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Journal Article
PloS one, ISSN 1932-6203, 08/2012, Volume 7, Issue 8, p. e44107
Background: Resveratrol is a natural polyphenolic compound that has cardioprotective, anticancer and anti-inflammatory properties. We investigated the capacity... 
SIGNALING PATHWAYS | INFLAMMATORY RESPONSES | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | LIPOPOLYSACCHARIDE | PROTEIN-KINASE PATHWAYS | INDUCED THP-1 CELLS | NF-KAPPA-B | TNF-ALPHA PRODUCTION | PHOSPHOINOSITIDE 3-KINASE | Dinoprostone - secretion | Sirtuin 1 - metabolism | Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Tumor Necrosis Factor-alpha - genetics | Stilbenes - chemistry | Interleukin-1beta - genetics | Stilbenes - pharmacology | RNA, Messenger - metabolism | Cyclooxygenase 2 - genetics | Inflammation Mediators - metabolism | Interleukin-1beta - biosynthesis | Phosphorylation - drug effects | Phosphatidylinositol 3-Kinase - metabolism | Cell Line | Cell Survival - drug effects | RNA, Messenger - genetics | Enzyme Activation - drug effects | Macrophages - enzymology | Gene Expression Regulation - drug effects | Nitric Oxide - secretion | Animals | MAP Kinase Signaling System - drug effects | Androstadienes - pharmacology | Nitric Oxide Synthase Type II - genetics | Signal Transduction - drug effects | Models, Biological | Cyclic AMP Response Element-Binding Protein - metabolism | Cyclooxygenase 2 - metabolism | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Mice | Tumor Necrosis Factor-alpha - biosynthesis | Mitogen-Activated Protein Kinases - metabolism | Nitric Oxide Synthase Type II - metabolism | Resveratrol | Physiological aspects | Inflammation | Genetic aspects | Research | Drug therapy | Lipopolysaccharides | Mitogen-activated protein kinases | Phosphorylation | Interleukin | Prostaglandin E2 | AKT protein | mRNA | Activation | Kinases | AMP-activated protein kinase | Macrophages | Inflammatory diseases | Cell adhesion & migration | Anticancer properties | Proteins | Signal transduction | Cell activation | Metabolites | Rodents | Cascades | Tumor necrosis factor-TNF | Inhibition | Enzyme-linked immunosorbent assay | Cytokines | Cyclic AMP | Bioavailability | Tumor necrosis factor-α | IL-1β | Metabolism | Cyclic AMP response element-binding protein | SIRT1 protein | Anti-inflammatory agents | Nitric-oxide synthase | 1-Phosphatidylinositol 3-kinase | Polymerase chain reaction | Signaling | Inhibitors | Nitric oxide | Cyclooxygenase-2 | Immunofluorescence
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 9, p. e73327
Journal Article
Journal Article
Trends in Immunology, ISSN 1471-4906, 2008, Volume 29, Issue 10, pp. 493 - 501
Mast cells play a central role in the initiation of inflammatory responses associated with asthma and other allergic disorders. Receptor-mediated mast cell... 
Allergy and Immunology | REGULATORY SUBUNIT | ACTIVATION | FC-EPSILON-RI | PHOSPHATIDYLINOSITOL 3-KINASE | IGE | C-BETA | KIT | CALCIUM MOBILIZATION | PROTEIN-TYROSINE KINASE | IMMUNOLOGY | INDUCED SECRETION | Forkhead Transcription Factors - immunology | Protein Kinases - metabolism | 1-Phosphatidylinositol 4-Kinase - immunology | Calcium - metabolism | Humans | Calcium - immunology | Proto-Oncogene Proteins c-kit - immunology | Proto-Oncogene Proteins c-kit - metabolism | 1-Phosphatidylinositol 4-Kinase - metabolism | Phosphoric Monoester Hydrolases - immunology | Protein Isoforms - metabolism | PTEN Phosphohydrolase - immunology | Forkhead Transcription Factors - metabolism | Receptors, IgE - immunology | Proto-Oncogene Proteins c-akt - metabolism | Protein-Serine-Threonine Kinases - metabolism | Cytokines - immunology | Mast Cells - immunology | Cytokines - metabolism | PTEN Phosphohydrolase - metabolism | Cell Degranulation - immunology | Mast Cells - enzymology | Animals | Proto-Oncogene Proteins c-akt - immunology | Protein Kinases - immunology | Receptors, IgE - metabolism | Protein-Serine-Threonine Kinases - immunology | TOR Serine-Threonine Kinases | Phosphoric Monoester Hydrolases - metabolism | Protein Isoforms - immunology | Conflicts of interest | Histamine | Cytokines | Kinases | Allergies | p110γ | Mast cells | FcεRI | PI3K | p110δ | Kit | Basic Medicine | Medicinska grundvetenskaper | PI3 Kinase | Immunologi inom det medicinska området | Immunology in the medical area | Cell signaling
Journal Article
The Journal of immunology (1950), ISSN 0022-1767, 11/2009, Volume 183, Issue 9, pp. 5673 - 5684
B-1 and marginal zone (MZ) B cells produce natural Abs, make Ab responses to microbial pathogens, and contribute to autoimmunity. Although the delta isoform of... 
