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Journal Article
Journal Article
Phytotherapy Research, ISSN 0951-418X, 11/2014, Volume 28, Issue 11, pp. 1671 - 1675
...)‐induced diabetic rats. Our objective was to examine the effect of UMB on adipogenesis by investigating its stimulatory effect on lipid accumulation and mRNA expression of adipogenic transcription factors and adipocyte‐specific genes in 3 T3... 
adipogenic transcription factor | adipogenesis | 3 T3‐L1 adipocyte | umbelliferone | Adipogenic transcription factor | Umbelliferone | 3 T3-L1 adipocyte | Adipogenesis | ADIPOGENIC GENES | CHEMISTRY, MEDICINAL | OBESITY | ADIPONECTIN | INFLAMMATION | COUMARIN | FAT | PHARMACOLOGY & PHARMACY | DIFFERENTIATION | DERIVATIVES | 3T3-L1 adipocyte | ADIPOSE-TISSUE
Journal Article
NMR in Biomedicine, ISSN 0952-3480, 10/2016, Volume 29, Issue 10, pp. 1391 - 1402
Obesity is a worldwide epidemic, and associated pathologies, including type 2 diabetes and cardiovascular alterations, are increasingly escalating morbidity... 
3 T3‐L1 white adipocytes | bile acids | nuclear magnetic resonance | chenodeoxycholic acid | obesity | Obesity | Bile acids | Nuclear magnetic resonance | 3T3-L1 white adipocytes | Chenodeoxycholic acid | 3 T3-L1 white adipocytes | FXR | ACTIVATION | TRIGLYCERIDE LEVELS | ASSAY | QUANTITATION | ENERGY-EXPENDITURE | BIOPHYSICS | SPECTROSCOPY | LIVER | DIFFERENTIATION | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | BROWN | CYCLE | Cell Survival - drug effects | Chenodeoxycholic Acid - administration & dosage | 3T3-L1 Cells | Carbon-13 Magnetic Resonance Spectroscopy - methods | Obesity - metabolism | Dose-Response Relationship, Drug | Animals | Adipose Tissue, White - physiology | Bile Acids and Salts - administration & dosage | Obesity - prevention & control | Glucose - metabolism | Metabolic Networks and Pathways - physiology | Mice | Metabolic Networks and Pathways - drug effects | Adipose Tissue, White - drug effects | Cell Survival - physiology | Adipose tissues | Fatty acids | Analysis | Deoxycholic acid | Adipocytes | Metabolism | Rodents | Bile | Epidemics | Adipose tissue | Nuclear magnetic resonance--NMR | Clinical trials | Reversing | Nervous system | Shivering | Magnetic resonance spectroscopy | Alterations | Pathways | Restoration | Oxidation | Adipose tissue (brown) | Diabetes mellitus (non-insulin dependent) | Medical research | Diabetes mellitus | Insulin | Morbidity | Thermogenesis | Acids | Weight reduction | Glycolysis | In vivo methods and tests | Metabolic pathways
Journal Article
Endocrine journal, ISSN 0918-8959, 2014, Volume 61, Issue 12, pp. 1153 - 1162
Journal Article
Neuroscience Research, ISSN 0168-0102, 2011, Volume 69, Issue 1, pp. 8 - 16
.... Using co-cultures of dorsal root ganglion (DRG)-derived cells and 3T3-L1 adipocytes, we have previously shown that the presence of fat cells enhances neurite outgrowth and number of synapses... 
ERK1/2 | Dorsal root ganglia | PACAP | 3T3-L1 adipocytes | ApoD | DRG/3T3-L1 cell co-culture | Neurite outgrowth in vitro | CYCLASE-ACTIVATING POLYPEPTIDE | NERVOUS-SYSTEM | VASOACTIVE-INTESTINAL-PEPTIDE | KINASES 1 | NEURONAL DIFFERENTIATION | NEUROBLASTOMA-CELLS | NEUROSCIENCES | SENSORY INNERVATION | WHITE ADIPOSE-TISSUE | BIOLOGICAL FUNCTIONS | GENE-EXPRESSION | Up-Regulation | 3T3-L1 Cells - metabolism | Apolipoproteins D - metabolism | Coculture Techniques | Adipocytes - drug effects | Angiopoietin-1 - metabolism | Apolipoproteins E - metabolism | Synapses - metabolism | 3T3-L1 Cells - drug effects | Pituitary Adenylate Cyclase-Activating Polypeptide - pharmacology | Receptors, Pituitary Adenylate Cyclase-Activating Polypeptide, Type I - metabolism | Neurites - drug effects | Rats, Inbred WF | Synapses - drug effects | Receptors, Vasoactive Intestinal Peptide, Type II - antagonists & inhibitors | Receptors, Vasoactive Intestinal Peptide, Type II - metabolism | Rats | Neurites - metabolism | Adipokines | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Adipocytes - metabolism | Pituitary Adenylate Cyclase-Activating Polypeptide - metabolism | Mice | Ganglia, Spinal - drug effects | Neurofilament Proteins - metabolism | Ganglia, Spinal - metabolism | Receptors, Pituitary Adenylate Cyclase-Activating Polypeptide, Type I - antagonists & inhibitors | Fluorescence | Messenger RNA | Peptides | Apolipoproteins | Analysis
Journal Article
Metabolism, ISSN 0026-0495, 2014, Volume 63, Issue 6, pp. 735 - 745
Abstract New-onset diabetes has been observed in clinical trials and meta-analyses involving statin therapy. To explain this association, three major... 
