELLS | GLYCANS | POLY-N-ACETYLLACTOSAMINE | ASPARAGINE-LINKED OLIGOSACCHARIDES | GLYCOSYLATION | HG-CD147 | c-jun | COLON-CANCER | ONCOLOGY | 3Gn-T8 | MICE | MELANOMA-CELLS | TUMOR-GROWTH | Transcription factors | Promoters (Genetics) | Innovations | Genetic aspects | Properties | Gene expression | Observations | Carcinogenesis | Computational biology
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14. Paclitaxel reversed trastuzumab resistance via regulating JUN in human gastric cancer cells identified by FAN analysis
by Wang, Zhe and Zou, Fei and Tian, Yingying and Xiang, Bowen and Qin, Baoli and Liu, Yefu
Future Oncology, ISSN 1479-6694, 11/2018, Volume 14, Issue 26, pp. 2701 - 2712
Aim: In this study, we aim to use bioinformatics approach to identify paclitaxel-targeted modulators potentially involved in the process of reversing the... 
trastuzumab resistance reversal | paclitaxel | JUN | FAN analysis | gastric cancer | bioinformatics | NETWORK | PROGNOSIS | ACQUIRED-RESISTANCE | THERAPY-RESISTANT | MECHANISMS | MCF7 CELLS | BREAST-CANCER | ONCOLOGY | PATHWAY | C-JUN | EXPRESSION
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15. Full Text Longikaurin A, a natural ent-kaurane, induces G2/M phase arrest via downregulation of Skp2 and apoptosis induction through ROS/JNK/c-Jun pathway in hepatocellular carcinoma cells
by Liao, Y.-J and Bai, H.-Y and Li, Z.-H and Zou, J and Chen, J.-W and Zheng, F and Zhang, J.-X and Mai, S.-J and Zeng, M.-S and Sun, H.-D and Pu, J.-X and Xie, D
Cell Death and Disease, ISSN 2041-4889, 2014, Volume 5, Issue 3, pp. e1137 - e1137
Hepatocellular carcinoma (HCC) is the most common form of primary liver cancer, and is also highly resistant to conventional chemotherapy treatments. In this... 
Cell cycle arrest | Hepatocellular carcinoma | Skp2 | Longikaurin A | Apoptosis | KINASE | CYCLE ARREST | P38 MAPK | apoptosis | KAPPA-B | cell cycle arrest | P21 | CELL BIOLOGY | ISODON-ERIOCALYX | COLON-CANCER CELLS | REACTIVE OXYGEN | SIGNALING PATHWAY | ROS | hepatocellular carcinoma | CDC2 Protein Kinase | Phosphorylation | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Cyclin B1 - metabolism | Dose-Response Relationship, Drug | Carcinoma, Hepatocellular - drug therapy | G2 Phase Cell Cycle Checkpoints - drug effects | Transfection | Time Factors | Carcinoma, Hepatocellular - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Liver Neoplasms - pathology | Cyclin-Dependent Kinases | Liver Neoplasms - enzymology | Diterpenes, Kaurane - pharmacology | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Liver Neoplasms - genetics | Down-Regulation | Liver Neoplasms - drug therapy | Carcinoma, Hepatocellular - enzymology | S-Phase Kinase-Associated Proteins - metabolism | Hep G2 Cells | Xenograft Model Antitumor Assays | Animals | Signal Transduction - drug effects | Tumor Burden - drug effects | Mice, Nude | Proto-Oncogene Proteins c-jun - metabolism | Carcinoma, Hepatocellular - pathology | Cyclin B - metabolism | S-Phase Kinase-Associated Proteins - genetics | Mice | Mice, Inbred BALB C | Protein Kinase Inhibitors - pharmacology | Antineoplastic Agents, Phytogenic - pharmacology | Original
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16. Full Text Transcription factor c-jun regulates β3Gn-T8 expression in gastric cancer cell line SGC-7901
by Jiang, Zhi and Liu, Zhenhua and Zou, Shitao and Ni, Jianlong and Shen, Li and Zhou, Yinghui and Hua, Dong and Wu, Shiliang
Oncology Reports, ISSN 1021-335X, 09/2016, Volume 36, Issue 3, pp. 1353 - 1360
Aberrant glycosylation, a common feature of malignant alteration, is partly due to changes in the expression of glycosyltransferases, including... 
