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index medicus (75) 75
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macrophages (34) 34
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cancer (27) 27
mice, inbred c57bl (25) 25
tumors (23) 23
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macrophages - immunology (21) 21
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macrophages - pathology (15) 15
angiogenesis (14) 14
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macrophages - metabolism (14) 14
tumor microenvironment (14) 14
tumor-associated macrophages (14) 14
biology and life sciences (13) 13
gene expression (13) 13
hematology (13) 13
cells (12) 12
development and progression (12) 12
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immunotherapy (12) 12
metastasis (12) 12
monocytes (12) 12
monocytes - immunology (12) 12
neoplasms - pathology (12) 12
myeloid-derived suppressor cells (11) 11
neoplasms - immunology (11) 11
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biology (10) 10
cancer research (10) 10
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bone marrow (9) 9
disease models, animal (9) 9
health aspects (9) 9
macrophage (9) 9
macrophage activation (9) 9
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care and treatment (8) 8
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colony-stimulating factor (8) 8
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t-cells (8) 8
abridged index medicus (7) 7
allergy and immunology (7) 7
antitumor immunity (7) 7
dendritic cells - immunology (7) 7
genetic aspects (7) 7
immune system (7) 7
macrophages - drug effects (7) 7
macrophages - physiology (7) 7
mice, inbred balb c (7) 7
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1. Full Text The wound healing chronicles
by Van Ginderachter, Jo A
Blood, ISSN 0006-4971, 07/2012, Volume 120, Issue 3, pp. 499 - 500
In this issue of Blood, Willenborg and colleagues uncover the timeline of monocyte/macrophage involvement during sequential phases of skin wound healing. The... 
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2. Full Text Impeding Macrophage Entry into Hypoxic Tumor Areas by Sema3A/Nrp1 Signaling Blockade Inhibits Angiogenesis and Restores Antitumor Immunity
by Casazza, Andrea and Laoui, Damya and Wenes, Mathias and Rizzolio, Sabrina and Bassani, Nicklas and Mambretti, Marco and Deschoemaeker, Sofie and Van Ginderachter, Jo A and Tamagnone, Luca and Mazzone, Massimiliano
Cancer Cell, ISSN 1535-6108, 12/2013, Volume 24, Issue 6, pp. 695 - 709
Recruitment of tumor-associated macrophages (TAMs) into avascular areas sustains tumor progression; however, the underlying guidance mechanisms are unknown.... 
NEUROPILIN-1 | ONCOLOGY | INFLAMMATION | REGULATORY T-CELLS | AXON GUIDANCE | GENE-EXPRESSION | THERAPEUTIC TARGETS | RECEPTOR | SEMAPHORIN 3A | CANCER | ENDOTHELIAL GROWTH-FACTOR | CELL BIOLOGY | Macrophages - physiology | Neuropilin-1 - genetics | Neuropilin-1 - antagonists & inhibitors | Mice, Knockout | Cell Hypoxia | Neoplasms, Experimental - pathology | Neuropilin-1 - physiology | Animals | Semaphorin-3A - physiology | Neoplasms, Experimental - immunology | Macrophages - drug effects | Neovascularization, Pathologic - prevention & control | Signal Transduction - physiology | Mice | Semaphorin-3A - antagonists & inhibitors | Neoplasms, Experimental - blood supply | Development and progression | Endothelial growth factors | Macrophages | Tumors
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3. Full Text Identification of discrete tumor-induced myeloid-derived suppressor cell subpopulations with distinct T cell-suppressive activity
by Movahedi, Kiavash and Guilliams, Martin and van den Bossche, Jan and van den Bergh, Rafael and Gysemans, Conny and Beschin, Alain and de Baetselier, Patrick and van Ginderachter, Jo A
Blood, ISSN 0006-4971, 2008, Volume 111, Issue 8, pp. 4233 - 4244
The induction of CD11b(+)Gr-1(+) myeloid-derived suppressor cells (MDSCs) is an important immune-evading mechanism used by tumors. However, the exact nature... 
