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American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 10/2016, Volume 311, Issue 4, pp. C537 - C543
Journal Article
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2014, Volume 68, Issue 100, pp. 66 - 77
Abstract Rett syndrome (RTT) is a rare neurodevelopmental disorder affecting almost exclusively females, caused in the overwhelming majority of the cases by... 
Neurology | Lipid peroxidation | Oxidative stress | Brain damage | Neurodevelopmental disorder | Murine models | Rett syndrome | F-4-NEUROPROSTANES | IRON | F-2-DIHOMO-ISOPROSTANES | NEUROSCIENCES | DEFICIENCY | CHOLESTEROL-METABOLISM | MECP2 | MOUSE MODEL | DISEASE | DYSFUNCTION | STRESS | Arachidonic Acid - metabolism | Rett Syndrome - complications | Gas Chromatography-Mass Spectrometry | Mice, Inbred C57BL | Neuroprostanes - metabolism | Oxidative Stress - physiology | Male | Mice, Transgenic | Aldehydes - metabolism | Mutation - genetics | Methyl-CpG-Binding Protein 2 - genetics | Animals | Isoprostanes - metabolism | Analysis of Variance | Nestin - genetics | Brain Injuries - blood | Female | Mice | Brain Injuries - etiology | Rett Syndrome - blood | Rett Syndrome - genetics | Brain Injuries - pathology | Disease Models, Animal | Docosahexaenoic Acids - metabolism | Models | Analysis | Protein binding | Organic chemistry | Chemical Sciences | OS, oxidative stress | Mecp2 | y, hemizygous null mice | NPBI, non-protein-bound iron | Mecp2, methyl-CpG-binding protein 2 — mouse protein | CRE, Cre-Recombinase | AdA, adrenic acid | AUs, arbitrary units | ARA, arachidonic acid | 4-HNE PAs, 4-HNE protein adducts | PSV, Preserved Speech Variant | Mecp2, methyl-CpG-binding protein 2 — mouse gene | wt, wild type | wt-Cre, wild type expressing Cre recombinase | MECP2, methyl-CpG-binding protein 2 — human gene | y, symptomatic Mecp2 308-mutated hemizygous males | ROS, reactive oxygen species | F2-IsoPs, F2-isoprostanes | MeCP2, methyl-CpG-binding protein 2 — human protein | y, Lox | F2-dihomo-IsoPs, F2-dihomo-isoprostanes | 4-HNE PAs, 4-hydroxy-2-nonenal protein adducts | Mecp2 stop | y NestinCre, rescued Lox | ASDs, autism spectrum disorders | F4-NeuroPs, F4-neuroprostanes | Mecp2 308 | stop pre-symptomatic hemizygous mice | x, symptomatic Mecp2 308-mutated females | DHA, docosahexaenoic acid | PUFAs, polyunsaturated fatty acids | BDNF, brain-derived neurotrophic factor | IsoPs, isoprostanes | RTT, Rett syndrome | stop mice (Mecp2 reactivated in the nervous tissue) | 4-HNE, 4-hydroxy-2-nonenal
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 10/2017, Volume 111, pp. 316 - 327
Journal Article
Current Drug Targets, ISSN 1389-4501, 01/2014, Volume 15, Issue 10, pp. 948 - 955
Journal Article
Journal Article
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 04/2011, Volume 121, Issue 4, pp. 487 - 498
Huntington's disease (HD) is an autosomal dominant neurodegenerative disorder. Oxidative damage has been associated with pathological neuronal loss in HD. The... 
Lipid peroxidation | Neuronal survival | Mitochondria | Huntington's disease | 4-Hydroxy-2-nonenal (4-HNE) | OXIDATIVE STRESS | REPEAT | ALZHEIMERS-DISEASE | NORDIHYDROGUAIARETIC ACID | PATHOLOGY | NEUROSCIENCES | NEURODEGENERATIVE DISEASES | MOUSE MODELS | CLINICAL NEUROLOGY | CELL-DEATH | FIBRILS IN-VITRO | CORTICAL-NEURONS | TRANSGENIC MICE | Neurons - pathology | Age Factors | Embryo, Mammalian | Microscopy, Electron, Transmission - methods | Humans | Huntington Disease - pathology | Imaging, Three-Dimensional - methods | Male | Synapses - pathology | Aldehydes - metabolism | Membrane Potential, Mitochondrial - drug effects | Cerebral Cortex - cytology | Mitochondria - ultrastructure | Dose-Response Relationship, Drug | Tetrazolium Salts | Neurons - ultrastructure | Neostriatum - drug effects | Adenosine Triphosphate - metabolism | Lipid Peroxidation - drug effects | Thiazoles | Membrane Potential, Mitochondrial - genetics | Neurons - metabolism | Indoles | Huntington Disease - drug therapy | Nuclear Proteins - genetics | Disease Models, Animal | Masoprocol - therapeutic use | Neostriatum - pathology | Synapses - drug effects | Glutamic Acid - pharmacology | Cells, Cultured | Oxidative Stress - genetics | Lipoxygenase Inhibitors - therapeutic use | Mice, Transgenic | Synapses - ultrastructure | In Situ Nick-End Labeling - methods | Mitochondria - drug effects | Mitochondria - pathology | Nerve Tissue Proteins - genetics | Lipid Peroxidation - physiology | Huntingtin Protein | Animals | Analysis of Variance | Huntington Disease - genetics | Trinucleotide Repeat Expansion - genetics | Mice | Oxidative Stress - drug effects | Antioxidants | Medical colleges | Nervous system diseases | Huntington's chorea | Cell death | Neurons | Mitochondrial DNA
Journal Article
Acta Neuropathologica, ISSN 0001-6322, 4/2011, Volume 121, Issue 4, pp. 487 - 498
Huntington’s disease (HD) is an autosomal dominant neurodegenerative disorder. Oxidative damage has been associated with pathological neuronal loss in HD. The... 
Pathology | Neurosciences | Mitochondria | Medicine & Public Health | 4-Hydroxy-2-nonenal (4-HNE) | Lipid peroxidation | Neuronal survival | Huntington’s disease
Journal Article