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1. Full Text Animal Models of Atherosclerosis
by Getz, Godfrey S and Reardon, Catherine A
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 05/2012, Volume 32, Issue 5, pp. 1104 - 1115
Atherosclerosis is a chronic inflammatory disorder that is the underlying cause of most cardiovascular disease. Both cells of the vessel wall and cells of the... 
Genetically altered mice | Atherosclerosis | atherosclerosis | FAMILIAL HYPERCHOLESTEROLEMIA | ACCELERATED ATHEROSCLEROSIS | LESION FORMATION | FOAM CELLS | CORONARY-ARTERIES | genetically altered mice | APOLIPOPROTEIN-E | MOUSE MODEL | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | E-DEFICIENT MICE | DENSITY-LIPOPROTEIN RECEPTOR | Animals | Atherosclerosis - immunology | Atherosclerosis - physiopathology | Lipoproteins - metabolism | Lipid Metabolism | Hemodynamics | Immunity, Innate | Atherosclerosis - metabolism | Disease Models, Animal
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2. Full Text Animal models of atherosclerosis
by Emini Veseli, Besa and Perrotta, Paola and De Meyer, Gregory R.A and De Meyer, Guido R.Y and Roth, Lynn and Van der Donckt, Carole and Martinet, Wim
European Journal of Pharmacology, ISSN 0014-2999, 12/2017, Volume 816, pp. 3 - 13
An ideal animal model of atherosclerosis resembles human anatomy and pathophysiology and has the potential to be used in medical and pharmaceutical research to... 
Plaque rupture | PCSK9 | Animal models | ApoE | Atherosclerosis | LDL receptor | MYOCARDIAL-INFARCTION | ACCELERATED ATHEROSCLEROSIS | KNOCKOUT MICE | MOUSE MODELS | SHEAR-STRESS | PHARMACOLOGY & PHARMACY | APOLIPOPROTEIN-E-DEFICIENT | GENE-TRANSFER | DENSITY-LIPOPROTEIN RECEPTOR | TRANSGENIC MICE | Atherosclerosis - pathology | Animals | Plaque, Atherosclerotic - genetics | Plaque, Atherosclerotic - metabolism | Atherosclerosis - genetics | Plaque, Atherosclerotic - pathology | Atherosclerosis - metabolism | Disease Models, Animal | Human anatomy | Genetic engineering | Apolipoproteins | Low density lipoproteins | Analysis
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3. Full Text Macrophages in the Pathogenesis of Atherosclerosis
by Moore, Kathryn J and Tabas, Ira
Cell, ISSN 0092-8674, 2011, Volume 145, Issue 3, pp. 341 - 355
In atherosclerosis, the accumulation of apolipoprotein B-lipoproteins in the matrix beneath the endothelial cell layer of blood vessels leads to the... 
DENDRITIC CELLS | CHOLESTEROL | FOAM CELL-FORMATION | PROMOTES COLLAGEN ACCUMULATION | ACCELERATED ATHEROSCLEROSIS | IMMUNE-RESPONSES | BIOCHEMISTRY & MOLECULAR BIOLOGY | APOPTOTIC CELLS | LOW-DENSITY-LIPOPROTEIN | MICE | ENDOPLASMIC-RETICULUM STRESS | CELL BIOLOGY | Animals | Atherosclerosis - immunology | Macrophages - pathology | Signal Transduction | Dendritic Cells - immunology | Humans | Monocytes - pathology | Atherosclerosis - metabolism | Macrophages - immunology | Dendritic cells | Macrophages | Atherosclerosis | Blood lipids
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4. Full Text Lupus Atherosclerosis Prevention Study (LAPS)
by Petri, M A and Kiani, A N and Post, W and Christopher-Stine, L and Magder, L S
Annals of the Rheumatic Diseases, ISSN 0003-4967, 05/2011, Volume 70, Issue 5, pp. 760 - 765
Background Cardiovascular disease is one of the major causes of death in systemic lupus erythematosus (SLE). A study was undertaken to investigate whether... 
