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Journal of Clinical Investigation, ISSN 0021-9738, 12/2011, Volume 121, Issue 12, pp. 4712 - 4721
Cushing disease is a condition in which the pituitary gland releases excessive adrenocorticotropic hormone (ACTH) as a result of an adenoma arising from the... 
NUCLEAR-LOCALIZATION | MEDICINE, RESEARCH & EXPERIMENTAL | DEPENDENT CUSHINGS-SYNDROME | GEFITINIB INDUCES APOPTOSIS | CYCLE INHIBITOR P27(KIP1) | EPIDERMAL-GROWTH-FACTOR | PROOPIOMELANOCORTIN GENE | CORTICOTROPH TUMORS | T-BOX FACTOR | FACTOR RECEPTOR | PROGNOSTIC VALUE | ACTH-Secreting Pituitary Adenoma - drug therapy | Neoplasm Transplantation | Humans | Cell Line, Tumor - drug effects | Molecular Targeted Therapy | Pituitary ACTH Hypersecretion - drug therapy | ACTH-Secreting Pituitary Adenoma - complications | Adenoma - enzymology | Adenoma - complications | Dog Diseases - drug therapy | Dog Diseases - etiology | Secretory Rate - drug effects | Adenoma - drug therapy | Corticosterone - blood | Gene Expression Regulation, Neoplastic - drug effects | Pro-Opiomelanocortin - genetics | Adenoma - veterinary | Promoter Regions, Genetic | Pituitary ACTH Hypersecretion - etiology | ACTH-Secreting Pituitary Adenoma - veterinary | Cell Line, Tumor - enzymology | Tumor Burden | Adrenocorticotropic Hormone - secretion | Pituitary ACTH Hypersecretion - blood | Adenoma - blood | Dog Diseases - blood | ACTH-Secreting Pituitary Adenoma - blood | Animals | Mice, Nude | Protein Kinase Inhibitors - therapeutic use | Quinazolines - therapeutic use | Dogs | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Mice | Protein Kinase Inhibitors - pharmacology | ACTH-Secreting Pituitary Adenoma - enzymology | Pituitary ACTH Hypersecretion - veterinary | Quinazolines - pharmacology | Drug Screening Assays, Antitumor | Care and treatment | Pituitary gland tumors | ACTH | Drug targeting | Adenoma | Diagnosis | Research | Properties | Index Medicus | Abridged Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 05/2013, Volume 110, Issue 21, pp. 8555 - 8560
Cushing disease (CD) is a life-threatening disorder attributed to excess pituitary tumor-derived adrenocorticotrophic hormone (ACTH) and adrenal steroid... 
Cell growth | Phosphorylation | Pituitary neoplasms | Secretion | Plasmids | Cell lines | Pituitary ACTH hypersecretion | Mice | Orphan nuclear receptors | Tumors | MANAGEMENT | ACID | PHOSPHORYLATION | PATHWAY | MULTIDISCIPLINARY SCIENCES | PROLIFERATION | MODULATOR | EXPRESSION | Transcriptional Activation - genetics | Cell Proliferation | Receptors, Steroid - metabolism | Humans | Neoplasm Proteins - metabolism | ACTH-Secreting Pituitary Adenoma - therapy | Adrenocorticotropic Hormone - genetics | Pituitary ACTH Hypersecretion - genetics | ACTH-Secreting Pituitary Adenoma - genetics | Receptors, Thyroid Hormone - genetics | Response Elements - genetics | Corticosterone - secretion | Neoplasm Proteins - genetics | Pro-Opiomelanocortin - genetics | Pituitary ACTH Hypersecretion - therapy | Gene Expression Regulation, Neoplastic - genetics | Pituitary ACTH Hypersecretion - pathology | ACTH-Secreting Pituitary Adenoma - metabolism | Corticosterone - genetics | Adrenocorticotropic Hormone - secretion | Receptors, Thyroid Hormone - metabolism | Animals | Pituitary ACTH Hypersecretion - metabolism | Receptors, Steroid - genetics | Pro-Opiomelanocortin - biosynthesis | Cell Line, Tumor | ACTH-Secreting Pituitary Adenoma - pathology | Physiological aspects | Pituitary gland | Research | Hydrocortisone | Health aspects | Causes and theories of causation | Diseases | Rodents | Genetics | T cell receptors | Binding sites | Index Medicus | Biological Sciences
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 05/2011, Volume 96, Issue 5, pp. 1213 - 1222
Ten cases of ectopic ACTH syndrome occurring in youngsters are described; the diagnostic and therapeutic algorithms recommended in adults can be used in this... 
PEDIATRIC CUSHINGS-SYNDROME | EWINGS-SARCOMA | DIFFERENTIAL-DIAGNOSIS | ISLET-CELL-CARCINOMA | ENDOCRINOLOGY & METABOLISM | ADRENOCORTICOTROPIN SYNDROME | CORTICOTROPIN SECRETION | CHILDHOOD | SECRETING WILMS-TUMOR | BLOOD-PRESSURE | STROMAL EPITHELIAL TUMOR | Prognosis | Humans | ACTH Syndrome, Ectopic - epidemiology | Cushing Syndrome - etiology | Hydrocortisone - urine | Male | Tomography, X-Ray Computed | Weight Gain - physiology | Positron-Emission Tomography | Pituitary ACTH Hypersecretion - complications | Radioimmunoassay | Female | Receptors, Somatostatin - metabolism | Child | Adrenocorticotropic Hormone - blood | Diagnosis, Differential | France - epidemiology | Dexamethasone | ACTH-Secreting Pituitary Adenoma - metabolism | Pituitary Gland - pathology | Pituitary Gland - diagnostic imaging | Treatment Outcome | ACTH Syndrome, Ectopic - diagnosis | ACTH-Secreting Pituitary Adenoma - diagnosis | Growth - physiology | Magnetic Resonance Imaging | Health Surveys | ACTH Syndrome, Ectopic - drug therapy | Adolescent | ACTH-Secreting Pituitary Adenoma - pathology | Index Medicus | Abridged Index Medicus | Growth | Hydrocortisone | Life Sciences | Pituitary ACTH Hypersecretion | Receptors, Somatostatin | Adrenocorticotropic Hormone | Weight Gain | France | ACTH-Secreting Pituitary Adenoma | ACTH Syndrome, Ectopic | Cushing Syndrome | Pituitary Gland | Cancer
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 08/2017, Volume 102, Issue 8, pp. 2836 - 2843
Context: Somatic mutations in the ubiquitin-specific protease 8 (USP8) gene have been recently identified as the most common genetic alteration in patients... 
