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1. Full Text HMG-coenzyme A reductase inhibition, type 2 diabetes, and bodyweight: evidence from genetic analysis and randomised trials
by Swerdlow, Daniel I, Dr and Preiss, David, Dr and Kuchenbaecker, Karoline B, MSc and Holmes, Michael V, PhD and Engmann, Jorgen E L, MSc and Shah, Tina, PhD and Sofat, Reecha, MD and Stender, Stefan, MD and Johnson, Paul C D, PhD and Scott, Robert A, PhD and Leusink, Maarten, MSc and Verweij, Niek, MD and Sharp, Stephen J, MSc and Guo, Yiran, PhD and Giambartolomei, Claudia, MSc and Chung, Christina, MSc and Peasey, Anne, PhD and Amuzu, Antoinette, MSc and Li, KaWah, MSc and Palmen, Jutta, MSc and Howard, Philip, MSc and Cooper, Jackie A, MSc and Drenos, Fotios, PhD and Li, Yun R, BSc and Lowe, Gordon, Prof and Gallacher, John, PhD and Stewart, Marlene C W, PhD and Tzoulaki, Ioanna, PhD and Buxbaum, Sarah G, PhD and van der A, Daphne L, PhD and Forouhi, Nita G, MRCP and Onland-Moret, N Charlotte, PhD and van der Schouw, Yvonne T, Prof and Schnabel, Renate B, MD and Hubacek, Jaroslav A, PhD and Kubinova, Ruzena, MD and Baceviciene, Migle, MD and Tamosiunas, Abdonas, Prof and Pajak, Andrzej, Prof and Topor-Madry, Romanvan, MD and Stepaniak, Urszula, PhD and Malyutina, Sofia, Prof and Baldassarre, Damiano, PhD and Sennblad, Bengt, PhD and Tremoli, Elena, Prof and de Faire, Ulf, Prof and Veglia, Fabrizio, PhD and Ford, Ian, Prof and Jukema, J Wouter, Prof and Westendorp, Rudi G J, Prof and de Borst, Gert Jan, MD and de Jong, Pim A, MD and Algra, Ale, Prof and Spiering, Wilko, MD and der Zee, Anke H Maitland-van, PhD and Klungel, Olaf H, PharmD and de Boer, Anthonius, Prof and Doevendans, Pieter A, Prof and Eaton, Charles B, Prof and Robinson, Jennifer G, Prof and Duggan, David, PhD and Kjekshus, John, Prof and Downs, John R, MD and Gotto, Antonio M, MD and Keech, Anthony C, MD and Marchioli, Roberto, MD and Tognoni, Gianni, MD and Sever, Peter S, Prof and Poulter, Neil R, Prof and Waters, David D, MD and Pedersen, Terje R, MD and Amarenco, Pierre, MD and Nakamura, Haruo, MD and McMurray, John J V, Prof and Lewsey, James D, PhD and Chasman, Daniel I, PhD and Ridker, Paul M, MD and Maggioni, Aldo P, Prof and Tavazzi, Luigi, Prof and Ray, Kausik K, Prof and Seshasai, Sreenivasa Rao Kondapally, MD and Manson, JoAnn E, Prof and Price, Jackie F, MD and Whincup, Peter H, Prof and Morris, Richard W, Prof and Lawlor, Debbie A, Prof and Smith, George Davey, Prof and Ben-Shlomo, Yoav, Prof and Schreiner, Pamela J, Prof and Fornage, Myriam, Prof and Siscovick, David S, Prof and Cushman, Mary, MD and Kumari, Meena, PhD and Wareham, Nick J, Prof and Verschuren, W M Monique, PhD and Redline, Susan, Prof and Patel, Sanjay R, MD and Whittaker, John C, Prof and Hamsten, Anders, Prof and Delaney, Joseph A, PhD and ... and MAGIC Consortium and InterAct Consortium and DIAGRAM Consortium
The Lancet, ISSN 0140-6736, 01/2015, Volume 385, Issue 9965, pp. 351 - 361
Summary Background Statins increase the risk of new-onset type 2 diabetes mellitus. We aimed to assess whether this increase in risk is a consequence of... 