c-jun | β3Gn-T8 | poly-LacNAc chains | gastric cancer | HG-CD147 | Poly-LacNAc chains | Gastric cancer | C-jun | Glycoproteins - genetics | Stomach Neoplasms - genetics | Proto-Oncogene Proteins c-jun - genetics | Humans | Cell Line, Tumor | N-Acetylglucosaminyltransferases - genetics | Up-Regulation - genetics | Glycosylation | Binding Sites - genetics | Transcription Factors - genetics | Promoter Regions, Genetic - genetics | Gene Expression Regulation, Neoplastic - genetics | Enzymes | Lymphatic system | Transcription factors | Laboratories | Plasmids | Cloning | Committees | Extracellular matrix | Metastasis | Mutation | Colon | Stomach cancer | Index Medicus
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17. Full Text Effects of c-Jun N-terminal kinase signaling pathway on severe acute pancreatitis-associated lung injury
by Liu, Mingdong and Shi, Liangliang and Chen, Min and Chen, Suyu and Zou, Xiaoping
Pancreas, ISSN 0885-3177, 04/2012, Volume 41, Issue 3, pp. 358 - 366
Objective: The objective of this study was to investigate the effects of the c-Jun N-terminal kinase (JNK) signaling pathway on rats' acute... 
c-Jun N-terminal kinase signaling pathway | severe acute pancreatitis-associated lung injury | SP600125 | ACTIVATION | NUCLEAR | JNK | FACTOR-KAPPA-B | RESPIRATORY-DISTRESS-SYNDROME | PATHOPHYSIOLOGY | LIPOPOLYSACCHARIDE | INHIBITOR | GASTROENTEROLOGY & HEPATOLOGY | INFLAMMATORY MEDIATORS | Tumor Necrosis Factor-alpha - metabolism | Amylases - blood | Lung Injury - pathology | Phosphorylation | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Pancreatitis - complications | Pancreatitis - drug therapy | Lung Injury - etiology | Lung - enzymology | Lung Injury - enzymology | Taurocholic Acid | Time Factors | Lung Injury - prevention & control | Disease Models, Animal | Pancreas - enzymology | Interleukin-1 - metabolism | Acute Disease | Lung - pathology | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Pancreas - drug effects | Pancreas - pathology | Rats | Pancreatitis - chemically induced | Rats, Sprague-Dawley | Anthracenes - pharmacology | Pancreatitis - enzymology | Intercellular Adhesion Molecule-1 - metabolism | Animals | Signal Transduction - drug effects | Lung - drug effects | Pancreatitis - pathology | Protein Kinase Inhibitors - pharmacology | Peroxidase - metabolism
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18. Sonic hedgehog-c-jun N-terminal kinase-zinc finger protein Gli1 signaling protects against high glucose concentration-induced reactive oxygen species generation in human fibroblasts
by Song, Na and Wang, Haijun and Gu, Tengteng and Qi, Jinbo and Yang, Jun and Qiu, Yanyan and Chen, Qiuyue and Zou, Yawen and Chen, Yinze and Hu, Qing and Ma, Xiaoyan and Zhao, Tiesuo and Feng, Zhiwei
Experimental and Therapeutic Medicine, ISSN 1792-0981, 06/2018, Volume 15, Issue 6, pp. 5084 - 5090
Diabetes mellitus (DM) complications affect patients and cause varying damage. Skin ulcers exhibit difficulties in wound healing, and the regulatory basis for... 