C-REACTIVE PROTEIN | INTIMA-MEDIA THICKNESS | CONTROLLED-TRIAL | RISK-FACTORS | MULTIPLE-SCLEROSIS | STATIN THERAPY | ACCELERATED ATHEROSCLEROSIS | CARDIOVASCULAR-DISEASE | RHEUMATOLOGY | LIPID-LOWERING THERAPY | CORONARY-ARTERY-DISEASE | Carotid Artery Diseases - prevention & control | Lupus Erythematosus, Systemic - complications | Heptanoic Acids - therapeutic use | Humans | Middle Aged | Male | Tomography, X-Ray Computed | Heptanoic Acids - adverse effects | Young Adult | Atherosclerosis - etiology | Carotid Artery Diseases - diagnostic imaging | Ultrasonography | Pyrroles - adverse effects | Adult | Female | Pyrroles - therapeutic use | Severity of Illness Index | Double-Blind Method | Coronary Artery Disease - prevention & control | Treatment Outcome | Atorvastatin Calcium | Coronary Artery Disease - diagnostic imaging | Hydroxymethylglutaryl-CoA Reductase Inhibitors - adverse effects | Hydroxymethylglutaryl-CoA Reductase Inhibitors - therapeutic use | Adolescent | Aged | Atherosclerosis - prevention & control | Care and treatment | Systemic lupus erythematosus | Research | Patient outcomes | Atherosclerosis | Lupus | Heart attacks | Health risk assessment | Low density lipoprotein | Statins | Veins & arteries
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5. Full Text Excess atherosclerosis in systemic lupus erythematosus, - A matter of renal involvement: Case control study of 281 SLE patients and 281 individually matched population controls
by Gustafsson, Johanna T and Herlitz Lindberg, Marie and Gunnarsson, Iva and Pettersson, Susanne and Elvin, Kerstin and Öhrvik, John and Larsson, Anders and Jensen-Urstad, Kerstin and Svenungsson, Elisabet and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Biokemisk struktur och funktion and Uppsala universitet and Institutionen för medicinska vetenskaper
PLoS ONE, ISSN 1932-6203, 04/2017, Volume 12, Issue 4, p. e0174572
Background Systemic lupus erythematosus (SLE), is a heterogeneous disease which predominantly affects young females (90%). SLE is associated with a shorter... 
INTIMA-MEDIA THICKNESS | RHEUMATOID-ARTHRITIS | WOMEN | ACCELERATED ATHEROSCLEROSIS | PREDICTORS | MULTIDISCIPLINARY SCIENCES | DISEASE | NEPHRITIS | ANTIPHOSPHOLIPID SYNDROME | CARDIOVASCULAR RISK-FACTORS | PROSPECTIVE COHORT | Cardiovascular Diseases - etiology | Carotid Stenosis - etiology | Lupus Erythematosus, Systemic - complications | Antibodies, Antiphospholipid - blood | Carotid Intima-Media Thickness | Humans | Middle Aged | Risk Factors | Male | Case-Control Studies | Plaque, Atherosclerotic - etiology | Atherosclerosis - etiology | Lupus Erythematosus, Systemic - immunology | Lupus Erythematosus, Systemic - classification | Adult | Female | Lupus Nephritis - complications | Care and treatment | Systemic lupus erythematosus | Patient outcomes | Atherosclerosis | Dosage and administration | Antiphospholipid antibodies | Risk factors | Heart | Nephritis | Sjogren's syndrome | Antibodies | Phospholipids | Epidemiology | Carotid arteries | Subgroups | Arteries | Immunology | Carotid artery | Age | Lupus | Antigens | Fasting | Life expectancy | Mortality | Diabetes mellitus | Age factors | Disease control | Patients | Studies | Chronic conditions | Cardiovascular diseases | Females | Autoimmune diseases | Health risk assessment | Rheumatology and Autoimmunity | Clinical Medicine | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Reumatologi och inflammation
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6. Full Text RAGE biology, atherosclerosis and diabetes
by Barlovic, Drazenka Pongrac and Soro-Paavonen, Aino and Jandeleit-Dahm, Karin A. M
Clinical Science, ISSN 0143-5221, 07/2011, Volume 121, Issue 2, pp. 43 - 55
Diabetes is characterized by accelerated atherosclerosis with widely distributed vascular lesions. An important mechanism by which hyperglycaemia contributes... 
Receptor for advanced glycation end-products (RAGE) | Vascular injury | Diabetes | Advanced glycation end-product (AGE) | Atherosclerosis | atherosclerosis | MEDICINE, RESEARCH & EXPERIMENTAL | receptor for advanced glycation end-products (RAGE) | ACCELERATED ATHEROSCLEROSIS | LOW-DENSITY-LIPOPROTEIN | FACTOR-KAPPA-B | advanced glycation end-product (AGE) | VASCULAR-DISEASE | RECEPTOR-DEFICIENT MICE | vascular injury | ENDOTHELIAL PROGENITOR CELLS | E-NULL MICE | SMOOTH-MUSCLE-CELLS | diabetes | GLYCATION END-PRODUCTS | SOLUBLE RECEPTOR | Glycation End Products, Advanced - physiology | Diabetic Angiopathies - physiopathology | Animals | Receptors, Immunologic - physiology | Atherosclerosis - physiopathology | Humans | Endothelium, Vascular - physiopathology | Rats | Ligands | Mice | Receptor for Advanced Glycation End Products | Disease Models, Animal
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7. Full Text Macrophage death and defective inflammation resolution in atherosclerosis
by Tabas, Ira
Nature Reviews Immunology, ISSN 1474-1733, 01/2010, Volume 10, Issue 1, pp. 36 - 46
A key event in atherosclerosis is a maladaptive inflammatory response to subendothelial lipoproteins. A crucial aspect of this response is a failure to resolve... 
