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10018 (25) 25
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index medicus (23) 23
hematology (16) 16
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10014 (5) 5
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heart (5) 5
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10012 (4) 4
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iso 10018 (3) 3
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permeability (2) 2
platelet activation (2) 2
platelet activation - drug effects (2) 2
platelet aggregation inhibitors - pharmacology (2) 2
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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2018, Volume 38, Issue 7, pp. 1468 - 1478
OBJECTIVE—The Wnt/β-catenin signaling is an ancient and evolutionarily conserved pathway that regulates essential aspects of cell differentiation,... 
atherosclerosis | β-catenin | macrophage | low-density lipoprotein receptor | inflammation | 10018 | 10188 | Basic Sciences
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2018, Volume 38, Issue 7, pp. 1427 - 1439
Journal Article
Circulation, ISSN 0009-7322, 03/2017, Volume 135, Issue 12, pp. 1160 - 1173
BACKGROUND:Elabela/toddler (ELA) is a critical cardiac developmental peptide that acts through the G-protein–coupled apelin receptor, despite lack of sequence... 
Cardiopulmonary | Receptors, G-protein-coupled | Elabela/Toddler | Pulmonary hypertension | Apelin | CARDIAC & CARDIOVASCULAR SYSTEMS | ELABELA | apelin | pulmonary hypertension | receptors, G-protein-coupled | HEART | IMMUNOCYTOCHEMICAL LOCALIZATION | PATHWAY | ENDOTHELIAL-CELLS | IN-VIVO | DISEASE | PERIPHERAL VASCULAR DISEASE | CARDIAC CONTRACTILITY | LIGAND | cardiopulmonary | Peptide Hormones - pharmacology | Humans | Hypertension, Pulmonary - physiopathology | Intercellular Signaling Peptides and Proteins - chemistry | Endothelium, Vascular - drug effects | Male | Peptide Hormones - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Peptide Hormones - chemistry | Hypertension, Pulmonary - drug therapy | Binding Sites | Disease Models, Animal | Protein Structure, Tertiary | Amino Acid Sequence | Catheterization | Rats | Down-Regulation - drug effects | Rats, Sprague-Dawley | Molecular Dynamics Simulation | Intercellular Signaling Peptides and Proteins - agonists | Animals | Endothelium, Vascular - metabolism | Intercellular Signaling Peptides and Proteins - pharmacology | Peptide Hormones - therapeutic use | Heart Ventricles - metabolism | Intercellular Signaling Peptides and Proteins - therapeutic use | Heart Ventricles - drug effects | Care and treatment | Research | Toddlers | Gene expression | Health aspects | 10129 | 10018 | 10041 | 10012 | Toddler | Elabela | 10113 | Original s
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2017, Volume 37, Issue 11, pp. 2087 - 2087
OBJECTIVE—Atherosclerosis develops near branches and bends of arteries that are exposed to low shear stress (mechanical drag). These sites are characterized by... 
atherosclerosis | endothelial cells | glycolysis | MOUSE AORTAS | DEFICIENT MICE | CELL METABOLISM | DISTURBED FLOW | apolipoproteins E | hypoxia-inducible factor 1 | LAMINAR SHEAR-STRESS | PERIPHERAL VASCULAR DISEASE | HIF-1-ALPHA | HEMATOLOGY | NF-KAPPA-B | TRANSCRIPTIONAL REGULATION | HIF-ALPHA | Inflammation - pathology | Up-Regulation | Cell Proliferation | Apolipoproteins E - deficiency | Human Umbilical Vein Endothelial Cells - metabolism | Atherosclerosis - genetics | Humans | Stress, Mechanical | NF-kappa B - metabolism | Oxygen - metabolism | Inflammation - metabolism | Ubiquitination | Transfection | RNA Interference | Time Factors | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Proteolysis | Inflammation Mediators - metabolism | Female | Protein Stability | Disease Models, Animal | Endopeptidases - metabolism | Atherosclerosis - pathology | Genetic Predisposition to Disease | Endothelial Cells - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Cells, Cultured | Enzyme Induction | Plaque, Atherosclerotic | Regional Blood Flow | Atherosclerosis - metabolism | Mice, Knockout | Mechanotransduction, Cellular | Phenotype | Animals | Apolipoproteins E - genetics | Glycolysis | Inflammation - genetics | Human Umbilical Vein Endothelial Cells - pathology | Endothelial Cells - pathology | Sus scrofa | 10018 | 10054 | 10024 | 10188 | 10014 | Basic Sciences
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2018, Volume 38, Issue 7, pp. 1479 - 1492
OBJECTIVE—Atherosclerosis is a chronic multifactorial and inflammatory disease of large and medium arteries and the leading cause of cardiovascular diseases... 