Fibroblasts | Skin | Damage | Hedgehog signalling | High-glucose | high-glucose | DIABETES-MELLITUS | MEDICINE, RESEARCH & EXPERIMENTAL | damage | fibroblasts | PATHWAY | hedgehog signalling | skin | PROLIFERATION | CANCER | EXPRESSION | PCR
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19. Full Text Follistatin-like protein 1 contributes to dendritic cell and T-lymphocyte activation in nasopharyngeal carcinoma patients by altering nuclear factor b and Jun N-terminal kinase expression
by Wang, H and Wu, SY and Huang, SP and Yin, SL and Zou, GL and Huang, KE and Zhang, Z and Tang, AZ and Wen, WS
CELL BIOCHEMISTRY AND FUNCTION, ISSN 0263-6484, 12/2016, Volume 34, Issue 8, pp. 554 - 562
Follistatin-like protein 1 (FSTL1) is a newly characterized protein that can regulate the immune response in various ways. Dendritic cells (DCs) are central to... 
APOPTOSIS | nasopharyngeal carcinoma | PROMOTES ARTHRITIS | NPC PATIENTS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PROLIFERATION | MODEL | IDENTIFICATION | CELL BIOLOGY | nuclear factor b | MESENCHYMAL STROMAL CELLS | THERAPY | follistatin-like protein 1 | INFLAMMATION | ENDOPLASMIC-RETICULUM | Jun N-terminal kinase | T lymphocyte | dendritic cell
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20. Full Text c-Jun NH2-terminal kinase-mediated up-regulation of death receptor 5 contributes to induction of apoptosis by the novel synthetic triterpenoid methyl-2-cyano-3,12-dioxooleana-1, 9-dien-28-oate in human lung cancer cells
by Zou, Wei and Liu, Xiangguo and Yue, Ping and Zhou, Zhongmei and Sporn, Michael B and Lotan, Reuben and Khuri, Fadlo R and Sun, Shi-Yong
Cancer Research, ISSN 0008-5472, 10/2004, Volume 64, Issue 20, pp. 7570 - 7578
Death receptor (DR) 4 or 5, on binding to its ligand, tumor necrosis factor-related apoptosis-inducing ligand (TRAIL), triggers apoptosis via activating the... 
TUMOR-NECROSIS-FACTOR | TRAIL | CARCINOMA CELLS | ONCOLOGY | CDDO INDUCES APOPTOSIS | RETINOID CD437 | LEUKEMIA-CELLS | KILLER/DR5 GENE | CASPASE-8 | LIGAND | MOUSE MACROPHAGES | Lung Neoplasms - drug therapy | Up-Regulation | Caspase 8 | Apoptosis - drug effects | Humans | Lung Neoplasms - metabolism | Oleanolic Acid - analogs & derivatives | Oleanolic Acid - pharmacology | JNK Mitogen-Activated Protein Kinases - metabolism | Lung Neoplasms - pathology | Receptors, TNF-Related Apoptosis-Inducing Ligand | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Caspases - metabolism | TNF-Related Apoptosis-Inducing Ligand | Receptors, Tumor Necrosis Factor - genetics | Carcinoma, Non-Small-Cell Lung - pathology | Signal Transduction | Membrane Glycoproteins - pharmacology | Carcinoma, Non-Small-Cell Lung - metabolism | Enzyme Activation - drug effects | Drug Synergism | Membrane Glycoproteins - administration & dosage | Tumor Necrosis Factor-alpha - pharmacology | Oleanolic Acid - administration & dosage | Apoptosis Regulatory Proteins | JNK Mitogen-Activated Protein Kinases - physiology | Cell Line, Tumor | Receptors, Tumor Necrosis Factor - biosynthesis | Tumor Necrosis Factor-alpha - administration & dosage | Apoptosis - physiology | Carcinoma, Non-Small-Cell Lung - drug therapy
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