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American journal of physiology. Heart and circulatory physiology, ISSN 0363-6135, 08/2019, Volume 317, Issue 4, pp. H840 - H850
Assessing right ventricular (RV) functional reserve is important for determining clinical status and prognosis in patients with pulmonary hypertension (PH). In... 
Journal Article
JOURNAL OF CLINICAL MEDICINE, ISSN 2077-0383, 08/2019, Volume 8, Issue 8, p. 1204
Pulmonary hypertension (PH) as a result of pulmonary vein stenosis (PVS) is extremely difficult to treat. The ideal therapy should not target the... 
EXERCISING SWINE | DIAGNOSIS | MEDICINE, GENERAL & INTERNAL | nitric oxide | pulmonary hypertension | HEART-FAILURE | GUIDELINES | INFANTS | RESISTANCE | exercise | pulmonary vascular resistance | EPIDEMIOLOGY
Journal Article
American Journal of Physiology, ISSN 0363-6135, 03/2018, Volume 314, Issue 3, p. H616
Chronic thromboembolic pulmonary hypertension (CTEPH) develops in 4% of patients after pulmonary embolism and is accompanied by an impaired exercise tolerance,... 
Heart | Embolisms | Infusion | Exercise | Arginine | Swine | Thromboembolism | Heart diseases | Cardiovascular system | Hypertension | Stroke | Pulmonary hypertension | Pulmonary artery | Nitric-oxide synthase | Embolism | Stroke volume | NG-Nitroarginine methyl ester | Hogs | Lungs | Perfusion | Nitric oxide | Ventilation | Embolization | Livestock | Hemodynamics | Ventricle | Hypertrophy
Journal Article
American Journal of Physiology, ISSN 0363-6135, 02/2018, Volume 314, Issue 2, p. H170
Myocardial infarction (MI) may result in pulmonary hypertension (PH). Inhibition of phosphodiesterase 5 (PDE5), the enzyme responsible for the breakdown of... 
Hypertension | Myocardial infarction | Cerebral infarction | Stroke | Heart attacks | Blood vessels | Muscles | Smooth muscle | Pharmacology | Pulmonary hypertension | Vasodilation | Stroke volume | Hogs | Exercise | Swine | Nitric oxide | Cyclic GMP | Infarction | Livestock | Hemodynamics | Inhibition | Phosphodiesterase
Journal Article
American Journal of Physiology : Heart and Circulatory Physiology, ISSN 0363-6135, 2016, Volume 311, Issue 6, pp. H1339 - H1351
Coronary microvascular dysfunction (CMD) has been proposed as an important component of diabetes mellitus (DM)-and hypercholesterolemia-associated coronary... 
Hypercholesterolemia | Diabetes | Swine | Coronary microvascular dysfunction | Endothelin-1 | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | RISK-FACTORS | ENDOTHELIAL DYSFUNCTION | RESISTANCE ARTERIES | VASCULAR DYSFUNCTION | ENDOGENOUS ENDOTHELIN | MEDIATED DILATION | EARLY ATHEROSCLEROSIS | INSULIN SENSITIVITY | swine | hyper-cholesterolemia | RESPONSES | coronary microvascular dysfunction | DISEASE | PERIPHERAL VASCULAR DISEASE | endothelin-1 | diabetes | Microvessels - physiopathology | Small-Conductance Calcium-Activated Potassium Channels - genetics | Intermediate-Conductance Calcium-Activated Potassium Channels - genetics | Coronary Vessels - physiopathology | Endothelin-1 - pharmacology | Diabetes Mellitus, Experimental - genetics | Microvessels - metabolism | Microvessels - pathology | Male | Coronary Vessels - metabolism | Vasoconstriction - physiology | Diet, High-Fat | Large-Conductance Calcium-Activated Potassium Channels - genetics | Diabetes Mellitus, Experimental - complications | Vasodilation - physiology | Diabetes Mellitus, Experimental - metabolism | Plaque, Atherosclerotic - pathology | Real-Time Polymerase Chain Reaction | Bradykinin - pharmacology | Diabetes Mellitus, Experimental - physiopathology | Coronary Vessels - drug effects | Coronary Vessels - pathology | Vasodilator Agents - pharmacology | Microvessels - drug effects | Receptor, Endothelin A - genetics | Vasoconstriction - drug effects | Receptor, Endothelin B - genetics | Animals | Hypercholesterolemia - complications | Hypercholesterolemia - physiopathology | Hypercholesterolemia - genetics | Vasodilation - drug effects | Nitric Oxide - metabolism | Hypercholesterolemia - metabolism | S-Nitroso-N-Acetylpenicillamine - pharmacology | Sus scrofa | Complications and side effects | Blood circulation disorders | Coronary vessels | Health aspects | Risk factors
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2017, Volume 12, Issue 9, p. e0183419
textabstractBackground: DM remains a risk factor for poor outcome after stent-implantation, but little is known if and how DM affects the vascular response to... 