Internal Medicine | LDL CHOLESTEROL | HIGH-DOSE ATORVASTATIN | METAANALYSIS | STATIN THERAPY | ASSOCIATION ANALYSES | DISEASE | PREVENTION | BODY-MASS INDEX | RISK | MENDELIAN RANDOMIZATION | MEDICINE, GENERAL & INTERNAL | Body Mass Index | Genetic Testing | Cholesterol, HDL - metabolism | Diabetes Mellitus, Type 2 - genetics | Humans | Middle Aged | Risk Factors | Male | Body Weight - genetics | Randomized Controlled Trials as Topic | Hydroxymethylglutaryl-CoA Reductase Inhibitors - adverse effects | Cholesterol, LDL - metabolism | Polymorphism, Single Nucleotide - genetics | Female | Aged | Hydroxymethylglutaryl CoA Reductases - genetics | Type 2 diabetes | Genetic research | Enzymes | Thiols | Diabetes | Low density lipoprotein | Statins | Cholesterol | Risk factors
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2. Full Text Association between alcohol and cardiovascular disease: Mendelian randomisation analysis based on individual participant data
by Holmes, Michael V and Dale, Caroline E and Zuccolo, Luisa and Silverwood, Richard J and Guo, Yiran and Ye, Zheng and Prieto-Merino, David and Dehghan, Abbas and Trompet, Stella and Wong, Andrew and Cavadino, Alana and Drogan, Dagmar and Padmanabhan, Sandosh and Li, Shanshan and Yesupriya, Ajay and Leusink, Maarten and Sundstrom, Johan and Hubacek, Jaroslav A and Pikhart, Hynek and Swerdlow, Daniel I and Panayiotou, Andrie G and Borinskaya, Svetlana A and Finan, Chris and Shah, Sonia and Kuchenbaecker, Karoline B and Shah, Tina and Engmann, Jorgen and Folkersen, Lasse and Eriksson, Per and Ricceri, Fulvio and Melander, Olle and Sacerdote, Carlotta and Gamble, Dale M and Rayaprolu, Sruti and Ross, Owen A and McLachlan, Stela and Vikhireva, Olga and Sluijs, Ivonne and Scott, Robert A and Adamkova, Vera and Flicker, Leon and Bockxmeer, Frank M van and Power, Christine and Marques-Vidal, Pedro and Meade, Tom and Marmot, Michael G and Ferro, Jose M and Paulos-Pinheiro, Sofia and Humphries, Steve E and Talmud, Philippa J and Leach, Irene Mateo and Verweij, Niek and Linneberg, Allan and Skaaby, Tea and Doevendans, Pieter A and Cramer, Maarten J and Harst, Pim van der and Klungel, Olaf H and Dowling, Nicole F and Dominiczak, Anna F and Kumari, Meena and Nicolaides, Andrew N and Weikert, Cornelia and Boeing, Heiner and Ebrahim, Shah and Gaunt, Tom R and Price, Jackie F and Lannfelt, Lars and Peasey, Anne and Kubinova, Ruzena and Pajak, Andrzej and Malyutina, Sofia and Voevoda, Mikhail I and Tamosiunas, Abdonas and Maitland-van der Zee, Anke H and Norman, Paul E and Hankey, Graeme J and Bergmann, Manuela M and Hofman, Albert and Franco, Oscar H and Cooper, Jackie and Palmen, Jutta and Spiering, Wilko and Jong, Pim A de and Kuh, Diana and Hardy, Rebecca and Uitterlinden, Andre G and Ikram, M Arfan and Ford, Ian and Hyppönen, Elina and Almeida, Osvaldo P and Wareham, Nicholas J and Khaw, Kay-Tee and Hamsten, Anders and Husemoen, Lise Lotte N and Tjønneland, Anne and Tolstrup, Janne S and Rimm, Eric and Beulens, Joline W J and Verschuren, W M Monique and ... and InterAct Consortium and IMPROVE Study Grp and on behalf of The InterAct Consortium and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Kardiologi and Medicinska och farmaceutiska vetenskapsområdet, centrumbildningar mm and Geriatrik and Uppsala universitet and Institutionen för medicinska vetenskaper and Institutionen för folkhälso- och vårdvetenskap and Uppsala kliniska forskningscentrum (UCR)
BMJ : British Medical Journal, ISSN 0959-8138, 7/2014, Volume 349, Issue jul10 6, pp. g4164 - g4164
Objective To use the rs1229984 variant in the alcohol dehydrogenase 1B gene (ADH1B) as an instrument to investigate the causal role of alcohol in... 
