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Frontiers in Immunology, ISSN 1664-3224, 2019, Volume 10, pp. 855 - 855
Multiple sclerosis (MS) is a demyelinating autoimmune disease in which innate and adaptive immune cells infiltrate the central nervous system (CNS) and damage... 
Neuroinflammation | Advanced glycation endproducts | Multiple sclerosis | α-dicarbonyl | Astrocytes | OXIDATION | BIOMARKER | multiple sclerosis | RECEPTOR | MECHANISMS | IMMUNOLOGY | advanced glycation endproducts | CEREBROSPINAL-FLUID | INDIVIDUALS | neuroinflammation | DISEASE | GENE-EXPRESSION | END-PRODUCTS AGES | CHOROID-PLEXUS | alpha-dicarbonyl | astrocytes
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2015, Volume 194, Issue 5, pp. 2099 - 2109
Journal Article
Drug Discovery Today: Disease Models, ISSN 1740-6757, 2017, Volume 25-26, pp. 69 - 74
Grey matter tissue damage is a characteristic pathological hallmark of multiple sclerosis. Although evidence is emerging that cortical atrophy is strongly... 
Journal Article
Brain, ISSN 0006-8950, 2012, Volume 135, Issue 3, pp. 886 - 899
Journal Article
Journal Article
Science, ISSN 0036-8075, 12/2011, Volume 334, Issue 6063, pp. 1727 - 1731
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 08/2013, Volume 126, Issue 4, pp. 483 - 492
Journal Article
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 8/2016, Volume 53, Issue 6, pp. 3976 - 3991
Multiple sclerosis (MS) pathology is characterized by neuroinflammation and demyelination. Recently, the inflammatory molecule S100B was identified in... 
Neurology | Neurosciences | Glial inflammatory response | Multiple sclerosis | Biomedicine | Human samples | Demyelination | Neurobiology | Cerebellar organotypic slice cultures | S100B | Cell Biology | S-100 PROTEIN | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | CEREBROSPINAL-FLUID | NEUROSCIENCES | IN-VITRO | TRAUMATIC BRAIN-INJURY | NEURON-SPECIFIC ENOLASE | CENTRAL-NERVOUS-SYSTEM | END-PRODUCTS RAGE | NEUROBIOCHEMICAL MARKERS | Inflammasomes - metabolism | White Matter - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Middle Aged | Astrocytes - pathology | Male | Neutralization Tests | Multiple Sclerosis, Relapsing-Remitting - diagnosis | Demyelinating Diseases - metabolism | Molecular Targeted Therapy | Case-Control Studies | S100 Calcium Binding Protein beta Subunit - blood | Young Adult | Aged, 80 and over | Antigens, Neoplasm - metabolism | Adult | Female | S100 Calcium Binding Protein beta Subunit - cerebrospinal fluid | Demyelinating Diseases - pathology | Cytokines - metabolism | Multiple Sclerosis, Relapsing-Remitting - blood | Lysophosphatidylcholines - metabolism | Antibodies, Neutralizing - pharmacology | Biomarkers - blood | White Matter - pathology | Gene Expression Regulation - drug effects | Aged | Cell Proliferation - drug effects | Biomarkers - cerebrospinal fluid | Multiple Sclerosis, Relapsing-Remitting - cerebrospinal fluid | Chronic Disease | Astrocytes - metabolism | Mitogen-Activated Protein Kinases - metabolism | Health aspects | Analysis | Molecular biology | Inflammation | Index Medicus
Journal Article
Antioxidants & Redox Signaling, ISSN 1523-0864, 09/2011, Volume 15, Issue 5, pp. 1167 - 1178
Cerebral amyloid angiopathy (CAA) is frequently observed in Alzheimer's disease (AD) and is characterized by deposition of amyloid beta (Aβ) in leptomeningeal... 
Forum Original Research Communication | PATHOGENESIS | NADPH OXIDASE | PROTEIN | OXYGEN SPECIES MEDIATE | PERMEABILITY | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOTHELIAL-CELLS | ENDOCRINOLOGY & METABOLISM | PATHOLOGY | PEPTIDE | BASEMENT-MEMBRANE | Microglia - metabolism | Reactive Oxygen Species - metabolism | Capillaries - pathology | Membrane Glycoproteins - metabolism | Humans | NADPH Oxidases - metabolism | Male | Cerebral Amyloid Angiopathy - metabolism | Occludin | Aged, 80 and over | Female | Membrane Proteins - metabolism | Cerebral Amyloid Angiopathy - genetics | Capillaries - metabolism | Tight Junctions - metabolism | Cell Line | Amyloid beta-Peptides - toxicity | Endothelial Cells - metabolism | Membrane Proteins - genetics | RNA, Messenger - genetics | Receptor for Advanced Glycation End Products - metabolism | Antioxidants - pharmacology | Phosphoproteins - genetics | Down-Regulation - drug effects | NADPH Oxidase 2 | Down-Regulation - genetics | Blood-Brain Barrier - metabolism | Gene Expression Regulation - drug effects | Cerebral Amyloid Angiopathy - pathology | Zonula Occludens-1 Protein | Aged | Oxidative Stress - drug effects | Tight Junctions - pathology | Endothelial Cells - drug effects | Blood-brain barrier | Amyloid beta-protein | Physiological aspects | Development and progression | Amyloidosis | Research | Alzheimer's disease | Index Medicus
Journal Article
Glia, ISSN 0894-1491, 2014, Volume 62, Issue 7, pp. 1125 - 1141
To ensure efficient energy supply to the high demanding brain, nutrients are transported into brain cells via specific glucose (GLUT) and monocarboxylate... 
neurodegeneration | nutrient transporters | reactive astrocytes | proliferator‐activated receptor gamma co‐activator 1‐alpha | Nutrient transporters | Neurodegeneration | Proliferator-activated receptor gamma co-activator 1-alpha | Reactive astrocytes | ACTIVATED RECEPTOR-GAMMA | NMR-SPECTROSCOPY | FUNCTIONAL-CHARACTERIZATION | CORTICAL ASTROCYTES | NEUROSCIENCES | proliferator-activated receptor gamma co-activator 1-alpha | MITOCHONDRIAL CHANGES | MAGNETIC-RESONANCE | CENTRAL-NERVOUS-SYSTEM | RAT-BRAIN | ENERGY-METABOLISM | NF-KAPPA-B | Glutamate Plasma Membrane Transport Proteins - metabolism | Leukocytes - pathology | Microglia - metabolism | White Matter - metabolism | Humans | Middle Aged | Astrocytes - pathology | Male | Glucose Transporter Type 3 - metabolism | Brain - metabolism | Brain - blood supply | Monocarboxylic Acid Transporters - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Aged, 80 and over | Microglia - pathology | Adult | Female | White Matter - blood supply | Multiple Sclerosis - metabolism | Cell Line | Endothelial Cells - metabolism | Axons - metabolism | Multiple Sclerosis, Chronic Progressive - pathology | Multiple Sclerosis, Chronic Progressive - metabolism | White Matter - pathology | Transcription Factors - metabolism | Axons - pathology | Brain - pathology | Multiple Sclerosis - pathology | Aged | Endothelial Cells - pathology | Leukocytes - metabolism | Astrocytes - metabolism | Proteins | Glucose metabolism | Brain | Multiple sclerosis | Physiological aspects | Brain damage | Development and progression | Glucose | Dextrose | Index Medicus
Journal Article