atherosclerosis | endothelial cells | macrophages | inflammation | sphingolipids | INDUCED ACTIVATION | MEDIATED EXPRESSION | NECROSIS-FACTOR-ALPHA | CERAMIDE | LIPOPROTEIN RETENTION | CELL-DEATH | SYNTHASE 2 | NRF2 | NEUTRAL SPHINGOMYELINASE-2 | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | UP-REGULATION | Life Sciences | Human health and pathology | 10018 | 10030 | 10035 | 10012 | Basic Sciences
Journal Article
Circulation Research, ISSN 0009-7330, 01/2019, Volume 124, Issue 1, pp. 52 - 65
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2018, Volume 38, Issue 7, pp. 1576 - 1593
OBJECTIVE—Vascular remodeling is associated with complex molecular changes, including increased Notch2, which promotes quiescence in human smooth muscle cells.... 
mass spectrometry | carotid arteries | muscle, smooth | vascular remodeling | proteome | MATRIX-METALLOPROTEINASE | CAROTID-ARTERY | MUTATIONS LEAD | CELL MIGRATION | VASCULAR SMOOTH-MUSCLE | DUCTUS-ARTERIOSUS | SURGICAL INJURY | muscle | GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | GROWTH-FACTOR | HEMATOLOGY | NF-KAPPA-B | smooth | 10018 | 10050 | 10193 | 10054 | Basic Sciences | 10017
Journal Article
Circulation Research, ISSN 0009-7330, 08/2017, Volume 121, Issue 4, pp. 392 - 410
RATIONALE:Mitochondria are important cellular organelles and play essential roles in maintaining cell structure and function. Emerging evidence indicates that... 
acetylation | tumor necrosis factor-alpha | heart failure | molecular dynamics simulation | mitochondrial dynamics | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | HEART-FAILURE | REPERFUSION INJURY | KAPPA-B | LYSINE ACETYLATION | MESENCHYMAL STEM-CELLS | FACTOR-ALPHA | TUMOR-NECROSIS-FACTOR | IN-VIVO | CARDIAC-HYPERTROPHY | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | Animals, Newborn | Protein Structure, Tertiary | Gene Expression | Protein Structure, Secondary | Receptors, Tumor Necrosis Factor, Type II - chemistry | Humans | Mice, Inbred C57BL | Cells, Cultured | NF-kappa B - metabolism | NF-kappa B - chemistry | STAT3 Transcription Factor - chemistry | Aortic Diseases - metabolism | Mice, Knockout | Mitochondrial Dynamics - physiology | GTP Phosphohydrolases - chemistry | Aortic Diseases - genetics | Animals | GTP Phosphohydrolases - genetics | Receptors, Tumor Necrosis Factor, Type II - metabolism | HEK293 Cells | Myocytes, Cardiac - metabolism | Mice | GTP Phosphohydrolases - biosynthesis | STAT3 Transcription Factor - metabolism | Heart | Tumor necrosis factor receptors | Neonates | Transcription | Cytology | Event-related potentials | Myocytes | Atrophy | Mitochondria | Rodents | Aorta | Membrane potential | Acetylation | Heart diseases | NF-κB protein | Stat3 protein | Oxygen consumption | Cardiomyocytes | Inflammation | siRNA | Organelles | Optic atrophy | Lysine | RelA protein | Apoptosis | Structure-function relationships | 10018 | 10094 | Cellular Biology | 10014
Journal Article
Journal Article
Circulation Research, ISSN 0009-7330, 01/2018, Volume 122, Issue 2, pp. 231 - 245
Journal Article