X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
animals (480) 480
humans (470) 470
index medicus (382) 382
5'-nucleotidase - metabolism (306) 306
5'-nucleotidase (286) 286
mice (262) 262
male (233) 233
immunology (208) 208
female (207) 207
adenosine (194) 194
5'-nucleotidase - genetics (143) 143
5'-nucleotidase - immunology (130) 130
cell biology (121) 121
mice, inbred c57bl (111) 111
expression (110) 110
adenosine - metabolism (108) 108
inflammation (104) 104
adult (101) 101
cd73 (97) 97
cells, cultured (97) 97
rats (96) 96
nucleotidases - metabolism (86) 86
research article (85) 85
enzymes (83) 83
middle aged (83) 83
mice, knockout (79) 79
oncology (78) 78
lymphocytes (70) 70
nucleotidases (70) 70
antigens, cd - metabolism (69) 69
apyrase - metabolism (67) 67
gene expression (67) 67
cd73 antigen (64) 64
proteins (64) 64
aged (63) 63
flow cytometry (61) 61
rodents (61) 61
t-lymphocytes - immunology (61) 61
biochemistry & molecular biology (60) 60
research (60) 60
article (59) 59
nucleotidase (57) 57
cancer (56) 56
activation (54) 54
medicine (53) 53
t cells (52) 52
analysis (51) 51
multidisciplinary sciences (50) 50
adenosine triphosphate - metabolism (49) 49
physiological aspects (49) 49
abridged index medicus (48) 48
lymphocytes - enzymology (48) 48
t-cells (47) 47
t-lymphocytes, regulatory - immunology (47) 47
cells (46) 46
5'-nucleotidase - analysis (45) 45
5'-nucleotidase - biosynthesis (45) 45
atp (45) 45
metabolism (45) 45
science (45) 45
cd39 (44) 44
immunohistochemistry (44) 44
signal transduction (44) 44
disease models, animal (43) 43
hematology (43) 43
adenosine deaminase - metabolism (42) 42
cell proliferation (42) 42
5'-nucleotidase - physiology (41) 41
cell line, tumor (41) 41
lymphocyte activation (41) 41
regulatory t-cells (41) 41
5′-nucleotidase (40) 40
gpi-linked proteins - metabolism (40) 40
5'-nucleotidase - blood (39) 39
health aspects (39) 39
life sciences (39) 39
medicine, research & experimental (39) 39
mice, inbred balb c (39) 39
neurosciences (39) 39
receptors (39) 39
t-lymphocytes - enzymology (39) 39
5'-nucleotidase - antagonists & inhibitors (38) 38
cytosolic 5'-nucleotidase 1a (38) 38
dendritic cells (38) 38
immunity (38) 38
5'-nucleotidase - deficiency (37) 37
biology (37) 37
nucleotides (37) 37
adenosine monophosphate - metabolism (36) 36
cell line (36) 36
phenotype (36) 36
ecto-5'-nucleotidase (35) 35
ecto-5'-nucleotidase cd73 (35) 35
tumors (35) 35
antigens, cd - immunology (34) 34
biochemistry (34) 34
cell differentiation (34) 34
immune system (34) 34
cell membrane - enzymology (33) 33
differentiation (33) 33
more...
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (808) 808
Russian (30) 30
Japanese (6) 6
Chinese (3) 3
French (2) 2
Polish (2) 2
Spanish (2) 2
Ukrainian (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Immunity, ISSN 1074-7613, 03/2013, Volume 38, Issue 3, pp. 555 - 569
Monocyte-derived macrophages are essential for recovery after spinal cord injury, but their homing mechanism is poorly understood. Here, we show that although... 
