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Journal Article
Matrix Biology, ISSN 0945-053X, 2004, Volume 23, Issue 4, pp. 251 - 257
Journal Article
International Journal of Experimental Pathology, ISSN 0959-9673, 02/2004, Volume 85, Issue 1, pp. A24 - A25
Introduction  The hallmark of fibrosis is an excessive accumulation of collagen, a process in which TGF‐β plays an important role. The deposited collagen shows... 
Journal Article
International Journal of Experimental Pathology, ISSN 0959-9673, 02/2004, Volume 85, Issue 1, pp. A24 - A25
Journal Article
European Journal of Human Genetics, ISSN 1018-4813, 10/1999, Volume 7, Issue 7, pp. 837 - 840
Journal Article
Biochimica et biophysica acta, ISSN 0006-3002, 04/2005, Volume 1740, Issue 1, p. 60
Fibrotic processes in humans are characterised by an excessive accumulation of collagen containing increased levels of hydroxyallysine-derived cross-links. The... 
Fibrosis - chemically induced | Skin - metabolism | Humans | Collagen - chemistry | 2-Aminoadipic Acid - chemistry | Fibrosis - metabolism | Collagen - metabolism | Animals | Female | Mice | Matrix Metalloproteinase 1 - chemistry | 2-Aminoadipic Acid - analogs & derivatives | Skin - pathology | Bleomycin - toxicity
Journal Article
Journal Article
Neuroscience Letters, ISSN 0304-3940, 2000, Volume 286, Issue 1, pp. 53 - 56
Tetraethylammonium (TEA) induces a form of long-term potentiation (LTP) that is independent on N-methyl--aspartate (NMDA) receptor activation (LTPK). LTPK may... 
Protein kinase C | RC3 | GAP-43/B-50 | Long-term potentiation | Ca 2+ channel | K + channel | channel | K+ channel | PHOSPHORYLATION STATE | Ca2+ channel | GAP-43 | B-50 | AREA CA1 | NEUROSCIENCES | protein kinase C | long-term potentiation | PLASTICITY | Tetraethylammonium - pharmacology | Phosphorylation | Synaptic Transmission - physiology | 2-Amino-5-phosphonovalerate - pharmacology | Neurogranin | Hippocampus - drug effects | Presynaptic Terminals - ultrastructure | Excitatory Postsynaptic Potentials - drug effects | Excitatory Postsynaptic Potentials - physiology | Potassium Channels - metabolism | Long-Term Potentiation - drug effects | Protein Kinase C - metabolism | Long-Term Potentiation - physiology | Synaptic Transmission - drug effects | Potassium Channel Blockers | 4-Aminopyridine - pharmacology | Nifedipine - pharmacology | Presynaptic Terminals - drug effects | GAP-43 Protein - metabolism | Protein Kinase C - drug effects | Rats | Calmodulin-Binding Proteins - metabolism | Excitatory Amino Acid Antagonists - pharmacology | Calcium Channel Blockers - pharmacology | Hippocampus - cytology | Potassium Channels - drug effects | Synaptic Membranes - ultrastructure | Nerve Tissue Proteins - metabolism | Hippocampus - metabolism | Synaptic Membranes - drug effects | Animals | Signal Transduction - drug effects | Presynaptic Terminals - metabolism | Signal Transduction - physiology | In Vitro Techniques | Synaptic Membranes - metabolism
Journal Article
Journal Article
Molecular & Biochemical Parasitology, ISSN 0166-6851, 2000, Volume 109, Issue 2, pp. 147 - 156
Apical membrane antigen 1 is a candidate vaccine component for malaria. It is encoded by a single copy gene and has been characterised in a number of malaria... 
Red cell invasion | Plasmodium falciparum | Plasmodium reichenowi | Malaria | Apical membrane antigen-1 | Polymorphism | KNOWLESI | LOCALIZATION | PHYLOGENY | CHABAUDI | polymorphism | FULL-LENGTH | malaria |