RECEPTOR-DEFICIENT MICE | INHIBITS ATHEROSCLEROSIS | DENDRITIC CELLS | MONOCYTE-DERIVED CELLS | ACCELERATED ATHEROSCLEROSIS | IN-VIVO | ENDOPLASMIC-RETICULUM STRESS | IMMUNOLOGY | APOPTOTIC CELL ACCUMULATION | MER RECEPTOR | LIVER-X-RECEPTOR | Inflammation - pathology | Atherosclerosis - pathology | Atherosclerosis - immunology | Macrophages - pathology | Macrophage Activation - immunology | Humans | Phagocytosis - immunology | Inflammation - immunology | Inflammation - therapy | Animals | Apoptosis - immunology | Atherosclerosis - therapy | Macrophages - immunology
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8. Full Text Receptor for Advanced Glycation End Products (RAGE) Deficiency Attenuates the Development of Atherosclerosis in Diabetes
by Aino Soro-Paavonen and Anna M.D. Watson and Jiaze Li and Karri Paavonen and Audrey Koitka and Anna C. Calkin and David Barit and Melinda T. Coughlan and Brian G. Drew and Graeme I. Lancaster and Merlin Thomas and Josephine M. Forbes and Peter P. Nawroth and Angelika Bierhaus and Mark E. Cooper and Karin A. Jandeleit-Dahm
Diabetes, ISSN 0012-1797, 09/2008, Volume 57, Issue 9, pp. 2461 - 2469
Receptor for Advanced Glycation End Products (RAGE) Deficiency Attenuates the Development of Atherosclerosis in Diabetes Aino Soro-Paavonen 1 , Anna M.D.... 
VASCULAR INJURY | CELLS | ACTIVATION | INHIBITION | ACCELERATED ATHEROSCLEROSIS | ENDOCRINOLOGY & METABOLISM | PREVENTION | MICE | MODEL | NEPHROPATHY | Vasculitis - physiopathology | Oxidative Stress - physiology | Vasculitis - pathology | Myocytes, Smooth Muscle - pathology | NADPH Oxidases - metabolism | Vasculitis - immunology | Diabetic Angiopathies - physiopathology | Diabetic Angiopathies - pathology | Gene Expression - physiology | T-Lymphocytes - pathology | Energy Metabolism - physiology | Atherosclerosis - pathology | Atherosclerosis - immunology | Macrophages - pathology | Atherosclerosis - physiopathology | Mice, Knockout | Collagen - metabolism | Animals | Apolipoproteins E - genetics | Diabetic Angiopathies - immunology | Glycation End Products, Advanced - metabolism | Receptor, Angiotensin, Type 1 - metabolism | Biomarkers | Myocytes, Smooth Muscle - immunology | Mitogen-Activated Protein Kinases - genetics | Mice | Matrix Metalloproteinases - metabolism | Mitogen-Activated Protein Kinases - metabolism | Diabetics | Immune response | Atherosclerosis | Physiological aspects | Development and progression | Research | Health aspects | Risk factors | Complications
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9. Full Text Atherosclerosis and systemic lupus erythematosus — mechanistic basis of the association
by McMahon, Maureen and Hahn, Bevra H
Current Opinion in Immunology, ISSN 0952-7915, 2007, Volume 19, Issue 6, pp. 633 - 639
As patients with Systemic Lupus Erythematosus (SLE) live longer due to improved therapies and preventive measures, death and disability from cardiovascular... 
Allergy and Immunology | TOLL-LIKE RECEPTOR-2 | TUMOR-NECROSIS-FACTOR | ACCELERATED ATHEROSCLEROSIS | ENDOTHELIAL-CELLS | LOW-DENSITY-LIPOPROTEIN | SMOOTH-MUSCLE-CELLS | OXIDIZED PHOSPHOLIPIDS | ADHESION MOLECULES | IMMUNOLOGY | MONOCYTE CHEMOTACTIC PROTEIN-1 | CORONARY-ARTERY-DISEASE | Autoimmunity | Lupus Erythematosus, Systemic - complications | Antibodies, Antiphospholipid - blood | Autoantibodies - blood | Humans | Antibodies, Antiphospholipid - immunology | Lupus Erythematosus, Systemic - blood | Monocytes - immunology | Antigen-Antibody Complex - blood | Inflammation - blood | Lupus Erythematosus, Systemic - immunology | Cytokines - blood | Cytokines - immunology | Immunity, Active | Atherosclerosis - immunology | Atherosclerosis - complications | Inflammation - immunology | Immunity, Innate | Cell Adhesion Molecules - metabolism | Atherosclerosis - blood | Autoantibodies - immunology | Animals | Cholesterol, HDL - blood | T-Lymphocytes - immunology | Antigen-Antibody Complex - immunology | Lipoproteins, LDL - blood | Lipoproteins, LDL - immunology | Lupus | Development and progression | Systemic lupus erythematosus | Low density lipoproteins | Risk factors | Atherosclerosis
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