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Journal Article
Journal Article
Nature, ISSN 0028-0836, 03/2016, Volume 531, Issue 7596, pp. 651 - 655
Journal Article
Archives of Toxicology, ISSN 0340-5761, 12/2016, Volume 90, Issue 12, pp. 3073 - 3085
The leading cause of drug-induced liver injury in the developed world is overdose with N-acetyl-p-aminophenol (APAP). A comparative metabonomic approach was... 
Biomedicine, general | N -acetyl- m -aminophenol (AMAP) | Biomedicine | Environmental Health | Nuclear magnetic resonance spectroscopy | Occupational Medicine/Industrial Medicine | Metabonomics/Metabolic Phenotyping | Pharmacology/Toxicology | N -acetyl- p -aminophenol (APAP) | Hepatotoxicity | N-acetyl-p-aminophenol (APAP) | N-acetyl-m-aminophenol (AMAP) | OXIDATIVE STRESS | NMR-SPECTROSCOPY | PARACETAMOL | Nuclear magnetic resonance spectroscopy | MOUSE-LIVER | INDUCED LIVER-INJURY | MASS-SPECTROMETRY | N-acetylm-aminophenol (AMAP) | NONHEPATOTOXIC REGIOISOMER | GAS-CHROMATOGRAPHY | MITOCHONDRIAL DYSFUNCTION | TOXICOLOGY | N-acetyl-p-aminophenol (APAP) | PROTEIN ADDUCTS | Acetaminophen - adverse effects | Liver - pathology | Biomarkers - urine | Analgesics, Non-Narcotic - pharmacokinetics | Glutathione - metabolism | Acetylcysteine - metabolism | Male | Isomerism | Necrosis | Tissue Distribution | Liver - drug effects | Acetylcysteine - analogs & derivatives | Analgesics, Non-Narcotic - adverse effects | Biotransformation | Liver - diagnostic imaging | Chemical and Drug Induced Liver Injury - pathology | Biomarkers - metabolism | Eosinophilia - etiology | Acetaminophen - analogs & derivatives | Specific Pathogen-Free Organisms | Glutathione - analogs & derivatives | Acetaminophen - chemistry | Liver - metabolism | Mice, Inbred C57BL | Glutathione - antagonists & inhibitors | Analgesics, Non-Narcotic - metabolism | Biomarkers - blood | Chemical and Drug Induced Liver Injury - physiopathology | Metabolomics - methods | Chemical and Drug Induced Liver Injury - diagnostic imaging | Acetylcysteine - urine | Magnetic Resonance Imaging | Animals | Chemical and Drug Induced Liver Injury - metabolism | Acetaminophen - pharmacokinetics | Oxidative Stress - drug effects | Analgesics, Non-Narcotic - chemistry | Energy Metabolism - drug effects | Sulfur compounds | Metabolites | Acetaminophen | Liver | Comparative analysis | Mass spectrometry | Histochemistry | Nuclear magnetic resonance--NMR | Toxicity | Spectrum analysis | Metabonomics | Organ Toxicity and Mechanisms | Metabolic Phenotyping
Journal Article
Molecular Cell, ISSN 1097-2765, 09/2016, Volume 63, Issue 6, pp. 1006 - 1020
While much research has examined the use of glucose and glutamine by tumor cells, many cancers instead prefer to metabolize fats. Despite the pervasiveness of... 
PROTEIN | HIF | MALONYL-COA | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION | ACETYL-COA CARBOXYLASE | ACUTE MYELOID-LEUKEMIA | PHARMACOLOGICAL INHIBITION | CELL-METABOLISM | PROLYL HYDROXYLASES | DEFICIENT CELLS | CELL BIOLOGY | Hypoxia-Inducible Factor-Proline Dioxygenases - chemistry | Neoplasm Transplantation | RNA, Small Interfering - genetics | Proline - metabolism | Humans | Leukemia, Myeloid, Acute - metabolism | Gene Expression Regulation, Neoplastic | Male | Acetyl-CoA Carboxylase - genetics | HEK293 Cells | Fatty Acids - metabolism | Acetyl-CoA Carboxylase - antagonists & inhibitors | Amino Acid Sequence | Hydroxylation | Oxidation-Reduction | Signal Transduction | Leukemia, Myeloid, Acute - pathology | Hypoxia-Inducible Factor-Proline Dioxygenases - genetics | Models, Molecular | Proline - chemistry | Leukemia, Myeloid, Acute - mortality | Metabolic Networks and Pathways - genetics | Animals | Hypoxia-Inducible Factor-Proline Dioxygenases - metabolism | K562 Cells | Survival Analysis | Cell Line, Tumor | Mice, Inbred NOD | Mice | Structural Homology, Protein | Acetyl-CoA Carboxylase - metabolism | Acetyl-CoA Carboxylase - chemistry | Leukemia, Myeloid, Acute - genetics | RNA, Small Interfering - metabolism | Medical colleges | Usage | Leukemia | Proline | Disease susceptibility | Glucose | Fatty acids | Dextrose | Proteins | Liability (Law) | Stem cells | Oxidation-reduction reaction | Research institutes | Glutamine
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2018, Volume 293, Issue 31, pp. 12259 - 12270
Certain dysregulated chondrocyte metabolic adaptive responses such as decreased activity of the master regulator of energy metabolism AMP-activated protein... 
TARGET | nucleocytosolic acetyl-CoA | BIOCHEMISTRY & MOLECULAR BIOLOGY | AMP-activated kinase (AMPK) | AUTOPHAGY | epigenetics | CARTILAGE | acetylation | PATHOGENESIS | chondrocyte | ACETYL-COENZYME | GROWTH | OSTEOARTHRITIS | BONE | acetyl coenzyme A (acetyl-CoA) | ATP-citrate lyase | hydroxycitric acid | CHONDROPROTECTION | Metabolism
Journal Article