RESEARCH | MORTALITY | MEDICINE, GENERAL & INTERNAL | DESIGN | RISK-FACTORS | METAANALYSIS | VARIANTS | HEALTH | CORONARY-HEART-DISEASE | CONSUMPTION | DEHYDROGENASE | DEPENDENCE | Coronary Disease - blood | Humans | Middle Aged | Coronary Disease - etiology | Genotype | Male | Biomarkers - blood | Genetic Markers | Models, Statistical | Alcohol Drinking - adverse effects | Alcohol Drinking - genetics | Stroke - genetics | Stroke - blood | Stroke - etiology | Coronary Disease - genetics | Alcohol Dehydrogenase - genetics | Mendelian Randomization Analysis | Adult | Female | Aged | Polymorphism, Single Nucleotide | Other Medical Sciences | Medical and Health Sciences | Medicin och hälsovetenskap | Annan medicin och hälsovetenskap
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3. Full Text Identification of 64 Novel Genetic Loci Provides an Expanded View on the Genetic Architecture of Coronary Artery Disease
by van der Harst, Pim and Verweij, Niek
Circulation research, ISSN 0009-7330, 02/2018, Volume 122, Issue 3, pp. 433 - 443
Rationale: Coronary artery disease (CAD) is a complex phenotype driven by genetic and environmental factors. Ninety-seven genetic risk loci have been... 
computational biology | coronary artery disease | REGULATOR | RISK LOCI | genetics | PROTEIN | EVENTS | genome-wide association study | sample size | VARIANT | DISCOVERY | GENOME-WIDE ASSOCIATION | Genetics | Genome-wide association study | Sample size | Coronary artery disease | Computational biology | CARDIAC & CARDIOVASCULAR SYSTEMS | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Genome-wide association studies | Research | Genetic variation | Coronary heart disease | Analysis | 10082 | 10081 | 10084 | 10053 | 10189 | Molecular Medicine
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4. Full Text Genome-wide association study identifies 74 loci associated with educational attainment
by Okbay, Aysu and Beauchamp, Jonathan and Fontana, M.A and Lee, James J and Pers, Tune and Rietveld, C.A and Turley, Patrick and Chen, G.-B and Emilsson, Valur and Meddens, S.F.W and Oskarsson, S and Pickrell, J.K and Thom, K and Timshel, P and Vlaming, Ronald and Abdellaoui, Abdel and Ahluwalia, Tarunveer Singh and Bacelis, Jonas and Baumbach, Clemens and Bjornsdottir, G and Brandsma, Johan and Pina Concas, M and Derringer, J and Furlotte, N.A and Galesloot, Tessel and Girotto, S and Gupta, R and Hall, Leanne M and Harris, Sarah and Hofer, E and Horikoshi, M and Huffman, Jennifer E and Kaasik, K and Kalafati, Ioanna-Panagiota and Karlsson, Robert and Kong, Augustine and Lahti, Jari and Lee, Sven and Deleeuw, C and Lind, Penelope and Lindgren, K.-O and Liu, T and Mangino, Massimo and Marten, Jonathan and Mihailov, Evelin and Miller, Mike and Most, Peter and Oldmeadow, Christopher and Payton, Antony and Pervjakova, Natalia and Peyrot, Wouter and Qian, Y and Raitakari, Olli and Rueedi, R and Salvi, E and Schmidt, B and Schraut, K.E and Shi, J and Smith, Albert Vernon and Poot, Raymond and St Pourcain, Beate and Teumer, Alexander and Thorleifsson, Gudmar and Verweij, Niek and Vuckovic, gana and Wellmann, J and Westra, Harm-Jan and Yang, J and Zhao, W and Zhu, Z and Alizadeh, Behrooz and Amin, Najaf and Bakshi, A and Baumeister, Sebastian and Biino, Ginevra and Bønnelykke, Klaus and Boyle, Patricia and Campbell, Harry and Cappuccio, F.P and Davies, Gail and Neve, Jan-Emmanuel and Deloukas, Panagiotis