CHOLESTEROL CRYSTALS | SYSTEM | PART I | OPTICAL COHERENCE TOMOGRAPHY | MULTIDISCIPLINARY SCIENCES | DISEASE | ATHEROSCLEROSIS | CORONARY-ARTERY MODEL | DYSFUNCTION | ABSORB | DRUG-ELUTING STENTS | Diabetes Mellitus - pathology | Absorbable Implants - adverse effects | Atherosclerosis - pathology | Coronary Vessels - pathology | Tissue Scaffolds - adverse effects | Tomography, Optical Coherence | Diabetes Mellitus - metabolism | Male | Neointima - metabolism | Atherosclerosis - metabolism | Atherosclerosis - etiology | Collagen - metabolism | Animals | Atherosclerosis - diagnostic imaging | Biopsy | Swine | Neointima - pathology | Biomarkers | Disease Models, Animal | Physiological aspects | Diabetics | Development and progression | Comparative analysis | Atherosclerosis | Calcification (ectopic) | Infrared spectroscopy | Surgical implants | Angiography | Streptozocin | Arteries | Risk factors | Biomedical materials | Spectrum analysis | Implants | Tomography | Biocompatibility | Cardiology | Stents | Food | Medical imaging | Diabetes mellitus | Implantation | I.R. radiation | Histology | Infrared spectra | Metabolism | Thrombosis | Cholesterol | Near infrared radiation | Optical Coherence Tomography | Arteriosclerosis | Coronary vessels | Collagen | Calcification | Livestock | Diabetes | Laboratory animals | Scaffolds | Veins & arteries
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 04/2018, Volume 314, Issue 3, pp. H627 - H642
Chronic thromboembolic pulmonary hypertension (CTEPH) develops in 4% of patients after pulmonary embolism and is accompanied by an impaired exercise tolerance,... 
Right ventricular remodeling | Chronic thromboembolic pulmonary hypertension | Animal model | Exercise | Vascular remodelling | EMBOLISM | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | right ventricular remodeling | exercise | vascular remodeling | AIR EMBOLIZATION | chronic thromboembolic pulmonary hypertension | TREADMILL EXERCISE | PIGLET MODEL | MORPHOMETRY | ARTERIAL-HYPERTENSION | NITRIC-OXIDE | RESISTANCE | PERIPHERAL VASCULAR DISEASE | RIGHT-VENTRICULAR RESERVE | animal model | ENDOTHELIAL GROWTH-FACTOR | Vascular Remodeling | Hypertension, Pulmonary - physiopathology | Male | Hypertrophy, Right Ventricular - etiology | Hypertrophy, Right Ventricular - physiopathology | Pulmonary Artery - metabolism | Ventricular Remodeling | Female | Ventricular Dysfunction, Right - physiopathology | Hypertrophy, Right Ventricular - metabolism | Ventricular Dysfunction, Right - metabolism | Physical Exertion | Disease Models, Animal | Pulmonary Embolism - complications | Endothelium, Vascular - physiopathology | Hypertension, Pulmonary - metabolism | Pulmonary Artery - physiopathology | Disease Progression | Animals | Endothelium, Vascular - metabolism | Ventricular Dysfunction, Right - etiology | Chronic Disease | Hypertension, Pulmonary - etiology | Sus scrofa | Ventricular Function, Right | Pulmonary Embolism - metabolism | Pulmonary Embolism - physiopathology | Physiological aspects | Health aspects | Pulmonary hypertension
Journal Article
American Journal of Physiology-Heart and Circulatory Physiology, ISSN 0363-6135, 11/2017, p. ajpheart.00380.