ECTO-5'-NUCLEOTIDASE CD73 | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | DENDRITIC CELLS | MICROGLIA | REGENERATION | MONOCYTES | CNS | CENTRAL-NERVOUS-SYSTEM | IMMUNOLOGY | DIVERGENT | EXPRESSION | 5'-Nucleotidase - antagonists & inhibitors | 5'-Nucleotidase - genetics | Leukocyte Common Antigens - metabolism | Meninges - immunology | Spinal Cord - metabolism | Choroid Plexus - metabolism | Choroid Plexus - immunology | Monocytes - metabolism | Green Fluorescent Proteins - genetics | Monocytes - immunology | Spinal Cord Injuries - genetics | Cell Movement - genetics | Meninges - metabolism | Cell Movement - immunology | Leukocyte Common Antigens - immunology | Flow Cytometry | 5'-Nucleotidase - immunology | Gene Expression - immunology | Antigens, Ly - metabolism | Vascular Cell Adhesion Molecule-1 - genetics | Receptors, Chemokine - genetics | Macrophages - immunology | Green Fluorescent Proteins - metabolism | Vascular Cell Adhesion Molecule-1 - immunology | Blood-Brain Barrier - immunology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Integrin alpha4beta1 - genetics | Receptors, Chemokine - immunology | Reverse Transcriptase Polymerase Chain Reaction | Adenosine Diphosphate - pharmacology | Blood-Brain Barrier - metabolism | Mice, Knockout | Integrin alpha4beta1 - immunology | Monocytes - drug effects | Spinal Cord - immunology | Microscopy, Confocal | Macrophages - metabolism | Animals | Antigens, Ly - immunology | Macrophages - drug effects | Spinal Cord Injuries - immunology | Mice | Adenosine Diphosphate - analogs & derivatives | CX3C Chemokine Receptor 1 | Spinal Cord Injuries - cerebrospinal fluid | Macrophages | Enzymes | Spinal cord injuries | Statistical analysis | Cytokines | Rodents | Recruitment
Journal Article
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 04/2016, Volume 75, Issue 4, pp. 696 - 701
Journal Article
Journal of Immunology, ISSN 0022-1767, 03/2015, Volume 194, Issue 6, pp. 2487 - 2492
Journal Article
Journal Article
CANCER RESEARCH, ISSN 0008-5472, 07/2018, Volume 78, Issue 13, pp. 3604 - 3618
The production of CD73-derived a denosine (Ado) by Tregs has been proposed as a resistance mechanism to anti-PD-1 therapy in murine tumor models. We reported... 
BREAST-CANCER | CD73 | T(H)17 CELLS | METASTASIS | HUMAN PERIPHERAL-BLOOD | ONCOLOGY | TNF-ALPHA | SUPPRESSION | EXPRESSION | PROMOTES | TH17 CELLS | T-Lymphocytes, Regulatory - metabolism | Breast Neoplasms - immunology | Humans | Ovarian Neoplasms - pathology | T-Lymphocytes, Regulatory - immunology | T-Lymphocytes, Helper-Inducer - immunology | Female | Lymphocytes, Tumor-Infiltrating - metabolism | Costimulatory and Inhibitory T-Cell Receptors - antagonists & inhibitors | Ovarian Neoplasms - drug therapy | Tumor Escape - immunology | T-Lymphocytes, Helper-Inducer - metabolism | Apyrase - metabolism | Drug Resistance, Neoplasm - immunology | Breast Neoplasms - drug therapy | GPI-Linked Proteins - metabolism | Antineoplastic Agents, Immunological - pharmacology | Interleukin-17 - metabolism | Antineoplastic Agents, Immunological - therapeutic use | Breast Neoplasms - pathology | Costimulatory and Inhibitory T-Cell Receptors - metabolism | Adenosine - metabolism | 5'-Nucleotidase - metabolism | Lymphocytes, Tumor-Infiltrating - immunology | Ovarian Neoplasms - immunology | Therapy | Animal models | PD-1 protein | Effector cells | Interleukin 22 | Lymphocytes T | Immunity | MDR1 protein | Lymphocytes | Interleukin 1 | CD73 antigen | Autocrine signalling | Biodegradation | Adenosine | AMP | Granulocyte-macrophage colony-stimulating factor | Breast cancer | CXCR3 protein | CCR6 protein | CD4 antigen | Immune checkpoint | γ-Interferon | Effectors | Breast | Ectonucleotidase | ATP | Adenosine triphosphatase | Cancer | Tumors | Life Sciences | Human health and pathology
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2010, Volume 285, Issue 36, pp. 27571 - 27580
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 641 - 12
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 05/2017, Volume 101, Issue 5, pp. 1263 - 1271
Down‐regulation of CD73 on B cells of viremic HIV patients correlates with B cell activation and disease progression. Recently, alterations of the T cell... 