and Demuth, Ilja and Ding, J and Eibich, P and Eisele, L and Eklund, Niina and Evans, David and Faul, Jessica D and Feitosa, Mary Furlan and Forstner, Aneas and Gandin, Ilaria and Gunnarsson, B and Halldorsson, Bjarni and Harris, Tamara and Holliday, Elizabeth and Heath, Anew C and Hocking, L.J and Homuth, Georg and Horan, Mike and ... and LifeLines Cohort Study and Humanistisk-samhällsvetenskapliga vetenskapsområdet and Uppsala universitet and Statsvetenskapliga institutionen and Samhällsvetenskapliga fakulteten
Nature: international weekly journal of science, ISSN 0028-0836, 05/2016, Volume 533, Issue 7604, pp. 539 - 542
textabstractEducational attainment is strongly influenced by social and other environmental factors, but genetic factors are estimated to account for at least... 
ACHIEVEMENT | TRAITS | MULTIDISCIPLINARY SCIENCES | HERITABILITY | Genome-Wide Association Study | Gene Expression Regulation - genetics | Humans | Molecular Sequence Annotation | Computational Biology | Fetus - metabolism | United Kingdom | Cognition | Educational Status | Bipolar Disorder - genetics | Gene-Environment Interaction | Brain - metabolism | Schizophrenia - genetics | Polymorphism, Single Nucleotide - genetics | Alzheimer Disease - genetics | Quantitative trait loci | Academic achievement | Genome-wide association studies | Genetic aspects | Observations | Studies | Genomes | Gene expression | Genes | Educational attainment | Meta-analysis | Quantitative Finance | Samhällsvetenskap | Biological Sciences | Social Sciences | Naturvetenskap | Natural Sciences | Biologiska vetenskaper
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5. Full Text Defining the role of common variation in the genomic and biological architecture of adult human height
by Wood, Anew R and Esko, Tonu and Yang, Jian and Vedantam, Sailaja and Pers, Tune H and Gustafsson, Stefan and Chu, Auey Y and Estrada, Karol and Luan, Jian'an and Kutalik, Zoltán and Amin, Najaf and Buchkovich, Martin L and Croteau-Chonka, Damien C and Day, Felix R and Duan, Yanan and Fall, Tove and Fehrmann, Rudolf and Ferreira, Teresa and Jackson, Anne U and Karjalainen, Juha and Lo, Ken Sin and Locke, Adam E and Mägi, Reedik and Mihailov, Evelin and Porcu, Eleonora and Randall, Joshua C and Scherag, Ané and Vinkhuyzen, Anna A. E and Westra, Harm-Jan and Winkler, Thomas W and Workalemahu, Tsegaselassie and Zhao, Jing Hua and Absher, Devin and Albrecht, Eva and Anderson, Denise and Baron, Jeffrey and Beekman, Marian and Demirkan, Ayse and Ehret, Georg B and Feenstra, Bjarke and Feitosa, Mary F and Fischer, Krista and Fraser, Ross M and Goel, Anuj and Gong, Jian and Justice, Anne E and Kanoni, Stavroula and van der Velde, Nathalie and Hovingh, G. Kees and Kastelein, John J. P and PAGE Consortium and MIGen Consortium and LifeLines Cohort Study and Elect Med Records & Genom eMERGE C and Electronic Medical Records and Genomics (eMEMERGEGE) Consortium and PAGEGE Consortium and The MIGen Consortium and The PAGE Consortium and The Electronic Medical Records and Genomics (eMERGE) Consortium and The LifeLines Cohort Study and Högskolan Dalarna and Akademin Utbildning, hälsa och samhälle and Medicinsk vetenskap
Nature genetics, ISSN 1061-4036, 2014, Volume 46, Issue 11, pp. 1173 - 1186
Using genome-wide data from 253,288 individuals, we identified 697 variants at genome-wide significance that together explained one-fifth of the heritability... 