BEARING HOST | CTL SUPPRESSION | IFN-GAMMA | IN-VIVO | GENE-EXPRESSION | MICE | DIFFERENTIATION | SEQUENCE-BINDING PROTEIN | HEMATOLOGY | ACTIVATED GRANULOCYTES | PROGRESSION | Myeloid Cells - cytology | Cell Line | CD11b Antigen - immunology | Signal Transduction | Mice, Inbred C57BL | Spleen - cytology | Stem Cells - cytology | Macrophages - cytology | Epitopes - immunology | Lymphocyte Activation - immunology | Phenotype | Animals | T-Lymphocytes - cytology | Forkhead Transcription Factors - metabolism | Myeloid Cells - immunology | Interferon-gamma - immunology | T-Lymphocytes, Regulatory - cytology | Biomarkers | Female | Leukocytes, Mononuclear - cytology | Cell Differentiation | Mice | Kinetics | Neoplasms - pathology
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4. Full Text Identifying the variables that drive tamoxifen‐independent CreERT2 recombination: implications for microglial fate mapping and gene deletions
by Van Hove, Hannah and Antunes, Ana Rita Pombo and De Vlaminck, Karen and Scheyltjens, Isabelle and Van Ginderachter, Jo A and Movahedi, Kiavash
European Journal of Immunology, ISSN 0014-2980, 11/2019
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5. Full Text Different tumor microenvironments contain functionally distinct subsets of macrophages derived from Ly6C(high) monocytes
by Movahedi, Kiavash and Laoui, Damya and Gysemans, Conny and Baeten, Martijn and Stangé, Geert and van den Bossche, Jan and Mack, Matthias and Pipeleers, Daniel and In't Veld, Peter and de Baetselier, Patrick and van Ginderachter, Jo A
Cancer research, ISSN 0008-5472, 2010, Volume 70, Issue 14, pp. 5728 - 5739
Tumor-associated macrophages (TAM) form a major component of the tumor stroma. However, important concepts such as TAM heterogeneity and the nature of the... 
CTL SUPPRESSION | ONCOLOGY | INFLAMMATORY DENDRITIC CELLS | TIE2-EXPRESSING MONOCYTES | BONE-MARROW | SUPPRESSOR-CELLS | BLOOD MONOCYTES | NF-KAPPA-B | BEARING MICE | CANCER | PROGRESSION | Mammary Neoplasms, Experimental - immunology | Adenocarcinoma - pathology | Macrophages - pathology | Lymphocyte Activation | Adenocarcinoma - immunology | Mice, Inbred C57BL | Carcinoma, Lewis Lung - immunology | Cell Differentiation - immunology | Animals | Antigens, Ly - immunology | Antigens, Ly - biosynthesis | Myeloid Cells - immunology | Mammary Neoplasms, Experimental - pathology | Carcinoma, Lewis Lung - pathology | Female | Granulocytes - pathology | T-Lymphocytes - immunology | Mice | Mice, Inbred BALB C | Myeloid Cells - pathology | T-Lymphocytes - pathology | Granulocytes - immunology | Macrophages - immunology
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6. Full Text HRG Inhibits Tumor Growth and Metastasis by Inducing Macrophage Polarization and Vessel Normalization through Downregulation of PlGF
by Rolny, Charlotte and Mazzone, Massimiliano and Tugues, Sònia and Laoui, Damya and Johansson, Irja and Coulon, Cathy and Squadrito, Mario Leonardo and Segura, Inmaculada and Li, Xiujuan and Knevels, Ellen and Costa, Sandra and Vinckier, Stefan and Dresselaer, Tom and Åkerud, Peter and De Mol, Maria and Salomäki, Henriikka and Phillipson, Mia and Wyns, Sabine and Larsson, Erik and Buysschaert, Ian and Botling, Johan and Himmelreich, Uwe and Van Ginderachter, Jo A and De Palma, Michele and Dewerchin, Mieke and Claesson-Welsh, Lena and Carmeliet, Peter
Cancer Cell, ISSN 1535-6108, 2011, Volume 19, Issue 1, pp. 31 - 44
Polarization of tumor-associated macrophages (TAMs) to a proangiogenic/immune-suppressive (M2-like) phenotype and abnormal, hypoperfused vessels are hallmarks... 
