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1. Full Text Synapse-Level Determination of Action Potential Duration by K+ Channel Clustering in Axons
by Rowan, Matthew J.M and DelCanto, Gina and Yu, Jianqing J and Kamasawa, Naomi and Christie, Jason M
Neuron, ISSN 0896-6273, 07/2016, Volume 91, Issue 2, pp. 370 - 383
In axons, an action potential (AP) is thought to be broadcast as an unwavering binary pulse over its arbor, driving neurotransmission uniformly at release... 
TRANSMITTER RELEASE | MAMMALIAN BRAIN | NERVE-TERMINALS | POTASSIUM CURRENTS | WAVE-FORM | NA+ CHANNELS | PRESYNAPTIC BOUTONS | INITIAL SEGMENT | FAST-SPIKING NEURONS | MOSSY FIBER BOUTONS | NEUROSCIENCES | Action Potentials - physiology | Interneurons - physiology | Animals | Synaptic Transmission - physiology | Synapses - physiology | Mice, Inbred C57BL | Potassium Channels - physiology | Axons - physiology | Presynaptic Terminals - physiology | Cerebellum - physiology | Patch-Clamp Techniques - methods | Neurosciences | Simulation | Microscopy | Plasmids | Lasers | Rodents | Morphology | Colleges & universities | Index Medicus
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2. Full Text FMRP Regulates Neurotransmitter Release and Synaptic Information Transmission by Modulating Action Potential Duration via BK Channels
by Deng, Pan-Yue and Rotman, Ziv and Blundon, Jay A and Cho, Yongcheol and Cui, Jianmin and Cavalli, Valeria and Zakharenko, Stanislav S and Klyachko, Vitaly A
Neuron, ISSN 0896-6273, 02/2013, Volume 77, Issue 4, pp. 696 - 711
Loss of FMRP causes fragile X syndrome (FXS), but the physiological functions of FMRP remain highly debatable. Here we show that FMRP regulates... 
TRANSMITTER RELEASE | MESSENGER-RNAS | POTASSIUM CHANNELS | MOUSE MODEL | BETA-SUBUNITS | MAMMALIAN CENTRAL NEURONS | NEUROSCIENCES | X MENTAL-RETARDATION | SHORT-TERM PLASTICITY | K+ CHANNELS | HIPPOCAMPAL SYNAPSES | Large-Conductance Calcium-Activated Potassium Channels - metabolism | Calcium - metabolism | Neurotransmitter Agents - metabolism | Fragile X Syndrome - metabolism | Fragile X Mental Retardation Protein - metabolism | Synaptic Transmission | Action Potentials - physiology | Excitatory Postsynaptic Potentials - physiology | Hippocampus - metabolism | Animals | Presynaptic Terminals | Mice | Neurons - metabolism | Fast food restaurants | Potassium channels | Analysis | Cells | Biomedical engineering | Proteins | Studies | Autism | Neurons | Information theory | Index Medicus | Action potential
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3. Full Text Strength–duration relationship for intra- versus extracellular stimulation with microelectrodes
by Rattay, F and Paredes, L.P and Leao, R.N and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Uppsala universitet and Genetisk utvecklingsbiologi and Institutionen för neurovetenskap
Neuroscience, ISSN 0306-4522, 2012, Volume 214, Issue 5, pp. 1 - 13
Highlights ► The excitability time constant chronaxie for electrical stimulation is analyzed. ► Chronaxie varies along the neural axis. ► Extracellular... 
