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1. Full Text Regulating excitability of peripheral afferents: Emerging ion channel targets
by Waxman, Stephen G and Zamponi, Gerald W
Nature Neuroscience, ISSN 1097-6256, 02/2014, Volume 17, Issue 2, pp. 153 - 163
The transmission and processing of pain signals relies critically on the activities of ion channels that are expressed in afferent pain fibers. This includes... 
GATED CATION CHANNELS | NEUROPATHIC PAIN | NA CHANNELS | RESISTANT SODIUM-CHANNEL | CA2+-ACTIVATED K+ CHANNELS | ACTIVATED POTASSIUM CHANNELS | INFLAMMATORY PAIN | RAT SENSORY NEURONS | DORSAL-ROOT GANGLION | NERVE INJURY | NEUROSCIENCES | Afferent Pathways - physiopathology | Ion Channels - physiology | Animals | Pain - pathology | Humans | Peripheral Nerves - physiopathology | Nociception | Physiological aspects | Neural transmission | Physiological research | Ion channels | Research
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2. Full Text Ion Channel Pharmacology
by Conte Camerino, Diana and Tricarico, Domenico and Desaphy, Jean-François
Neurotherapeutics, ISSN 1933-7213, 2007, Volume 4, Issue 2, pp. 184 - 198
Because ion channels are involved in many cellular processes, drugs acting on ion channels have long been used for the treatment of many diseases, especially... 
neurotransmitter-gated | drug therapy | ion channel | channelopathy | pharmacogenetics | Voltage-gated | Neurology | Neurosciences | Biomedicine | Neurobiology | Neurosurgery | voltage-gated | GATED SODIUM-CHANNELS | NA+ CHANNELS | ACTIVATED POTASSIUM CHANNELS | HYPERKALEMIC PERIODIC PARALYSIS | NEUROSCIENCES | SKELETAL-MUSCLE | UNION-OF-PHARMACOLOGY | MOLECULAR RELATIONSHIPS | K-ATP CHANNEL | PHARMACOLOGY & PHARMACY | STATE-DEPENDENT BLOCK | CARBONIC-ANHYDRASE INHIBITORS | Channelopathies - genetics | Pharmacogenetics | Animals | Ion Channel Gating - genetics | Channelopathies - drug therapy | Ion Channels - classification | Drug Therapy - methods | Humans | Ion Channels - genetics | Ion Channels - drug effects | Mutation | Ion Channel Gating - drug effects | Pharmacology | Proteins
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3. Full Text Ca2+-dependent phospholipid scrambling by a reconstituted TMEM16 ion channel
by Malvezzi, Mattia and Chalat, Madhavan and Janjusevic, Radmila and Picollo, Alessandra and Terashima, Hiroyuki and Menon, Anant K and Accardi, Alessio
Nature Communications, ISSN 2041-1723, 2013, Volume 4, Issue 1, pp. 2367 - 2367
Phospholipid (PL) scramblases disrupt the lipid asymmetry of the plasma membrane, externalizing phosphatidylserine to trigger blood coagulation and mark... 
FLIPPASES | MULTIDISCIPLINARY SCIENCES | FUNCTIONAL RECONSTITUTION | ENDOPLASMIC-RETICULUM | COMPONENT | PROTEINS | CA2+-ACTIVATED CL-CHANNEL | ANOCTAMIN 6 | FAMILY | Mammals - metabolism | Calcium Channels - metabolism | Calcium - metabolism | Humans | Ions | Lipid Metabolism | Phospholipid Transfer Proteins - metabolism | Animals | Chlorides - metabolism | Aspergillus fumigatus - metabolism | Ion Transport | Phospholipids - metabolism | Fungal Proteins - isolation & purification | Binding Sites | Ion Channel Gating | Fungal Proteins - metabolism
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4. Full Text Flufenamic acid as an ion channel modulator
by Guinamard, Romain and Simard, Christophe and Del Negro, Christopher
Pharmacology and Therapeutics, ISSN 0163-7258, 05/2013, Volume 138, Issue 2, pp. 272 - 284
Flufenamic acid has been known since the 1960s to have anti-inflammatory properties attributable to the reduction of prostaglandin synthesis. Thirty years... 