SNPS | GENETIC-VARIATION | MUTATIONS | COMPLEX TRAITS | HERITABILITY | GENETICS & HEREDITY | Genetics, Population | European Continental Ancestry Group - genetics | Analysis of Variance | Oligonucleotide Array Sequence Analysis | Humans | Polymorphism, Single Nucleotide - genetics | Adult | Genetic Variation - genetics | Genome-Wide Association Study - methods | Body Height - genetics | Stature | Genome-wide association studies | Genetic variation | Genes | Genetic aspects | Research | Identification and classification | Studies | Genomes | Genealogy | Architecture | Statistics | Clinical Medicine | Hälsa och välfärd | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Health and Welfare | complex traits | mutations | snps | genetic-variation | heritability
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6. Full Text Eosinophil Count Is a Common Factor for Complex Metabolic and Pulmonary Traits and Diseases: The LifeLines Cohort Study
by Amini, Marzyeh and Bashirova, Dinara and Prins, Bram P and Corpeleijn, Eva and Bruinenberg, Marcel and Franke, Lude and van der Harst, Pim and Navis, Gerjan and Wolffenbuttel, Bruce H. R and Stolk, Ronald P and Wijmenga, Cisca and Postma, Dirkje S and Koppelman, Gerard H and Boezen, H. Marike and Vonk, Judith and Snieder, Harold and Alizadeh, Behrooz Z and LifeLines Cohort Study
PLoS ONE, ISSN 1932-6203, 12/2016, Volume 11, Issue 12, p. e0168480
There is ongoing debate on the association between eosinophil count and diseases, as previous studies were inconsistent. We studied the relationship of... 
HEART-DISEASE | SEVERE ASTHMA | DIFFERENTIAL LEUKOCYTE COUNTS | INFLAMMATION | EXACERBATIONS | BODY-MASS INDEX | AIRWAY EOSINOPHILIA | SPUTUM EOSINOPHILS | BLOOD-CELL COUNTS | GENETIC-VARIANTS | DETERMINANTS | MULTIDISCIPLINARY SCIENCES | ASTHMA | INTERLEUKIN-5 | Body Mass Index | Multivariate Analysis | Asthma - metabolism | Lipoproteins, HDL - blood | Glycated Hemoglobin A - metabolism | Humans | Middle Aged | Logistic Models | Male | Metabolic Syndrome - metabolism | Obesity - blood | Obesity - metabolism | Eosinophils - physiology | Metabolic Syndrome - blood | Regression Analysis | Asthma - blood | Pulmonary Disease, Chronic Obstructive - blood | Sex Factors | Adult | Female | Aged | Leukocyte Count | Pulmonary Disease, Chronic Obstructive - metabolism | Cohort Studies | Measurement | Physiological aspects | Metabolic diseases | Respiratory tract diseases | Research | Diagnosis | Eosinophils | Lipoproteins (low density) | Obesity | Pediatrics | Statistical analysis | Lung diseases | Population studies | Regression analysis | Metabolic syndrome | Epidemiology | Coronary artery disease | Asthma | Diseases | Body mass index | Mens health | Body mass | Womens health | Men | Population | Chronic obstructive pulmonary disease | Blood pressure | Heart diseases | Lipoproteins (high density) | Metabolic disorders | Smoking
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7. Full Text Healthy aging and disease: Role for telomere biology?