Neurology | electrical stimulation | chronaxie | microelectrode | activating function | strength–duration curve | Chronaxie | Activating function | Electrical stimulation | Microelectrode | Strength-duration curve | NERVOUS-SYSTEM | ELECTRODE CONFIGURATION | RESTORE VISION | SPINAL-CORD | ACTION-POTENTIAL INITIATION | NEUROSCIENCES | strength-duration curve | RETINAL GANGLION-CELLS | IN-VITRO | ELECTRICAL-STIMULATION | NEOCORTICAL PYRAMIDAL NEURONS | DENDRITIC EXCITABILITY | Extracellular Fluid - physiology | Microelectrodes | Animals | Chronaxy - physiology | Time Factors | Mice, Inbred C57BL | Mice | Intracellular Fluid - physiology | Hippocampus - physiology | Membrane Potentials - physiology | Electric Stimulation - methods | Neurosciences | Analysis | Index Medicus | VSD, voltage sensitive dye | AIS, axon initial segments | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Neurovetenskaper
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4. Full Text Coupling of smoothened to inhibitory G proteins reduces voltage-gated K + currents in cardiomyocytes and prolongs cardiac action potential duration
by Cheng, Lan and Al-Owais, Moza and Covarrubias, Manuel L and Koch, Walter J and Manning, David R and Peers, Chris and Riobo-Del Galdo, Natalia A
Journal of Biological Chemistry, ISSN 0021-9258, 2018, Volume 293, Issue 28, pp. 11022 - 11032
SMO (Smoothened), the central transducer of Hedgehog signaling, is coupled to heterotrimeric G(i) proteins in many cell types, including cardiomyocytes. In... 
G(I) | HEDGEHOG PATHWAY | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | noncanonical | cardiovascular | action potential | heart | Smoothened | arrhythmias | ion channel | G protein-coupled receptor (GPCR) | Hedgehog signaling pathway | potassium channel | CHANNELS | G protein | GTP-Binding Protein alpha Subunits, Gi-Go - genetics | Potassium - metabolism | Myocytes, Cardiac - cytology | Cells, Cultured | Hedgehog Proteins - metabolism | Mice, Transgenic | Smoothened Receptor - metabolism | Smoothened Receptor - genetics | Action Potentials - physiology | Animals | Hedgehog Proteins - genetics | Potassium Channels, Voltage-Gated - metabolism | Myocytes, Cardiac - metabolism | Mice | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism | Ion Channel Gating | Index Medicus | Signal Transduction
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5. Full Text Action Potential Duration Restitution and Alternans in Rabbit Ventricular Myocytes: The Key Role of Intracellular Calcium Cycling
by Goldhaber, Joshua I and Xie, Lai-Hua and Duong, Tan and Motter, Christi and Khuu, Kien and Weiss, James N
Circulation Research, ISSN 0009-7330, 03/2005, Volume 96, Issue 4, pp. 459 - 466
Action potential duration (APD) restitution properties and repolarization alternans are thought to be important arrhythmogenic factors. We investigated the... 
Alternans | Arrhythmia | Action potential restitution | Transients | FIBRILLATION | CELLS | action potential restitution | CARDIAC & CARDIOVASCULAR SYSTEMS | MECHANISM | ELECTRICAL RESTITUTION | STABILITY | WAVE DYNAMICS | CARDIAC TISSUE | MODEL | EXCITABILITY | arrhythmia | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | alternans | Ca2+ transients | PURKINJE | Humans | Calcium Channels - physiology | Calcium - physiology | Time Factors | Heart Conduction System - physiology | Myocardium - metabolism | Egtazic Acid - analogs & derivatives | Caffeine - pharmacology | Ion Transport - drug effects | Sarcoplasmic Reticulum - metabolism | Action Potentials - drug effects | Ryanodine - pharmacology | Calcium Channels - drug effects | Rabbits | Cells, Cultured - drug effects | Models, Cardiovascular | Calcium Channels, L-Type - physiology | Heart Conduction System - drug effects | Cardiac Pacing, Artificial | Action Potentials - physiology | Patch-Clamp Techniques | Tachycardia, Ventricular - physiopathology | Animals | Egtazic Acid - pharmacology | Myocytes, Cardiac - drug effects | Heart Conduction System - physiopathology | Calcium Signaling - drug effects | Calcium Channels, L-Type - drug effects | Myocytes, Cardiac - metabolism | Thapsigargin - pharmacology | Cells, Cultured - metabolism | Index Medicus
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6. Full Text Prolonged action potential duration and dynamic transmural action potential duration heterogeneity underlie vulnerability to ventricular tachycardia in patients undergoing ventricular tachycardia ablation
by Srinivasan, Neil T and Orini, Michele and Providencia, Rui and Dhinoja, Mehul B and Lowe, Martin D and Ahsan, Syed Y and Chow, Anthony W and Hunter, Ross J and Schilling, Richard J and Taggart, Peter and Lambiase, Pier D
EP Europace, ISSN 1099-5129, 04/2019, Volume 21, Issue 4, pp. 616 - 625
Abstract Aims Differences of action potential duration (APD) in regions of myocardial scar and their borderzones are poorly defined in the intact human heart.... 
