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Biological Psychiatry, ISSN 0006-3223, 05/2019, Volume 85, Issue 9, pp. e45 - e46
Journal Article
Molecular Autism, ISSN 2040-2392, 05/2019, Volume 10, Issue 1, pp. 23 - 9
textabstractBackground: Angelman syndrome (AS) is a severe neurodevelopmental disorder caused by mutations affecting UBE3A gene expression. Previous studies in... 
Angelman syndrome | Phenotype | Mouse model | Seizure | Ube3a | Autism spectrum disorder | AUTISM | GENE | CRE | GENETICS & HEREDITY | NEUROSCIENCES | INSIGHTS | Usage | Electroencephalography | Research | Gene expression | Autism | Medical research | Analysis | Genes | Medicine, Experimental | Genetic aspects | Seizures (Medicine) | Phosphorylation | Brain research | Laboratories | Comorbidity | Rodents | Mutation | Females | Experiments
Journal Article
The EMBO Journal, ISSN 0261-4189, 12/2019, Volume 38, Issue 23, p. n/a
Tunneling nanotubes (TNTs) are actin‐based transient tubular connections that allow direct communication between distant cells. TNTs play an important role in... 
intercellular communication | tunneling nanotubes | CaMKII | α‐synuclein | Wnt pathway
Journal Article
Journal Article
Frontiers in Molecular Neuroscience, ISSN 1662-5099, 08/2019, Volume 12
textabstractNeuronal gap junctions formed by connexin36 (Cx36) and chemical synapses share striking similarities in terms of plasticity.... 
connexin36 | antibody | CaMKII | retina | bipolar cell | cross-reactivity | gap junction | electrical synapse
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2015, Volume 125, Issue 11, pp. 4305 - 15
Angelman syndrome (AS) is a severe neurological disorder that is associated with prominent movement and balance impairments that are widely considered to be... 
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2015, Volume 125, Issue 5, pp. 2069 - 2069
Angelman syndrome (AS) is a severe neurodevelopmental disorder that results from loss of function of the maternal ubiquitin protein ligase E3A (UBE3A) allele.... 
Journal Article
Journal of clinical investigation, ISSN 0021-9738, 2015, Volume 125, Issue 5, pp. 2069 - 2076
Angelman syndrome (AS) is a severe neurodevelopmental disorder that results from loss of function of the maternal ubiquitin protein ligase E3A (UBE3A) allele.... 
MEDICINE, RESEARCH & EXPERIMENTAL | MEMORY | MOUSE MODEL | MICE | PHENOTYPES | LONG-TERM POTENTIATION | INHIBITORS | MUTATIONS | DEFICITS | DISC1 | REVERSAL | Genomic Imprinting | Age Factors | Male | Epilepsy - physiopathology | Ubiquitin-Protein Ligases - physiology | Angelman Syndrome - therapy | Cerebral Cortex - physiopathology | Cerebellum - embryology | Anxiety - therapy | Gene Expression Regulation, Developmental | Angelman Syndrome - genetics | Epilepsy - genetics | Female | Disease Models, Animal | Recombinant Fusion Proteins - biosynthesis | Stereotyped Behavior - physiology | Hippocampus - embryology | Genes, Synthetic | Anxiety - physiopathology | Cerebellum - physiopathology | Ubiquitin-Protein Ligases - biosynthesis | Anxiety - genetics | Phenotype | Animals | Movement Disorders - physiopathology | Cerebral Cortex - embryology | Movement Disorders - therapy | Neuronal Plasticity | Angelman Syndrome - embryology | Tamoxifen - pharmacology | Recombinant Fusion Proteins - genetics | Ubiquitin-Protein Ligases - deficiency | Angelman Syndrome - physiopathology | Mice | Movement Disorders - genetics | Ubiquitin-Protein Ligases - genetics | Hippocampus - physiopathology | Epilepsy - therapy | Physiological aspects | Research | Ligases | Genes | Proteins | Studies | Software | Behavior | Gene expression | Epilepsy | Development | Genetics | Neuroscience
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 09/2018, Volume 38, Issue 37, pp. 8011 - 8030
Journal Article
Journal Article
The Journal of Neuroscience, ISSN 0270-6474, 09/2018, Volume 38, Issue 37, p. 8011
E3 ubiquitin ligase (UBE3A) levels in the brain need to be tightly regulated, as loss of functional UBE3A protein is responsible for the severe... 
Ubiquitin | Brain | Cortex (prefrontal) | Phenotypes | Interneurons | Neurons | Brain slice preparation | Excitability | Reversing | Action potential | Activation | Neurodevelopmental disorders | Proteins | Autism | Pyramidal cells | Reinstatement | Reaction kinetics | Physiology | Kinetics | Ubiquitin-protein ligase
Journal Article
Frontiers in Cellular Neuroscience, ISSN 1662-5102, 2014, Volume 8
Calcium/Calmodulin-dependent kinase type II (CaMKII) is essential for various forms of synaptic plasticity. The predominant α- and βCaMKII isoforms have both... 
Journal Article