by Zhu, Haidong and Belcher, Matthew and Van Der Harst, Pim
Clinical Science, ISSN 0143-5221, 05/2011, Volume 120, Issue 10, pp. 427 - 440
Aging is a biological process that affects most cells, organisms and species. Human aging is associated with increased susceptibility to a variety of chronic... 
Heart failure | Parkinson's disease | Atherosclerosis | Aging | Longevity | Alzheimer's disease | Telomerase | Telomere | atherosclerosis | MEDICINE, RESEARCH & EXPERIMENTAL | heart failure | CHRONIC HEART-FAILURE | OXIDATIVE STRESS | REPEAT-CONTAINING RNA | telomere | BLOOD MONONUCLEAR-CELLS | ENDOTHELIAL PROGENITOR CELLS | telomerase | REVERSE-TRANSCRIPTASE HTERT | CARDIOVASCULAR-DISEASE | HEMODIALYSIS-PATIENTS | longevity | SMOOTH-MUSCLE-CELLS | aging | GENOME-WIDE ASSOCIATION | Aging - genetics | Telomerase - physiology | Telomere - ultrastructure | Diabetes Mellitus - genetics | Humans | Stem Cells - physiology | Telomere - physiology | Cardiovascular Diseases - genetics | Chronic Disease | T2D, Type 2 diabetes | CVD, cardiovascular disease | Review | T1D, Type 1 diabetes | TERC, telomerase RNA component | LDL, low-density lipoprotein | PBMC, peripheral blood mononuclear cell | EPA, eicosapentaenoic acid | TERT, telomerase reverse transcriptase | SHR, spontaneous hypertensive rats | DHA, docosahexaenoic acid | PD, Parkinson's disease | AD, Alzheimer's disease | TERRA, telomeric repeat-containing RNA | TRF, telomeric repeat-binding factor | ACEI, angiotensin-converting enzyme inhibitor | CRP, C-reactive protein | CHF, chronic heart failure
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8. Full Text Translational Perspective on Epigenetics in Cardiovascular Disease
by van der Harst, Pim and de Windt, Leon J and Chambers, John C
Journal of the American College of Cardiology, ISSN 0735-1097, 08/2017, Volume 70, Issue 5, pp. 590 - 606
A plethora of environmental and behavioral factors interact, resulting in changes in gene expression and providing a basis for the development and progression... 
HAT | histones | EWAS | RNA | methylation | HDAC | CARDIAC & CARDIOVASCULAR SYSTEMS | DNA METHYLATION | BLOOD-LIPIDS | INHIBITION | LONG NONCODING RNA | CPT1A LOCUS | CARDIAC-HYPERTROPHY | BODY-MASS INDEX | HUMAN HEART | EPIGENOME-WIDE ASSOCIATION | HISTONE DEACETYLASE | Epigenomics - methods | Cardiovascular Diseases - genetics | Epigenesis, Genetic | Humans | Translational Medical Research | Epigenetic inheritance | Genes | Genomics | Development and progression | Cardiovascular diseases | Cardiology | Gene expression | Epidemiology | Methyltransferases | Histones | Nucleic acids | DNA binding proteins | Methylation | Heart failure | Heterogeneity | Atherosclerosis | DNA methylation | Epigenetics | Cell division | Cardiovascular disease | Heredity | Genomes | Deoxyribonucleic acid--DNA
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