MYOCARDIUM | CELLS | CARDIAC & CARDIOVASCULAR SYSTEMS | Transmural dispersion of repolarization | DISPERSION | Ventricular tachycardia | Dispersion of repolarization | Action potential duration | CURRENTS | REPOLARIZATION | Clinical Research
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7. Full Text Compound muscle action potential duration in critical illness neuromyopathy
by Kramer, Christopher L and Boon, Andrea J and Harper, C. Michel and Goodman, Brent P
Muscle & Nerve, ISSN 0148-639X, 03/2018, Volume 57, Issue 3, pp. 395 - 400
IntroductionWe sought to determine the specificity of compound muscle action potential (CMAP) durations and amplitudes in a large critical illness... 
compound muscle action potential duration | compound muscle action potential amplitude | critical illness neuropathy | critical illness neuromyopathy | nerve conduction study | ICU acquired weakness | critical illness myopathy | POLYNEUROPATHY | MYOPATHY | WEAKNESS | MODEL | NEUROSCIENCES | CLINICAL NEUROLOGY | EXCITABILITY | Action Potentials - physiology | Young Adult | Humans | Middle Aged | Muscle, Skeletal - physiopathology | Aged, 80 and over | Adult | Female | Male | Aged | Retrospective Studies | Neuromuscular Diseases - physiopathology | Amplitudes | Nerves | Muscles | Action potential | Peripheral neuropathy | Patients | Myopathy | Index Medicus
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8. Full Text Transient outward K + current can strongly modulate action potential duration and initiate alternans in the human atrium
by Ni, Haibo and Zhang, Henggui and Grandi, Eleonora and Narayan, Sanjiv M and Giles, Wayne R
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 03/2019, Volume 316, Issue 3, pp. H527 - H542
Efforts to identify the mechanisms for the initiation and maintenance of human atrial fibrillation (AF) often focus on changes in specific elements of the... 
currents | Transient outward K | channel isoform switch | Action potential restitution | Atrial fibrillation | Action potential alternans | Mathematical modeling | K | Human atrial myocyte | action potential restitution | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | action potential alternans | HEART-FAILURE | POTASSIUM CURRENT | VENTRICULAR MYOCYTES | FIBRILLATION INITIATION | K+ channel isoform switch | RATE DEPENDENCE | atrial fibrillation | ELECTRICAL ALTERNANS | REPOLARIZATION ALTERNANS | mathematical modeling | CARDIAC PURKINJE | transient outward K+ currents | CHANNEL GENE-EXPRESSION | PERIPHERAL VASCULAR DISEASE | human atrial myocyte | VOLTAGE-CLAMP | Atrium | Stability | Calcium | Arrhythmia | Action potential | Activation | Pharmacology | Substrates | Properties (attributes) | Fibrillation | Clonal deletion | Dynamic stability | Isoforms | Reaction kinetics | Deletion | Potassium channels (voltage-gated) | Mathematical models | Kinetics | Calcium ions
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9. Full Text Increased seizure duration and slowed potassium kinetics in mice lacking aquaporin‐4 water channels
by Binder, Devin K and Yao, Xiaoming and Zador, Zsolt and Sick, Thomas J and Verkman, Alan S and Manley, Geoffrey T
Glia, ISSN 0894-1491, 04/2006, Volume 53, Issue 6, pp. 631 - 636
The glial water channel aquaporin‐4 (AQP4) has been hypothesized to modulate water and potassium fluxes associated with neuronal activity. In this study, we... 