ADHESION | HEART-DISEASE | ACTIVATION | PYK2 TYROSINE PHOSPHORYLATION | ATHEROSCLEROSIS | INJURY | OXIDATION-SPECIFIC EPITOPES | ISOFORM | MOLECULAR MIMICRY | IMMUNOLOGY | DEPENDENT ANTIGEN | Isoantibodies - biosynthesis | Spleen - immunology | Rats, Inbred Lew | Phosphatidylinositol 3-Kinases - metabolism | Spleen - drug effects | Autoantibodies - biosynthesis | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Antibodies, Bacterial - biosynthesis | Spleen - enzymology | Peritoneum - cytology | Peritoneum - immunology | B-Lymphocyte Subsets - immunology | Female | Quinazolines - administration & dosage | Homeostasis - drug effects | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | B-Lymphocyte Subsets - drug effects | Adenine - analogs & derivatives | Administration, Oral | Mice, Inbred C57BL | Cells, Cultured | Homeostasis - immunology | Rats | Spleen - cytology | B-Lymphocyte Subsets - enzymology | Adenine - administration & dosage | Gene Knock-In Techniques | Animals | B-Lymphocyte Subsets - microbiology | Phosphatidylinositol 3-Kinases - physiology | Mice | Peritoneum - metabolism | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Bacterial | cytology | Phosphorylation | Antibodies | Homeostasis | Oral | Isoantibodies | Quinazolines | B-Lymphocyte Subsets | administration & dosage | biosynthesis | Cultured | immunology | Spleen | Administration | 1-Phosphatidylinositol 3-Kinase | Autoantibodies | physiology | Adenine | microbiology | drug effects | antagonists & inhibitors | enzymology | Cells | Inbred C57BL | Inbred Lew | Patobiologi | Proto-Oncogene Proteins c-akt | Pathobiology | analogs & derivatives | metabolism | Peritoneum
Journal Article
Journal of Autoimmunity, ISSN 0896-8411, 2012, Volume 38, Issue 4, pp. 381 - 391
.... Since loss of Lyn confers hyper-activation of phosphoinositide 3-kinase (PI3K) signaling, we studied the effect of down-modulating PI3K in Lyn-deficient mice... 
Allergy and Immunology | Lyn tyrosine kinase | Autoimmune disease | Systemic lupus erythematosus | Phosphoinositide 3-kinase p110δ | Autoimmune disease therapy | B-CELL DEVELOPMENT | SYSTEMIC-LUPUS-ERYTHEMATOSUS | PHOSPHATIDYLINOSITOL 3-KINASE | TYROSINE KINASE | ALLERGIC AIRWAY INFLAMMATION | IMMUNOLOGY | NEGATIVE REGULATOR | MUTANT MICE | Phosphoinositide 3-kinase p110 delta | LYN(-/-) MICE | T-CELLS | LYN-DEFICIENT MICE | Kidney - pathology | Antibodies, Antinuclear - immunology | Hyperplasia | Autoimmune Diseases - mortality | Phosphatidylinositol 3-Kinases - metabolism | src-Family Kinases - deficiency | Autoimmune Diseases - genetics | Kidney - metabolism | Lymphocyte Activation - genetics | Lymphocyte Activation - immunology | T-Lymphocytes - metabolism | Myeloid Cells - immunology | Proto-Oncogene Proteins c-akt - metabolism | B-Lymphocytes - metabolism | Haploinsufficiency - genetics | Signal Transduction | Mice, Inbred C57BL | Autoimmune Diseases - immunology | Genotype | Receptors, Antigen, B-Cell - immunology | Mice, Knockout | Antibodies, Antinuclear - genetics | Phosphatidylinositol 3-Kinases - genetics | Animals | B-Lymphocytes - immunology | Alleles | Myeloid Cells - metabolism | T-Lymphocytes - immunology | Mice | Myeloid Cells - pathology | src-Family Kinases - genetics | Plasma Cells - cytology | Haploinsufficiency - immunology | Mitogen-Activated Protein Kinases - metabolism | Plasma Cells - immunology | Glomerulonephritis | Enzymes | Animal models | Immunoglobulins | Autoantibodies | Inflammation | Lymphocytes T | Kidney | phosphoinositides | 1-Phosphatidylinositol 3-kinase | Cell activation | haploinsufficiency | Lymphocytes B | Plasma cells | Lyn protein | Autoimmune diseases
Journal Article
Journal Article