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Circulation Research, ISSN 0009-7330, 03/2015, Volume 116, Issue 6, pp. 960 - 975
Blockers of the renin–angiotensin–aldosterone system (RAAS), that is, renin inhibitors, angiotensin (Ang)-converting enzyme (ACE) inhibitors, Ang II type 1... 
remodeling | gender | aldosterone | AT2 receptor | hyperkalemia | resistant hypertension | receptor | renin | primary aldosteronism | reactive oxygen species | angiotensin | angiotensin-(1-7) | prorenin | angiotensin-(1–7) | CARDIAC & CARDIOVASCULAR SYSTEMS | BLOOD-PRESSURE RESPONSES | PLASMA-RENIN | CONVERTING ENZYME-INHIBITORS | CORONARY VASCULAR BED | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | II TYPE-2 RECEPTOR | HEMATOLOGY | MYOCARDIAL-INFARCTION | AT 2 receptor | CELL MINERALOCORTICOID RECEPTORS | SEX-DIFFERENCES | CONGESTIVE-HEART-FAILURE | Antihypertensive Agents - pharmacology | Drugs, Investigational - pharmacology | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Humans | Drugs, Investigational - therapeutic use | Endothelium, Vascular - drug effects | Hypertension - drug therapy | Male | Ion Channels - physiology | Sex Chromosomes | Molecular Targeted Therapy | Gonadal Steroid Hormones - physiology | Vasoconstriction - physiology | Drug Interactions | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Hyperaldosteronism - physiopathology | Female | Hypertension - genetics | Drug Evaluation, Preclinical | Drug Resistance | Precision Medicine | Aldosterone - physiology | Endothelium, Vascular - physiopathology | Hyperaldosteronism - genetics | Mice, Transgenic | Renin-Angiotensin System - genetics | Renin-Angiotensin System - physiology | Clinical Trials as Topic | Sex Characteristics | Antihypertensive Agents - therapeutic use | Vasoconstriction - drug effects | Hyperaldosteronism - drug therapy | Hypertension - physiopathology | Mice, Knockout | Mineralocorticoid Receptor Antagonists - therapeutic use | Animals | Renin-Angiotensin System - drug effects | Mice | Therapeutic Equivalency
Journal Article
Journal Article
Journal of Hypertension, ISSN 0263-6352, 08/2016, Volume 34, Issue 8, pp. 1654 - 1655
Journal Article
Journal of Hypertension, ISSN 0263-6352, 08/2016, Volume 34, Issue 8, pp. 1654 - 1655
Journal Article
Journal of Hypertension, ISSN 0263-6352, 08/2016, Volume 34, Issue 8, pp. 1654 - 1655
Journal Article
Journal of Hypertension, ISSN 0263-6352, 09/2016, Volume 34, pp. e468 - e469
Journal Article
Journal of hypertension, 08/2016, Volume 34, Issue 8, p. 1654
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 02/2010, Volume 119, Issue 2, p. 211
  Alzheimer's disease (AD) is characterized by pathological lesions such as amyloid-β (Aβ) plaques and cerebral amyloid angiopathy. Both these lesions consist... 
Immunohistochemistry | Viral antibodies | Care and treatment | Neurosciences | Antibodies | Sulfates | Alzheimer's disease | Antigenic determinants
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 2014, Volume 306, Issue 10, pp. F1179 - F1189
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2011, Volume 6, Issue 8, p. e23411
textabstractMedial degeneration is a key feature of aneurysm disease and aortic dissection. In a murine aneurysm model we investigated the structural and... 
PATHOGENESIS | ARTERIAL | GROWTH-FACTOR-BETA | RENIN | MULTIDISCIPLINARY SCIENCES | MARFAN-SYNDROME | MOUSE MODEL | SMOOTH-MUSCLE-CELLS | MECHANISMS | MEDIAL DEGENERATION | EXPRESSION | Immunohistochemistry | Angiotensin II - genetics | Receptor, Angiotensin, Type 1 - physiology | Oligonucleotide Array Sequence Analysis | Extracellular Matrix Proteins - deficiency | Humans | Transcriptome | Male | Mice, 129 Strain | Angiotensin II Type 1 Receptor Blockers - pharmacology | Vasoconstriction - physiology | Phenylephrine - pharmacology | Smad2 Protein - genetics | Female | Aorta, Thoracic - pathology | Aortic Aneurysm - prevention & control | Animals, Newborn | Vasoconstriction - genetics | Vasoconstrictor Agents - pharmacology | Angiotensin II - pharmacology | Angiotensin II - metabolism | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Aortic Aneurysm - physiopathology | Losartan - pharmacology | Smad2 Protein - metabolism | Aorta, Thoracic - metabolism | Aorta, Thoracic - physiopathology | Vasoconstriction - drug effects | Mice, Knockout | Pregnancy | Animals | Transforming Growth Factor beta - genetics | Mice | Aortic Aneurysm - genetics | In Vitro Techniques | Transforming Growth Factor beta - metabolism | Analysis | Elastin | Angiotensin | Physiological aspects | Aneurysms | Bone morphogenetic proteins | Transforming growth factors | Health aspects | Medicine, Preventive | Preventive health services | Phosphorylation | Marfan's syndrome | Disease | Pathogenesis | Genomics | Medical services | Aneurysm | Smooth muscle | Biology | Insulin-like growth factors | Genomes | Marfan syndrome | Fragmentation | Renin | Rodents | Smad2 protein | Network analysis | Angiotensin AT1 receptors | Extracellular matrix | Aorta | Degeneration | Angiotensin II | Cardiology | Dissection | Bioinformatics | Hypertension | Vascular surgery | Internal medicine | Medical treatment | Contractility | Muscles | Media | Pharmacology | Gene expression | Muscle contraction | Medicine | Pathology | Signaling | Life span | Protein synthesis | Coronary vessels | Mutation | Laboratory animals | Cancer | Structure-function relationships
Journal Article
Journal of the American Society of Hypertension, ISSN 1933-1711, 2016, Volume 10, Issue 4, pp. e15 - e16
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 2/2010, Volume 119, Issue 2, pp. 211 - 220
Alzheimer’s disease (AD) is characterized by pathological lesions such as amyloid-β (Aβ) plaques and cerebral amyloid angiopathy. Both these lesions consist... 
Amyloid-β plaques | Heparan sulfate | Pathology | Neurosciences | Medicine & Public Health | Alzheimer’s disease | Sulfation
Journal Article
American Journal of Physiology (Consolidated), ISSN 0002-9513, 05/2014, Volume 306, Issue 5, p. F1179
Dual renin-angiotensin system (RAS) blockade in diabetic nephropathy is no longer feasible because of the profit/side effect imbalance. (Pro)renin receptor... 
Kidneys | Peptides | Physiological aspects | Diabetes | Renin-angiotensin system | Health aspects
Journal Article