epilepsy | glial cell | potassium | seizure | extracellular space | aquaporin | astrocyte | Glial cell | Epilepsy | Seizure | Astrocyte | Potassium | Aquaporin | Extracellular space | Potassium - metabolism | Action Potentials - genetics | Electric Stimulation | Seizures - genetics | Brain Ischemia - genetics | Brain Ischemia - metabolism | Extracellular Space - genetics | Male | Seizures - metabolism | Cell Membrane - genetics | Electroencephalography | Brain - metabolism | Brain Edema - genetics | Potassium Channels - metabolism | Seizures - physiopathology | Extracellular Space - metabolism | Aquaporin 4 - genetics | Cell Membrane - metabolism | Neurons - metabolism | Water-Electrolyte Imbalance - genetics | Disease Models, Animal | Brain Edema - metabolism | Brain - physiopathology | Brain Ischemia - physiopathology | Signal Transduction - genetics | Mice, Knockout | Potassium Channels - genetics | Water-Electrolyte Imbalance - metabolism | Hippocampus - metabolism | Animals | Brain Edema - physiopathology | Water-Electrolyte Imbalance - physiopathology | Mice | Hippocampus - physiopathology | Homeostasis - genetics | Astrocytes - metabolism | Index Medicus
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10. Full Text Prolongation of Action Potential Duration and QT Interval During Epilepsy Linked to Increased Contribution of Neuronal Sodium Channels to Cardiac Late Na+ Current: Potential Mechanism for Sudden Death in Epilepsy
by Biet, Michael and Morin, Nathalie and Lessard-Beaudoin, Melissa and Graham, Rona K and Duss, Sandra and Gagné, Jonathan and Sanon, Nathalie T and Carmant, Lionel and Dumaine, Robert
Circulation: Arrhythmia and Electrophysiology, ISSN 1941-3149, 08/2015, Volume 8, Issue 4, pp. 912 - 920
BACKGROUND—Arrhythmias associated with QT prolongation on the ECG often lead to sudden unexpected death in epilepsy. The mechanism causing a prolongation of... 
epilepsy | DOG VENTRICLE | CARDIAC & CARDIOVASCULAR SYSTEMS | death | action potentials | UNEXPECTED DEATH | BLOCK | STATUS-EPILEPTICUS | PARTIAL SEIZURES | PURKINJE-FIBERS | HEART | arrhythmias | electrophysiology | sodium channels | INCREASED SUSCEPTIBILITY | sudden | I-NA | cardiac | SUBUNIT | Arrhythmias, Cardiac - metabolism | Death, Sudden - etiology | Gene Expression Regulation | Rats | Epilepsy - metabolism | Male | Epilepsy - mortality | Blotting, Western | Epilepsy - complications | Action Potentials - physiology | DNA - genetics | Patch-Clamp Techniques | Animals | Myocytes, Cardiac - metabolism | Arrhythmias, Cardiac - complications | NAV1.5 Voltage-Gated Sodium Channel - genetics | NAV1.5 Voltage-Gated Sodium Channel - biosynthesis | Real-Time Polymerase Chain Reaction | Arrhythmias, Cardiac - genetics | Index Medicus
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11. Full Text Purkinje-cell plasticity and cerebellar motor learning are graded by complex-spike duration
by Yang, Yan and Lisberger, Stephen G
Nature, ISSN 0028-0836, 2014, Volume 510, Issue 7506, pp. 529 - 532
Behavioural learning is mediated by cellular plasticity, such as changes in the strength of synapses at specific sites in neural circuits. The theory of... 