Flufenamic acid | Flufenamate | Chloride channels | Non-selective cation channel | TRP | Channel blockers | MEFENAMIC-ACID | CARDIAC VENTRICULAR MYOCYTES | VOLTAGE-GATED SODIUM | CL-CHANNEL | TRANSMEMBRANE CONDUCTANCE REGULATOR | NIFLUMIC ACID | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | FUNCTIONAL EXPRESSION | PHARMACOLOGY & PHARMACY | ACTIVATED CHLORIDE CHANNELS | NONSELECTIVE CATION CHANNELS | Cell Line | Flufenamic Acid - pharmacology | Patch-Clamp Techniques | Animals | Flufenamic Acid - chemistry | Humans | Ion Channels - agonists | Ion Channels - genetics | Protein Binding | Anti-Inflammatory Agents, Non-Steroidal - chemistry | Ion Channels - antagonists & inhibitors | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | GABA | Methyl aspartate | Aspartate | channel blockers | flufenamate | non-selective cation channel | chloride channels
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5. Full Text Ion Channel Modulators in Cystic Fibrosis
by Gentzsch, Martina and Mall, Marcus A
Chest, ISSN 0012-3692, 08/2018, Volume 154, Issue 2, pp. 383 - 393
Cystic fibrosis (CF) is caused by mutations in the cystic fibrosis transmembrane conductance regulator ( ) gene and remains one of the most common... 
translating basic research | cystic fibrosis | pharmacotherapy | CHRONIC-BRONCHITIS | CONTROLLED-TRIAL | ACTIVATED CHLORIDE CHANNEL | TEZACAFTOR-IVACAFTOR | CLINICAL DEVELOPMENT | LUMACAFTOR-IVACAFTOR | CFTR POTENTIATOR | TRANSMEMBRANE CONDUCTANCE REGULATOR | G551D MUTATION | RESPIRATORY SYSTEM | PHE508DEL CFTR | CRITICAL CARE MEDICINE | Usage | Gene mutations | Cystic fibrosis | Genetic aspects | Research | High-throughput screening (Biochemical assaying) | Genetic regulation | TMEM16A, transmembrane protein member 16A | CF, cystic fibrosis | SLC26A9, solute carrier 26 family member 9 | CFTR, cystic fibrosis transmembrane conductance regulator | ENaC, epithelial sodium channel | Translating Basic Research Into Clinical Practice
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6. Full Text Dendritic ion channel trafficking and plasticity
by Shah, Mala M and Hammond, Rebecca S and Hoffman, Dax A
Trends in Neurosciences, ISSN 0166-2236, 2010, Volume 33, Issue 7, pp. 307 - 316
Dendritic ion channels are essential for the regulation of intrinsic excitability as well as modulating the shape and integration of synaptic signals. Changes... 
Neurology | RECTIFYING POTASSIUM CHANNELS | INTRINSIC EXCITABILITY | I-H | EXCITATORY SYNAPTIC-TRANSMISSION | K+-CHANNEL | LONG-TERM POTENTIATION | BASAL DENDRITES | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | ACTIVATED CATION CHANNELS | CA1 PYRAMIDAL NEURONS | Ion Channel Gating - physiology | Animals | Neuronal Plasticity - physiology | Neurons - ultrastructure | Synapses - physiology | Dendrites - physiology | Neurons - physiology | Ion Channels - genetics | Ion Channels - physiology | Protein Processing, Post-Translational | Ion Channels - chemistry | Excitability
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7. Full Text The ion channel TRPA1 is required for chronic itch
by Wilson, Sarah R and Nelson, Aislyn M and Batia, Lyn and Morita, Takeshi and Estandian, Daniel and Owens, David M and Lumpkin, Ellen A and Bautista, Diana M
Journal of Neuroscience, ISSN 0270-6474, 05/2013, Volume 33, Issue 22, pp. 9283 - 9294
Chronic itch is a debilitating condition that affects one in 10 people. Little is known about the molecules that mediate chronic itch in primary sensory... 
