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2004, Progress in inflammation research, ISBN 3764370017, xi, [296]
Book
1998, Progress in inflammation research, ISBN 9783764358532, xii, 236
Book
Cell and Tissue Research, ISSN 0302-766X, 01/2014, Volume 355, Issue 1, pp. 163 - 171
Lysyl hydroxylase 2b (LH2b) is known to increase pyridinoline cross-links, making collagen less susceptible to enzymatic degradation. Previously, we observed a... 
PATHWAYS | HUMAN-DISEASE | INDUCTION | Synovium | OSTEOPHYTE FORMATION | CELL BIOLOGY | OVEREXPRESSION | TGF-beta | GROWTH-FACTOR-BETA | Fibrosis | Lysyl hydroxylase 2b | Osteoarthritis | EXPRESSION | PYRIDINOLINE CROSS-LINKS | TRANSIENT FIBROSIS | TGF-BETA-1 | Collagen | Analysis | Heat shock proteins | Bone morphogenetic proteins | Hydroxylases | Transforming growth factors | Gene expression
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 03/2017, Volume 76, Issue Suppl 1, pp. A62 - A62
BackgroundNo disease-modifying drugs are available for osteoarthritis (OA). Although the aetiology is still unknown, inflammation is indicated to be involved... 
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 04/2016, Volume 68, Issue 4, p. 795
Objective The prevalence of periodontitis is increased in patients with rheumatoid arthritis (RA), and the severity of periodontitis can affect the level of... 
Journal Article
Cell Metabolism, ISSN 1550-4131, 2010, Volume 12, Issue 6, pp. 593 - 605
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 01/2014, Volume 370, Issue 5, pp. 433 - 443
Journal Article
Arthritis Research & Therapy, ISSN 1478-6354, 2005, Volume 7, Issue 1, pp. 29 - 37
Interleukin-17 (IL-17) is a T cell cytokine spontaneously produced by cultures of rheumatoid arthritis (RA) synovial membranes. High levels have been detected... 
Inflammation | Osteoclast | Autoimmune diseases | Interleukin-23 | Receptor activator of NF-κB ligand | Cytokine | autoimmune diseases | cytokine | RHEUMATOID-ARTHRITIS | COLLAGEN-INDUCED ARTHRITIS | BONE EROSION | NECROSIS-FACTOR-ALPHA | RHEUMATOLOGY | receptor activator of NF-kappa B ligand | interleukin-23 | NITRIC-OXIDE PRODUCTION | MESSENGER-RNA | inflammation | ARTICULAR-CARTILAGE | IN-VIVO | HUMAN OSTEOARTHRITIC CHONDROCYTES | osteoclast | NF-KAPPA-B | Autoimmune Diseases - physiopathology | Bone and Bones - pathology | Arthritis, Experimental - drug therapy | Receptor Activator of Nuclear Factor-kappa B | Species Specificity | Humans | Interleukin-17 - physiology | Synovial Fluid - metabolism | RANK Ligand | Receptors, Interleukin-17 | Arthritis, Experimental - pathology | Membrane Glycoproteins - physiology | Interleukins - physiology | Arthritis, Rheumatoid - drug therapy | Antirheumatic Agents - pharmacology | Cytokines - genetics | Arthritis, Rheumatoid - physiopathology | Antirheumatic Agents - therapeutic use | Neutrophils - pathology | Carrier Proteins - physiology | Osteoclasts - pathology | Receptors, Interleukin - physiology | T-Lymphocytes - secretion | Arthritis, Experimental - physiopathology | Arthritis, Rheumatoid - etiology | Gene Expression Regulation - physiology | Rats | Arthritis, Rheumatoid - pathology | Cartilage, Articular - pathology | Animals | Interleukin-17 - antagonists & inhibitors | Tumor Necrosis Factor-alpha - physiology | Mice | Cytokines - biosynthesis | Inflammation - physiopathology | receptor activator of NF-κB ligand | Review
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 03/2014, Volume 73, Issue Suppl 1, pp. A24 - A24
Background and Objective T helper-17 (Th17) cells are important mediators of inflammatory diseases, and are the main pathogenic cell type in many animal models... 
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 03/2014, Volume 73, Issue Suppl 1, pp. A68 - A69
Background and Objectives Rheumatoid arthritis (RA) is a chronic destructive autoimmune disease with periods of exacerbation and remission. An attractive... 
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 03/2014, Volume 73, Issue 3, p. A9
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 02/2012, Volume 71, Issue Suppl 1, pp. A74 - A75
Background and objectives The IL-1 receptor antagonist knockout mice (IL-1Ra-/-) spontaneously develop a T cell driven arthritis with profound IL-17... 
Journal Article