CD39 | ADO | 5′‐ectonucleotidase | Ig class switch | elite controller | Elite controller | 5’-ectonucleotidase | CLASS SWITCH RECOMBINATION | 5 '-ectonucleotidase | IMMUNOLOGY | HUMAN-IMMUNODEFICIENCY-VIRUS | CELL BIOLOGY | ADENOSINE GENERATION | IN-VITRO | IMMUNE ACTIVATION | INFECTION | HEMATOLOGY | EXPRESSION | T-CELLS | GP120 IMPAIRS | LYMPHOCYTES | 5'-Nucleotidase - genetics | Antigens, CD - immunology | Cell Proliferation | Lymph Nodes - pathology | Humans | Middle Aged | Receptors, Complement 3d - immunology | Male | Antigens, CD - genetics | Case-Control Studies | HIV Infections - immunology | Ki-67 Antigen - immunology | HIV Infections - pathology | Receptors, Complement 3d - genetics | 5'-Nucleotidase - immunology | Aged, 80 and over | B-Lymphocytes - virology | Adult | Anti-HIV Agents - therapeutic use | Female | Lymph Nodes - drug effects | Cell Differentiation | B-Lymphocytes - pathology | Tumor Necrosis Factor Receptor Superfamily, Member 7 - immunology | GPI-Linked Proteins - immunology | Gene Expression Regulation - immunology | Lymphocyte Activation | HIV Infections - virology | Apyrase - immunology | CD4 Lymphocyte Count | Lymph Nodes - virology | Lymph Nodes - immunology | Tumor Necrosis Factor Receptor Superfamily, Member 7 - genetics | Viremia - drug therapy | Disease Progression | B-Lymphocytes - drug effects | B-Lymphocytes - immunology | Ki-67 Antigen - genetics | Viremia - pathology | Viremia - virology | Apyrase - genetics | HIV Infections - drug therapy | Immunologic Memory | Viremia - immunology | Aged | Programmed Cell Death 1 Receptor - immunology | Programmed Cell Death 1 Receptor - genetics | GPI-Linked Proteins - genetics | CD27 antigen | Lymphocyte receptors | Flow cytometry | Correlation | Memory cells | Immunological memory | Lymphocytes T | T cell receptors | Activation | Patients | Antiretroviral therapy | Lymph nodes | CD4 antigen | Cytometry | Control | Cell activation | Lymphocytes B | Antiretroviral agents | Human immunodeficiency virus--HIV | Exhaustion | CD73 antigen | In vivo methods and tests | Apoptosis
Journal Article
The Journal of Immunology, ISSN 0022-1767, 05/2008, Volume 180, Issue 9, pp. 6288 - 6296
After an inflammatory stimulus, lymphocyte migration into draining lymph nodes increases dramatically to facilitate the encounter of naive T cells with... 
ECTO-5'-NUCLEOTIDASE CD73 | IMMUNE-RESPONSE | L-SELECTIN | HUMAN DENDRITIC CELLS | VASCULAR ADHESION | BARRIER FUNCTION | RECEPTOR | DEFICIENT MICE | IMMUNOLOGY | HIGH ENDOTHELIAL VENULES | T-CELLS | Tumor Necrosis Factor-alpha - metabolism | 5'-Nucleotidase - genetics | Adenosine Monophosphate - genetics | T-Lymphocytes - enzymology | Venules - immunology | Dendritic Cells - immunology | Tumor Necrosis Factor-alpha - genetics | Venules - enzymology | Endothelium, Vascular - enzymology | Cell Movement - genetics | Adenosine - genetics | L-Selectin - metabolism | Cell Movement - immunology | L-Selectin - immunology | Receptor, Adenosine A2B - immunology | 5'-Nucleotidase - immunology | Tumor Necrosis Factor-alpha - immunology | Endothelium, Vascular - immunology | Adenosine - immunology | Adenosine Monophosphate - metabolism | Up-Regulation - genetics | Inflammation - immunology | Adenosine Monophosphate - immunology | Lymph Nodes - immunology | Receptor, Adenosine A2B - metabolism | Adenosine A2 Receptor Agonists | Mice, Knockout | Up-Regulation - drug effects | Cell Movement - drug effects | Animals | Up-Regulation - immunology | Dendritic Cells - enzymology | Aminopyridines - pharmacology | Lipopolysaccharides - pharmacology | Adenosine - metabolism | Inflammation - genetics | T-Lymphocytes - immunology | Mice | 5'-Nucleotidase - metabolism | Inflammation - enzymology
Journal Article
Blood, ISSN 0006-4971, 02/2012, Volume 119, Issue 6, pp. 1428 - 1439
Adenosine acts as anti-inflammatory mediator on the immune system and has been described in regulatory T cell (Treg)-mediated suppression. In the absence of... 