Clodronic Acid - pharmacology | Gene Expression - genetics | Dendritic Cells - immunology | Humans | Pregnancy Proteins - genetics | Microvessels - pathology | Pregnancy Proteins - metabolism | Chemotactic Factors - metabolism | Neoplasm Metastasis - immunology | Antibodies - immunology | Neovascularization, Pathologic - immunology | Macrophages, Peritoneal - drug effects | Macrophages - pathology | Neoplasms - blood supply | Down-Regulation - genetics | Mice, Knockout | Macrophages - metabolism | Neoplasms - immunology | Cell Line, Tumor | Placenta Growth Factor | Mice | Mice, Inbred BALB C | CD8-Positive T-Lymphocytes - immunology | Macrophages, Peritoneal - metabolism | Neoplasms - metabolism | CD8-Positive T-Lymphocytes - pathology | Gene Expression - drug effects | Regional Blood Flow - genetics | Dendritic Cells - pathology | Culture Media, Conditioned - pharmacology | Lung Neoplasms - pathology | Hypoxia - metabolism | Neovascularization, Pathologic - pathology | Regional Blood Flow - drug effects | Transfection | Lung Neoplasms - secondary | Neoplasms - genetics | Dendritic Cells - metabolism | Macrophages - immunology | Lung Neoplasms - genetics | Microvessels - ultrastructure | Cytokines - metabolism | Mice, Inbred C57BL | Microvessels - drug effects | Pregnancy Proteins - immunology | Antibodies - pharmacology | Proteins - genetics | Neoplasm Metastasis - genetics | Hypoxia - genetics | Lung Neoplasms - immunology | Animals | Proteins - metabolism | Endothelial Cells - cytology | Neoplasm Metastasis - pathology | Macrophages - drug effects | Neovascularization, Pathologic - genetics | Cell Proliferation - drug effects | Proteins - pharmacology | Neoplasms - pathology | Endothelial Cells - drug effects
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7. Full Text Macrophage Activation and Polarization: Nomenclature and Experimental Guidelines
by Murray, Peter J and Allen, Judith E and Biswas, Subhra K and Fisher, Edward A and Gilroy, Derek W and Goerdt, Sergij and Gordon, Siamon and Hamilton, John A and Ivashkiv, Lionel B and Lawrence, Toby and Locati, Massimo and Mantovani, Alberto and Martinez, Fernando O and Mege, Jean-Louis and Mosser, David M and Natoli, Gioacchino and Saeij, Jeroen P and Schultze, Joachim L and Shirey, Kari Ann and Sica, Antonio and Suttles, Jill and Udalova, Irina and van Ginderachter, Jo A and Vogel, Stefanie N and Wynn, Thomas A
Immunity, ISSN 1074-7613, 07/2014, Volume 41, Issue 1, pp. 14 - 20
Description of macrophage activation is currently contentious and confusing. Like the biblical Tower of Babel, macrophage activation encompasses a panoply of... 