PARALLEL FIBERS | DENDRITES | CLIMBING FIBERS | MODEL | OSCILLATIONS | MULTIDISCIPLINARY SCIENCES | Motor Skills - physiology | Calcium - metabolism | Olivary Nucleus - physiology | Male | Axons - physiology | Macaca mulatta | Long-Term Synaptic Depression | Purkinje Cells - physiology | Action Potentials | Learning - physiology | Animals | Purkinje Cells - cytology | Neuronal Plasticity | Neuroplasticity | Physiological aspects | Neural circuitry | Observations | Motor ability | Neurology | Cognition & reasoning | Laboratory animals | Neurons | Experiments | Index Medicus
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12. Full Text Restitution slope is principally determined by steady-state action potential duration
by Shattock, Michael J and Park, Kyung Chan and Yang, Hsiang-Yu and Lee, Angela W. C and Niederer, Steven and MacLeod, Kenneth T and Winter, James
Cardiovascular Research, ISSN 0008-6363, 2017, Volume 113, Issue 7, pp. 817 - 828
Aims The steepness of the action potential duration (APD) restitution curve and local tissue refractoriness are both thought to play important roles in... 
Cardiac memory | Restitution | Electrical restitution | Action potential duration | FIBRILLATION | CARDIAC & CARDIOVASCULAR SYSTEMS | ALTERNANS | DISPERSION | HEART-FAILURE | I-KR | GUINEA-PIG | REPOLARIZATION | RATE DEPENDENCE | RABBIT VENTRICULAR MYOCYTES | Rabbits | Guinea Pigs | Ventricular Fibrillation - metabolism | Calcium - metabolism | Isolated Heart Preparation | Models, Cardiovascular | Risk Factors | Cardiomegaly - physiopathology | Male | Ventricular Fibrillation - etiology | Heart Rate - drug effects | Animals | Myocytes, Cardiac - drug effects | Calcium Signaling - drug effects | Myocytes, Cardiac - metabolism | Ventricular Fibrillation - physiopathology | Calcium Channel Agonists - pharmacology | Calcium Chelating Agents - pharmacology | Kinetics | Action Potentials - drug effects | Refractory Period, Electrophysiological - drug effects | Cardiomegaly - metabolism | Index Medicus | Original | Editor's Choice
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13. Full Text Mechanism of the medium‐duration afterhyperpolarization in rat serotonergic neurons
by Alix, Philippe and Venkatesan, Kumar and Scuvée‐Moreau, Jacqueline and Massotte, Laurent and Nguyen Trung, Mai‐Linh and Cornil, Charlotte A and Seutin, Vincent
European Journal of Neuroscience, ISSN 0953-816X, 01/2014, Volume 39, Issue 2, pp. 186 - 196
Most serotonergic neurons display a prominent medium‐duration afterhyperpolarization ( mAHP ), which is mediated by small‐conductance C a 2+ ‐activated K + (... 
bicuculline methiodide | channels | apamin | voltage‐dependent | Bicuculline methiodide | Apamin | Voltage-dependent Ca | SK channels | NMDA RECEPTORS | CA2+-ACTIVATED K+ CHANNELS | HIPPOCAMPAL PYRAMIDAL NEURONS | HYPOGLOSSAL MOTONEURONS | ACTIVATED POTASSIUM CHANNELS | NEUROSCIENCES | DORSAL RAPHE NEURONS | FIRING BEHAVIOR | DOPAMINERGIC MIDBRAIN NEURONS | CALCIUM-CHANNELS | voltage-dependent Ca2+ channels | SMALL-CONDUCTANCE | Immunohistochemistry | Membrane Potentials - drug effects | Serotonergic Neurons - physiology | Rats, Wistar | Serotonergic Neurons - drug effects | Calcium - metabolism | Brain Stem - growth & development | Rats | Male | Calcium Channel Blockers - pharmacology | Calcium Channels, N-Type - metabolism | Brain Stem - physiology | Brain Stem - drug effects | Small-Conductance Calcium-Activated Potassium Channels - metabolism | Small-Conductance Calcium-Activated Potassium Channels - antagonists & inhibitors | Patch-Clamp Techniques | Animals | Calcium Channels, T-Type - metabolism | Potassium Channels, Calcium-Activated - metabolism | Female | Potassium Channels, Calcium-Activated - antagonists & inhibitors | In Vitro Techniques | Action Potentials - drug effects | Serotonin | Neurons | Index Medicus | Action potential
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