DEAMINASE DEFICIENCY | SEVERE COMBINED IMMUNODEFICIENCY | IMMUNE SUPPRESSION | TOLERANCE | REGULATORY T-CELLS | OMENN SYNDROME | GENE-THERAPY | GENERATION | HEMATOLOGY | EXPRESSION | TRANSPLANTATION | Forkhead Transcription Factors - immunology | Immunohistochemistry | Antigens, CD - immunology | T-Lymphocytes, Regulatory - metabolism | Agammaglobulinemia - immunology | Agammaglobulinemia - genetics | Adenosine Deaminase - metabolism | Humans | Child, Preschool | Infant | Male | Polyethylene Glycols - chemistry | Hypothyroidism - immunology | Antigens, CD - metabolism | T-Lymphocytes, Regulatory - immunology | Forkhead Transcription Factors - metabolism | 5'-Nucleotidase - immunology | Adenosine Deaminase - therapeutic use | Adult | Female | Hypothyroidism - enzymology | Hypothyroidism - genetics | Adenosine Deaminase - deficiency | Child | Adenosine - immunology | Adenosine Deaminase - genetics | Agammaglobulinemia - therapy | Apyrase - metabolism | Apyrase - immunology | Severe Combined Immunodeficiency - therapy | Severe Combined Immunodeficiency - immunology | Mice, Knockout | Severe Combined Immunodeficiency - genetics | Autoantibodies - immunology | Animals | Adolescent | Adenosine - metabolism | Mice | Hematopoietic Stem Cell Transplantation - methods | 5'-Nucleotidase - metabolism | Adenosine Deaminase - immunology | Genetic Therapy - methods | Immunobiology
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2016, Volume 11, Issue 2, pp. e0150078 - e0150078
γδ T cells can either enhance or inhibit an adaptive immune response, but the mechanisms involved are not fully understood. Given that CD73 is the main enzyme... 
Eye Proteins - immunology | T-Lymphocyte Subsets - immunology | 5'-Nucleotidase - genetics | Uveitis - enzymology | Peptide Fragments - toxicity | Dendritic Cells - immunology | 5'-Nucleotidase - deficiency | Th1 Cells - immunology | T-Lymphocytes, Regulatory - immunology | Peptide Fragments - immunology | Female | Retinol-Binding Proteins - toxicity | Interferon-gamma - deficiency | 5'-Nucleotidase - biosynthesis | Interleukin-17 - blood | Adenosine Monophosphate - metabolism | Gene Expression Regulation - immunology | Lymphocyte Activation | Nervous System Autoimmune Disease, Experimental - enzymology | Mice, Inbred C57BL | Nervous System Autoimmune Disease, Experimental - immunology | Cells, Cultured | Eye Proteins - toxicity | Receptors, Antigen, T-Cell, gamma-delta - deficiency | Mice, Knockout | Animals | Receptors, Antigen, T-Cell, gamma-delta - analysis | T-Lymphocyte Subsets - enzymology | Uveitis - immunology | 5'-Nucleotidase - physiology | Adenosine - metabolism | Th17 Cells - immunology | Mice | Interferon-gamma - blood | T-Lymphocytes, Regulatory - enzymology | Retinol-Binding Proteins - immunology | Adenosine | Biomedical research | Immune response | AMP | Laboratories | Helper cells | Lymphocytes T | Inflammation | T cell receptors | Metabolism | Experimental autoimmune uveitis | Medicine | Immunosuppression | Metabolites | Lymphocytes | Rodents | Uveitis | Hypoxia | CD73 antigen | Autoimmune diseases | Experimental autoimmune uveoretinitis | Tumors | Immune system | Index Medicus
Journal Article