NITRIC-OXIDE PRODUCTION | MECHANISM | INFLAMMATION | GENE-EXPRESSION | ATHEROSCLEROSIS | DIVERSITY | DIFFERENTIATION | IMMUNOLOGY | MOUSE MACROPHAGES | ALTERNATIVE ACTIVATION | PLASTICITY | Guidelines as Topic | Animals | Granulocyte-Macrophage Colony-Stimulating Factor - immunology | Macrophage Activation - immunology | Humans | Terminology as Topic | Mice | Research | Macrophages - immunology | Macrophage Colony-Stimulating Factor - immunology | Macrophages | Studies | Regulatory agencies | Disease | Cytokines | Pathogenesis | Epigenetics | Autoimmune diseases | Pharmaceutical industry | Gene expression | Metabolic disorders
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8. E-cadherin: From epithelial glue to immunological regulator
by Jan Bossche and Jo A. Van Ginderachter
European Journal of Immunology, ISSN 0014-2980, 01/2013, Volume 43, Issue 1, p. 34
  E-cadherin is best known as a central molecule in adherens junctions, joining adjacent epithelial cells together, thereby safeguarding epithelial barrier... 
Proteins | Cellular biology | Cells
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9. Full Text Macrophage metabolism as therapeutic target for cancer, atherosclerosis, and obesity
by Geeraerts, Xenia and Bolli, Evangelia and Fendt, Sarah-Maria and Van Ginderachter, Jo A
Frontiers in Immunology, ISSN 1664-3224, 03/2017, Volume 8, p. 289
Macrophages are not only essential components of innate immunity that contribute to host defense against infections, but also tumor growth and the maintenance... 
Immunometabolism | M1-M2 macrophage polarization | Metabolic therapy | Metabolic reprogramming | Microenvironment | Inflammatory diseases | Tumor-associated macrophages | Cancer | LIPID-METABOLISM | TUMOR PROGRESSION | MITOCHONDRIAL CITRATE CARRIER | inflammatory diseases | IMMUNOLOGY | CELL METABOLISM | tumor-associated macrophages | microenvironment | SUCCINATE-DEHYDROGENASE | immunometabolism | FATTY-ACID OXIDATION | GLUCOSE-METABOLISM | metabolic therapy | NITRIC-OXIDE | cancer | TNF-ALPHA | metabolic reprogramming | ALTERNATIVE ACTIVATION | Physiological aspects | Cell metabolism | Macrophages | Health aspects
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10. Full Text Nanobody-based targeting of the macrophage mannose receptor for effective in vivo imaging of tumor-associated macrophages
by Movahedi, Kiavash and Schoonooghe, Steve and Laoui, Damya and Houbracken, Isabelle and Waelput, Wim and Breckpot, Karine and Bouwens, Luc and Lahoutte, Tony and De Baetselier, Patrick and Raes, Geert and Devoogdt, Nick and Van Ginderachter, Jo A
Cancer Research, ISSN 0008-5472, 08/2012, Volume 72, Issue 16, pp. 4165 - 4177
Tumor-associated macrophages (TAM) are an important component of the tumor stroma and exert several tumor-promoting activities. Strongly pro-angiogenic TAMs... 
DOMAIN ANTIBODY FRAGMENTS | DENDRITIC CELLS | ONCOLOGY | INFLAMMATION | RADIOIMMUNOTHERAPY | PINHOLE SPECT/MICRO-CT | MOLECULAR-SIZE | MICE | EXPRESSION | CANCER-THERAPY | MURINE | Macrophages - chemistry | Cell Hypoxia - physiology | Tomography, Emission-Computed, Single-Photon - methods | Single-Domain Antibodies - metabolism | Carcinoma, Lewis Lung - immunology | Lectins, C-Type - immunology | Molecular Sequence Data | Lectins, C-Type - chemistry | Macrophages - diagnostic imaging | Single-Domain Antibodies - immunology | Tissue Distribution | Carcinoma, Lewis Lung - diagnostic imaging | Female | Receptors, Cell Surface - chemistry | Macrophages - immunology | Technetium - chemistry | Amino Acid Sequence | Mice, Inbred C57BL | Carcinoma, Lewis Lung - metabolism | Receptors, Cell Surface - immunology | Mannose-Binding Lectins - chemistry | Animals | Mannose-Binding Lectins - immunology | Single-Domain Antibodies - chemistry | Mice | Mice, Inbred BALB C
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11. Full Text The Ontogeny and Microenvironmental Regulation of Tumor-Associated Macrophages
by Movahedi, Kiavash and Van Ginderachter, Jo A
Antioxidants & Redox Signaling, ISSN 1523-0864, 11/2016, Volume 25, Issue 14, pp. 775 - 791
Significance: Tumor progression is supported by non-cancerous stromal cells, of which tumor-associated macrophages (TAMs) are prominent constituents. These... 