SCRATCHING BEHAVIOR | PROTEINASE-ACTIVATED RECEPTOR-2 | ATOPIC-DERMATITIS | PRIMARY AFFERENT NEURONS | PROBE LEVEL DATA | SUPERFICIAL DORSAL-HORN | TRANSEPIDERMAL WATER-LOSS | DRY SKIN | NEUROSCIENCES | ACETONE-TREATED MICE | SENSORY NEURONS | Data Interpretation, Statistical | Gene Expression | Mice, Inbred C57BL | Male | Pruritus - genetics | Hyperplasia - pathology | Pruritus - physiopathology | Sensory Receptor Cells | Animals | Microarray Analysis | Transient Receptor Potential Channels - genetics | Homeostasis - physiology | TRPA1 Cation Channel | Skin - innervation | Transient Receptor Potential Channels - physiology | Mice | Pruritus - pathology | Chronic Disease | Real-Time Polymerase Chain Reaction | Skin - pathology
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8. Full Text Ion channel dysfunction in cerebellar ataxia
by Bushart, David D and Shakkottai, Vikram G
Neuroscience Letters, ISSN 0304-3940, 01/2019, Volume 688, pp. 41 - 48
Cerebellar ataxias constitute a heterogeneous group of disorders that result in impaired speech, uncoordinated limb movements, and impaired balance, often... 
Electrophysiology | Channel activator | Ion channel | Potassium channel | Spinocerebellar ataxia | Purkinje neuron | PURKINJE-CELLS | OLIVOCEREBELLAR CIRCUIT | ACTIVATED POTASSIUM CHANNELS | NEUROSCIENCES | LONG-TERM DEPRESSION | MOUSE MODEL | K+-CHANNEL | THERAPEUTIC TARGETS | SEVERE MYOCLONIC EPILEPSY | RESURGENT SODIUM CURRENT | Cerebellar Ataxia - physiopathology | Animals | Cerebellar Ataxia - genetics | Humans | Ion Channels - genetics | Ion Channels - physiology | Cerebellar Ataxia - drug therapy | Models, Neurological | Mutation | Molecular Targeted Therapy - methods | Purkinje Cells - physiology | Potassium channels | Neurons | Analysis | Cerebellar ataxia
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9. Full Text Structure of acid-sensing ion channel 1 at 1.9A resolution and low pH
by Jasti, J and Furukawa, H and Gonzales, EB and Gouaux, E
NATURE, ISSN 0028-0836, 09/2007, Volume 449, Issue 7160, pp. 316 - 316
Acid-sensing ion channels (ASICs) are voltage-independent, proton-activated receptors that belong to the epithelial sodium channel/degenerin family of ion... 
MULTIDISCIPLINARY SCIENCES | GATED NA+ CHANNEL | EXTRACELLULAR DOMAIN | ION CHANNELS | EPITHELIAL SODIUM-CHANNEL | SUBUNIT STOICHIOMETRY | HIPPOCAMPAL-NEURONS | ACTIVATED CURRENTS | ACETYLCHOLINE-RECEPTOR | SENSORY NEURONS | POTASSIUM CHANNEL
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10. Full Text Ion channel regulation by protein S-acylation
by Shipston, Michael J
Journal of General Physiology, ISSN 0022-1295, 2014, Volume 143, Issue 6, pp. 659 - 678
Protein S-acylation, the reversible covalent fatty-acid modification of cysteine residues, has emerged as a dynamic posttranslational modification (PTM) that... 
FATTY-ACID ACYLATION | PHYSIOLOGY | NITRIC-OXIDE SYNTHASE | CELL-SURFACE EXPRESSION | PALMITOYLATION-DEPENDENT REGULATION | ACTIVATED POTASSIUM CHANNELS | POSTTRANSLATIONAL MODIFICATIONS | EPITHELIAL SODIUM-CHANNEL | SITE-SPECIFIC ATTACHMENT | NEURONAL CEROID-LIPOFUSCINOSIS | ACTIVITY-BASED PROBES | Aminoacylation | Ion Channel Gating - physiology | Animals | Fatty Acids - chemistry | Models, Biological | Humans | Cysteine - metabolism | Ion Channels - physiology | Structure-Activity Relationship | Cysteine - chemistry | Fatty Acids - metabolism | Ion Channels - chemistry | Usage | Cysteine | Cell membranes | Fatty acids | Analysis | Review
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11. Full Text Mutations in ANO3 Cause Dominant Craniocervical Dystonia: Ion Channel Implicated in Pathogenesis
by Charlesworth, Gavin and Plagnol, Vincent and Holmström, Kira M and Bras, Jose and Sheerin, Una-Marie and