Endocrinology & Metabolism | Ca2 + channels | HMG-CoA inhibitors | Diabetes mellitus | Adipocytes | GLUT4 | Lipophilicity | Cholesterol | Adiponectin | Isoprenoids | COA REDUCTASE INHIBITORS | CHOLESTEROL ACCUMULATION | DRUG-INTERACTIONS | INSULIN-SECRETION | BETA-CELL FUNCTION | MEVALONIC ACID | Ca2+ channels | ENDOCRINOLOGY & METABOLISM | PPAR-GAMMA | PRAVASTATIN | GLUCOSE-UPTAKE | CORONARY-ARTERY-DISEASE | Hydroxymethylglutaryl-CoA Reductase Inhibitors - administration & dosage | Calcium Channels - metabolism | Glucose Transporter Type 4 - metabolism | Humans | Leptin - metabolism | MicroRNAs - metabolism | Caveolins - metabolism | Adipocytes - drug effects | Insulin-Secreting Cells - metabolism | Hyperglycemia - chemically induced | Terpenes - antagonists & inhibitors | Mitochondrial Proteins - metabolism | Adiponectin - metabolism | Insulin Secretion | Calcium Channels - drug effects | Hyperinsulinism - metabolism | Ubiquinone - analogs & derivatives | Insulin Resistance | Diabetes Mellitus - metabolism | Ubiquinone - antagonists & inhibitors | Hyperglycemia - metabolism | Hyperinsulinism - chemically induced | Insulin - metabolism | Animals | Dolichol - antagonists & inhibitors | Hydroxymethylglutaryl-CoA Reductase Inhibitors - adverse effects | Insulin-Secreting Cells - drug effects | Ion Channels - metabolism | Cell Differentiation - drug effects | Diabetes Mellitus - chemically induced | Uncoupling Protein 3 | Phosphates | Enzymes | Pancreatic beta cells | Development and progression | Glucose | Dextrose | Cardiovascular agents | Glucose metabolism | Analysis | Leptin | Cellular signal transduction | Diabetes | Statins | Protein binding | Diabetes therapy
Journal Article
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, p. e76199
Aims: Ephrin-B1 (EfnB1) was selected among genes of unknown function in adipocytes or adipose tissue and subjected to thorough analysis to understand its role in the development of obesity... 
METABOLIC SYNDROME | OBESITY | IMPACT | INSULIN-RESISTANCE | TYROSINE KINASE | MULTIDISCIPLINARY SCIENCES | TISSUE | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | ENDOPLASMIC-RETICULUM STRESS | EPH | EXPRESSION | Male | Monocytes - metabolism | RNA, Messenger - metabolism | Obesity - genetics | Gene Knockdown Techniques | Panniculitis - genetics | Adipose Tissue - metabolism | Inflammation - metabolism | Cell Line | Cell Adhesion - genetics | Ephrin-B1 - genetics | RNA, Messenger - genetics | Adipose Tissue - pathology | Gene Expression Regulation | Ephrin-B1 - metabolism | Obesity - metabolism | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Models, Biological | Adipocytes - metabolism | Inflammation - genetics | Mice | Enzyme Activation | Panniculitis - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Adipose tissues | Obesity | RNA | Tumor necrosis factor | Adenoviruses | Genes | Cell culture | Adipose tissue | Inflammatory response | mRNA | Adipocytes | Kinases | Metabolic syndrome | Blood | Proteins | Reduction | Cell activation | Rodents | Animal tissues | Atherosclerosis | Tumor necrosis factor-TNF | Departments | Cytokines | Extracellular signal-regulated kinase | Inflammation | Tumor necrosis factor-α | Gene expression | Adhesion | Medicine | Monocyte chemoattractant protein | Insulin resistance | Diabetes | Laboratory animals | Chemokines | Monocyte chemoattractant protein 1
Journal Article