Journal Article
Cardiovascular Research, ISSN 0008-6363, 06/2018, Volume 114, Issue 7, pp. 954 - 964
Abstract Aims More than 50% of patients with heart failure have preserved ejection fraction characterized by diastolic dysfunction. The prevalance of diastolic... 
Heart failure | Translational studies | Coronary circulation | Oxidant stress | Endothelium/nitric oxide | POPULATION | METABOLIC SYNDROME | PRESSURE-OVERLOAD | CARDIAC & CARDIOVASCULAR SYSTEMS | HEART-FAILURE | PRESERVED EJECTION FRACTION | MECHANISMS | MODEL | STIFFNESS | INFLAMMATION | NONCARDIAC COMORBIDITIES | Diastole | Oxidative Stress | Ventricular Function, Left | Coronary Vessels - physiopathology | Hypertension, Renovascular - physiopathology | Hypertension, Renovascular - metabolism | Coronary Vessels - metabolism | Microcirculation | Coronary Artery Disease - physiopathology | Myocardium - metabolism | Ventricular Dysfunction, Left - pathology | Female | Diabetes Mellitus, Experimental - complications | Diabetes Mellitus, Experimental - metabolism | Diabetes Mellitus, Experimental - physiopathology | Coronary Circulation | Coronary Artery Disease - etiology | Comorbidity | Coronary Artery Disease - metabolism | Risk Factors | Ventricular Dysfunction, Left - etiology | Myocardium - pathology | Ventricular Dysfunction, Left - physiopathology | Stroke Volume | Ventricular Dysfunction, Left - metabolism | Animals | Hypertension, Renovascular - complications | Fibrosis | Hypercholesterolemia - complications | Hypercholesterolemia - physiopathology | Hypercholesterolemia - metabolism | Sus scrofa | nitric oxide | Original | Endothelium
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 02/2018, Volume 314, Issue 2, pp. H170 - H179
Myocardial infarction (MI) may result in pulmonary hypertension (PH). Inhibition of phosphodiesterase 5 (PDE5), the enzyme responsible for the breakdown of... 
Myocardial infarction | Phosphodiesterase 5 | Nitric oxide | Pulmonary hypertension | CHRONIC HEART-FAILURE | EXERCISING SWINE | nitric oxide | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ENDOTHELIAL DYSFUNCTION | pulmonary hypertension | myocardial infarction | BLOOD-FLOW | NATRIURETIC PEPTIDE SYSTEM | CYCLIC-GMP PRODUCTION | GUANYLATE-CYCLASE | phosphodiesterase 5 | LEFT-VENTRICULAR DYSFUNCTION | EJECTION FRACTION | ARTERIAL-HYPERTENSION | PERIPHERAL VASCULAR DISEASE | Antihypertensive Agents - pharmacology | Hypertension, Pulmonary - physiopathology | Male | Arterial Pressure - drug effects | Vascular Resistance - drug effects | Female | Myocardial Infarction - physiopathology | Hypertension, Pulmonary - drug therapy | Phosphodiesterase 5 Inhibitors - pharmacology | Cyclic Nucleotide Phosphodiesterases, Type 5 - metabolism | Hypertension, Pulmonary - enzymology | Disease Models, Animal | Myocardial Infarction - enzymology | Vasodilator Agents - pharmacology | Cardiac Output - drug effects | Pulmonary Artery - physiopathology | Pulmonary Artery - drug effects | Pulmonary Artery - enzymology | Myocardial Infarction - complications | Animals | Signal Transduction - drug effects | Vasodilation - drug effects | Nitric Oxide - metabolism | Hypertension, Pulmonary - etiology | Sus scrofa | Complications and side effects | Dilatation | Causes of | Physiological aspects | Blood vessels | Physiological research | Research | Phosphodiesterases | Heart attack
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 04/2018, Volume 314, Issue 3, pp. H603 - H615
Pulmonary vascular disease (PVD) represents an underestimated and increasing clinical burden not only in the neonatal period but also later in life, when... 