TARGET | UBPY | ADOLESCENTS | ENDOCRINOLOGY & METABOLISM | RESISTANCE | CANCER | EGFR | DEUBIQUITINATING ENZYMES | PITUITARY-ADENOMAS | CHILDREN | Adenoma - surgery | Prognosis | Adenoma - genetics | Humans | Child, Preschool | Endosomal Sorting Complexes Required for Transport - genetics | Hydrocortisone - metabolism | Male | Adenoma - metabolism | Pituitary ACTH Hypersecretion - genetics | ACTH-Secreting Pituitary Adenoma - genetics | ACTH-Secreting Pituitary Adenoma - surgery | Pituitary ACTH Hypersecretion - surgery | Female | Retrospective Studies | Neuroendoscopy | Child | Adrenocorticotropic Hormone - metabolism | Cavernous Sinus - pathology | Pituitary ACTH Hypersecretion - pathology | ACTH-Secreting Pituitary Adenoma - metabolism | Neoplasm Invasiveness | Tumor Burden | Ubiquitin Thiolesterase - genetics | Endopeptidases - genetics | Pituitary ACTH Hypersecretion - metabolism | Adolescent | Age of Onset | Adenoma - pathology | Neoplasm Recurrence, Local - genetics | Mutation | ACTH-Secreting Pituitary Adenoma - pathology | Ubiquitin | Pediatrics | Nervous system diseases | Ubiquitin-specific proteinase | Nucleotide sequence | Hydrocortisone | Gene deletion | Patients | 14-3-3 protein | Adrenocorticotropic hormone | Clonal deletion | Sinus | Pituitary | Deletion | Deoxyribonucleic acid--DNA | Index Medicus | Abridged Index Medicus
Journal Article
1987, ISBN 0124472133, Volume 13., x, 202
Book
Journal Article
Alimentary Pharmacology & Therapeutics, ISSN 0269-2813, 06/2019, Volume 49, Issue 12, pp. 1539 - 1539
Linked Content This article is linked to Philpott et al and Rodríguez Manchón et al papers. To view these articles, visit https://doi.org/10.1111/apt.14573 and... 
ACTH | Corticosteroids
Journal Article
Alimentary Pharmacology & Therapeutics, ISSN 0269-2813, 06/2019, Volume 49, Issue 12, pp. 1537 - 1538
Linked Content This article is linked to Philpott et al papers. To view these articles, visit https://doi.org/10.1111/apt.14573 and... 
ACTH | Corticosteroids
Journal Article
Journal Article
Journal Article
European Journal of Endocrinology, ISSN 0804-4643, 2013, Volume 169, Issue 3, pp. 329 - 337
Journal Article
Clinical Endocrinology, ISSN 0300-0664, 02/2015, Volume 82, Issue 2, pp. 260 - 266
Journal Article
Nature Genetics, ISSN 1061-4036, 12/2014, Volume 47, Issue 1, pp. 31 - 38
Cushing's disease is caused by corticotroph adenomas of the pituitary. To explore the molecular mechanisms of endocrine autonomy in these tumors, we performed... 
UBPY | ENDOSOMES | DOMAIN | PROTEIN | EPIDERMAL-GROWTH-FACTOR | GENETICS & HEREDITY | UBIQUITYLATION | DEGRADATION | RECEPTOR DOWN-REGULATION | ENDOCYTIC TRAFFICKING | PITUITARY-ADENOMAS | Adenoma - genetics | Humans | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Cercopithecus aethiops | Endosomal Sorting Complexes Required for Transport - genetics | Molecular Sequence Data | Pituitary Neoplasms - complications | ACTH-Secreting Pituitary Adenoma - complications | Pituitary ACTH Hypersecretion - genetics | ACTH-Secreting Pituitary Adenoma - genetics | Receptor, Epidermal Growth Factor - metabolism | Pituitary Neoplasms - secretion | Neoplasm Proteins - genetics | Pro-Opiomelanocortin - genetics | Amino Acid Sequence | ACTH-Secreting Pituitary Adenoma - metabolism | Pituitary Neoplasms - genetics | Ubiquitin Thiolesterase - physiology | Pituitary Neoplasms - metabolism | Ubiquitin Thiolesterase - genetics | 14-3-3 Proteins - metabolism | Adrenocorticotropic Hormone - secretion | Sequence Homology, Amino Acid | Exome - genetics | Sequence Alignment | Animals | Endopeptidases - genetics | ACTH-Secreting Pituitary Adenoma - secretion | Pro-Opiomelanocortin - biosynthesis | Mutation | COS Cells | Endosomal Sorting Complexes Required for Transport - physiology | Endopeptidases - physiology | Gene mutations | Cushing syndrome | Physiological aspects | Genetic aspects | Research | Ubiquitin-proteasome system | Health aspects | Risk factors | Pituitary gland | Kinases | Rodents | Genes | Tumors | Index Medicus
Journal Article