Forum Review Articles | hypoxia | cancer | tumor microenvironment | inflammation | Macrophage | DENDRITIC CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | macrophage | GENE-EXPRESSION ANALYSIS | MYELOID CELLS | MAMMARY-TUMORS | HEMATOPOIETIC STEM-CELLS | IN-VIVO | ENDOCRINOLOGY & METABOLISM | TISSUE-RESIDENT MACROPHAGES | NF-KAPPA-B | COLONY-STIMULATING FACTOR-1 | ALTERNATIVE ACTIVATION | Macrophages - physiology | Tumor Microenvironment | Cell Differentiation | Neoplasms - pathology | Humans | Development and progression | Research | Macrophages | Tumors | Phenotypes | Therapeutic applications | Blood vessels | Ontogeny | Heterogeneity | Monocytes | Chemotherapy | Immunosuppression | Immunotherapy | Stromal cells | Growth factors
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12. Full Text Tissue-resident versus monocyte-derived macrophages in the tumor microenvironment
by Lahmar, Qods and Keirsse, Jiri and Laoui, Damya and Movahedi, Kiavash and Van Overmeire, Eva and Van Ginderachter, Jo A
BBA - Reviews on Cancer, ISSN 0304-419X, 01/2016, Volume 1865, Issue 1, pp. 23 - 34
The tumor-promoting role of macrophages has been firmly established in most cancer types. However, macrophage identity has been a matter of debate, since... 
Kupffer cell | Glioma | Lung cancer | Tissue-resident macrophages | Pancreatic adenocarcinoma | Hepatocellular carcinoma | Breast cancer | Monocyte-derived macrophages | Tumor-associated macrophages | Microglia | ACTIVATED MONOCYTES | BIOCHEMISTRY & MOLECULAR BIOLOGY | PANCREATIC-CANCER | MYELOID CELLS | STIMULATING FACTOR-I | CD8(+) T-CELLS | CELL LUNG-CANCER | BREAST-CANCER | PROMOTE EXPANSION | BIOPHYSICS | ONCOLOGY | MAMMARY-GLAND DEVELOPMENT | TIE2-EXPRESSING MONOCYTES | Macrophages | Analysis | Tumors | Liver cancer | Gliomas | Pancreatic cancer | Liver
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13. Full Text Tumor hypoxia does not drive differentiation of tumor-associated macrophages but rather fine-tunes the M2-like macrophage population
by Laoui, Damya and Van Overmeire, Eva and Conza, Giusy Di and Aldeni, Chiara and Keirsse, Jiri and Morias, Yannick and Movahedi, Kiavash and Houbracken, Isabelle and Schouppe, Elio and Elkrim, Yvon and Karroum, Oussama and Jordan, Bénédicte and Carmeliet, Peter and Gysemans, Conny and DeBaetselier, Patrick and Mazzone, Massimiliano and Van Ginderachter, Jo A
Cancer Research, ISSN 0008-5472, 01/2014, Volume 74, Issue 1, pp. 24 - 30
Tumor-associated macrophages (TAM) are exposed to multiple microenvironmental cues in tumors, which collaborate to endow these cells with protumoral... 
ONCOLOGY | EXPRESSION | METASTASIS | Neoplasms - metabolism | Cell Hypoxia - physiology | Macrophages - pathology | Mice, Inbred C57BL | Transcriptome | Mice, Transgenic | Macrophages - metabolism | Animals | Neoplasms - genetics | Female | Mice | Neoplasms - pathology | Cell Differentiation - physiology | Disease Models, Animal
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