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Journal Article
NATURE COMMUNICATIONS, ISSN 2041-1723, 07/2019, Volume 10, Issue 1, pp. 3287 - 16
Journal Article
Molecular Cell, ISSN 1097-2765, 10/2010, Volume 40, Issue 1, pp. 34 - 49
Following genotoxic stress, cells activate a complex kinase-based signaling network to arrest the cell cycle and initiate DNA repair. p53-defective tumor cells... 
CANCER-CELLS | CHK1 | P38 | AU-RICH ELEMENTS | MEDIATE ACTIVATION | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MAPKAP KINASE-2 | NUCLEAR EXPORT | ONCOGENE-INDUCED SENESCENCE | KINASE ACTIVATION | CELL BIOLOGY | Protein Kinases - metabolism | Cell Cycle - genetics | Phosphorylation | Antibiotics, Antineoplastic - pharmacology | RNA Processing, Post-Transcriptional - radiation effects | Mitosis | Humans | Intracellular Signaling Peptides and Proteins - metabolism | RNA Stability - drug effects | RNA, Messenger - metabolism | Cell Nucleus - enzymology | Transfection | Ultraviolet Rays | RNA Interference | Time Factors | Cell Cycle Proteins - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Active Transport, Cell Nucleus | Head and Neck Neoplasms - enzymology | cdc25 Phosphatases - metabolism | 3' Untranslated Regions | Nuclear Proteins - genetics | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Cytoplasm - enzymology | Signal Transduction | RNA Processing, Post-Transcriptional - drug effects | Cell Cycle - radiation effects | Protein-Serine-Threonine Kinases - genetics | Heterogeneous-Nuclear Ribonucleoproteins - metabolism | Feedback, Physiological | DNA Repair | Checkpoint Kinase 1 | Head and Neck Neoplasms - genetics | DNA Damage | HeLa Cells | Cell Cycle - drug effects | RNA Stability - radiation effects | Doxorubicin - pharmacology | Exoribonucleases - metabolism | RNA-Binding Proteins - metabolism | Messenger RNA | Oncology, Experimental | DNA | Genetics | Automated teller machines | Research | Universities and colleges | Tumor proteins | Gene expression | Cancer | Tumors
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 02/2017, Volume 216, Issue 2, pp. 295 - 297
Phosphoglycerate mutase 1 (PGAM1) functions in glycolysis. In this issue, Qu et al. (2017. J. Cell Biol. https://doi.org/10.1083/jcb.201607008) show that PGAM1... 
DAMAGE | PHOSPHOGLYCERATE MUTASE | CELL BIOLOGY | Glycolysis | Recombinational DNA Repair | Phosphoglycerate Mutase - genetics | DNA Repair | Humans | Neoplasms
Journal Article
Cell Cycle, ISSN 1538-4101, 08/2019, Volume 18, Issue 16, pp. 1830 - 1848
Recent studies have shown that genomic instability in tumor cells leads to activation of inflammatory signaling through the cGAS/STING pathway. In this review,... 
cGAS | STING | inflammatory signaling | cytoplasmic DNA | Genomic instability | interferon response | CYCLIC GMP-AMP | DEPENDENT ANTITUMOR IMMUNITY | CYTOSOLIC DNA SENSOR | TUMOR-REGRESSION | I INTERFERON | CHROMOSOMAL INSTABILITY | CELL BIOLOGY | BREAST-CANCER | AICARDI-GOUTIERES-SYNDROME | INTRACELLULAR DNA | INNATE IMMUNE SENSOR
Journal Article
Journal Article
Advances in Protein Chemistry and Structural Biology, ISSN 1876-1623, 01/2019, Volume 115, pp. 157 - 201
Genomically instable cancers are characterized by progressive loss and gain of chromosomal fragments, and the acquisition of complex genomic rearrangements.... 
Resolvases | Ultra-fine bridges | Checkpoint | WEE1 | CHK1 | Mitosis | Replication stress | Cell cycle | DNA repair | ATR | Cancer
Journal Article
Cell, ISSN 0092-8674, 2007, Volume 129, Issue 7, pp. 1415 - 1426
Protein kinases control cellular decision processes by phosphorylating specific substrates. Thousands of in vivo phosphorylation sites have been identified,... 
CELLBIO | ACTIVATION | DNA-DAMAGE RESPONSE | 53BP1 | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | SPECTROMETRY-BASED PROTEOMICS | ATM | EUKARYOTIC PROTEINS | PROTEIN-PHOSPHORYLATION | MASS-SPECTROMETRY | SIGNALING NETWORKS | CELL BIOLOGY | Protein Kinases - metabolism | Phosphorylation | Protein Kinases - genetics | Humans | DNA Repair Enzymes - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Phosphoproteins - metabolism | CDC2 Protein Kinase - metabolism | DNA-Binding Proteins - metabolism | Tumor Suppressor Proteins - genetics | DNA Repair Enzymes - metabolism | Cell Cycle Proteins - genetics | DNA Damage - genetics | Proteomics - methods | Intracellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Repressor Proteins - metabolism | CDC2 Protein Kinase - genetics | Computational Biology - methods | Tumor Suppressor Proteins - metabolism | Cell Cycle Proteins - metabolism | Protein-Serine-Threonine Kinases - genetics | Repressor Proteins - genetics | Ataxia Telangiectasia Mutated Proteins | Binding Sites - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Glycogen Synthase Kinase 3 - metabolism | Transcription Factors - metabolism | Glycogen Synthase Kinase 3 - genetics | Signal Transduction - physiology | Software | Tumor Suppressor p53-Binding Protein 1
Journal Article