Hypoxia | Pulmonary vascular disease | Exercise | Sex | Perinatal programming | hypoxia | LUNG | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | CIRCULATION | sex | EARLY-STAGE | exercise | EXERCISE CAPACITY | CLINICAL-FEATURES | perinatal programming | pulmonary vascular disease | BRONCHOPULMONARY DYSPLASIA | EXTRA-UTERINE LIFE | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | OUTCOMES | HYPERTENSION | Vascular Remodeling | Hyperoxia - physiopathology | Hypertension, Pulmonary - diagnostic imaging | Hypertension, Pulmonary - physiopathology | Male | Hypertrophy, Right Ventricular - etiology | Pulmonary Artery - diagnostic imaging | Hypertrophy, Right Ventricular - physiopathology | Arterial Pressure | Time Factors | Hyperoxia - complications | Female | Hypertrophy, Right Ventricular - pathology | Ventricular Dysfunction, Right - physiopathology | Disease Models, Animal | Animals, Newborn | Echocardiography | Pulmonary Artery - physiopathology | Disease Progression | Hyperoxia - pathology | Animals | Ventricular Dysfunction, Right - etiology | Exercise Tolerance | Sex Factors | Hypertension, Pulmonary - etiology | Hypertension, Pulmonary - pathology | Sus scrofa | Pulmonary Artery - pathology | Ventricular Dysfunction, Right - pathology | Ventricular Function, Right | Infants (Newborn) | Physiological aspects | Lung diseases | Diseases
Journal Article
International journal of cardiology, 11/2017
We studied the effect of bioresorbable vascular scaffold (BVS) implantation on distal coronary endothelial function, in swine on a high fat diet without (HFD)... 
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2013, Volume 305, Issue 7, pp. H1104 - H1110
textabstractThe objective of this study was to compare heart-specific fatty acid binding protein (hFABP) and highsensitivity troponin I (hsTnI) via serial... 
High-sensitivity troponin I | STEMI model | No reflow | Acute myocardial infarction | Heart-specific fatty acid binding protein | Markers of myocardial necrosis | heart-specific fatty acid binding protein | DIAGNOSIS | IMPROVE | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | MARKERS | INJURY | REPERFUSION | ISCHEMIA | CARDIAC TROPONIN | high-sensitivity troponin I | PERCUTANEOUS CORONARY INTERVENTION | markers of myocardial necrosis | MYOGLOBIN RELEASE | ACID-BINDING PROTEIN | no reflow | PERIPHERAL VASCULAR DISEASE | acute myocardial infarction | Staining and Labeling - methods | Myocardial Infarction - blood | Predictive Value of Tests | Up-Regulation | Male | Necrosis | Troponin I - blood | Tetrazolium Salts | Myocardial Infarction - therapy | Time Factors | No-Reflow Phenomenon - blood | Swine | Myocardial Infarction - pathology | Myocardium - metabolism | Fatty Acid-Binding Proteins - blood | Thiazoles | No-Reflow Phenomenon - etiology | Female | Myocardial Infarction - physiopathology | No-Reflow Phenomenon - physiopathology | Disease Models, Animal | Coronary Circulation | Risk Factors | Myocardium - pathology | Biomarkers - blood | Myocardial Reperfusion - adverse effects | Animals | No-Reflow Phenomenon - pathology | Hemodynamics | Physiological aspects | Research | Fatty acid metabolism | Heart attack | Protein binding
Journal Article
American Journal of Physiology, ISSN 0363-6135, 10/2013, Volume 305, Issue 7, p. H1104
  The objective of this study was to compare heart-specific fatty acid binding protein (hFABP) and high-sensitivity troponin I (hsTnI) via serial measurements... 
Proteins | Size | Comparative studies | Cardiovascular disease | Fatty acids
Journal Article